Chest Pain Non Trauma

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    Chest Pain

    (Non - Trauma)Oleh :

    M. Samsul Arifin 0810710072

    Nur Hidayati Azar 0810710088

    Peppy Tria 0810710092Tita Luthfia S 0810710107

    Anantika Putri 0810713004

    Arrasyid Indra 0710713025

    Pembimbing :

    dr. Munsifah Z., SpEM

    FK UNIV. BRAWIJAYA/RSU DR. SAIFUL ANWAR

    MALANG2013

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    Introduction

    Chest painmany

    symptoms overlap

    Goal in ED is to r/o life

    threatening causesof chestpain

    Need appropriate history,

    physical exam, and ancillary

    tests

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    LIFE-THREATENING CAUSES

    Acute myocardial infarction

    Unstable angina

    Aortic dissection

    Pulmonary embolism

    Tension pneumothorax

    Oesophageal rupture

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    NON LIFE-THREATENING CAUSES

    Cardiac Stable angina

    Prinzmetal angina

    Pericarditis/myocarditis

    Respiratory Simple pneumothoraxPneumonia with pleurisy

    Gastrointestinal Reflux oesophagitis

    Oesophageal spasm

    Referred pain Gastritis/PUD

    Biliary disease

    Subphrenic abscess/inflammation

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    Time & character ofonset

    Quality

    Location Radiation

    Associated Symptoms

    Chest Pain - History

    Aggravating symptoms

    Alleviating symptoms

    Prior episodes

    Severity Review risk factors

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    Time & Character of Onset

    Abrupt onset with greatest intensity at start :

    Aortic dissection, PTX, Occasionally PE will present in this

    manner

    Chest pain lasting seconds or constant over weeks is not

    likely to be due to ischemia

    Pleuritic Pain: PE, Pleurisy, Pneumonia, Pericarditis, PTX

    Esophageal: Burning

    MI: squeezing, tightness, pressure, heavy weight on chest,

    can also be burning

    Sharp, tearing, ripping pain: Aortic Dissection

    Quality

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    If very localized, consider chest wall pain or pain of pleural

    origin

    Location

    Associated Symptoms

    Fevers, chills, URI symptoms, productive cough : Pneumonia Nausea, vomiting, diaphoresis, shortness of breath: MI Shortness of breath: PE, PTX, MI, Pneumonia, COPD/Asthma Asymmetric leg swelling: DVTPE

    With new onset neurologic findings or limb ischemia:consider dissection Pain with swallowing, acid taste in mouth: Esophageal

    disease

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    Activity: Consider ischemic heart disease Food: Consider esophageal disease

    Position: If worse with laying back, consider pericarditis.

    Swallowing: Esophageal disease

    Movement: Chest wall pain Respiration: PE, PTX, Pneumonia, pleurisy

    Palpation: Chest Wall Pain

    Aggravating Symptoms

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    Alleviating Symptoms

    Rest/ Cessation of Activity: Ischemic Sitting up: Pericarditis

    Antacids: Usually GI system

    Prior Episodes

    Have they had this kind of pain before Does this feel like prior cardiac pain, esophageal pain, etc

    What diagnostic work-up have they had so far? Last ECG,echo, last stress test, last cath, etc

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    Risk Factor

    Hypertension, DM, high cholesterol, tobacco, family history Long plane trips, car rides, recent surgery or immobility,

    hypercoagulable state: PE

    Uncontrolled HTN/ Marfans: Dissection

    Rheumatic Diseases: Pleurisy Smoking: COPD, Ischemia

    Severity

    Severity of chest pain

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    Chest PainPhysical Examination

    Review vital signs* Fever: Pericarditis, Pneumonia

    * Check BP in both arms: Dissection

    * Decreased sats: More commonly in pneumonia, PE, COPD

    * Unexplained sinus tachy: consider PE

    Neck

    * Look for tracheal deviation: PTX

    * Look for JVD: Tension PTX, Tamponade, (CHF)

    * Look for accessory muscle use: Respiratory Distress(COPD/ASTHMA)

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    Chest PainPhysical Examination

    Chest wall exam* Look for lesions: Herpes Zoster

    * Palpate for localized tenderness: Likely musculoskeletal cause

    Lung exam* Decreased breath sounds/hyperresonance: PTX

    * Look for Rhonchi: Pneumonia

    * Listen for wheezing/prolonged expiration: COPD

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    Chest PainPhysical Examination

    Cardiovascular Exam* Assess heart rate

    * Listen for murmurs, S3/S4

    * Pericardial friction rub: pericarditis

    * Muffled heart sounds: Tamponade* Assess distal pulses

    Abdominal Exam

    * Assess RUQ and epigastrium

    NEURO EXAM

    * Chest pain +neurologic findings: consider dissection

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    Chest PainAncillary Tests

    LABS CBC, PT/PTT, D dimer (PE), Blood cultures(pneumonia), Sputum cultures (pneumonia), Peakflow (Asthma), ABG, Cardiac Enzymes (MI), ESR(pericarditis)

