Common Cases in Vascular Surgery

72
Miss Kaji Sritharan Specialist Registrar in General Surgery Northwest Thames, London Deanery Dec 2009

Transcript of Common Cases in Vascular Surgery

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Miss Kaji SritharanSpecialist Registrar in General Surgery Northwest Thames, London Deanery 

Dec 2009

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Common History Cases: Lower Limb PVD AAA

Carotid Disease

Short Cases Varicose Veins

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Establish:

Whether symptoms: 

Acute

Acute on

 chronic

Chronic

Viability of the limb

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Acute Limb Ischaemia Pain

Pale or white

Perishingly cold

Pulseless

Paraesthesiae

Paralysis Dictates

urgency

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Remember:

60% ‐ thrombotic occlusion of pre‐existing stenotic arterial segment

30% ‐ embolus (80% from left atrial appendage in assoc AF)

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Management of Acute Limb Ischaemia

Management of Acute Limb Ischaemia

Paralysis & ParaesthesiaParalysis & ParaesthesiaSensation &

Movement

intact

Sensation &

Movement

intact

1. Resuscitate

2. IV heparin

3. Urgent surgery – embolectomy/ bypass

1. Resuscitate

2. IV heparin

3. Urgent surgery – embolectomy/ bypass

1. Optimise patient

2. IV heparin

3. Arteriogram – plan for bypass

4. Observe limb for deterioration

1. Optimise patient

2. IV heparin

3. Arteriogram – plan for bypass

4. Observe limb for deterioration

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History Claudication 

(?deteriorated) 

Rest pain

Tissue Loss

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Pain calf, thigh or buttock,

after walking predictable distance

resolution of pain after rest

Not while standing or sitting.

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Pain in the toes/forefoot at rest.

Initially only at night, relieved by

dependency

Progresses to constant pain

Can occur in areas of tissue loss

elsewhere

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Dry/wet gangrene, usually painful 

NB: diabetic foot wounds (not always painful)

Ulcers – can  be of mixed aetiology

Amputations

‐ when and why? Diabetic? Was 

revascularisation attempted 

 before?

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Arterial Ulcers or Gangrene

ORRest pain of 2 weeks or more requiring Opiate 

Analgesia

AND 

Absolute Ankle Pressure < 50mmHg 

or Toe Pressure <30mmHg

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Do you smoke or have you ever smoked? If yes, estimate pack year history, record how long cessation. 

Are you diabetic? If yes, what do you use to control your  blood sugar levels? 

Do you have, or take medicine to control, high cholesterol or high  blood pressure? 

Have you ever suffered angina, had a heart attack, 

treatment of

 heart

 disease

 (angioplasty

 or

 CABG)

Have you ever suffered a stroke or ministroke ʹTIAʹ. 

Have any

 of

 your

 close

 family

 suffered

 from

 heart

 

disease or PVD?

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General approach

Inspect

- General- Focussed: look for evidence of adequate orinadequate perfusion

Palpate/Auscultate the major pulses to

work out the likely level of the problem

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Wash/gel

Introduce

Permission

Explain

Position

Expose

Tender?

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You must listen to the examiner as the

instructions may be more or lessexplicit about what is required

Even if the instruction is to examinethe lower limbs you must make a

reference to how you would usuallystart your examination at the hands

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Look for clues around the  bed

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INSPECT FOR:

Nicotine staining

Pallor

Muscle wastingSplinter haemorrhage

Venous guttering

Scars

Fistula

Tissue loss

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PALPATE FOR:

WarmthCapillary refill

Radial pulse(AF? R-R delay?)

Ulnar pulse

Allen’s Test

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Brachial pulse

Axillary pulse

Subclavian abnormality

Carotid bruit

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Look

Colour 

mottling, marbled, pallor, venous 

guttering

Trophic changes hair loss, thin skin, 

muscle atrophy

Scars 

Amputations

Ulcers + Gangrene

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Scars to note:

Carotid end‐arterectomyCABG

Thoraco‐abdominalMidline laparotomyVertical groinAbove‐knee medialBelow‐knee medial

LSV 

distributionLateral calfFoot

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Scars to note:

Fasciotomy

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Feel

Temperature 

difference CRT <2sec

Pulses 

Femorals

Popliteals

Posterior Tibial

Dorsalis pedis

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Auscultate Bruits

Buergers Test/Angle 

angle foot goes white

< 20 degrees – severe ischaemia

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To Finish: Examine

 Neuro

 lower

 limb

 + Fundoscopy

Examine the remainder Peripheral Vascular System

Examine Abdomen AAA

Measure ABPIs

Dipstick Urine

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Simple: Blood tests: Cholesterol,

HbA1c, U&E

Duplex Ultrasound(CTA or MRA depending on localskills)

Angiography (like cardiac like to

perform intervention at same sitting)

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Management of 

PVD

Intermittent

claudicant

Critical

ischaemia

Medical therapies

Encourage exercise

STOP SMOKING

BP control

Statins & aspirin

Diabetic control

Watch retino/nephopathy

Revascularisation

Angioplasty +/- stenting

Bypass procedure

Revascularisation

Angioplasty +/- stenting

Bypass procedure

Sepsis control

Antibiotics + DM control

DebridementAmputation

+ Medical therapies

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Definition: necrosis oftissue with mummificationor putrefaction

