Wooden Tongue

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WOODEN TONGUE Wooden tongue is an infection caused the rod-shaped bacterium, Actinobacillus lignieresii, which lives only in the presence of oxygen . The bacteria, which live in the mouth, invade tissue through breaks in the lining of the mouth. Any rough feed can cause mouth abrasions which allow entry of infection. Wooden tongue occurs almost entirely in soft tissue with the tongue and lymph nodes of the head most often affected. The disease starts suddenly with the tongue becoming hard, swollen and painful. Affected animals drool saliva and may appear to be chewing gently. The tongue often protrudes between the lips and nodules and ulcers may be observed on the tongue. They are unable to eat or drink and rapidly lose condition. The disease is progressive and often fatal unless treated. It is important to begin treatment early as early treatment is usually successful, but advanced cases may fail to respond. The most common treatments are iodine therapy or tetracyclines. Advanced cases may require surgical drainage and irrigation with iodine solution for several days. Treated animals should be observed regularly, as relapses can occur.

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Transcript of Wooden Tongue

WOODEN TONGUEWooden tongue is an infection caused the rod-shaped bacterium,Actinobacillus lignieresii,which lives only in the presence of oxygen.The bacteria, which live in the mouth, invade tissue through breaks in the lining of the mouth. Any rough feed can cause mouth abrasions which allow entry of infection. Wooden tongue occurs almost entirely in soft tissue with the tongue and lymph nodes of the head most often affected. The disease starts suddenly with the tongue becoming hard, swollen and painful. Affected animals drool saliva and may appear to be chewing gently. The tongue often protrudes between the lips and nodules and ulcers may be observed on the tongue. They are unable to eat or drink and rapidly lose condition. The disease is progressive and often fatal unless treated.It is important to begin treatment early as early treatment is usually successful, but advanced cases may fail to respond. The most common treatments are iodine therapy or tetracyclines. Advanced cases may require surgical drainage and irrigation with iodine solution for several days. Treated animals should be observed regularly, as relapses can occur.

WARTSWarts in cattle are caused by the contagious viruspapillomavirus. Four types of the virus are known to produce warts on cattle.Calves are most susceptible with few cases of warts seen in cattle over 2 years of age. Warts appear 1 to 6 months after infection with the virus. Not all animals carrying the virus will have warts. It can be transmitted from the unapparent carrier to the susceptible calf.Warts are usually more of an appearance problem than a physical problem. Warts usually shrink and drop off after a few months. This spontaneous recovery is probably the basis for the alleged effectiveness of many home treatments including rubbing with various kinds of oil, toothpaste of various brands, etc.If there is a severe outbreat in the herd an autogenous vaccine can be prepared from chemically treated warts taken from animals in a herd. This autogenous vaccine is more apt to have the strain or type ofpapillomaviruscausing the wart problem in the herd than the commercial vaccines. Warts can also be removed surgically with a scissors or a side cutter.

RINGWORMRingworm is a transmissible infectious skin disease caused most often byTrichophyton verrucosum, a spore forming fungi. The spores can remain alive for years in a dry environment. It occurs in all species of mammals including cattle and man. Although unsightly, fungal infections cause little permanent damage or economic loss. Direct contact with infected animals is the most common method of spreading the infection.Spores germinate and attack the shafts of the hair and the surface layers of the skin. Exudates ooze from the damaged skin and mix with debris from skin and hair forming a crusty scab. The grayish-white scab is noticeably higher than the surrounding skin. Ringworm is most frequent on the head and neck, but it may be found over the entire body in severe cases. Infection spreads from the center outwards and resulting in a circular lesion. Scabs fall from older lesions leaving a ring with a hairless area in the center. Hence, the name ringworm.Ringworm will usually cure itself without treatment. Common treatments include topical application of a 2% solution of iodine, thiabendazole paste or any fungicide used to treat athlete's foot in man.

