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THE BASICS OF CXR
INTERPRETATION
Mir M. Alikhan, MDPulmonary & Critical Care Medicine
drbeen.com
Disclosures● None
● All sources and credits for images will be provided at the end of this presentation
Introduction
● CXR is probably the most common imaging test
● Few providers (including MDs) are comfortable interpreting
their own films
● Clinical decisions are too often made based on reports
from non-clinicians
● Having a systematic and repetitive approach is the key
Objectives
● By the end of this lecture, the learner will be able to:
○ Develop an understanding of the normal CXR appearance
○ Assess the technical quality of a CXR
○ Utilize a systematic and reliable approach in CXR evaluation
○ Identify common CXR findings
○ Correlate basic CXR findings with clinical evaluation in order
to reach a diagnosis
Understanding the Normal CXR
Is this a normal CXR?
● Develop confidence with the
normal appearance
● Type of CXR will have an
impact on what is
considered “normal”
● Absence/aberration of
normal findings should raise
red flags
● Know which structures
should be present or absent
Different Types of CXR
Postero-anterior (PA; standard)
Patient is usually standing with anterior chest against the x-ray plate
X-ray beam originates from 5-6 feet behind the patient
Beam penetrates from posterior to anterior chest
Antero-posterior (AP; portable)
Patient is usually in bed and leaning with back against the x-ray plate
X-ray beam originates from 2-4 feet in front of the patient
Beam penetrates from anterior to posterior chest
Heart
Diaphragm
Trachea
Gastric air bubble
Aortic knob
Right PA/hilumLeft PA/hilum
Carina
Utilizing a Systematic Approach
RIP - ABCDE - LUNGS
Assessing Technical Quality
● R - rotation● I - inspiration● P - penetration
Rotation
● Ideally CXR beam should be transmitted perpendicular to
the chest
● Abnormal angles will distort the image by creating an
oblique view
● Clavicular heads should be equidistant from vertebral
spinous processes
Inspiration
● Assessment of inspiratory effort and lung volumes
● Ideally 7-9 ribs should be visible
● < 7 suggests poor effort by the patient and/or low lung
volumes (restrictive lung disease, atelectasis, etc.)
● 10 or more ribs typically suggests hyperinflation (COPD,
asthma, bronchiectasis)
Penetration
● Exposure quality of the film
● Over-penetration will make structures more radiolucent
which could lessen significance of opacities
● Under-penetration will make structures more radioopaque
which may lead to “over-calling” certain findings
Evaluation of Structures
● A - airway● B - bones (and soft tissues)● C - cardiac silhouette● D - diaphragm● E - everything else (hardware)
Airway● Trachea
○ Deviation
○ Caliber
● Carina
○ Typical angle
○ Splaying
● Mainstem and lobar bronchi
○ Right mainstem is more straightly aligned with trachea
Bones (and soft tissues)● Scan all bony structures
○ Fractures
○ Pins/rods/staples/wires
○ Thoracic cage deformities (scoliosis, etc.)
● Look for foreign bodies
Cardiac ● Evaluate the size and shape of the cardiac silhouette
○ Cardiomegaly - width of the silhouette is greater than ½ the thoracic cage width
■ Can be exaggerated or “over-called” on AP (portable) films
○ Aortic knob
○ Left atrium
○ Pulmonary arteries
○ Shift of mediastinal structures
○ Cardiac borders
○ Pericardial effusion
Diaphragm● Diaphragmatic line should be clearly demarcated
● Evaluate costophrenic and cardiophrenic angles
● Retrocardiac space
● Elevation or flattening of the hemidiaphragms
● Also look at structures immediately beneath diaphragm (liver, gastric bubble,
free air in the abdomen)
● Things that obscure the diaphragm:
○ Pleural effusion
○ Atelectasis
○ Lower lobe infiltrates or mass
Everything Else (Hardware) ● Endotracheal tube
○ Tip should be 2-4 cm from the carina
● Central line
○ Tip of catheter should lie in the cavo-atrial junction
● Pacemaker or defibrillator
○ Know how to tell the difference
○ Leads can be placed in the atria or ventricles
● Chest tubes
○ Always identify the “sentinel” hole to make sure it is within the pleural space
Lungs
● Make a conscious effort to evaluate the lung parenchyma
last!!
