Joints - Mr Hoover's...
Transcript of Joints - Mr Hoover's...
Types of Joints-classified by structure or function
Cartilaginous joint-body of one bone connects to thebody of another by means of cartilage-slight movement is possible-intervertebral discs-hard outer ring with soft core
Fibrous joint-bound tightly together by connective tissue-allow no movement-joints between interlocking bones of the skull
Synovial joint-allow the most movement-bony surfaces are separated by lubricating fluid (synovia)-joined by ligaments-tough bands of elastic tissue that enclose the ends of articulating bones and form the capsule containing the synovial membrane
The Characteristics of a Synovial Joint
Bone
Blood vessels
Nerve
Synovial membrane
Joint cavity (filled with synovial fluid)
Fibrous capsule
Joint capsule
Bursa
Tendon sheath
Tendon
Articular cartilage-located on the ends of bones that comein contact-protects the ends of bone-allows for a smooth contact surface -shock absorber
Fibrous layer
Membranous layer Periosteum
Synovial Membrane-allows certain nutrients to pass through
Fibrous capsule-keep synovial fluid from leaking
Bursa-small, flattened fluid filled sacs found at friction points between tendons, ligaments and bones
Ligament-Intrinsic-thick bands of fibrous connective tissue that help thicken and reinforce joint capsule-Extrinsic-separate from joint capsule, attach bones together
Types of Synovial Joints
• There are three basic types of synovial joints: • unilateral (rotation only about one axis)
• biaxial joints (movement about two perpendicular axes)
• multiaxial joints (movement about all three perpendicular axes)
Types of Synovial Joints
Ball-and-socket joint-movement around 3 axis- multiaxial joints
Hinge joint- unilateral -convex portion of one bone fits into concave portion of another-movement in one plane
Saddle joint- biaxial joints-movement in 2 planes
Gliding jointunilateral-connects flat or slightly curved bones
Pivot joint- unilateral -rotation in one plane
Ellipsoid jointbiaxial joints-movement in 2 planes
Tissue Properties Tendons:
Composed of collagen (bundles of white, fibrous protein)
Attach muscle to bone
Greater stretching range
Dynamic stabilizer Vascular
Ligaments: Tough bands of white, fibrous tissue
Attach bone to bone
Less rigid than bones Allow certain amount of stretch, but do not have same stretch as tendons
Tear when reach threshold
Static stabilizers of joint Avascular-no blood supply
Strengthen with proper training/conditioning
Common Sport Injuries
Strains, pulls, and tears
Terms used to describe injuries to all joint tissue types Sprains-tendons and ligaments
Pulls and strains-muscles
First, second and third degree
Tendinitis
Inflammation of a tendon caused by irritation due to prolonged or abnormal use
Rest, cold/heat therapy, cast, splint or anti-inflammatories
Dislocations
Bone displaced from its original location
Often in finger joints
Damage to joint capsule and ligaments Symptoms
Joint look awkward or deformed
Joint is painful when touched or moved
Joint is not usable
Tendinitis
Bursa
Common Sport Injuries
Separations
Fibrous ligaments that bind the bones tear and separate
Ex. Acromioclavicular and Sternoclavicular
Cartilage
Torn cartilage
Avascular-long time to heal
Shin splints
Tearing of the interosseous membrane or the periosteum
Result of overuse without adequate rest
Occurs on medial or lateral side of tibia (shaft)
Risk factors include training surface or regimen, changes in frequency, duration or intensity or old/worn out footware
Proper Treatment of an Injury
S.H.A.R.P – signs of injury P.I.E.R. Principle
Swelling: instantly or over time Pressure: tensor wrap
Heat: increased temperature in the
areaIce: placed on affected area
Altered: tissue will not function
properlyElevate: to reduce swelling
Red: in colour Restrict: tensors, slings, or crutches
Painful: to touch or move
The Shoulder Joint
Clavicle
Coracoclavicular ligament
Coracoid process
Scapula
Acromioclavicular ligament
Acromion
Coracoacromial ligament
Glenohumeral ligaments and joint capsule
