NULON LIFTER FREE AND TUNE UP - Part Info Lifter Free and Tune Up MSDS.pdf · NULON LIFTER FREE AND...

NULON LIFTER FREE AND TUNE UPChemwatch Safety Data Sheet (Conforms to Regulation (EC) No 1907/2006)Issue Date: 24-Dec-2008 CHEMWATCH 4957-25NA160TC Version No:5

CD 2008/3 of 22

Section 1 - CHEMICAL PRODUCT AND COMPANY IDENTIFICATION

PRODUCT NAMENULON LIFTER FREE AND TUNE UP

SUPPLIERCompany: Nulon Products Australia Pty LtdAddress:17 Yulong CloseMoorebankNSW, 2170AUSTelephone: +61 2 9986 7800Fax: +61 2 9601 4700

PRODUCT USEAdditive added to engine oil for removal of gum, carbon and varnish from internal parts

Section 2 - HAZARDS IDENTIFICATION

STATEMENT OF HAZARDOUS NATURECONSIDERED A DANGEROUS SUBSTANCE ACCORDING TO DIRECTIVE 1999/45/EC AND ITS AMENDMENTS.

HAZARD RATINGS

Flammability Toxicity

Body Contact Reactivity

Chronic

SCALE: Min/Nil=0 Low=1 Moderate=2 High=3 Extreme=4

RISKRisk Codes Risk PhrasesR18 In use, may form flammable/ explosivevapour- air mixture.R19 May form explosive peroxides.R20/21/22 Harmful by inhalation, in contact with skin and if swallowed.R37/38 Irritating to respiratory system and skin.R41 Risk of serious damage to eyes.R52/53 Harmful to aquatic organisms, may causelong- term adverse effects

in the aquaticenvironment.R65 HARMFUL- May cause lung damage if swallowed.R67 Vapours may cause drowsiness and dizziness.

Section 3 - COMPOSITION / INFORMATION ON INGREDIENTS

NAME CAS RN INT HAZ %mineral oil Not avail. None 10-30ethylene glycol monobutyl ether 111-76-2 Xn 10-30 EC NO: 203-905-0 R CODES: R20/21/22, R36/38

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CD 2008/3 of 22Section 3 - COMPOSITION / INFORMATION ON INGREDIENTS

aromatic hydrocarbon solvent 64742-95-6. T 10-30 R CODES: R45, R65isopropanol 67-63-0 F,Xi 1-5 EC NO: 200-661-7 R CODES: R11, R36, R67nonylphenol, ethoxylated 9016-45-9 F,Xn,N 1-10 EC NO: 500-024-6 R CODES: R11, R22, R41, R51/53, R66additives unregulated 1-10

Section 4 - FIRST AID MEASURES

SWALLOWED• If swallowed do NOT induce vomiting.• If vomiting occurs, lean patient forward or place on left side (head-down position, if possible) to maintain open airway and prevent aspiration.• Observe the patient carefully.• Never give liquid to a person showing signs of being sleepy or with reduced awareness; i.e. becoming unconscious.• Give water to rinse out mouth, then provide liquid slowly and as much as casualty can comfortably drink.• Seek medical advice.Avoid giving milk or oils.Avoid giving alcohol.• If spontaneous vomiting appears imminent or occurs, hold patient's head down, lower than their hips to help avoid possible aspiration of vomitus.

EYEIf this product comes in contact with the eyes:• Wash out immediately with fresh running water.• Ensure complete irrigation of the eye by keeping eyelids apart and away from eye and moving the eyelids by occasionally lifting the upper and lower lids.• If pain persists or recurs seek medical attention.• Removal of contact lenses after an eye injury should only be undertaken by skilled personnel.

SKINIf skin contact occurs:• Immediately remove all contaminated clothing, including footwear.• Flush skin and hair with running water (and soap if available).• Seek medical attention in event of irritation.

INHALED• If fumes or combustion products are inhaled remove from contaminated area.• Lay patient down. Keep warm and rested.• Prostheses such as false teeth, which may block airway, should be removed, where possible, prior to initiating first aid procedures.• Apply artificial respiration if not breathing, preferably with a demand valve resuscitator, bag-valve mask device, or pocket mask as trained. Perform CPR if necessary.• Transport to hospital, or doctor, without delay.

NOTES TO PHYSICIANAny material aspirated during vomiting may produce lung injury. Therefore emesis shouldnot be induced mechanically or pharmacologically. Mechanical means should be used if itis considered necessary to evacuate the stomach contents; these include gastric lavageafter endotracheal intubation. If spontaneous vomiting has occurred after ingestion, thepatient should be monitored for difficult breathing, as adverse effects of aspirationinto the lungs may be delayed up to 48 hours.For acute or short term repeated exposures to petroleum distillates or related

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CD 2008/3 of 22Section 4 - FIRST AID MEASURES

hydrocarbons:• Primary threat to life, from pure petroleum distillate ingestion and/or inhalation, isrespiratory failure.• Patients should be quickly evaluated for signs of respiratory distress (e.g. cyanosis,tachypnoea, intercostal retraction, obtundation) and given oxygen. Patients withinadequate tidal volumes or poor arterial blood gases (pO2 50 mm Hg) should be intubated.• Arrhythmias complicate some hydrocarbon ingestion and/or inhalation andelectrocardiographic evidence of myocardial injury has been reported; intravenous linesand cardiac monitors should be established in obviously symptomatic patients. The lungsexcrete inhaled solvents, so that hyperventilation improves clearance.• A chest x-ray should be taken immediately after stabilisation of breathing andcirculation to document aspiration and detect the presence of pneumothorax.• Epinephrine (adrenalin) is not recommended for treatment of bronchospasm because ofpotential myocardial sensitisation to catecholamines. Inhaled cardioselectivebronchodilators (e.g. Alupent, Salbutamol) are the preferred agents, with aminophylline asecond choice.• Lavage is indicated in patients who require decontamination; ensure use of cuffedendotracheal tube in adult patients. [Ellenhorn and Barceloux: Medical Toxicology].Treat symptomatically.Followed acute or short term repeated exposures to ethylene glycol monoalkyl ethers andtheir acetates:• Hepatic metabolism produces ethylene glycol as a metabolite.• Clinical presentation, following severe intoxication, resembles that of ethylene glycolexposures.• Monitoring the urinary excretion of the alkoxyacetic acid metabolites may be a usefulindication of exposure.[Ellenhorn and Barceloux: Medical Toxicology].For acute or short term repeated exposures to ethylene glycol:• Early treatment of ingestion is important. Ensure emesis is satisfactory.• Test and correct for metabolic acidosis and hypocalcaemia.• Apply sustained diuresis when possible with hypertonic mannitol.• Evaluate renal status and begin haemodialysis if indicated. [I.L.O]• Rapid absorption is an indication that emesis or lavage is effective only in the firstfew hours. Cathartics and charcoal are generally not effective.• Correct acidosis, fluid/electrolyte balance and respiratory depression in the usualmanner. Systemic acidosis (below 7.2) can be treated with intravenous sodium bicarbonatesolution.• Ethanol therapy prolongs the half-life of ethylene glycol and reduces the formation oftoxic metabolites.• Pyridoxine and thiamine are cofactors for ethylene glycol metabolism and should begiven (50 to 100 mg respectively) intramuscularly, four times per day for 2 days.• Magnesium is also a cofactor and should be replenished. The status of 4-methylpyrazole,in the treatment regime, is still uncertain. For clearance of the material and itsmetabolites, haemodialysis is much superior to peritoneal dialysis. [Ellenhorn andBarceloux: Medical Toxicology]It has been suggested that there is a need for establishinga new biological exposure limit before a workshift that is clearly below 100 mmol ethoxy-acetic acids per mole creatinine in morning urine of people occupationally exposed toethylene glycol ethers. This arises from the finding that an increase in urinary stonesmay be associated with such exposures.Laitinen J., et al: Occupational & Environmental Medicine 1996; 53, 595-600.• Heavy and persistent skin contamination over many years may lead to dysplastic changes.Pre-existing skin disorders may be aggravated by exposure to this product.• In general, emesis induction is unnecessary with high viscosity, low volatilityproducts, i.e. most oils and greases.• High pressure accidental injection through the skin should be assessed for possibleincision, irrigation and/or debridement.NOTE: Injuries may not seem serious at first, butwithin a few hours tissue may become swollen, discoloured and extremely painful withextensive subcutaneous necrosis. Product may be forced through considerable distancesalong tissue planes.