    CXR - Rib fractures

    - Hamptons Hump/Westermarkssign: PE

    - Infiltrates: Pneumonia

    - Widened mediastinum: Aortic dissection

    - Pneumothorax

    - Cardiac size: enlarged silhouette without CHF:pericardial effusion

    ECG MI

    CT Scan CT Scan Thorax if suspect PE or Aortic Dissection

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    Management Non Traumatic

    Chest Pain

    Ensure vital sign are stable. If unstable, patient in distress

    and diaphoretic, bring patient to resuscitation area

    immediately

    Put patient on oxygen supplementation, pulse oximetry,continuous ECG monitoring, blood pressure monitoring

    Set up IV line and take blood test

    Give pain relief depending on provisional diagnosis

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    ACUTE MYOCARDIAL INFARCTION (AMI)

    Definisi

    Sering disebut serangan jantung, merupakan akibat darigangguan aliran darah ke bagian jantung, menyebabkan

    kematian sel jantung mati.

    Tanda dan Gejala

    Nyeri dada tiba-tiba (menyebar ke lengan kiri atau lehersebelah kiri), sesak nafas, nausea, vomiting, palpitasi,berkeringat, dan cemas.

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    Management AMI

    O2Aspirin 300-

    320 mgCPG 300

    mg

    S/L GTN 1 tabstat, repeatECG after 5minutes (to

    exclude ECGchanges dtcoronary

    spasm)

    Morphine iv

    2-5 mgslow bolus

    (ifnecessary)

    IV GTN 20-200microgram/min, increase by 5-

    10microgram/min

    at 5-10 minintervals (ifnecessary)

    Considermyocardial

    salvagetherapy

    Considerthrombolytic

    therapy

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    Indikasi Terapi Thrombolytic

    Typical chest pain of AMI

    ST elevation of at least 1 mm in at least 2 inferior ECG

    leads or elevation of at least 2 mm in at least 2

    contiguous anterior leads

    < 12 h from chest pain onset

    < 75 y.o of age

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    UNSTABLE ANGINA PECTORIS

    Unstableanginaresults

    from thesudden

    rupture of aplaque

    Rapidaccumulationof platelets at

    the rupturesite and asudden

    increase inobstruction toblood flow inthe coronary

    artery

    Accumulation

    of plateletsandobstruction toblood flow can

    result in aheart attack

    Risk of heartattack remains

    even if theunstableangina

    symptomslessen ordisappear

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    Tanda dan Gejala UAP

    Bisa berlangsung selama 5-20 menit. Gejala yang dirasakan :

    Nyeri atau tertekan

    Rasa berat dan tidak nyaman pada dada, leher,

    kerongkongan, bahu dan lengan

    Rasa terbakar atau indigestion Sesak

    Unstable angina terjadi tanpa didahului tanda awal dan

    terjadi saat istirahat sehingga sering mengakibatkan ansietas.

    Gejala lain yang bisa terjadi : Mual

    Nyeri kepala

    Keringat berlebihan

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    Management UAP

    Nitrates

    - Dilatasi pembuluh darah

    - Mengurangi resistensi pembuluh darahmengurangi

    kerja jantung (workload) Beta-blockers

    - Memperlambat denyut jantung dan mengurangi tekanan

    kontraksi otot jantung

    Calcium channel blockers

    - Dilatasi pembuluh darah dan mengurangi tekanan darah

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    PULMONARY EMBOLISM (PE)

    PE is a blockage of themain artery of the lungor

    one of its branches by a

    substance that has

    travelled from elsewherein the body through the

    bloodstream (embolism)

    Origin >> DVT

    Virchows Triad(Endothelial Injury, Stasis,

    Hypercoagulability)

    http://en.wikipedia.org/wiki/Pulmonary_arteryhttp://en.wikipedia.org/wiki/Embolismhttp://en.wikipedia.org/wiki/Embolismhttp://en.wikipedia.org/wiki/Pulmonary_artery
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    Clinical Symptoms of PE

    Clinical symptoms suggestive of PE: Dyspnea

    Chest pain (Pleuritic/non pleuritic)

    Cough

    Orthopnea Calf and/or thigh pain or swelling

    Wheezing

    Common signs:

    Tachypnea

    Tachycardia

    Rales

    Decreased breath sounds Jugular venous distension

    Accentuated pulmonic

    component of second

    heart sound

    Symptoms/ signs of lower extremity DVT include :

    edema, erythema, tenderness or a palpable cord.

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    PE Management

    Initiate Heparin- Unfractionated Heparin: 80 Units/Kg bolus IV, then

    18units/kg/hr

    - Fractionated Heparin (Lovenox): 1mg/kg SubQ BID

    - If high pre-test probability for PE, initiate empiric heparinwhile waiting for imaging

    - Make sure no intraparenchymal brain hemorrhage or GI

    hemorrhage prior to initiating heparin.

    Consider Fibrinolytic Therapy:

    - Especially if PE + hypotension

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    PE Management

    Surgery and Other Prosedure Consider Clot removal. For a very large clot in lung and in

    shock, doctor may thread a thin flexible tube (catheter)

    through blood vessels and suction out the clot.

