CVS REport
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Transcript of CVS REport
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VALERA, ERLLYNESOSA, DIANE
REBONQUIN, MARIANEPONFERRADO, MARK ANGELO
REGULATION OFBLOOD PRESSURE
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Maintenance of Resting BloodPressure
BLOOD PRESSURE HOMEOSTASIS
Situational adjustment of BloodPressure
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Barorecetor Re!e"
#$emorecetor Re!e"
Brain %#&S'Isc$aemicResonse
Renal(Bod) *luid#ontrol
S)stem
INTERMEDIATE
LONG-TERM
BLOOD PRESSURE REGULATORYMECHANISM
#atec$olamines
Renin(Angiotensin
(AldosteroneS)stem
+asoressin
IMMEDIATE
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IntegratngCenter
Recetor
E,ectors
A,erent
E,erent
Bran Ste!"Me#$%%a
S&na% '(r#"SYMPATHETICNERVOUSSYSTEM
H)&(t*a%a!$+
Barorecetors
#$emorecetors
Heart- Blood.essels
Adrenal
medulla/#atec$olamines
0idne)/Acti.ation of
RAA s)stem
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BLOOD PRESSURE
Car#a' O$t&$t
a% Per&*era% Re++ta
CO TPR BP
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DIRECT CARDIOVASCULAREFFECTS
SYMPATHETIC TONE S)!&at*et'
($t.(/
HR
VENOCONSTRICTI
ON
ARTERIOLAR
CONSTRICTION
VENOUSRETURN
SV
CAPILLARYPRESSURE
ISFFORMATION
PLASMALOSS
CO TPR BP
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NEUROENDOCRINE EFFECTS
SYMPATHETIC TONE S)!&at*et'
($t.(/
ADRENAL
MEDULLA
0UTAGLOMERULA
R CELLS INKIDNEY
CARDIOVASCULAREFFECTS
SECERETION OFCATECHOLAMINES ACTIVATION OF
RENIN-ANGIOTENSIN-
ALDOSTERONESYSTEMVOLUMEEFFECTS
BLOOD PRESSURE
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BARORECEPTOR REFLE
Short-Term &Intermediate
Blood Pressure
Regulation
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BARORECEPTORS
ATRI
A
LO1 PRESSUREBARORE#EPTORS
+olume recetorsAtrium
HI2H PRESSUREBARORE#EPTORS
Arteries %#arotid sinus-
Aortic Arc$
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CHEMORECEPTORREFLE
Short-Term BloodPressure
Regulation
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ARTERIALCHEMORECEPTORS
BLOOD FLO1
O2 De%3er) t( t++$e+4STAGNANT HYPOIA5
CO2 $&ta6e 7r(! t++$e+
STAGNATION
BP
O2
CO2St!$%ate VMC
S)!&at*et'#+'*argeBLOODPRESSURE
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CNS ISCHAEMICRESPONSE
Short-Term BloodPressure
Regulation
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CNS ISCHAEMIC RESPONSE
BLOOD FLO1
O2 De%3er) t( t++$e+4STAGNANT HYPOIA5
CO2 $&ta6e 7r(! t++$e+
STAGNATION
BP
O2
CO2
S)!&at*et'#+'*argeBLOODPRESSURE
Cere8ra%
VMC
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RENIN-ANGIOTENSIN-
ALDOSTERONESYSTEM
IntermediateBlood PressureRegulation
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RENIN-ANGIOTENSIN-ALDOSTERONESYSTEM
Rena% Per7$+(nPre++$re
0$9tag%(!er$%ar 'e%%+ na:erent arter(%ar !$+'%e
n K#ne)
Bar(re'e&t(rRe.e9
BLOODVOLUME;PRESSUR
E
Ang(ten+n-C(n3ertngEn
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+asoconstriction
*acilitates releaseof &oreine$rine
Release of
+asoressin
Increase 1aterinta3e
Renal rea4sortionof Sodium 5 1ater
ANGIOTENSIN II
TP
R
#OBLOOD+OLUME
B
P
+ascularsmoot$muscle
S)mat$eticner.eendings
H)ot$alamus
Secretion of
ALDOSTERO&E
Adrenal#orte"
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CATECHOLAMINES
IntermediateBlood PressureRegulation
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N(re&ne&*rne
CATECHOLAMINES
= >re'e&t(r+
? re'e&t(r+
VASOCONSTRICTION
E&ne&*rne
CARDIOSTIMULATORY EFFECTS"
D(&a!ne
BLOOD PRESSURE
Automaticit) %SA &odal
Disc$arge'#onducti.it)E"cita4ilit)M)ocardial contractilit)
TPR
HR
SV
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RENAL-FLUID BLOODREGULATION
REBO&6UI&- MARIA&E
Long -TermBlood Pressure
Regulation
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1$en 4lood .olume and BP is increased-0ID&E7S e"crete e"cess !uid 4)
PRESSURE DIURESIS- Increased urine formation as a result of
increased glomerular 8ltration due to raisedrenal erfusion ressure
PRESSURE &ATRIURESIS (Increased urinar) e"cretion of sodium as a
result of increased glomerular 8ltration of
sodium due to raised renal erfusion ressure
RENAL REGULATION OF B@P
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1$en 4lood .olume and BP is DE#REASED/
Decrease glomerular caillar) H)drostaticPressure (9 Decreased 2lomerular *iltration Rate
OLI2URIA( decreased urine formation
A&URIA( renal s$utdo:n( no urine formation
RENAL REGULATION OF B@P
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RESTORATION OF FLUID VOLUME OF THE BODY BY ADH IN VOLUME DEPLETION STATE.[NTS: NUCLEUS TRACTUS SOLITARIOUS]
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SEQUENTIAL EVENTS BY !I"! IN"REASE# SALT INTA$EIN"REASES T!E ARTERIAL PRESSURE%
Increased sa! "n!a#e
Increased e$!race%ar &'%(e
Increased ar!er"a )ress%re
Decreased ren"n and an*"'!ens"n
Decreased rena re!en!"'n '+ sa! and ,a!er
Re!%rn '+ e$!race%ar &'%(e a('s! !'n'r(a
Re!%rn '+ ar!er"a ress%re a('s! !' n'r(a
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Than'ou(