Tendon of biceps brachii (long head)
Humerus
The Shoulder Joint
Intrinsic unstable
Instability versatility Adduction/abduction
Flexion/extension
Elevation/depression
Protraction/retraction
Medial/Lateral rotation
Circumduction
Ball and socket-humerus and scapula
The Shoulder Joint
Ball and socket Humerus and Scapula-directly
Humeral head articulates with glenoid fossa of scapula
Held in place by ligaments
Clavicle-indirectly
Shoulder Joint Injuries
Biceps tendinitis
Caused by overuse of the biceps brachii muscle
Pain to proximal end of bicep
Shoulder separation
Tearing of the acromioclavicular ligament
Result of falls directly on shoulder
Shoulder dislocation
Occurs when the humerus “pops out” of the glenoid fossa
Result of hit or fall resulting in tear to the glenohumeral ligaments and joint capsule
Rotator cuff tears
An injury to one of the rotator cuff tendons
Difficulty laterally/medially rotating as well as abduction
Shoulder separation
The Knee Joint
• Articulation between femur and tibia• Femur does not come into contact with fibula
• Modified hinge• Flexion and extension
• Modified ellipsoid joint• Ability to slightly rotate medially and laterally
The Knee Joint – Anterior
Patella
Medial (Tibial) collateral ligament
Patellar ligament
Tibial tuberosity
Tibia
Quadriceps tendon
Fibula
The Knee Joint Anterior (deep)
Femur
Lateral (Fibular) collateral ligament removed
Medial (Tibial) collateral ligamentremoved
Lateral Meniscus
Tibial Tuberosity
Fibula
Lateral Condyle Medial Condyle
Medial Meniscus
Tibia
Posterior cruciate ligament
Anterior cruciate ligament
The Knee Joint• Articulating cartitilage at distal end of femur
• Meniscus on proximal end of each tibial condyle
• Cruciate Ligaments• ACL
• PCL
• Collateral Ligaments• MCL
• LCL
• Muscles stabilize the knee• Anterior side-quadriceps
• Posterior-gastrocnemius
The Knee Joint – Posterior
Femur
Adductor magnus tendon
Medial head of gastrocnemius tendon
Semimembranosus tendon
Medial (Tibial) collateral ligament
Lateral (Fibular) collateral ligament
Fibular head
Lateral head of gastrocnemius tendon
Oblique popliteal ligament
Fibula
Tibia
The Knee Joint – Posterior (deep)
Anterior cruciate ligament
Popliteal tendon
Lateral meniscus
Lateral (Fibular) collateral ligament
Medial (Tibial) collateralligament
Medial meniscus
Posterior cruciate
Femur
Fibula
Tibia
Posterior meniscofemoral ligament
Knee Joint Injuries
Knee ligament tearsBlows to lateral side of knee
Severity of blow determines degree of tear
Knee damage will happen to the medial side
Tear order Joint capsule
MCL, medial meniscus, ACL
Q-angle may contribute to the predisposition
of ACL tears
Knee Joint Injuries
Osgood-Schlatter syndrome Result of a condition known as osteochondritis-disease
of ossification centers
Standard explanation is growing pains for children
Affects the epiphyseal plate of the tibial tuberosity that has been overloaded or overused
Stress on patellar tendon or ligament causes plate to become inflamed-swelling and discomfort
Does not affect growth of the child
More common in males
PIER
Osgood-Schlatter syndrome
Knee Joint Injuries
Patellofemoral Syndrome (PFS) Gradual onset of anterior knee pain/pain around the patella
More common in adolscents, young adults and women
Aggravated by sports
Result of increased or misdirected force between kneecap and femur
Overuse, overload or misuse
PIER
The Ankle Joint – Medial View
Tibia
Medial malleolus
Calcaneal (Achilles) tendon
Long plantar ligament
Deltoid ligament
The Ankle Joint
• Modified hinge joint
• Distal ends of tibia, fibula resting on the talus
• Responsible for plantar flexion and dorsiflexion
The Ankle Joint – Lateral View
Tibia
Fibula
Posterior tibiofibular ligament
Lateral malleolus
Anterior tibiofibular ligament
Anterior talofibular ligament
Calcaneus
Posterior talofibular ligament
Anterior talofibular ligament
Ankle Joint Injuries
Inversion sprains
Ankle is weakest when plantar flexed
“twisted ankle” or “rolling”
Low or high
PIER
Eversion sprains
Occurs to the deltoid ligament Attaches the medial malleolus to 3 bones of
the foot.