    Vein filter.Filter insertion is typically reserved for peoplewho can't take anticoagulant drugs or when anticoagulant

    drugs don't work well enough

    Surgery.IThis happens infrequently, and the goal is to

    remove as many blood clots as possible, especially if there'sa large clot in main (central) pulmonary artery.

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    AORTIC DISSECTION

    Aortic dissection is an acute event where blood enters

    the aortic wall through a tear of the intima followed by

    extravasation of blood into the media.

    Currently believed the process begins with an intramuralhematoma

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    Etiology

    Degenerative

    Hypertension

    Pregnancy

    Skeletal (scoliosis) Connective tissue (Marfans)

    Mycotic aneurysm

    Takayasu (giant cell) arteritis

    Aortic laceration/coarctation

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    Aortic Dissection

    Stanford Classification

    Type A -involves ascending

    aorta

    Type Binvolves

    descending aorta

    DeBakey Classification

    Type Iascending, arch &

    descending aorta

    Type IIascending only

    Type IIIdescending only

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    Aortic Dissection

    ClinicalFeatures

    >85%abrupt,

    severe painin chest or

    b/wscapula

    50%ripping or

    tearing

    Pain inanteriorchest

    ascendingaorta (70%)

    Back pain(less

    common)descendingaorta (63%)

    Ifdissection

    into carotidclassic

    neurosymptoms

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    Aortic Dissection

    Physical Exam

    Usually normal heart and lung exam

    May have aortic insufficiency

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    Aortic Dissection

    T

    reathype

    rtension-blocker

    Esmolol 500g/kgIV bolus over 1minute then 50-

    150 g/kg minute

    Metoprolol 5mgq2min x3 IV then 2-5mg/hr

    Propranolol 20mgIV then 40mg, 8-mg q10min to

    300mg totalCalcium channel

    blocker if -blockercontraindicated

    Va

    sodilatorNitroprusside 0.3

    g/kg/min IV

    SurgeryOR for ascending

    aortic dissection

    Descending aorticdissection worsesurgical risks

    controversial forrepair

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    Trachea deviates to contralateral side

    Mediastinum shifts to contralateral side

    Decreased breath sounds and hyperresonance on affectedside

    JVD

    Treatment: Emergent needle decompression followed bychest tube insertion

    TENSION PNEUMOTHORAX

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    NEEDLE DECOMPRESSION

    Insert large bore needle (14 or 16 Gauge) with catheter inthe 2nd intercostal space mid-clavicular line. Removeneedle and leave catheter in place. Should hear air.

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    Nyeri dada pleuritik

    Lokasinya posterior atau lateral. Sifatnya tajam dan

    seperti ditusuk.

    Bertambah nyeri bila batuk atau bernafas dalam dan

    berkurang bila menahan nafas atau sisi dada yang sakit

    digerakan.

    Nyeri berasal dari dinding dada, otot, iga, pleura

    perietalis, saluran nafas besar, diafragma, mediastinum

    dan saraf interkostalis.

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    Nyeri dada Non- pleuritik

    Lokasinya sentral, menetap atau dapat menyebar ke

    tempat lain.

    Sering disebabkan oleh kelainan di luar paru.

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    Nyeri Dada

    Non

    Pleuritik

    Kardial

    Perikardial

    Aorta

    Muskulo

    skleletal

    Fungsional

    Pulmonal

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    Trauma lokal atau radang dari rongga dada otot, tulangkartilago sering menyebabkan nyeri dada setempat.

    Nyeri biasanya timbul setelah aktivitas fisik.

    Muskulo

    skeletal

    Kecemasan dapat menyebabkan nyeri substernal atauprekordinal, rasa tidak enak di dada, palpilasi, dispnea,using dan rasa takut mati.

    Fungsional

    Obstruksi saluran nafas atas seperti pada penderitainfeksi laring kronis dapat menyebakan nyeri dada,terutama terjadi pada waktu menelan.

    Pada emboli paru akut nyeri dada menyerupai infarkmiokard akut dan substernal.

    Nyeri dada merupakan keluhan utama pada kanker paruyang menyebar ke pleura, organ medianal atau dindingdada.

    Pulmonal

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    Angina stabil (Angina klasik, Angina of Effort), Angina takstabil (Angina preinfark, Insufisiensi koroner akut) ,Infark miokard

    Kardial

    Saraf sensoris untuk nyeri terdapat pada perikardiumparietalis diatas diafragma. Nyeri perikardila lokasinya di

    daerah sternal dan area preokordinal, tetapi dapatmenyebar ke epigastrium, leher, bahu dan punggung

    Nyeri bisanya seperti ditusuk dan timbul pada aktumenarik nafas dalam, menelan, miring atau bergerak.

    Perikardial

    Penderita hipertensi, koartasio aorta, trauma dindingdada merupakan resiko tinggi untuk pendesakan aorta.

    Diagnosa dicurigai bila rasa nyeri dada depan yang hebattimbul tiba- tiba atau nyeri interskapuler

    Aorta

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