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CD 2008/3 of 22

Section 5 - FIRE FIGHTING MEASURES

EXTINGUISHING MEDIA• Foam.• Dry chemical powder.• BCF (where regulations permit).• Carbon dioxide.• Water spray or fog - Large fires only.

FIRE FIGHTING• Alert Fire Brigade and tell them location and nature of hazard.• Wear full body protective clothing with breathing apparatus.• Prevent, by any means available, spillage from entering drains or water course.• Use water delivered as a fine spray to control fire and cool adjacent area.• Avoid spraying water onto liquid pools.• DO NOT approach containers suspected to be hot.• Cool fire exposed containers with water spray from a protected location.• If safe to do so, remove containers from path of fire.

FIRE/EXPLOSION HAZARDWARNING: In use may form flammable/ explosive vapour-air mixtures.• Combustible.• Slight fire hazard when exposed to heat or flame.• Heating may cause expansion or decomposition leading to violent rupture of containers.• On combustion, may emit toxic fumes of carbon monoxide (CO).• May emit acrid smoke.• Mists containing combustible materials may be explosive.Combustion products include: carbon dioxide (CO2), other pyrolysis products typical ofburning organic material.May emit poisonous fumes.May emit corrosive fumes.

FIRE INCOMPATIBILITY• Avoid contamination with oxidising agents i.e. nitrates, oxidising acids, chlorinebleaches, pool chlorine etc. as ignition may result.

PERSONAL PROTECTIONGlasses:Chemical goggles.Gloves:PVC chemical resistant type.Respirator:Type ANO- P Filter of sufficient capacity

Section 6 - ACCIDENTAL RELEASE MEASURES

MINOR SPILLSSlippery when spilt.• Remove all ignition sources.• Clean up all spills immediately.• Avoid breathing vapours and contact with skin and eyes.• Control personal contact by using protective equipment.• Contain and absorb spill with sand, earth, inert material or vermiculite.• Wipe up.• Place in a suitable labelled container for waste disposal.

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CD 2008/3 of 22Section 6 - ACCIDENTAL RELEASE MEASURES

MAJOR SPILLSChemical Class: aliphatic hydrocarbons

For release onto land: recommended sorbents listed in order of priority.

SORBENT TYPE RANK APPLICATION COLLECTION LIMITATIONS

LAND SPILL - SMALL

cross- linked 1 shovel shovel R, W, SSpolymer -particulatecross- linked 1 throw pitchfork R, DGC, RTpolymer - pillowwood fiber - 2 throw pitchfork R, P, DGC, RTpillowtreated wood 2 throw pitchfork DGC, RTfibre- pillowsorbent clay - 3 shovel shovel R, I, Pparticulatefoamed glass - 3 throw pitchfork R, P, DGC, RTpillow

LAND SPILL - MEDIUM

cross- linked 1 blower skiploader R, W, SSpolymer -particulatecross- linked 2 throw skiploader R, DGC, RTpolymer - pillowsorbent clay - 3 blower skiploader R, I, Pparticulatepolypropylene - 3 blower skiploader W, SS, DGCparticulateexpanded mineral 4 blower skiploader R, I, W, P, DGC- particulatepolypropylene - 4 throw skiploader DGC, RTmat

LegendDGC: Not effective where ground cover is denseR; Not reusableI: Not incinerableP: Effectiveness reduced when rainyRT:Not effective where terrain is ruggedSS: Not for use within environmentally sensitive sitesW: Effectiveness reduced when windy

Reference: Sorbents for Liquid Hazardous Substance Cleanup and Control;R.W Melvold et al: Pollution Technology Review No. 150: Noyes Data Corporation 1988.Slippery when spilt.Moderate hazard.• Clear area of personnel and move upwind.• Alert Fire Brigade and tell them location and nature of hazard.• Wear breathing apparatus plus protective gloves.• Prevent, by any means available, spillage from entering drains or water course.

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CD 2008/3 of 22Section 6 - ACCIDENTAL RELEASE MEASURES

• No smoking, naked lights or ignition sources.• Increase ventilation.• Stop leak if safe to do so.• Contain spill with sand, earth or vermiculite.• Collect recoverable product into labelled containers for recycling.• Absorb remaining product with sand, earth or vermiculite.• Collect solid residues and seal in labelled drums for disposal.• Wash area and prevent runoff into drains.• If contamination of drains or waterways occurs, advise emergency services.

Section 7 - HANDLING AND STORAGE

PROCEDURE FOR HANDLING• Containers, even those that have been emptied, may contain explosive vapours.• Do NOT cut, drill, grind, weld or perform similar operations on or near containers.• DO NOT allow clothing wet with material to stay in contact with skin.The substance accumulates peroxides which may become hazardous only if it evaporates or is distilled or otherwise treated to concentrate the peroxides. The substance may concentrate around the container opening for example.Purchases of peroxidisable chemicals should be restricted to ensure that the chemical is used completely before it can become peroxidised.• A responsible person should maintain an inventory of peroxidisable chemicals or annotate the general chemical inventory to indicate which chemicals are subject to peroxidation. An expiration date should be determined. The chemical should either be treated to remove peroxides or disposed of before this date.• The person or laboratory receiving the chemical should record a receipt date on the bottle. The individual opening the container should add an opening date.• Unopened containers received from the supplier should be safe to store for 18 months.• Opened containers should not be stored for more than 12 months.• Electrostatic discharge may be generated during pumping - this may result in fire.• Ensure electrical continuity by bonding and grounding (earthing) all equipment.• Restrict line velocity during pumping in order to avoid generation of electrostatic discharge (<=1 m/sec until fill pipe submerged to twice its diameter, then <= 7 m/sec).• Avoid splash filling.• Do NOT use compressed air for filling discharging or handling operations.• Avoid all personal contact, including inhalation.• Wear protective clothing when risk of exposure occurs.• Use in a well-ventilated area.• Prevent concentration in hollows and sumps.• DO NOT enter confined spaces until atmosphere has been checked.• Avoid smoking, naked lights or ignition sources.• Avoid contact with incompatible materials.• When handling, DO NOT eat, drink or smoke.• Keep containers securely sealed when not in use.• Avoid physical damage to containers.• Always wash hands with soap and water after handling.• Work clothes should be laundered separately.• Use good occupational work practice.• Observe manufacturer's storing and handling recommendations.• Atmosphere should be regularly checked against established exposure standards to ensure safe working conditions.

SUITABLE CONTAINER• DO NOT use aluminium or galvanised containers.• Metal can or drum• Packaging as recommended by manufacturer.

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CD 2008/3 of 22Section 7 - HANDLING AND STORAGE

• Check all containers are clearly labelled and free from leaks.

STORAGE INCOMPATIBILITY• Glycol ethers may form peroxides under certain conditions.• In the presence of strong bases or the salts of strong bases, at elevated temperatures, the potential exists for runaway reactions.• Contact with aluminium should be avoided; release of hydrogen gas may result- will corrode scratched aluminium surfaces.CARE: Water in contact with heated material may cause foaming or a steam explosion with possible severe burns from wide scattering of hot material. Resultant overflow of containers may result in fire.• Avoid reaction with oxidising agents.

