The First 3 Months of Acl Rehabilitation Factors That Influence Success-paine

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    Russ Paine, PTMemorial Hermann Ironman

    Sportsmedicine Institute

    Houston, Texas

    ACL Rehabilitation

    Adrian Peterson NFL Running Back

    NFL MVP 2012 2,097 yds.

    6th fastest player toreach 8,000 rushingyards

    150 yds. 7 games

    ACL PTG, MCLIII8 mos prior to beginof season

    Jointcondition/intensity.genetics

    Phase I ACL Rehab. ROM and

    Strength

    ACL/MCL injuryon December 24,2011

    Grade III MCL

    Surgery: Dec. 30,2011 Dr.Andrews

    Returned toHouston for rehab

    on January 10

    Quad Atrophy Still Enigma

    Rehabilitation

    AJSM March2005

    VL, VI smallerin ACL def.knee

    VMO stilldifficult toactivate

    Pre-op and Immediate Post-op

    Training Quad Re-cruitment

    biofeedback 10

    Stimulate Proprioception(gait training, balancingactivities) cone amb.

    Extension ROM techniques

    ERMI device SLRs up to 10# 5

    Flexion contracture = locatepain

    Ant. Or Post.

    Ant = fat pad impingement

    Post = capsular scarring or hsspasm

    Ligament Unloading Exercises

    Quad setting, SLR kneein full extension - Drez,Paine Journal of KneeSurgery

    Knee extension limitingROM 90 - 40 Grood -JBJS 84

    Leg Press = 90 - 40 =decreased strain in thisROM no need forextension = decreasedquad contraction

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    ACL Tear = Loss of

    Proprioceptive Function must be

    Restored before return to sports Primary goal

    Rehabilitation

    Enhance muscle

    reaction time

    Stabilization/Recruit

    ment

    Proprioception

    Carter et al, Br J Sports

    Med, 1997 Loss of proprioception after

    ACL rupture

    Roberts et al, J Orthop Res,

    2000

    Loss of proprioception after

    ACL reconstruction

    Gait Abnormalities

    Acute ACL Tears

    Decreased NMinput

    Delayed quadfiring heelstrike

    Result = flexedknee gait pattern

    Increased PFJRF

    Force patient tosqueeze quad atheel strikeTreadmill amb.

    Revascularization of ACL Graft

    4 phases Avascular graft

    Revascularization

    Cellular repopulation

    Remodeling &Reorganizationcollagen fibers(ligamentizationAmiel 86 AJSM)

    Recent studies =intraarticular portionrevascularizes fasterthan bone interface

    sitesNtoulia AJSM2011

    Early Phase

    Inflammatory response:macrophages, cytokines

    produce scar betweengraft and bone 4-10

    days 6 weeks graft hasvascular synovialenvelope

    20 weeks intrinsic graftrevascularization

    Fat pad, synovial tissue,bone tunnels =vascularity

    Animal Studies Bone Tunnel Healing

    12 weeks Sharpeysfibers into graft = bonehealing

    Pull out failure prior to12 weeks = tunnelfailure, after 12 weeks= midsubstanceRodeoJBJS 93

    BPTB 6 weekscompleteincorporationPapageorgiou AJSM2001

    Bracing 4-6 hyperflexion

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    Immobilization

    Decrease in

    proteoglycan content

    Reduces stiffness of

    cartilage

    Cant withstand

    shearing forces

    Compression & Fluid Flow

    Matrix - CPM As compression

    increases,resistance to fluid

    flow in matrixincreases

    GAGs slow downfluid flow in &out of matrix

    Result = increasedstiffness ofcartilage

    Stiffness = allowsresistance tocompression

    Passive ACL Strain

    Passive ROM 0% strain 0-

    60, 70-120 1-2% strain in

    normal ACL

    Push extension wait on

    flexion (swelling)