● Look at each side independently and then compare the
two sides
● Point out features that seem abnormal
● Always describe before diagnosing!
Lungs● Common descriptive terms
○ Opacities/Infiltrates
○ Mass/Nodule
● Defining the descriptions
○ Consolidation
○ Effusion
○ Atelectasis
○ Edema
○ Fibrosis
Lungs
● Opacities
○ Something which appears relatively radio-opaque compared to normal lung
○ Alveolar opacity
○ Interstitial opacity
● Mass/Nodule
○ Discrete appearance with apparent borders
○ Nodule < 3 cm
○ Can be pleural-based or parenchymal
Lungs● Consolidation
○ Focal confluence of alveolar opacities○ Air bronchograms○ Obliteration of vessels
● Atelectasis vs. Effusion○ Look for discrete lines or lobar distribution for atelectasis○ Effusions are usually dependent which causes gradation from base upwards
● Edema○ Alveolar vs. Interstitial patterns
● Fibrosis○ Septal thickening○ Honeycombing
Clinical Correlations
A Common Radiology Report ...
● “The lung fields demonstrate non-specific hazy, discrete interstitial and alveolar infiltrates or opacities that could represent any of the following: consolidation, effusion, atelectasis, or mass. These findings could suggest pneumonia, lung malignancy, or absolutely nothing. Please correlate clinically.”
Case #1
60 y/o M with recent pneumonia returns with severe shortness of breath and cough. Also complains of L-sided chest pain when taking a breath.
T 101.3 HR 115 BP 120/70 RR 30 O2Sat 87%
Exam: tachypneic, diminished breath sounds in L hemithorax
Labs: WBC 21.2 BUN 28 Cr 1.4
Case #1What is the most likely diagnosis?
A) Recurrent pneumonia
B) Mucus plugging leading to atelectasis
C) Parapneumonic pleural effusion
D) Hemothorax
E) Tension pneumothorax
Case #1What is the most likely diagnosis?
A) Recurrent pneumonia
B) Mucus plugging leading to atelectasis
C) Parapneumonic pleural effusion
D) Hemothorax
E) Tension pneumothorax
Case #2
68 y/o male with history of smoking presents with intermittent night sweats, weight loss, and hemoptysis.
T 99.0 HR 96 BP 118/80 RR 18 O2Sat 98%
Exam: no distress; breath sounds clear bilaterally
Labs: WBC 11.5 Hgb 9.5
Case #2What is the most likely diagnosis?
A) Aspergilloma
B) Tuberculosis
C) Lung adenocarcinoma
D) Sarcoidosis
Case #2What is the most likely diagnosis?
A) Aspergilloma
B) Tuberculosis
C) Lung adenocarcinoma
D) Sarcoidosis
Case #3
42 y/o male with history of CAD presents with sudden onset of chest pain and severe shortness of breath. The pain radiates to the back and he finds it hard to take a deep breath. He also was in a car accident 2 days ago and has been under a lot of stress.
T 98.0 HR 130 BP 85/50 RR 35 O2Sat 80%
Exam: severe resp distress, diminished breath sounds, mild tracheal deviation
Labs: WBC 14.5
EKG: sinus tachycardia, T wave inversions
Case #3What is the most likely diagnosis?
A) Aspiration pneumonia
B) Myocardial infarction
C) Aortic dissection
D) Hemothorax
E) Tension pneumothorax
Case #3What is the most likely diagnosis?
A) Aspiration pneumonia
B) Myocardial infarction
C) Aortic dissection
D) Hemothorax
E) Tension pneumothorax
Case #4What do you think of this CXR?
A) Lobar pneumonia
B) Atelectasis
C) Pleural effusion
D) Cardiomegaly
Case #4What do you think of this CXR?
A) Lobar pneumonia
B) Atelectasis
C) Pleural effusion
D) Cardiomegaly
Case #5
40 y/o male with recent influenza infection presenting with shortness of breath, recurrent fever, cough, and yellow-green sputum production.
T 102.3 HR 110 BP 105/65 RR 24 O2Sat 93%
Exam: diminished breath sounds and some scattered crackles/rhonchi
Labs: WBC 22.5 BUN 30 Cr 1.6 BNP 45
Case #5What is the most likely diagnosis?