STORAGE REQUIREMENTS• Store in original containers.• Keep containers securely sealed.• No smoking, naked lights or ignition sources.• Store in a cool, dry, well-ventilated area.• Store away from incompatible materials and foodstuff containers.• Protect containers against physical damage and check regularly for leaks.• Observe manufacturer's storing and handling recommendations.

Section 8 - EXPOSURE CONTROLS / PERSONAL PROTECTION

EXPOSURE CONTROLSSource Material TWA ppm TWA mg/m³ STEL ppm STEL mg/m³__________________ __________________ _______ _______ _______ _______EU Consolidated List of ethylene glycol 20 98 50 246Indicative Occupational monobutyl ether (2-Exposure Limit Values Butoxyethanol)(IOELVs)European Union (EU) ethylene glycol 20 98 50 246First List of Indicative monobutyl ether (2-Occupational Exposure Butoxyethanol)Limit Values (IOELVs)UK Workplace Exposure ethylene glycol 25 50Limits (WELs) monobutyl ether (2-

Butoxyethanol)UK Workplace Exposure isopropanol (Propan- 2- 400 999 1250 500Limits (WELs) ol)

The following materials had no OELs on our records• aromatic hydrocarbon solvent: CAS:64742- 95- 6• nonylphenol, ethoxylated: CAS:9016- 45- 9 CAS:26027- 38- 3

EMERGENCY EXPOSURE LIMITSMaterial Revised IDLH Value (mg/m3) Revised IDLH Value (ppm)ethylene glycol monobutyl ether 700 [Unch]isopropanol 2, 000 [LEL]

NOTESValues marked LEL indicate that the IDLH was based on 10% of the lower explosive limitfor safety considerations even though the relevant toxicological data indicated thatirreversible health effects or impairment of escape existed only at higher concentrations.

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CD 2008/3 of 22Section 8 - EXPOSURE CONTROLS / PERSONAL PROTECTION

MATERIAL DATASensory irritants are chemicals that produce temporary and undesirable side-effects onthe eyes, nose or throat. Historically occupational exposure standards for theseirritants have been based on observation of workers' responses to various airborneconcentrations. Present day expectations require that nearly every individual should beprotected against even minor sensory irritation and exposure standards are establishedusing uncertainty factors or safety factors of 5 to 10 or more. On occasion animal no-observable-effect-levels (NOEL) are used to determine these limits where human resultsare unavailable. An additional approach, typically used by the TLV committee (USA) indetermining respiratory standards for this group of chemicals, has been to assign ceilingvalues (TLV C) to rapidly acting irritants and to assign short-term exposure limits (TLVSTELs) when the weight of evidence from irritation, bioaccumulation and other endpointscombine to warrant such a limit. In contrast the MAK Commission (Germany) uses a five-category system based on intensive odour, local irritation, and elimination half-life.However this system is being replaced to be consistent with the European Union (EU)Scientific Committee for Occupational Exposure Limits (SCOEL); this is more closelyallied to that of the USA.OSHA (USA) concluded that exposure to sensory irritants can:• cause inflammation• cause increased susceptibility to other irritants and infectious agents• lead to permanent injury or dysfunction• permit greater absorption of hazardous substances and• acclimate the worker to the irritant warning properties of these substances thusincreasing the risk of overexposure.

INGREDIENT DATAAROMATIC HYDROCARBON SOLVENT:ISOPROPANOL:NONYLPHENOL, ETHOXYLATED:

Sensory irritants are chemicals that produce temporary and undesirable side-effects onthe eyes, nose or throat. Historically occupational exposure standards for theseirritants have been based on observation of workers' responses to various airborneconcentrations. Present day expectations require that nearly every individual should beprotected against even minor sensory irritation and exposure standards are establishedusing uncertainty factors or safety factors of 5 to 10 or more. On occasion animal no-observable-effect-levels (NOEL) are used to determine these limits where human resultsare unavailable. An additional approach, typically used by the TLV committee (USA) indetermining respiratory standards for this group of chemicals, has been to assign ceilingvalues (TLV C) to rapidly acting irritants and to assign short-term exposure limits (TLVSTELs) when the weight of evidence from irritation, bioaccumulation and other endpointscombine to warrant such a limit. In contrast the MAK Commission (Germany) uses a five-category system based on intensive odour, local irritation, and elimination half-life.However this system is being replaced to be consistent with the European Union (EU)Scientific Committee for Occupational Exposure Limits (SCOEL); this is more closelyallied to that of the USA.

OSHA (USA) concluded that exposure to sensory irritants can:• cause inflammation• cause increased susceptibility to other irritants and infectious agents• lead to permanent injury or dysfunction• permit greater absorption of hazardous substances and• acclimate the worker to the irritant warning properties of these substances thus

increasing the risk of overexposure.

MINERAL OIL:ES TWA: 5 mg/m3 refined mineral oil mist.Human exposure to oil mist alone has not been demonstrated to cause health effects

except at levels above 5 mg/m3 (this applies to particulates sampled by a method thatdoes not collect vapour). It is not advisable to apply this standard to oils containing

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unknown concentrations and types of additive.

AROMATIC HYDROCARBON SOLVENT:

CEL TWA: 50 ppm, 250 mg/m3 as total hydrocarbons [Manufacturer]

ISOPROPANOL:Odour Threshold Value: 3.3 ppm (detection), 7.6 ppm (recognition)Exposure at or below the recommended isopropanol TLV-TWA and STEL is thought to

minimise the potential for inducing narcotic effects or significant irritation of theeyes or upper respiratory tract. It is believed, in the absence of hard evidence, thatthis limit also provides protection against the development of chronic health effects.The limit is intermediate to that set for ethanol, which is less toxic, and n-propylalcohol, which is more toxic, than isopropanol.

PERSONAL PROTECTION

EYE• Safety glasses with side shields.• Chemical goggles.• Contact lenses may pose a special hazard; soft contact lenses may absorb andconcentrate irritants. A written policy document, describing the wearing of lens orrestrictions on use, should be created for each workplace or task. This should include areview of lens absorption and adsorption for the class of chemicals in use and an accountof injury experience. Medical and first-aid personnel should be trained in their removaland suitable equipment should be readily available. In the event of chemical exposure,begin eye irrigation immediately and remove contact lens as soon as practicable. Lensshould be removed at the first signs of eye redness or irritation - lens should beremoved in a clean environment only after workers have washed hands thoroughly. [CDCNIOSH Current Intelligence Bulletin 59].

HANDS/FEET• Wear chemical protective gloves, eg. PVC.• Wear safety footwear or safety gumboots, eg. Rubber.Suitability and durability of glove type is dependent on usage. Factors such as:• frequency and duration of contact,• chemical resistance of glove material,• glove thickness and• dexterity,are important in the selection of gloves.

OTHER• Overalls.• P.V.C. apron.• Barrier cream.• Skin cleansing cream.• Eye wash unit.

RESPIRATORRespiratory protection may be required when ANY "Worst Case" vapour-phase concentrationis exceeded (see Computer Prediction in "Exposure Standards").

Protection Factor (Min) Half- Face Respirator Full- face Respirator10 x ES ANO- P- - AUS -

ANO- P- - PAPR- AUS -20 x ES - ANO- P- - AUS

- ANO- P- - PAPR- AUS100 x ES - ANO- P- - 2

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- ANO- P- - PAPR- 2

^ - Full-face.

The local concentration of material, quantity and conditions of use determine the type ofpersonal protective equipment required. For further information consult site specificCHEMWATCH data (if available), or your Occupational Health and Safety Advisor.

ENGINEERING CONTROLSLocal exhaust ventilation usually required. If risk of overexposure exists, wear approvedrespirator. Correct fit is essential to obtain adequate protection. Supplied-air typerespirator may be required in special circumstances. Correct fit is essential to ensureadequate protection.An approved self contained breathing apparatus (SCBA) may be required in some situations.Provide adequate ventilation in warehouse or closed storage area. Air contaminantsgenerated in the workplace possess varying "escape" velocities which, in turn, determinethe "capture velocities" of fresh circulating air required to effectively remove thecontaminant.

