K11 - Patologi Paru
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Lung PathologyRespiratory System
Block
Dr. H. Soekimin, SpPADr. T. Ibnu Alferraly,
SpPADepartemen Patologi
AnatomiFakultas Kedokteran – USU
2008
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TUBERCULOSIS
– ETIO : M. TUBERCULOSE– LOC : - LUNG >>
- ETC– CLINIC : - VARIATION - DYSPNOE
- LOSS BODY WEIGHT
- FEBRIS - DISTRESS
- SWEATING - COUGH
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TUBERCULOSIS
• TYPE : - PRIMAIR
- SECUNDAIR
- MILIER
• Dx CLINICAL SIGN
• LAB : - SPUTUM - MANTOUX
- BLOOD
• RADIOLOGY
• IMMUNISATION BCG
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TUBERCULOSIS
• PRIMARY :
- FIRST CONTACT
- PRIMAIR LESION (GHON LESION) +
REG. LYMPHNODE (GHON COMPLEX)
- FIBROCALCIFICATION, BACIL (+)
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TUBERCULOSIS
• SECOUNDARY :
- REACTIVATION (PRIMAIR)
- LOC APEX ( +/- BILATERAL )
- FIBROCALCIFICATION
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TUBERCULOSIS• MILIER
- PRIMARY / SECOUNDARY- IMMUNITY <- ORGAN
* MENINGES * KIDNEY * BRAIN * LIVER
* OSTEO * LYMPHA
- GRANULOMA M. TUBERCULOSA (+)
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Ghon Complex
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Typical cavitating granuloma
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Miliary TB• Millet like – grain.• Extensive micro
spread.• Through blood or
bronchial spread• Low immunity• Pulmonary or
Systemic types.
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Miliary TB
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Miliary spread
TB
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Miliary TB Lung
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Cavitary Tuberculosis• When necrotic
tissue is coughed up cavity.
• Cavitation is typical for large granulomas.
• Cavitation is more common in the secondary reactivation tuberculosis - upper lobes.
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Tuberculous Granulomas
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Caseation Necrosis
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Epitheloid cells in Granuloma
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Cells in Granuloma
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Cavitary Secondary TB
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Systemic Miliary TB
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Adrenal TB - Addison Disease
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Testes TB Orchitis.
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TB Peritonitis + liver Miliary TB
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TB Brain – Caudate n.
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TB Intestine
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Prostate TB
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Spinal TB - Potts Disease
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Granuloma or LH giant cell is
not pathagnomonic of TB…!
• Foreign body granuloma.
• Fat necrosis.• Fungal infections.• Sarcoidosis.• Crohns disease.
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PNEUMONIA
• ALVEOLAR INFLAMMATION
• HIGH PROTEIN EXUDATE
• PMN,LYMPHOCYTE & MACROPHAGE INFILTRATION
• LOBAR & BRONCHOPNEUMONIA
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PNEUMONIA
– CLINIC : - PRIMAIR
- SECUNDARY– ETIO :
- BACTERIAL* STREP. PNEUMONIA * STAPH. AUREUS* M. TUBERCULOSA, ETC - VIRAL * INFLUENZAE, MEASLESS - YEAST* CRYPTOCOCCUS, CANDIDA,
ASPERGILLUS
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PNEUMONIA
• ETIO : OTHERS PNEUMOCYSTIS CARINII, MYCOPLASMA,
ASPIRA-TION, LIPID & EOSINIPHYLIC
• HOST REACTION : - FIBROUS - SUPURATIVE
• ANATOMIC : - BRONCHOPNEUMONIA - PNEUMONIA LOBARIS
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BRONCHOPNEUMONIA (PATH)
• CONSOLIDATION PLAQUE BRONCHIOLUS & BRONCHUS AROUND ALVEOLI
• INFANT & OLD & WEAKNESS
PATIENT ( CA, CARDIAC FAILURE,
CHRONIC KIDNEY FAILURE, TRAUMA-
TIC CEREBROVASCULAR),
ACUTE BRONCHITIS,
CHRONIC OBSTR. RESP. TRACT,
OR CYSTIC FIBROSIS & POST OP.