Types :1. Dry – well demarcated, auto-

amputate

2. Wet – due to trauma, acuteischaemia & infection. Poorlydemarcated and spreading.

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Vascular (thrombosis, embolus, critical 

ischaemia, 

Buerger’s 

disease, 

Raynaud’s 

disease)

Diabetes

Trauma – cold, heat, pressure Drug induced e.g. ergot poisoning

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Indications: 

“DEAD, DYING OR DANGEROUS”

Vascular (80

‐90%)

Infection (Osteomyelitis, Gas gangrene)

Trauma (Burns, Frostbite)

Malignancy

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Definition

Break 

in 

continuity 

of 

an 

epithelial 

surface

Aetiology

Vascular (arterial,

 venous

 or

 mixed)

Neuropathic

Traumatic

Malignant

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Site

Size Shape

Edge

Base

Depth

Surrounding Tissue

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Site: overlies lateral malleolus Edge: punched out

Base: deep; often lacks granulation tissue;necrotic

Minimal exudate

Painful +/- cellulitis Gangrene

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Medical

‐ Pain control‐ Optimise risk factors 

‐ ?intravenous prostaglandins

‐ Antibiotics if infection

Surgical

‐ Debridement (surgical,dressings,maggots)/amputation‐ Improve  blood supply 

(lumbar sympathectomy,

 angioplasty,

 BPG)

 

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Site: gaiter area; lower 3rd medial aspect leg

Shape: varies – can be very large, irregular Edge: sloping and shallow

Base: often pink granulation tissue +/-seropurulant discharge

Surrounding skin: induration, pigmentation,

lipodermatosclerosis Painful

NB. Examine for VVs, check ABPI’s

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Exclude arterial component

If mixed – correct arterial factor

Non-Surgical – high success (80-90% at 1 year)

Rest + elevate leg Four layer compression bandaging

Once healed – grade II compression hosiery

Surgical Exclude malignancy

Skin grafting if clean

Treat primary varicose veins

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Site: pressure areas

Edge: even wound margins; callousaround ulcer

Base: granulation tissue present (unlessco-existing PVD); low to moderateexudate

Absence of pain Peripheral pulses present

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Common: 10-20% population

Women > men

Definition

Tortuous, dilated, elongated veins of the superficial venous system

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Superficial veins Long saphenous vein (LSV)

Short saphenous vein (SSV)

Deep veins

Perforator veins Giacomini vein

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Aetiology

Acquired

Valve Muscle pump Venous return

Congenital

IncompetenceDeep vein thrombosis Immobility Pregnancy

Abdo/ pelvic mass

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Focussed history

‘Primary LSV' +/- signs at ankle

Ulcer of unknown aetiology(venous/arterial/mixed)

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Age, Occupation

How long have you had varicose veins? How do your veins trouble you?

Cosmesis

Swelling – typically end of day Aching

Pruritis

Cramps Ulcers +/- infection - periostitis

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Age

Female Family history (uncertain as to why!!!)

Pregnancy (impaired venous return as well as hormonal effect on vein wall)

PMH of DVT or long bone fracture

Contributing factors: HRT, OCP, obesity, sedentary lifestyles, and

professions that require prolonged standing or sitting

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Listen to the examiner’s instructions

Wash/gel

Introduce

Consent

Explain

Position/Expose Pain or tenderness anywhere?

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Inspect front and back for:

Obvious varicosities and their distribution

Signs (skin changes) at the ankle/calf 

Signs of previous surgery

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Saphenovarix

- Assess for coughimpulse

Feel- Tap test

- Temperature- Tethering

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Tourniquet test versus Trendelenberg test

GIVE CLEAR INSTRUCTIONS

Elevate the limb, milk the veins

Apply tourniquet to upper thigh

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Immediate filling of veins, release the tourniquetand tell the examiner:

'the filling of the varicose veins is not controllable atthe level of the SFJ'

OR

Veins not immediately filled, very slow filling =

undo the tourniquet and tell the examiner:'the filling of the varicose veins is controlled at thelevel of the SFJ'

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Auscultate the varicosities that do not emptylying flat ‘machinery murmur’ of AVM

Offer to palpate lower limb pulses +/- ABPIs

Perthes test

Offer to perform Abdo/Pelvic/Scrotal/rectal

examinations

Wash hands, ensure patient re-covered

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Hand helddoppler

Duplex imaging

Venography

Abdo/Pelvisultrasound

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Please examine this patients superficialvenous system in the lower limb.

TrendelenbergTrendelenberg

and tourniquetand tourniquet

test positivetest positive

How would you treat varicose veins?How would you treat varicose veins?

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Leg elevation

Regular walking to improve calf muscle

pump Class II support stockings – above or below

knee

Skin changes require - 4 layer bandaging(Charing cross)

Eczema – topical emolliants Thrombophlebitis – NSAIDs

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Open Surgery:- High tie and strip – ligation of SFJ

+/- avulsions – removal of varicosities

Foam injections

Sclerotherapy:

- 1% Sodium tetradecyl sulphate

EVLT or VNUS

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Accumulation fluid in interstitiumdue to problem with lymphatic

drainage Typically bilateral and non-pitting

Aetiology:

Primary: Milroy’s disease Secondary:

○ Lymphadenectomy

○ Malignancy

○ Post radiotherapy

○ Infections: Filiarisis