Type of Contaminant: Air Speed:solvent, vapours, degreasing etc., evaporating from tank (in 0.25- 0.5 m/s (50- 100still air). f/min.)aerosols, fumes from pouring operations, intermittent container 0.5- 1 m/s (100- 200filling, low speed conveyer transfers, welding, spray drift, f/min.)plating acid fumes, pickling (released at low velocity into zoneof active generation)direct spray, spray painting in shallow booths, drum filling, 1- 2.5 m/s (200- 500conveyer loading, crusher dusts, gas discharge (active generation f/min.)into zone of rapid air motion)grinding, abrasive blasting, tumbling, high speed wheel generated 2.5- 10 m/s (500- 2000dusts (released at high initial velocity into zone of very high f/min.)rapid air motion).

Within each range the appropriate value depends on:

Lower end of the range Upper end of the range1: Room air currents minimal or favourable to 1: Disturbing room air currentscapture2: Contaminants of low toxicity or of nuisance value 2: Contaminants of high toxicityonly.3: Intermittent, low production. 3: High production, heavy use4: Large hood or large air mass in motion 4: Small hood- local control only

Simple theory shows that air velocity falls rapidly with distance away from the openingof a simple extraction pipe. Velocity generally decreases with the square of distancefrom the extraction point (in simple cases). Therefore the air speed at the extractionpoint should be adjusted, accordingly, after reference to distance from the contaminatingsource. The air velocity at the extraction fan, for example, should be a minimum of 1-2m/s (200-400 f/min) for extraction of solvents generated in a tank 2 meters distant fromthe extraction point. Other mechanical considerations, producing performance deficitswithin the extraction apparatus, make it essential that theoretical air velocities aremultiplied by factors of 10 or more when extraction systems are installed or used.

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CD 2008/3 of 22

Section 9 - PHYSICAL AND CHEMICAL PROPERTIES

PHYSICAL PROPERTIESLiquid.

Molecular Weight: Not Applicable Boiling Range (°C): Not Av ailableMelting Range (°C): Not Available Specific Gravity (water= 1): 0.96- 0.98Solubility in water (g/L): Partly Miscible pH (as supplied): Not ApplicablepH (1% solution): Not Applicable Vapour Pressure (kPa): Not AvailableVolatile Component (%vol): Not Available Evaporation Rate: SlowRelative Vapour Density (air=1): >1 Flash Point (°C): >62Lower Explosive Limit (%): Not Available Upper Explosive Limit (%): Not AvailableAutoignition Temp (°C): Not Available Decomposition Temp ( °C): Not AvailableState: Liquid Viscosity: Not Available

APPEARANCEClear amber liquid, emulsifies in water. hydrocarbon solvent odour

Material ValueETHYLENE GLYCOL MONOBUTYL ETHER:log Kow (Prager 1995) 0.83log Kow (Sangster 1997) 0.8log Kow 0.76- 0.83ISOPROPANOL:log Kow (Sangster 1997) 0.05log Kow - 0.16- 0.28

Section 10 - CHEMICAL STABILITY AND REACTIVITY INFORMATION

CONDITIONS CONTRIBUTING TO INSTABILITY• Presence of incompatible materials.• Product is considered stable.• Hazardous polymerisation will not occur.For incompatible materials - refer to Section 7 - Handling and Storage.

Section 11 - TOXICOLOGICAL INFORMATION

POTENTIAL HEALTH EFFECTS

ACUTE HEALTH EFFECTS

SWALLOWEDLimited evidence exists that exposure to the material may produce irreversible damage(other than carcinogenesis, mutagenesis and teratogenesis) following a single exposure byswallowing.Accidental ingestion of the material may be harmful; animal experiments indicate thatingestion of less than 150 gram may be fatal or may produce serious damage to the healthof the individual.Considered an unlikely route of entry in commercial/industrial environments The liquidmay produce considerable gastrointestinal discomfort and may be harmful or toxic ifswallowed. Ingestion may result in nausea, pain and vomiting. Vomit entering the lungs by

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CD 2008/3 of 22Section 11 - TOXICOLOGICAL INFORMATION

aspiration may cause potentially lethal chemical pneumonitis.Ingestion of petroleum hydrocarbons may produce irritation of the pharynx, oesophagus,stomach and small intestine with oedema and mucosal ulceration resulting; symptomsinclude a burning sensation in the mouth and throat. Large amounts may produce narcosiswith nausea and vomiting, weakness or dizziness, slow and shallow respiration, swellingof the abdomen, unconsciousness and convulsions. Myocardial injury may producearrhythmias, ventricular fibrillation and electrocardiographic changes. Central nervoussystem depression may also occur. Light aromatic hydrocarbons produce a warm, sharp,tingling sensation on contact with taste buds and may anaesthetise the tongue. Aspirationinto the lungs may produce coughing, gagging and a chemical pneumonitis with pulmonaryoedema and haemorrhage.

EYEWhen applied to the eye(s) of animals, the material produces severe ocular lesions whichare present twenty-four hours or more after instillation.Evidence exists, or practical experience predicts, that the material may cause severe eyeirritation in a substantial number of individuals and/or may produce significant ocularlesions which are present twenty-four hours or more after instillation into the eye(s) ofexperimental animals. Eye contact may cause significant inflammation with pain. Cornealinjury may occur; permanent impairment of vision may result unless treatment is promptand adequate. Repeated or prolonged exposure to irritants may cause inflammationcharacterised by a temporary redness (similar to windburn) of the conjunctiva(conjunctivitis); temporary impairment of vision and/or other transient eyedamage/ulceration may occur.The liquid produces a high level of eye discomfort and is capable of causing pain andsevere conjunctivitis. Corneal injury may develop, with possible permanent impairment ofvision, if not promptly and adequately treated.Petroleum hydrocarbons may produce pain after direct contact with the eyes. Slight, buttransient disturbances of the corneal epithelium may also result. The aromatic fractionmay produce irritation and lachrymation.

SKINEvidence exists, or practical experience predicts, that the material either producesinflammation of the skin in a substantial number of individuals following direct contact,and/or produces significant inflammation when applied to the healthy intact skin ofanimals, for up to four hours, such inflammation being present twenty-four hours or moreafter the end of the exposure period. Skin irritation may also be present after prolongedor repeated exposure; this may result in a form of contact dermatitis (nonallergic). Thedermatitis is often characterised by skin redness (erythema) and swelling (oedema) whichmay progress to blistering (vesiculation), scaling and thickening of the epidermis. Atthe microscopic level there may be intercellular oedema of the spongy layer of the skin(spongiosis) and intracellular oedema of the epidermis.Limited evidence exists that exposure to the material may produce irreversible damage(other than carcinogenesis, mutagenesis and teratogenesis) following a single exposure byskin contact.The material may accentuate any pre-existing dermatitis condition.Entry into the blood-stream through, for example, cuts, abrasions, puncture wounds orlesions, may produce systemic injury with harmful effects. Examine the skin prior to theuse of the material and ensure that any external damage is suitably protected.Skin contact with the material may be harmful; systemic effects may result followingabsorption.Aromatic hydrocarbons may produce skin irritation, vasodilation with erythema and changesin endothelial cell permeability. Systemic intoxication, resulting from contact with thelight aromatics, is unlikely due to the slow rate of permeation. Branching of the sidechain appears to increase percutaneous absorption.

INHALEDEvidence shows, or practical experience predicts, that the material produces irritationof the respiratory system, in a substantial number of individuals, following inhalation.