Pott’s Fracture
A force on the medial side of ankle causing the deltoid ligament to rip off the tip of the medial malleolus; and a break of the fibula
Inversion sprain
The Three Energy Nutrients-Macronutrients Carbohydrates
4.1 calories per gram
Proteins
4.3 calories per gram
Fats 9.3 calories per gram
Bioenergetic Conversion Taking the food we eat and converting it
into fats, carbs and protein
Allows our bodies to function and carry out physical activity
The Chemistry of Energy Production
• Energy in the human body is derived from the breakdown of complex nutrients like carbohydrates, fats, and proteins.
• The end result of this breakdown is production of the adenosine triphosphate (ATP) molecule.
• ATP provides energy necessary for body functions
Carbohydrates
Fats
Proteins
ATP
Muscular Work
Digesting Food
Thermoregulation
Breakdown of Energy currency Biochemical processes
The Role of Carbohydrates
Carbohydrates are the most abundant organic substances in nature, and they
are essential for human and animal life.
Come from foods that originate from plants and grain
Veggies, fruits, breads and pastas
Formed by green plants by photosynthesis
Reactants-water and carbon dioxide
Products-oxygen and glucose
Glucose
Primary carb used/assimilated by humans
Stored as glycogen in human body (liver and skeletal muscle)
Glycogen is broken down and carried through blood and used as an
energy source
MetabolismHighly complex
Process in which energy is supplied throughout the body
Energy rich materials are assimilated through the body-energy renewal
Adenosine Triphosphate (ATP)-final form of free energy
Adenosine triphosphate (ATP)
Made in the mitochondrion
Captures chemical energy from food breakdown
Fuel various cell processes
Resynthesized in two ways
Aerobically
Anaerobically
ATP ADP + P + ENERGY
Two Energy Systems
Anaerobic System
Without the use of oxygen (O2)
None of its metabolic activity will involve O2
Utilizes chemicals and enzymes
Occurs in the muscle fibre
Occurs quickly
Required for powerful but short-lived physical actions
Aerobic System
In the presence of oxygen (O2)
All of its metabolic activity will involve O2
Occurs in the mitochondria
Involves many enzymes and several sub-pathways
Leads to the complete breakdown of glucose
Three Metabolic Pathways
ATP-PC System
(anaerobic alactic)
Glycolysis
(anaerobic lactic)
Cellular respiration
(aerobic)
PC + ADP ATP + CREATINE
ATP-PC System
ATP-PC System (anaerobic alactic)
First of two anaerobic energy pathways
Relies on the action of stored ATP and phosphocreatine
Yields enough ATP for 10–15 seconds of energy-short and powerful burts
Use stored ATP
Synthesize ATP
Provides highest rate of ATP synthesis
Limited amount PC Needs to be regenerated using ATP
during recovery time
No by-product No lactic acid produced (alactic)
Glycolysis Glycolysis (anaerobic lactic)
Second anaerobic energy pathway
Provides additional 1–3 minutes in high-level performance
Ideal back-up because glucose is plentiful
First sequence in complete breakdown of glucose
Partially breaks down glucose
Involves 11 separate biochemical reactions
Uses glucose and glycogen to make ATP
Transfer energy from glucose and rejoins phosphate and ADP
Yields twice as much ATP
By product is lactic acid (LA)
C6H12O6 + 2ADP + 2Pi 2C3H6O3 + 2ATP + 2H2O
(Glucose) (Lactate)
Pyruvate and Lactic Acid
• Aerobic condition• Oxygen available• Beginning of the third
energy system (aerobic system)
• Leads to complete breakdown of glucose and synthesis of large amounts of ATP
• Anaerobic condition• No oxygen available• Intense exercise or high
altitudes• Pyruvate is converted
into lactic acid• Build-up of lactic acid
hampers the break-down of glucose and decreases ability of muscles to contract
• Exhaustion or painful muscle agony begins
The main product of glycolysis is pyruvate!