PINKEYEPinkeye (infectious bovine keratoconjunctivitis) is a common infectious disease affecting the eyes of cattle. The name describes the redness and inflammation of the lining of the eyelid and eyeball. Although pinkeye is non-fatal, it has a marked economic impact on the cattle industry. It is known to occur at all seasons of the year and in all breeds of cattle. Pinkeye and foot rot are the two most prevalent conditions affecting all breeding beef femalesOne or both eyes may be involved. Excessive weeping of the affected eye and closure due to pain are the two signs most commonly observed. As the disease progresses, the cornea becomes cloudy or white. An ulcer (eroded circular spot) frequently develops near the center of the cornea. Cattle with pinkeye keep the affected eye or eyes closed because of pain and to avoid bright sunlight. They lose weight because they are reluctant to forage for feed and water. The course of the infection may run for 4 to 8 weeks, or even longer.As the eye begins to heal, white scar tissue infiltrates the cornea. In most cases this scar will gradually disappear as healing progresses and vision will be restored. However, in severely affected eyes, a white scar often persists and interferes with vision. If the ulceration is severe enough to penetrate all layers of cells forming the cornea, the fluid in the eyeball will escape. This results in the iris and/or lens protruding partially or entirely through the ulceration. If this occurs, there will be permanent blindness in the affected eye.Pinkeye is caused by a combination of factors. A good control program should incorporate procedures to reduce initial eye irritation.An intensive fly control program is essential to limit the spread of pinkeye in a herd of cattle. The insecticide-impregnated plastic ear tags are effective in controlling the horn fly and face fly. These ear tags are also an aid in controlling the stable fly and house fly, and remain effective for up to 5 months. Also sprays, charged backrubbers, and dusts bags are products that can provide chemical control. Manure, weed, and brush management are necessary for total fly control.Cattle often have grass or weed seeds in their eyes, and these materials no doubt irritate the eye and contribute to the development of pinkeye. Clipping pastures to reduce the amount of tall grass and weeds can be an important management technique in controlling pinkeye.Ultraviolet light (sun light) - breed for eyelid pigmentation, introduce Brahman influence into the herd, provide shade or tree rows with ample room to prevent overcrowding.Cattle with pinkeye can be helped by prompt treatment. Most antibiotics in eye sprays are effective in reducing the infection. Many eye sprays also contain an anesthetic to relieve the intense pain due to infection. A dye to act as a filter for some of the light rays is also commonly included and probably gives some protection to the injured eye. The aerosol pinkeye sprays are most effective if applied several times a day.

LUMPY JAWActinomycosis or lumpy jaw produces immovable hard swellings on the upper and lower jawbones of cattle, commonly at the central molar level. It is caused by an anaerobic micro-organism,Actinomyces bovis.The bacterium invades tissue through breaks in the lining of the mouth caused by eating rough forage. The tumor-like swellings develop slowly and may take several months to reach a noticeable size. Lumpy jaw may be well advanced before external signs are visible. The lumps consist of honeycombed masses of thin bone filled with yellow pus. If neglected the swellings may become very large. In advanced cases openings develop and discharge small amounts of sticky pus containing gritty yellow granules.

Difficult breathing due to involvement of the nasal bones may be the first sign. As the disease progresses, chewing becomes more difficult and painful, resulting in loss of condition. Occasionally, the soft tissues of the head and alimentary tract can be involved. Lesions in the alimentary tract give vague symptoms of indigestion, often with chronic bloat.The most common treatments are iodine therapy or tetracyclines. Treatment is often ineffective. If the disease is detected early, it may be better to dispose of the animal while it is still in good condition. Only the head should be condemned by meat inspectors, unless the lesions have spread elsewhere in the body.

FOOTROTFusobacterium necrophorumandBacteroides melaninogenicusare the predominant bacteria isolated from footrot. Footrot occurs in cattle of all ages, but it is most common in adults. The disease is seen year-round, but there is increased incidence in the wet summer and fall months.Bacteria gain entrance through lesions on the lower part of the foot; the bacteria do not penetrate normal skin. Anything that can damage the skin between the claws should be considered as predisposing to the disease. Wet manure and mud can soften the skin between the claws and permit infection. Dried or frozen mud, stones, and stubble can bruise the tissues sufficiently to lower their resistance to disease.Lameness appears suddenly; usually only one foot is affected. An animal will put little weight on the affected leg, but will place weight on the limb while walking or running. A moderate fever (103-104 F.) may accompany the early signs. The typical early lesion is a break in the skin between the claws. Pus may be present, but not in large amounts. Edges of the break are covered with necrotic material, and the lesion has a characteristic foul odor. The foot is swollen and the animal is in acute pain.Spontaneous recovery may occur, but if the animal is not treated, the lameness may persist for several weeks. Penicillin, tetracyclines, sodium sulfadimidine, sulfabromomethazine, and other antibacterial agents are used for systemic therapy. Daily treatment begun immediately after onset of lameness usually will give excellent recovery in two to four days. Treated animals should be maintained on a dry surface until recovered. Recent research has shown that dietary zinc supplementation is effective in treating and preventing footrot in cattle. Feeding chlortetracycline (CTC) at the rate of 0.5 mg / lb. body weight daily may also help prevent foot rot. Many mineral mixes and commercial supplements are formulated to contain the correct amount of chlortetracycline (CTC).