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In contrast to most organs, the lung is able to respond to a chemical insult by firstremoving or neutralising the irritant and then repairing the damage. The repair process,which initially evolved to protect mammalian lungs from foreign matter and antigens, mayhowever, produce further lung damage resulting in the impairment of gas exchange, theprimary function of the lungs. Respiratory tract irritation often results in aninflammatory response involving the recruitment and activation of many cell types, mainlyderived from the vascular system.Limited evidence exists that exposure to the material may produce irreversible damage(other than carcinogenesis, mutagenesis and teratogenesis) following a single exposure byinhalation.Inhalation hazard is increased at higher temperatures.If exposure to highly concentrated solvent atmosphere is prolonged this may lead tonarcosis, unconsciousness, even coma and possible death.Inhalation of aerosols (mists, fumes), generated by the material during the course ofnormal handling, may be harmful.Xylene is a central nervous system depressant. Central nervous system (CNS) depressionmay include nonspecific discomfort, symptoms of giddiness, headache, dizziness, nausea,anaesthetic effects, slowed reaction time, slurred speech and may progress tounconsciousness. Serious poisonings may result in respiratory depression and may be fatal.

CHRONIC HEALTH EFFECTSLimited evidence suggests that repeated or long-term occupational exposure may producecumulative health effects involving organs or biochemical systems.There is some evidence that human exposure to the material may result in developmentaltoxicity. This evidence is based on animal studies where effects have been observed inthe absence of marked maternal toxicity, or at around the same dose levels as other toxiceffects but which are not secondary non-specific consequences of the other toxic effects.Exposure to the material may cause concerns for human fertility, on the basis thatsimilar materials provide some evidence of impaired fertility in the absence of toxiceffects, or evidence of impaired fertility occurring at around the same dose levels asother toxic effects, but which are not a secondary non-specific consequence of othertoxic effects..On the basis, primarily, of animal experiments, concern has been expressed that thematerial may produce carcinogenic or mutagenic effects; in respect of the availableinformation, however, there presently exists inadequate data for making a satisfactoryassessment.Exposure to the material for prolonged periods may cause physical defects in thedeveloping embryo (teratogenesis).Principal route of exposure is by skin contact; lesser exposures include inhalation offumes from hot oils, oil mists or droplets. Prolonged contact with mineral oils carrieswith it the risk of skin conditions such as oil folliculitis, eczematous dermatitis,pigmentation of the face (melanosis) and warts on the sole of the foot (plantar warts).With highly refined mineral oils no appreciable systemic effects appear to result throughskin absorption.Exposure to oil mists frequently elicits respiratory conditions, such as asthma; theprovoking agent is probably an additive. High oil mist concentrations may produce lipoidpneumonia although clinical evidence is equivocal. In animals exposed to concentrationsof 100 mg/m3 oil mist, for periods of 12 to 26 months, the activity of lung and serumalkaline phosphatase enzyme was raised; 5 mg/m3 oil mist did not produce this response.These enzyme changes are sensitive early indicators of lung damage. Workers exposed tovapours of mineral oil and kerosene for 5 to 35 years showed an increased prevalence ofslight basal lung fibrosis.Many studies have linked cancers of the skin and scrotum with mineral oil exposure.Contaminants in the form of additives and the polycyclic aromatic hydrocarbons (PAHs - asin the crude base stock) are probably responsible. PAH levels are higher in aromaticprocess oils/used/reclaimed motor oils. Subchronic 90-day feeding studies conducted onmale and female rats on highly refined white mineral oils and waxes found that highermolecular-weight hydrocarbons (microcrystalline waxes and the higher viscosity oils) werewithout biological effects. Paraffin waxes and low- to mid viscosity oils produced

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biological effects that were inversely proportional to molecular weight, viscosity andmelting point: oil-type and processing did not appear to be determinants. Biologicaleffects were more pronounced in females than in males. Effects occurred mainly in theliver and mesenteric lymph nodes and included increased organ weights, microscopicinflammatory changes, and evidence for the presence of saturated mineral hydrocarbons inaffected tissues. Inflammation of the cardiac mitral valve was also observed at highdoses in rats treated with paraffin waxes.Smith J.H., et al: Toxicologic Pathology: 24, 2, 214-230, 1996.

Nulon Lifter Free and Tune Up

TOXICITY AND IRRITATIONunless otherwise specified data extracted from RTECS - Register of Toxic Effects of Chemical Substances.

Asthma-like symptoms may continue for months or even years after exposure to the material ceases. This maybe due to a non-allergenic condition known as reactive airways dysfunction syndrome (RADS) which can occurfollowing exposure to high levels of highly irritating compound. Key criteria for the diagnosis of RADSinclude the absence of preceding respiratory disease, in a non-atopic individual, with abrupt onset ofpersistent asthma-like symptoms within minutes to hours of a documented exposure to the irritant. Areversible airflow pattern, on spirometry, with the presence of moderate to severe bronchialhyperreactivity on methacholine challenge testing and the lack of minimal lymphocytic inflammation, withouteosinophilia, have also been included in the criteria for diagnosis of RADS. RADS (or asthma) following anirritating inhalation is an infrequent disorder with rates related to the concentration of and duration ofexposure to the irritating substance. Industrial bronchitis, on the other hand, is a disorder that occursas result of exposure due to high concentrations of irritating substance (often particulate in nature) andis completely reversible after exposure ceases. The disorder is characterised by dyspnea, cough and mucusproduction.The material may produce severe irritation to the eye causing pronounced inflammation. Repeated orprolonged exposure to irritants may produce conjunctivitis.The material may cause skin irritation after prolonged or repeated exposure and may produce a contactdermatitis (nonallergic). This form of dermatitis is often characterised by skin redness (erythema) andswelling epidermis. Histologically there may be intercellular oedema of the spongy layer (spongiosis) andintracellular oedema of the epidermis.

MINERAL OIL:unless otherwise specified data extracted from RTECS - Register of Toxic Effects of Chemical Substances.

Toxicity and Irritation data for petroleum-based mineral oils are related to chemical components and varyas does the composition and source of the original crude.A small but definite risk of occupational skin cancer occurs in workers exposed to persistent skincontamination by oils over a period of years. This risk has been attributed to the presence of certainpolycyclic aromatic hydrocarbons (PAH) (typified by benz[a]pyrene).Petroleum oils which are solvent refined/extracted or severely hydrotreated, contain very lowconcentrations of both.

ETHYLENE GLYCOL MONOBUTYL ETHER:unless otherwise specified data extracted from RTECS - Register of Toxic Effects of Chemical Substances.

TOXICITY IRRITATIONOral (rat) LD50: 470 mg/kg Skin (rabbit): 500 mg, open; MildDermal (rabbit) LD50: 220 mg/kg Eye (rabbit): 100 mg/24h- ModerateInhalation (human) TCLo: 100 ppm Eye (rabbit): 100 mg SEVEREInhalation (human) TCLo: 195 ppm/8h * [UnionCarbide]Inhalation (Rat) LC50: 450 ppm *The material may produce severe irritation to the eye causing pronounced inflammation. Repeated orprolonged exposure to irritants may produce conjunctivitis.The material may cause skin irritation after prolonged or repeated exposure and may produce a contact

