Spinal Cord Injuries
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Transcript of Spinal Cord Injuries
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http://www.rexdonald.com/facts.html
http://www.cureparalysis.org/statistics/
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Spinal Cord InjuriesLife expectancy greatly increased since
WW II. Intermittent catheterization Medications, equipment, etc
Cause of premature death in QUADS is usually related to COMPROMISED RESPIRATORY FUNCTION
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Spinal Cord InjuriesWho’s at risk?
ADULT MEN BETWEEN 15 AND 30 YEARS
Anyone in a risk-taking occupation or lifestyle
SCI in older clients increasing largely due to MVAs
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Spinal Cord InjuriesCauses (in order of frequency)
MVA Gunshot wounds/acts of violence Falls Sports injuries
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Spinal and Neurogenic ShockBelow site of injury:
Total lack of function Decreased or absent reflexes and flaccid
paralysis Lasts from a week to several months after
onset. End of spinal shock signaled by muscular
spasticity, reflex bladder emptying, hyperreflexia
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Classification of SCIMechanism of injury
Flexion (bending forward) Hyperextension (backward) Rotation (either flexion- or extension-
rotation) Compression (downward motion)
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Pathophysiology of SCI Insert stuff here Insert picture here
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Classification of SCI Level or Injury
Cervical (C-1 through ??) Thoracic (T-1through ??) Lumbar (L-1through ??)
Degree of Injury Complete
Total paralysis and loss of sensory and motor function although arms or rarely completely paralyzed
Incomplete or partial
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http://www.sci-recovery.org/sci.htm
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Degree of Injury Complete transection
Total paralysis and loss of sensory and motor function although arms or rarely completely paralyzed
Incomplete (partial transection) Mixed loss of voluntary motor activity and
sensation Four patterns or syndromes
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Incomplete cord patterns Insert picture of cord here Central cord syndrome More common in
older clients Frequently from hyperextension of spine Weakness in upper and lower ext, but greater
in upper. Anterior cord syndrome Posterior cord syndrome Brown-Sequard syndrome
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Anterior cord syndromeCompression of the ant. Cord, usually
a flexion injurySudden, complete motor paralysis at
lesion and below; decreased sensation (including pain) and loss of temperature sensation below site.
Touch, position, vibration and motion remain intact.
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Posterior cord syndromeAssoc with cervical hyperextension
injuriesDorsal area of cord is damaged
resulting in loss of proprioceptionPain, temperature sensation and motor
function remain intact.
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Brown-Sequard syndrome Damage to one half of the cord on either side. Caused by penetrating trauma or ruptured disk.
ischemia (obstruction of a blood vessel), or infectious or inflammatory diseases such as tuberculosis, or multiple sclerosisBSS may be caused by a spinal cord tumor, trauma (such as a puncture wound to the neck or back),.
a rare SCI syndrome which results in weakness or paralysis (hemiparaplegia) on one side of
the body and a loss of sensation (hemianesthesia) on the opposite
side.
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Clinical manifestations of SCIDepend on the LEVEL and DEGREE of
the injury!Quadriplegia occurs with C-1 through
C-8 injuries.Paraplegia occurs with T-1 thru L-4.SEE TABLE 57-3 ON PAGE 1725!
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Clinical Manifestations of SCIRespiratory
C1 – C3: Absence of ability to breathe independently.
C4 – poor cough, diaphragmatic breathing, hypoventilation
C5 – T6: decreased respiratory reserve T6 or T7 – L4: functional respiratory
system with adequate reserve.
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What is the phrenic nerve? The phrenic nerve stimulates the diaphragm
to contract. Two phrenic nerves (right and left) - injury to
one or the other paralyzes contraction of only one half of the diaphragm but even hemi- (half) paralysis can significantly interfere with breathing for patients with lung disease.
The nerve arises from branches of the C3,4, and 5 nerve roots.
The phrenic nerve can be damaged by procedures exploring the neck & upper back
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Loss of the phrenic nerve on either side results in paralysis of the diaphragm on that side.
Paralysis of the diaphragm on one side results in less inflation of the lung on that side.