    Renstrom AJSM 86

    Motion Complications Increasing flexion

    Restricted flexion =increased PF Compression

    Flexionator

    Provides hydraulicresistance

    5 min. on 5 min.off, repeat

    6 x per day

    HS Biofeedback ProneHangs

    Steps To Avoid Extension LOM

    Recognize early factors:

    Poor quad function no SLR

    Flexed knee ambulation

    Poor pain tolerance

    MCL/Meniscus repair

    Reversing Lack of

    Extension

    1cm = 1 HHD Daniel

    AJSM 89

    Infrapatellar Fat Pad Tunnel Pain -

    Activity Source of pain

    post-op ACL

    ATS portals passthrough fat pad

    Cause fibrotic

    hematoma in somepatients

    May affect p.tracking with ATS

    Early intervention= US,Laser,manualsoft tissuerelease,scar

    Injection tunnel

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    ACL/MCL

    Combined ligament injury

    Limits ROM and

    WB 1st 4-6 weeks

    Locked inextension for 1week, increasedflexion 30d perweek

    WB with crutchesgradual increase,D/C crutches 4-6weeks

    Stability = No DJD? In a 2004 study of male former

    soccer players 14 years after anACL injury, Swedishinvestigators found nodifference between surgicallytreated and conservativelytreated athletes with regard toradiographicallydetected OA

    ACL reconstruction did notaffect prevalence of knee OAin female soccer players 12years after ACL injury

    Lohmander LS, Ostenberg A, Englund M, Roos H. High prevalence of

    knee osteoarthritis, pain, and functional limitations in female soccer

    players twelve years after anterior cruciate ligament injury. Arthritis

    Rheum 2004;50(10):3145-3152.

    ACL Deficient Rehabilitation

    Risks

    Increased articular cartilage loss

    long-term

    Murrell AJSM 01

    130 pts. ACL tears

    ATS exam. Reconstruction

    Time = increased cartilage &

    meniscus loss

    9 x greater cartilage damage 2 yrs.

    Vs. 1 mo. ACL rec.

    P-F Cyclic compression exercises

    Difficulty with post-op recovery

    Cycling - properseat ht., 90-100Rpm's, dont standon pedals

    Total gym -allows bodyweight to bereduced

    Pool therapy -load, unload

    EFX ?

    Step 2

    Begin Gradual Loading Exercises

    Leg Press Best MR Leg Press

    supine squat low load

    high endurance

    activity 60 secondcontractions

    Begin with 5Kg

    Quad EMG WB & NWB

    Wilk AJSM knee extension =highest EMG = 25 degrees

    Wilk AJSM leg press =highest EMG = 85 degrees

    Knee extension = requires highEMG due to lack of patellarheight near extension

    Huberti = Fpt > Fquad 1st 20d.45d Fquad > Fpt = Carefulwith minisquats

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    Step 3

    Body Weight Control

    Functional Squat

    MR Systems

    Squat Control

    Chair Squats

    3x20

    New Device

    Primal 7

    Allows

    patient toassume

    normal

    body

    positions

    early

    using

    assisted

    strapping

    Testing Functional Hip

    Abductor Strength

    Crossley AJSM 2011

    A- participant demonstratesgood performance

    B- participant demonstratespoor overall and trunkperformance

    C- participant demonstratespoor pelvis and hipperformance

    D- participant demonstrates

    poor hip and kneeperformance

    Chris Powers JOSPT

    2003

    Controlling

    femoral position

    Hip ABDuctor,

    G.Max, Lateral

    Rotation,

    NWB routine

    Hewitt AJSM

    07 pelvic

    control - ACL

    Powers: Controlling Hip Internal

    Rotation, Knee Adducton

    WB routine

    ABD, GMAX,

    Lateral rotation

    Core Strengthening = Strengthens

    Kinetic Chain

    Rotational plyoball: hold

    VMO contraction Lower Abdominal Trunk

    flexion/extension usingSwiss Ball

    Hewitt AJSM 07 Corecontrol may be assoc.with ACL tear = pelviccontrol = kinetic chain =PF control

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    Hip Flexor Stretch

    Sidelying

    Safe and unsafe closed vs open

    kinetic chain

    Is closed chain safer?

    challenged with strain-gaugestudies

    What is primary reason for

    description of open and

    closed chain?