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dermatitis (nonallergic). This form of dermatitis is often characterised by skin redness (erythema) andswelling epidermis. Histologically there may be intercellular oedema of the spongy layer (spongiosis) andintracellular oedema of the epidermis.For ethylene glycol:Ethylene glycol is quickly and extensively absorbed through the gastrointestinal tract. Limited informationsuggests that it is also absorbed through the respiratory tract; dermal absorption is apparently slow.Following absorption, ethylene glycol is distributed throughout the body according to total body water. Inmost mammalian species, including humans, ethylene glycol is initially metabolised by alcohol.dehydrogenase to form glycolaldehyde, which is rapidly converted to glycolic acid and glyoxal by aldehydeoxidase and aldehyde dehydrogenase. These metabolites are oxidised to glyoxylate; glyoxylate may be furthermetabolised to formic acid, oxalic acid, and glycine. Breakdown of both glycine and formic acid cangenerate CO2, which is one of the major elimination products of ethylene glycol. In addition to exhaled CO2,ethylene glycol is eliminated in the urine as both the parent compound and glycolic acid. Elimination ofethylene glycol from the plasma in both humans and laboratory animals is rapid after oral exposure;elimination half-lives are in the range of 1-4 hours in most species tested.Respiratory Effects. Respiratory system involvement occurs 12-24 hours after ingestion of sufficientamounts of ethylene glycol and is considered to be part of a second stage in ethylene glycol poisoning Thesymptoms include hyperventilation, shallow rapid breathing, and generalized pulmonary edema with calciumoxalate crystals occasionally present in the lung parenchyma. Respiratory system involvement appears to bedose-dependent and occurs concomitantly with cardiovascular changes. Pulmonary infiltrates and otherchanges compatible with adult respiratory distress syndrome (ARDS) may characterise the second stage ofethylene glycol poisoning Pulmonary oedema can be secondary to cardiac failure, ARDS, or aspiration ofgastric contents. Symptoms related to acidosis such as hyperpnea and tachypnea are frequently observed;however, major respiratory morbidities such as pulmonary edema and bronchopneumonia are relatively rare andusually only observed with extreme poisoning (e.g., in only 5 of 36 severely poisoned cases).Cardiovascular Effects. Cardiovascular system involvement in humans occurs at the same time as respiratorysystem involvement, during the second phase of oral ethylene glycol poisoning, which is 12- 24 hours afteracute exposure. The symptoms of cardiac involvement include tachycardia, ventricular gallop and cardiacenlargement. Ingestion of ethylene glycol may also cause hypertension or hypotension, which may progress tocardiogenic shock. Myocarditis has been observed at autopsy in cases of people who died following acuteingestion of ethylene glycol . As in the case of respiratory effects, cardiovascular involvement occurswith ingestion of relatively high doses of ethylene glycol.Nevertheless, circulatory disturbances are a rare occurrence, having been reported in only 8 of 36 severelypoisoned cases .Therefore, it appears that acute exposure to high levels of ethylene glycol can causeserious cardiovascular effects in humans. The effects of a long-term, low-dose exposure are unknown.Gastrointestinal Effects. Nausea, vomiting with or without blood, pyrosis, and abdominal cramping and painare common early effects of acute ethylene glycol ingestion. Acute effects of ethylene glycol ingestion inone patient included intermittent diarrhea and abdominal pain, which were attributed to mild colonicischaemia; severe abdominal pain secondary to colonic stricture and perforation developed 3 months afteringestion, and histology of the resected colon showed birefringent crystals highly suggestive of oxalatedeposition.Musculoskeletal Effects. Reported musculoskeletal effects in cases of acute ethylene glycol poisoning haveincluded diffuse muscle tenderness and myalgias associated with elevated serum creatinine phosphokinaselevels, and myoclonic jerks and tetanic contractions associated with hypocalcaemia.Hepatic Effects. Central hydropic or fatty degeneration, parenchymal necrosis, and calcium oxalate crystalsin the liver have been observed at autopsy in cases of people who died following acute ingestion ofethylene glycol.Renal Effects. Adverse renal effects after ethylene glycol ingestion in humans can be observed during thethird stage of ethylene glycol toxicity 24-72 hours after acute exposure. The hallmark of renal toxicity isthe presence of birefringent calcium oxalate monohydrate crystals deposited in renal tubules and theirpresence in urine after ingestion of relatively high amounts of ethylene glycol. Other signs ofnephrotoxicity can include tubular cell degeneration and necrosis and tubular interstitial inflammation. Ifuntreated, the degree of renal damage caused by high doses of ethylene glycol progresses and leads tohaematuria, proteinuria, decreased renal function, oliguria, anuria , and ultimately renal failure. Thesechanges in the kidney are linked to acute tubular necrosis but normal or near normal renal function canreturn with adequate supportive therapy.Metabolic Effects. One of the major adverse effects following acute oral exposure of humans to ethyleneglycol involves metabolic changes. These changes occur as early as 12 hours after ethylene glycol exposure.Ethylene glycol intoxication is accompanied by metabolic acidosis which is manifested by decreased pH and

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bicarbonate content of serum and other bodily fluids caused by accumulation of excess glycolic acid. Othercharacteristic metabolic effects of ethylene glycol poisoning are increased serum anion gap, increasedosmolal gap, and hypocalcaemia. Serum anion gap is calculated from concentrations of sodium, chloride, andbicarbonate, is normally 12-16 mM, and is typically elevated after ethylene glycol ingestion due toincreases in unmeasured metabolite anions (mainly glycolate).Neurological Effects: Adverse neurological reactions are among the first symptoms to appear in humans afterethylene glycol ingestion. These early neurotoxic effects are also the only symptoms attributed tounmetabolised ethylene glycol. Together with metabolic changes, they occur during the period of 30 minutesto 12 hours after exposure and are considered to be part of the first stage in ethylene glycolintoxication. In cases of acute intoxication, in which a large amount of ethylene glycol is ingested over avery short time period, there is a progression of neurological manifestations which, if not treated, maylead to generalized seizures and coma. Ataxia, slurred speech, confusion, and somnolence are common duringthe initial phase of ethylene glycol intoxication as are irritation, restlessness, and disorientation.Cerebral edema and crystalline deposits of calcium oxalate in the walls of small blood vessels in the brainwere found at autopsy in people who died after acute ethylene glycol ingestion.Effects on cranial nerves appear late (generally 5-20 days post-ingestion), are relatively rare, andaccording to some investigators constitute a fourth, late cerebral phase in ethylene glycol intoxication.Clinical manifestations of the cranial neuropathy commonly involve lower motor neurons of the facial andbulbar nerves and are reversible over many months.Reproductive Effects: Reproductive function after intermediate-duration oral exposure to ethylene glycolhas been tested in three multi-generation studies (one in rats and two in mice) and several shorter studies(15-20 days in rats and mice). In these studies, effects on fertility, foetal viability, and malereproductive organs were observed in mice, while the only effect in rats was an increase in gestationalduration.Developmental Effects: The developmental toxicity of ethylene glycol has been assessed in several acute-duration studies using mice, rats, and rabbits. Available studies indicate that malformations, especiallyskeletal malformations occur in both mice and rats exposed during gestation; mice are apparently moresensitive to the developmental effects of ethylene glycol. Other evidence of embyrotoxicity in laboratoryanimals exposed to ethylene glycol exposure includes reduction in foetal body weight.Cancer: No studies were located regarding cancer effects in humans or animals after dermal exposure toethylene glycol.Genotoxic Effects: Studies in humans have not addressed the genotoxic effects of ethylene glycol. However,available in vivo and in vitro laboratory studies provide consistently negative genotoxicity results forethylene glycol.NOTE: Changes in kidney, liver, spleen and lungs are observed in animals exposed to high concentrationsthis substance by all routes.

AROMATIC HYDROCARBON SOLVENT:The material may be irritating to the eye, with prolonged contact causing inflammation. Repeated orprolonged exposure to irritants may produce conjunctivitis.The material may cause skin irritation after prolonged or repeated exposure and may produce a contactdermatitis (nonallergic). This form of dermatitis is often characterised by skin redness (erythema) andswelling the epidermis. Histologically there may be intercellular oedema of the spongy layer (spongiosis)and intracellular oedema of the epidermis.Asthma-like symptoms may continue for months or even years after exposure to the material ceases. This maybe due to a non-allergenic condition known as reactive airways dysfunction syndrome (RADS) which can occurfollowing exposure to high levels of highly irritating compound. Key criteria for the diagnosis of RADSinclude the absence of preceding respiratory disease, in a non-atopic individual, with abrupt onset ofpersistent asthma-like symptoms within minutes to hours of a documented exposure to the irritant. Areversible airflow pattern, on spirometry, with the presence of moderate to severe bronchialhyperreactivity on methacholine challenge testing and the lack of minimal lymphocytic inflammation, withouteosinophilia, have also been included in the criteria for diagnosis of RADS. RADS (or asthma) following anirritating inhalation is an infrequent disorder with rates related to the concentration of and duration ofexposure to the irritating substance. Industrial bronchitis, on the other hand, is a disorder that occursas result of exposure due to high concentrations of irritating substance (often particulate in nature) andis completely reversible after exposure ceases. The disorder is characterised by dyspnea, cough and mucusproduction.