The Aerobic System
Aerobic system (cellular respiration)
Third energy system
Glycolysis Same as anerobic lactic
Pyruvate is converted to Acetyl Co-A
Krebs cycle or Citric Acid Cycle
8 reactions
Synthesize compounds that can store “high-energy” electrons
Electron transport chain Electrons pass down the chain resulting in the production of large amounts of ATP
Uses glucose, glycogen, fats (exercise longer than 20 minutes), and protein (starvation) to make ATP
After 90 seconds of activity
Endurance type activity Sustain activity for very long time
Complete breakdown of glucose
C6H12O6 + 6O2 + 36ADP + 36Pi 6CO2 + 36ATP + 6H2O
Interaction of Energy System
• Rest• Small amounts of energy are needed
• Supplied almost exclusively by aerobic metabolism
• Beginning of exercise• Slight energy demand
• Continue with aerobic
• Immediate and high demand• ATP-PC system until either the aerobic system catches up or
anaerobic glycolysis kicks in• Proves that warm-up is necessary
Interaction of Energy System
• Steady state exercise• Supply of oxygen had met the demand
• Aerobic pathway-oxidative complete breakdown of glucose
• Strenuous exercise• Energy demand is rapid and expected for extended period
of time
• Anaerobic pathway is the main producer of ATP if oxygen demand cannot be met
• Production of lactic acid results in muscle fatigue or failure
Lactic Acid• Blood Lactate (Anaerobic) Threshold
• Point (exercise intensity) at which blood lactate levels increase abruptly beyond resting level
• Production is greater than removal
• Exercising below the LT allows any lactate produced by the muscles to be removed without building up
• Point where aerobic system cannot supply enough ATP for the body’s needs forcing anaerobic system to increase their contribution
• Lower the threshold indicates oxidative energy system are not working well or are being over-taxed
• Untrained athletes have a lower threshold
Energy from Fats and Protein Aerobic system (cellular
respiration)
Third energy system
Glycolysis
Krebs cycle
Electron transport chain
Uses glucose, glycogen, fats, and protein to make ATP
Lasts 120 seconds and beyond
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Energy from Fats
• Contain large quantities of stored energy• Twice as much as carbs and protein (slide 2)
• Primary type in muscle cells and adipose tissue-fatty acid
• Stored as triglycerides
• Lipolysis• Breakdown of TG to produce fatty acids
• Available as an energy source
Energy from Fats
• Fatty acids enter the energy system at the KREB CYCLE
• First need to be converted to acetyl-CoA through beta-oxidation
• Similar to pyruvate from glycolysis
• Drive the production ATP in the ETC
• Fat balance• Amount necessary as energy source and excessive
amounts leading to unhealthy body
Energy from Protein
• Energy equal to carb and half that of fat (slide 2)
• No protein reserves in the body• All part of existing body tissue or actively engaged in
metabolic activity
• Comprised of 20 amino acids• Nine essential
• Must be in a.a form to be used as energy
• Endurance activity or glycogen stores are low-energy back-up
Slow-Twitch and Fast-Twitch Muscles
Slow-twitch muscle fibres:
Most active during: long-distance running, swimming, and cycling
Red or dark in colour
Generate and relax tension slowly; able to maintain a lower level of tension for long durations
Low levels of myosin ATPase and glycolytic enzymes (anerobic)
High levels of oxidative enzymes (aerobic)
Most active during long-distance swimming, running and cycling
Fast-twitch muscle fibres:
Ideal for: short sprints, powerlifting, and explosive jumping
Pale in colour
Ability to tense and relax quickly; generate large amounts of tension with low endurance levels
Two to three times faster than slow twitch fibres
High levels of myosin ATPase and glycolytic enzymes
Ideal for fast, powerful contraction needed for sprinting, power lifting or jumping
Importance of Myoglobin-dependency of muscles fibres on oxygen
• Difference in muscle fibre types is due the extent at which a particular muscle relies on oxygen
• Myoglobin• Oxygen storage unit that delivers oxygen to working muscles