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BRONCHOPNEUMONIA (PATH)
- LESION : - FOCAL (CENTRE OF RESPIRATORY TRACT) /
PLAQUE- BILATERAL ( BASAL )- AUSCULTATION CREPITATION
- ETIO : - Staphylococcus - Streptococcus- H. influenzae - Coliform, Yeast
- HP : - ACUTE INFLAMMATION + EXUDATE
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LOBAR PNEUMONIA
• ALL OF LOBUS• INFANT & OLD PATIENT <<• AGE : 20 – 50 YRS• MAN > WOMEN• 90 % STREP. PNEUMONIA
(PNEUMOCOCCUS)• CLINIC COUGH RUSHTY SPUTUM
FEBRIS (40OC), INSPIRATION PAIN, BRONCHIAL RESPIRATION
• KLEBSIELLA OLD, DM, ALKOHOLIC
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PNEUMONIA (STADIUM)
• CONGESTION :- I 24 HRS
- EXUDATE (PROTEIN) ALVEOLI SPACE - OEDEMA PULMONAL - RED COLOUR
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PNEUMONIA (STADIUM)
• RED HEPATISATION
- > 24 HRS DAYS
- ACCUMULATION (LYMPHOCYTE,
MACROPHAGE) ALVEOLAR
- EXTRAVASATION RED CELLS
- FIBRINOUS EXUDATE (PLEURAL)
- GAS (-) , CONSOLIDATION (HEPAR)
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PNEUMONIA (STADIUM)
• GRAY HEPATISATION
- FEW DAYS (STAD II)
- FIBRINE (ACCUMULATION)
- WHITE & RED CELLS (LYSIS)
- DARK GRAY
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PNEUMONIA (STADIUM)
• RESOLUTION :
- 8 – 10 DAYS UNTREATED
- EXUDATE & INFILTRATION DEBRIS (ABSORB)
- ALVEOLUS WALL (N)
- ALL OF CASE RECOVERY (+)
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SPECIAL PNEUMONIA
• NORMAL HOST- MYCOPLASMA & VIRAL- LEGIONNAIRES
• ABNORMAL HOST (IMMUNE)- PNEUMOCYSTIS CARINII- CANDIDA & ASPERGILLUS- CYTOMEGALO & MEASLESS
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PNEUMONIA NON INFECTION
• ASPIRATION
- LIQUID / FOOD CONSOLIDATION INFLAMMATION (SECONDAIRY)
- RISK FACTOR : POST OP, COMA, STUPOR
LARYNX CA, ETC- LESION : POSITION !!
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PNEUMONIA NON INFECTION
• LIPID PNEUMONIA
- ENDOGEN OBSTRUCTION (MACROPHAGE
GIANT CELL)- EXOGEN
PARAFFIN LIQUID INTERSTITIAL FIBROSIS
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PNEUMONIA NON INFECTION
• EOSINIPHYLIC PNEUMONIA
- EOSINOPHYL >> INTERSTITIAL & ALVEOLI
(ASTHMA, ASPERGILLUS, MICROPHYLARIA),
LOEFFLER SYNDROME
(IDIOPATIC)
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OBSTRUCTION LUNG DISEASE
• LOCAL
• DIFUSE ( CHRONIC )
- CHRONIC BRONCHITIS
- EMPHYSEMA
- ASTHMA
- BRONCHIECTASIS
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LOCAL OBSTRUCTION LUNG
DISEASE
• MECHANIC FACTOR OBSTRUCTION (C. AL, TUMOR) COLLAPS /
EXPANSIVE• COMPLICATION ( LIPID, INF.,
PNEUMONIA)• FUNCTION TEST NORMAL
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DIFUSE OBSTRUCTION LUNG DISEASE
• CHRONIC BRONCHITIS
• EMPHYSEMA
• ASTHMA
• BRONCHIECTASE
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CHRONIC BRONCHITIS
• ETIO : - SMOKERS >>,
- POLUTION
STREP. PNEUMONIA
H. INFLUENZAE & VIRAL
SEVERE HYPERCAPNIA, HYPOXIA & CYANOSIS (BLUE
BLOATERS)
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• Chronic Bronchitis • Definition : Persistent cough with sputum production for at least 3
months in at least 2 consecutive years• Cause : Initiated by smoking (by causing Chronic irritation of the
bronchial mucosa)– infections are secondary
• Pathology: Hypertrophy of mucus glands Hyper secretion of mucus
• Reid Index = ratio of thickness of mucous gland layer (CD) to the thickness between the epithelium and the cartilage (AB) (normally 0.4). The closer to 1 means there’s an increase in thickness and correlated to progression of disease
NormalCD/AB = 0.4
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Chronic Bronchitis
• Clinical course• Bronchi & bronchioles are obstructed by mucus plugs
• bronchiolitis obliterans.