ISOPROPANOL:

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unless otherwise specified data extracted from RTECS - Register of Toxic Effects of Chemical Substances.

TOXICITY IRRITATIONOral (human) LDLo: 3570 mg/kg Skin (rabbit): 500 mg - MildOral (human) TDLo: 223 mg/kg Eye (rabbit): 10 mg - ModerateOral (man) TDLo: 14432 mg/kg Eye (rabbit): 100mg/24hr- ModerateOral (rat) LD50: 5045 mg/kg Eye (rabbit): 100 mg - SEVEREDermal (rabbit) LD50: 12800 mg/kgThe material may cause skin irritation after prolonged or repeated exposure and may produce a contactdermatitis (nonallergic). This form of dermatitis is often characterised by skin redness (erythema) andswelling epidermis. Histologically there may be intercellular oedema of the spongy layer (spongiosis) andintracellular oedema of the epidermis.The substance is classified by IARC as Group 3:

NOT classifiable as to its carcinogenicity to humans.Evidence of carcinogenicity may be inadequate or limited in animal testing.

NONYLPHENOL, ETHOXYLATED:unless otherwise specified data extracted from RTECS - Register of Toxic Effects of Chemical Substances.

TOXICITY IRRITATIONOral (rat) LD50: >2000 mg/kg Skin (human): 15 mg/3D MildDermal (rabbit) LD50: 2830 ul/kg Skin (rabbit): 500 mg Mild

Eye (rabbit): 5 mg SEVEREThe material may produce severe irritation to the eye causing pronounced inflammation. Repeated orprolonged exposure to irritants may produce conjunctivitis.The material may cause skin irritation after prolonged or repeated exposure and may produce a contactdermatitis (nonallergic). This form of dermatitis is often characterised by skin redness (erythema) andswelling epidermis. Histologically there may be intercellular oedema of the spongy layer (spongiosis) andintracellular oedema of the epidermis.

MATERIAL CARCINOGEN MUTAGEN REPROTOXIN SENSITISER SKIN___________ ____________ __________ __________ __________ __________ethylene IARC:3 IOELVglycolmonobutyletheraromatic ATP:Carc.hydrocarbon Cat. 2solventisopropanol IARC:3

CARCINOGENIARC: International Agency for Research on Cancer (IARC)Carcinogens: ethylene glycol monobutyl ether Category:Thesubstance is classified by IARC as Group 3: NOT classifiable as toits carcinogenicity to humans. Evidence of carcinogenicity may beinadequate or limited in animal testing.

SKINIOELV: European Union (EU) First List of Indicative OccupationalExposure Limit Values (IOELVs) - Skin: ethylene glycol monobutylether

CARCINOGENATP: European Union (EU) List of Dangerous Substances (Annex I) -up to the 29th ATP: aromatic hydrocarbon solvent Category: Carc.Cat. 2; R45;Xn; R65

CARCINOGENIARC: International Agency for Research on Cancer (IARC)Carcinogens: isopropanol Category:The substance is classified byIARC as Group 3: NOT classifiable as to its carcinogenicity to

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humans. Evidence of carcinogenicity may be inadequate or limitedin animal testing.

Section 12 - ECOLOGICAL INFORMATION

Harmful to aquatic organisms, may cause long-term adverse effects in the aquaticenvironment.

Section 13 - DISPOSAL CONSIDERATIONS

• Containers may still present a chemical hazard/ danger when empty.• Return to supplier for reuse/ recycling if possible.Otherwise:• If container can not be cleaned sufficiently well to ensure that residuals do not remain or if the container cannot be used to store the same product, then puncture containers, to prevent re-use, and bury at an authorised landfill.• Where possible retain label warnings and MSDS and observe all notices pertaining to the product.• Recycle wherever possible or consult manufacturer for recycling options.• Consult State Land Waste Authority for disposal.• Bury or incinerate residue at an approved site.• Recycle containers if possible, or dispose of in an authorised landfill.

According to the European Waste Catalogue, Waste Codes are not product specific but application specific. Waste Codes should be assigned by the User based on the application in which the product is used.

Section 14 - TRANSPORTATION INFORMATION

HAZCHEM: None

NOT REGULATED FOR TRANSPORT OF DANGEROUS GOODS: ADR, IATA, IMDG

Section 15 - REGULATORY INFORMATION

ANNEX 1Ingredient Annex 1 67/548/EECethylene glycol monobutyl ether 603-014-00-0aromatic hydrocarbon solvent 649-356-00-4isopropanol 603-117-00-0

RISKRisk Codes Risk PhrasesR18 In use, may form flammable/ explosivevapour- air mixture.R19 May form explosive peroxides.

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CD 2008/3 of 22Section 15 - REGULATORY INFORMATION

R20/21/22 Harmful by inhalation, in contact with skin and ifswallowed.

R37/38 Irritating to respiratory system and skin.R41 Risk of serious damage to eyes.R52/53 Harmful to aquatic organisms, may causelong- term adverse

effects in the aquaticenvironment.R65 HARMFUL- May cause lung damage if swallowed.R67 Vapours may cause drowsiness and dizziness.

SAFETYSafety Codes Safety PhrasesS23 Do not breathe gas/fumes/vapour/spray.S51 Use only in well ventilated areas.S09 Keep container in a well ventilatedplace.S53 Avoid exposure - obtain specialinstructions before use.S401 To clean the floor and all objectscontaminated by this

material, use water and detergent.S07 Keep container tightly closed.S13 Keep away from food, drink andanimal feeding stuffs.S46 If swallowed, IMMEDIATELY contact Doctoror Poisons

Information Centre.(show this container or label).

ANNEX 2: Indications of DangerXn Harmful

REGULATIONSNulon Lifter Free and Tune Up (CAS: None):No regulations applicable

ethylene glycol monobutyl ether (CAS: 111-76-2) is found on the following regulatory lists; EU Consolidated List of Indicative Occupational Exposure Limit Values (IOELVs) EU REACH Regulation (EC) No 1907/2006 - Candidate List of Very High Concern - List of Substance Subject to Authorization European Customs Inventory of Chemical Substances (English) European Union - European Inventory of Existing Commercial Chemical Substances (EINECS) (English) European Union (EU) First List of Indicative Occupational Exposure Limit Values (IOELVs) European Union (EU) Inventory of Ingredients used in Cosmetic Products European Union (EU) List of Dangerous Substances (Annex I) - up to the 29th ATP GESAMP/EHS Composite List of Hazard Profiles - Hazard evaluation of substances transported by ships IMO IBC Code Chapter 17: Summary of minimum requirements IMO MARPOL 73/78 (Annex II) - List of Other Liquid Substances International Agency for Research on Cancer (IARC) Carcinogens OECD Representative List of High Production Volume (HPV) Chemicals UK Workplace Exposure Limits (WELs) UK Workplace Exposure Limits (WELs) - Biological Monitoring Guidance Values

aromatic hydrocarbon solvent (CAS: 64742-95-6) is found on the following regulatory lists; EU REACH Regulation (EC) No 1907/2006 - Candidate List of Very High Concern - List of Substance Subject to Authorization European Union - European Inventory of Existing Commercial Chemical Substances (EINECS) (English) European Union (EU) Annex I to Directive 67/548/EEC on Classification and Labelling of Dangerous Substances - Final proposal of the TC C&L for the 30th ATP European Union (EU) Carcinogenic Substances European Union (EU) Control of Major Accident Hazards Involving Dangerous Substances - Seveso Category European Union (EU) List of Dangerous Substances (Annex I) - up to the 29th ATP European Union (EU) Restrictions on the Marketing and Use of Certain Dangerous Substances and Preparations International Council of Chemical Associations (ICCA) - High Production Volume List OECD Representative List of High Production Volume (HPV) Chemicals