    Terminology should describe

    what is happening to specific

    anatomical structure

    Ligament loading exercises

    ACL

    Knee extension 30 to 0 degrees with

    resistance

    Pivoting, twisting activities -

    Markolf - JBJS - no protection

    Squatting - Beynnon - 97 AJSM-

    loads comparable to open chain - no

    increase in strain with increased

    load during squat. Knee extension

    = increased strain with increasedload

    Ligament Loads

    Peak Loads

    ACL

    Exercise

    -ACL = 1725-2160N beforefailure

    -PCL = 4000N

    Graft load

    sharing?

    ACL tear = loss of HS reflex

    Rehab. = re-gain

    Tsuda AJSM 01

    Stim. ACL = reflex HSactivity

    Response = Humans

    versus Solomonow =

    animal model

    High Speed Biodex

    Speeds 180-300deg./sec.

    May stimulate NMcontrol

    Rapid reversal fromext./flexion

    HS curls, goodmornings

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    Strength

    Load and Volume

    Set load and

    volume to meet

    goals of phase

    Periodization

    Model

    Strength Preparation for plyometrics and sport

    specific skills

    Vary Load and Volume Involve The Core

    MAKE IT PROPRIOCEPTIVELY

    CHALLENGING!!!!!

    Phase II Functional StrengtheningRunners Pose

    Cone Reach ACL Knee Bracing

    Used

    primarily

    during

    rehabilitation

    Ave. time for

    use after

    return = 8

    weeks.

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    Combination Core/LE/UE

    Slide Board End Stage RehabWhen to Run?

    PTG 2 - 3 mos.

    Treadmill

    Allograft ACL 3-

    4 mos.

    Timeframes

    dependent on

    quad strength and

    symptoms

    Phase III Plyometrics and Sports

    Specific Training

    Plyometrics Develop:

    Strength

    Speed

    Power

    Good ProprioceptiveTraining

    Injury Prevention

    Not everyone needs tojump!

    Plyometrics

    Jumps

    2 footed landing

    Hops

    1 foot landing

    Bounds

    Jumping form

    one foot to the

    other

    Proprioceptive

    When To Start

    Full PROM

    Normalized Quadriceps

    Control Appropriate Time Period

    Controlled Joint Effusion

    Normal LigamentousExam

    Quad Re-education

    Take-Off

    Recruitment =plyometric

    routine, singleleg plyos

    Sportsmetric

    FunctionalDrills

    Isokineticstrength, leg

    press 90-40

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    Bilateral Cone Hop Stick Broad Jumps

    Progression to

    Single Leg Hop Maintain

    mechanics

    Mustundergodouble leg

    jumps

    Must haveadequatecore/quadcontrol

    Agilities/Sports Specific

    The ability to change

    direction rapidly

    without loss of body

    control

    Uses

    Technique

    Conditioning

    Functional Assessment Return to

    Sports

    Single leg hop for distance = 90%normal

    Figure of 8 = 9.5s (male normal)

    Isokinetic test = 85% normal Proprioceptive function = restored

    ROM = 90% normal decreasedmotion = articular cartilage stress

    KT-1000 within normal limits(

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    Return to full competition in NFL

    8 mos

    Timetables werenot unusual

    Level of playwas veryunusual

    Skill position,ability tocut/pivot with nofear superhumaneffort

    Conclusions

    Rehabilitation = team

    effort

    Must know healingconstraints

    Progression = strength,

    proprioception/balance,

    plyometrics/agilities

    Never progress patient

    until ready for next phase