A) Pleural effusion
B) Pulmonary edema
C) Multifocal pneumonia
D) Atelectasis due to mucus plugging
E) Interstitial lung disease
Case #5What is the most likely diagnosis?
A) Pleural effusion
B) Pulmonary edema
C) Multifocal pneumonia
D) Atelectasis due to mucus plugging
E) Interstitial lung disease
Case #6
70 y/o male with history of ETOH abuse is admitted with acute pancreatitis. He is admitted to the hospital for aggressive IV fluid resuscitation and pain control. Over the next 24 hours he develops worsening respiratory distress?
T 100.2 HR 120 BP 160/95 RR 35 O2Sat 86%
Exam: diaphoretic, tachypneic, inspiratory crackles in lower lung fields
Case #6What is the most likely diagnosis?
A) Hospital-acquired pneumonia
B) ARDS
C) Myocardial infarction leading to pulmonary edema
D) Aspiration pneumonitis
Case #6What is the most likely diagnosis?
A) Hospital-acquired pneumonia
B) ARDS
C) Myocardial infarction leading to pulmonary edema
D) Aspiration pneumonitis
Summary
● Look at all your films - even the “normal” ones
● Use a repetitive & reliable system to avoid missing details
● RIP - ABCDE - LUNGS
● Describe findings first before considering a diagnosis
● Clinical correlation is ALWAYS necessary to arrive at a
diagnosis
Sources
● By Frank Gaillard - http://images.radiopaedia.org/images/4195/5e7cfb6d90bbcf70e73493819e691a.jpg, CC BY-SA 3.0, https://commons.wikimedia.org/w/index.php?curid=14698169
● By The original uploader was Pabloes at Spanish Wikipedia - Transferred from es.wikipedia to Commons., CC BY-SA 3.0, https://commons.wikimedia.org/w/index.php?curid=1753377
● By James Heilman, MD - Own work, CC BY-SA 3.0, https://commons.wikimedia.org/w/index.php?curid=4639778● By Hellerhoff - Own work, CC BY-SA 3.0, https://commons.wikimedia.org/w/index.php?curid=17766964● By James Heilman, MD - Own work, CC BY-SA 3.0, https://commons.wikimedia.org/w/index.php?curid=4646165● By Samir 04:51, 17 September 2007 (UTC). Modified by Delldot 07:55, 28 April 2008 (UTC) - http://en.wikipedia.
org/wiki/Image:Noncardiogenic_pulmonary_edema.JPG, CC BY-SA 3.0, https://commons.wikimedia.org/w/index.php?curid=3954240
● By Photographed by User Clinical Cases 00:42, 7 November 2006 - Originally from tension pneumthorax page on clinicalcases.orgTransferred from en.wikipedia to Commons.; description page is/was here; uploader to en Wiki was Clinical Cases at en.wikipedia, CC BY-SA 2.5, https://commons.wikimedia.org/w/index.php?curid=2235891
Sources
● By Clinical_Cases: I made the photo myself, licensed under Creative Commons license. - http://en.wikipedia.org/wiki/Image:Left-sided_Pleural_Effusion.jpg originally http://clinicalcases.blogspot.com/2004/02/massive-left-sided-pleural-effusion.html, CC BY-SA 2.5, https://commons.wikimedia.org/w/index.php?curid=2294191
● By James Heilman, MD - Own work, CC BY-SA 3.0, https://commons.wikimedia.org/w/index.php?curid=18076955● By Gregory Marcus, MD, MAS, FACC - http://knol.google.com/k/-/-/hCjLTV2A/bdmV3w/ICD.CXR.jpg , embedded in
http://knol.google.com/k/gregory-marcus-md-mas-facc/implantable-cardioverter-defibrillators/hCjLTV2A/qeyBbw?domain=knol.google.com&locale=en#, CC BY 3.0, https://commons.wikimedia.org/w/index.php?curid=5857447
● By Christaras A - Converted from anonymized dicom image, CC BY 2.5, https://commons.wikimedia.org/w/index.php?curid=1247722
● By Jtechr - Own work, CC BY-SA 3.0, https://commons.wikimedia.org/w/index.php?curid=17385153● By James Heilman, MD - Own work, CC BY-SA 3.0, https://commons.wikimedia.org/w/index.php?curid=11110205
Extras!!