• In long-standing cases,
• squamous metaplasia & dysplasia (precancerous)
• predisposes for squamous cell carcinoma
??
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EMPHYSEMA
• ALVEOLUS DILATATION + ELASTICITY (<<)
• FORM : - CENTRILOBULAR EMPHYSEMA- PANLOBULAR EMPHYSEMA
- PARASEPTAL EMPHYSEMA - IRREGULAR EMPHYSEMA
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EMPHYSEMA• OTHER FORM
- BULOSA EMPHYSEMA- INTERSTITIAL EMPHYSEMA- SENILE EMPHYSEMA
• CLINIC : - DYSPNOE
- COUGH - SPUTUM
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ASTHMA
• BRONCHUS IRRITABLE (+) BRONCHUS SPASM
MUCOUS (>>) OBSTRUCTION DYSPNOE
• TYPE : - ATOPIC - NON ATOPIC - ASPIRINE INDUCED - OCCUPATIONAL - ALLERGIC (ASPERGILLUS)
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Bronchial Asthma
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NON ATYPIC ASTHMA
• T. RESP. INFECTION CHRONIC BRONCHITIS
• ALLERGEN TEST (-)• LOCAL IRRITATION BRONCHUS
CONSTRICTION
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ASPIRINE INDUCED ASTHMA
• MECHANISM (?)
+/- PROSTAGLANDINE DECREASE / LEUKORINE INCREASE RESP. TR. IRRITABLE
• RHINITIS, NASAL POLYPS,
URTICARIA (+)
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OCCUPATIONAL ASTHMA
• REACTIVE HYPERSENSIVITY (ALLERGEN)
DYSPNOE COUGH (CHRONIC)
• ALLERGEN :
- WOOD
- CHEMICAL
- ETC
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ASPERGILLUS BRONCHITIS ALLERGY
• SPORA ASPERGILLUS FUMIGATUS
• HYPERSENSITIVITAS REAC.
• DYSPNOE
• MUCOUS GLOBULE ASPERGILLUS HYPAE (+)
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BROCHIECTASIS.
• ETIO : - BRONCHUS OBSTRUCTION
- INFECTION (SEVERE) - CONGENITAL (<<<)
• BRONCHUS & BRONCHIOLUS DILATATION
• COUGH (CHRONIC), DYSPNOE, SPUTUM (>>>) + BLOOD
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BRONCHIECTASIS• CLINIC :
- LOBUS INFERIOR + INFECTION - CLUBBING FINGER
• COMPLICATION PNEUMONIA, EMPIEMA, SEPTICAEMIA, MENINGITIS, ABSCESS METASTASIS
(CEREBRAL), AMYLOID (+)
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Bronchiectasis Gross
• Distended peripheral bronchi (Due to weakening of wall)
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LUNG NEOPLASMA• PRIMARY LUNG CA
• ANOTHER LUNG NEOPLASMA
- BENIGN
- MALIGNANT
• SECONDARY LUNG NEOPLASMA
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"It is nice to have money and the "It is nice to have money and the things that money can buy, but it's things that money can buy, but it's important to make sure you important to make sure you haven't lost the things money can't haven't lost the things money can't buy."buy."
George Lorimer1867-1937, Editor of "Saturday Evening Post"