isopropanol (CAS: 67-63-0) is found on the following regulatory lists; EU Directive 2002/72/EC Plastic materials and articles intended to come into contact with foodstuffs - Annex II Section A: List of authorised monomers and other starting substances EU Directive 2002/72/EC Plastic materials and articles intended to come into contact with foodstuffs - Annex III Section A Incomplete list of additives fully harmonised at Community level European Customs Inventory of Chemical Substances (English) European Union - European Inventory of Existing Commercial Chemical Substances (EINECS) (English) European Union (EU) Annex I to Directive 67/548/EEC on Classification and Labelling of Dangerous Substances - Final proposal of the TC C&L for the 30th ATP European Union (EU) Control of Major Accident Hazards Involving Dangerous Substances - Seveso Category European Union (EU) Inventory of Fragrance Ingredients (Perfume and Aromatic Raw Materials) European Union (EU) Inventory of Ingredients used in Cosmetic Products European Union (EU) List of Dangerous Substances (Annex I) - up to the 29th ATP European Union (EU) Restrictions on the Marketing and Use of Certain Dangerous Substances and Preparations GESAMP/EHS Composite List of Hazard Profiles - Hazard evaluation of substances transported by ships

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CD 2008/3 of 22Section 15 - REGULATORY INFORMATION

IMO IBC Code Chapter 18: List of products to which the Code does not apply IMO MARPOL 73/78 (Annex II) - List of Other Liquid Substances IMO Provisional Categorization of Liquid Substances - List 1: Pure or technically pure products IMO Provisional Categorization of Liquid Substances - List 2: Pollutant only mixtures containing at least 99% by weight of components already assessed by IMO International Agency for Research on Cancer (IARC) Carcinogens International Air Transport Association (IATA) Dangerous Goods Regulations OECD Representative List of High Production Volume (HPV) Chemicals UK Workplace Exposure Limits (WELs)

nonylphenol, ethoxylated (CAS: 9016-45-9) is found on the following regulatory lists; European Customs Inventory of Chemical Substances (English) European Union (EU) Inventory of Ingredients used in Cosmetic Products European Union (EU) No-Longer Polymers List (NLP) (67/548/EEC) OECD Representative List of High Production Volume (HPV) Chemicals OSPAR List of Chemicals for Priority Action OSPAR List of Substances of Possible Concern Scotland Pollution Inventorynonylphenol, ethoxylated (CAS: 26027-38-3) is found on the following regulatory lists; European Union (EU) No-Longer Polymers List (NLP) (67/548/EEC) GESAMP/EHS Composite List of Hazard Profiles - Hazard evaluation of substances transported by ships IMO Provisional Categorization of Liquid Substances - List 1: Pure or technically pure products OSPAR List of Chemicals for Priority Action Scotland Pollution Inventory

No data available for mineral oil as CAS: Not avail.

This safety data sheet is in compliance with the following EU legislation and its adaptations – as far asapplicable - : 67/548/EEC, 1999/45/EC, 76/769/EEC, 98/24/EC, 92/85/EEC, 94/33/EC, 91/689/EEC, 1999/13/EC, aswell as the following British legislation:- The Control of Substances Hazardous to Health Regulations (COSHH) 2002- COSHH Essentials- The Management of Health and Safety at Work Regulations 1999

Section 16 - OTHER INFORMATION

RISK

Explanation of risk codes used on this MSDSRisk Codes Risk PhrasesR11 Highly flammable.R18 In use, may form flammable/ explosivevapour- air mixture.R19 May form explosive peroxides.R20/21/22 Harmful by inhalation, in contact with skin and if

swallowed.R22 Harmful if swallowed.R36/38 Irritating to eyes and skin.R36 Irritating to eyes.R37/38 Irritating to respiratory system and skin.R41 Risk of serious damage to eyes.R45 May cause CANCER.R51/53 Toxic to aquatic organisms, may causelong- term adverse

effects in the aquaticenvironment.R52/53 Harmful to aquatic organisms, may causelong- term adverse

effects in the aquaticenvironment.R65 HARMFUL- May cause lung damage if swallowed.R66 Repeated exposure may cause skin dryness and cracking.R67 Vapours may cause drowsiness and dizziness.

ANNEX 2: Indications of DangerF Highly FlammableN Dangerous for the environmentT ToxicXi Irritant

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CD 2008/3 of 22Section 16 - OTHER INFORMATION

Xn Harmful

INGREDIENTS WITH MULTIPLE CAS NUMBERSIngredient Name CASnonylphenol, 9016- 45- 9, 26027- 38- 3ethoxylated

REPRODUCTIVE HEALTH GUIDELINESIngredient ORG UF Endpoint CR Adeq

TLVethylene glycol monobutyl 3.6 mg/m3 100 D NA -etherThese exposure guidelines have been derived from a screening level of risk assessment andshould not be construed as unequivocally safe limits. ORGS represent an 8-hour time-weighted average unless specified otherwise.CR = Cancer Risk/10000; UF = Uncertainty factor:TLV believed to be adequate to protect reproductive health:LOD: Limit of detectionToxic endpoints have also been identified as:D = Developmental; R = Reproductive; TC = Transplacental carcinogenJankovic J., Drake F.: A Screening Method for Occupational ReproductiveAmerican Industrial Hygiene Association Journal 57: 641-649 (1996).

EXPOSURE STANDARD FOR MIXTURES"Worst Case" computer-aided prediction of vapour components/concentrations:Composite Exposure Standard for Mixture (TWA) (mg/m3): 250 mg/m³If the breathing zone concentration of ANY of the components listed below is exceeded,"Worst Case" considerations deem the individual to be overexposed.Component Breathing Zone ppm Breathing Zone mg/m3 Mixture Conc: (%).

Component Breathing zone Breathing Zone Mixture Conc(ppm) (mg/m³) (%)

aromatic hydrocarbon solvent 50.00 250.0000 30.0

Operations which produce a spray/mist or fume/dust, introduce particulates to thebreathing zone.If the breathing zone concentration of ANY of the components listed belowis exceeded, "Worst Case" considerations deem the individual to be overexposed.At the "Composite Exposure Standard for Mixture" (TWA) (mg/m3): 250 mg/m³

MSDS SECTION CHANGESThe following table displays the version number of and date on which each section was last changed.Section Name Version Date Section Name Version Date Section Name Version DateEnvironmental 5 24- Dec- 2008

Classification of the preparation and its individual components has drawn on official and authoritative sources as well as independent review by the Chemwatch Classification committee using available literature references.A list of reference resources used to assist the committee may be found at: www.chemwatch.net/references.

The (M)SDS is a Hazard Communication tool and should be used to assist in the Risk Assessment. Many factors determine whether the reported Hazards are Risks in the workplace or other settings. Risks may be determined by reference to Exposures Scenarios. Scale of use, frequency of use and current or available engineering controls must be considered.

For detailed advice on Personal Protective Equipment, refer to the following EU CEN Standards:EN 16 Personal eye-protection

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CD 2008/3 of 22Section 16 - OTHER INFORMATION

EN 340 Protective clothingEN 374 Protective gloves against chemicals and micro-organismsEN 13832 Footwear protecting against chemicalsEN 133 Respiratory protective devices.

This document is copyright. Apart from any fair dealing for the purposes of private study, research, review orcriticism, as permitted under the Copyright Act, no part may be reproduced by any process without written permissionfrom CHEMWATCH. TEL (+61 3) 9572 4700.

Issue Date: 24-Dec-2008Print Date: 24-Dec-2008

NULON LIFTER FREE AND TUNE UP - Part Info Lifter Free and Tune Up MSDS.pdf · NULON LIFTER FREE AND...

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