• Ability to sustain long-term activity is related to a muscle use of aerobic processes
Importance of Myoglobin-dependency of muscles fibres on oxygen
• Slow-twitch, red muscle fibres• High in myoglobin
• Ideal for endurance activity
• Fast-twitch fibres• Low in myoglobin
• Adapted for shorter bursts of energy
Three Fibre Types
Type I or Slow-Oxidative (SO)
Generate energy slowly
Fatigue-resistant
Depend on aerobic processes
Type IIA or Fast-Oxidative Glycolytic (FOG)
Intermediate-type muscle fibres
Allow for high-speed energy release
Allow for glycolytic capacity
Type IIB of Fast-Glycolytic (FG)
Store glycogen and high levels of enzymes
Allow for quick contraction without the need for oxygen
Research indicates that type IIB fibres can become type IIA with aerobic endurance training
Distribution of Muscle Fibre Types
• Muscle fibre makeup generally determine function and vice versa
• Tonic Muscles• Assist body with maintaining posture or stability
• Standing, walking and throwing
• High % of Type I fibre• Slow-twitch with little ability for explosiveness
• Considerable endurance
• Example: Soleus• Dorsiflexes the foot and ensures body posture
Distribution of Muscle Fibre Types
• Phasic Muscles• High % of Type IIA and IIB
• Example: bicep
• Muscle biopsy is the only way to determine the % of muscle fibre type
Anaerobic Alactic (ATP-PC)
• All training for this system should be powerful (100% intensity) and short (6-12s).
• The recovery time for this system should be minimal (15s-120s).
ATP-PC Examples
• Running – 10 repetitions of 30m sprints with 30s recovery
• Strength Training – 4 repetitions of 90% maximum load with 30s recovery
• Swim – 8 repetitions of 25m sprints with 30s recovery
Effects of training on Anaerobic Alactic system:
• 20-40% increase of creatine phosphate stores• increase of ATP stores
• increase in creatine kinase function
Anaerobic lactic (Glycolysis)
• Training for this system should be fairly powerful (70-95% intensity) and a longer timeframe than immediate alactic training (12s-3 min.).
• The recovery time for this system is also longer (45s-180s).
Examples
• Running – 5 repetitions of 300m sprints with 60s recovery
• Strength Training – 10 repetitions of 70% maximum load with 60s recovery
• Swim – 5 repetitions of 200m sprints with 60s recovery
Why does lactic acid inhibit muscle contraction?• An increase in lactic acid causes a release of hydrogen ions, which
inhibit calcium from binding to tropomysosin, so actin binding sites are still blocked.
•
Why does lactic acid inhibit muscle contraction?• The hydrogen ions cause the muscles to become acidic. The acidity
slows the breakdown of glucose, and aggravates nerve endings.
• The pain and irritation reaches the central nervous system and is the reason why some athletes feel disoriented or nauseous after exercise.
• Do not confuse this with exhaustion.
When does this occur?
• Inhibited muscle contraction occurs when anaerobic threshold is reached. Anaerobic Threshold (AT) is the point at which the lactic acid is accumulating in the blood stream.
• The production of lactic acid is too great for the removal of the lactic acid via the Cori cycle to keep up.
• When this point is reached, the release of hydrogen ions will cause the acidity in the muscle. Trained athletes have a higher AT (at 80-90% of max. effort) while untrained athletes have a lower AT (~55% of max. effort).
Effects of training on short term lactic acid system:
• Enables oxygen system to be utilized sooner to limit lactic acid production
• Increases lactic acid removal from muscles
• Increases speed of conversion of lactic acid into glucose
Aerobic (Cellular Respiration)
• Training for this system should be low to moderate intensity (40-70% intensity) and have a long timeframe (3 min.- 180 min.).
• The recovery time for this system is the longest (90s – 12 hours).
Examples:
• Running – 5k run or 30 minute continuous run
• Strength Training – Circuit training at a low intensity
• Swim – 1500m swim or 30 minute continuous swim
Effects of training on aerobic system:
• Increase in vascularization (# of blood vessels) within muscles. (Angiogenesis)