Whether this is physiologically significant (producing respiratory distress, hypoventilation/hypercapnia) depends on other aspects of a patient's pulmonary physiology (namely underlying chronic obstructive pulmonary disease [emphysema, bronchitis], pneumonia, etc.).
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Cardiovascular system C1 – T5 shows decreased or absent SNS
influence. BRADYCARDIA AND HYPOTENSION
(due to vasodilation)
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What is the VAGUS nerve?The longest of the cranial nerves- exits
out of the medulla and ends in the abdomen
It supplies sensory and motor function to the pharyngx
Supplies motor function to the muscles of the abdominal organs
Provides parasympathetic activity to the heart, lungs, and most of the digestive system
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Urinary SystemAtonic bladder with RETENTION in
spinal shock.Post acute phase – irritability causing
dribbling or frequent urination.Urinary infection and calculi from
retention and distention. INTERMITTENT CATHETERIZATION!
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GI system Decreased motility Paralytic ileus Gastric distention – intermittent NG suctioning Increased H2 – administer H2 inhibitors such
as Zantac or Pepcid in initial stages Carafate and antacids later as prophyaxis Intraabdominal bleeding! Remember, no pain
or tenderness to warn you. Watch for H/H decrease and impactions
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Integumentary SystemPressure ulcers!Muscle atrophy in flaccid paralysisContractures in spastic paralysisPoikilothermism – the adjustment of
body temp to room temperatureDecreased ability to sweat below lesion
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Peripheral vascular systemDVT common but not detected easilyPulmonary embolism a significant
cause of death.Doppler studies, measurement of
extremity girth, impedance plethysmography (what the heck is this?)
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Post Injury Assessment Goals are to
Sustain life Prevent further cord damage
Assessment of muscle groups; motor status Against gravity Against resistance Both sides of the body Ask to move legs, hands, fingers, wrists, then
shrug shoulders
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Post injury assessment (p.1726)Thorough motor examination including
position sense and vibration.Sensory examination
Pinprick starting at toes and working upward
ALWAYS HAVE CLIENT CLOSE EYES OR LOOK AWAY! If he can see what you’re doing, he will answer accordingly.
Assess for head injury and ICPX-ray, CT scan, EMG
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Surgical TherapyReduces injury and stabilizes the SCDone for
Compression Bony fragments in the cord Compound fracture Penetrating trauma
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Drug TherapyVasopressors (Dopamine) to keep
mean arterial pressure greater than 80mm to 900mm/Hg so that PERFUSION TO CORD is improved.
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Methylprednisolone (Solu-medrol)
Increases the recovery of function and is the SOC! IV bolus then continuous IV over a 23 hour period.
Improves blood flow and reduces edema in the SC
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Other drug therapySymptom-reducing drugs for
GI problems - zantac, tagamet, pepcid Bradycardia - atropine Hypotension - vasopressors bladder spasticity - anticholinergics autonomic dysreflexia – blood pressure
reduction
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Function of Motor NeuronsUpper motor neurons
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Function of Motor NeuronsLower motor neurons
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Diagnoses and Interventions Impaired Gas Exchange r/t muscle
fatigue and weakness Decreased Pao2, increased PaCO2 Fatigue Diminished breath sounds
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Impaired gas exchangeMaintain patent airwayAssess respiratory status q 2 hoursMonitor ABGsProvide aggressive pulmonary toilet;
chest PT and quad-assist coughingAssess strength of coughSuction secretions
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Inability to sustain spontaneous ventilation
Related to diaphragmatic fatigue or paralysis evidenced by Dyspnea Use of accessory muscles Abnormal ABGS
Provide chest PTAssist with mechanical ventilationProvide emotional support
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Decreased cardiac outputRelated to venous pooling of blood and
immobility as evidenced by Hypotension Tachycardia Restlessness Oliguria Decreased pulmonary artery pressures
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Decreased cardiac outputMonitor blood pressure, pulse and
cardiac rhythmAdminister vasopressors to maintain
MAP at 800mm/Hg or aboveApply pneumatic compression boots or
stockings Perform ROM at least q8h to aid in
muscle contraction and venous return
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Impaired skin integrityRelated to immobility and poor tissue
perfusion Inspect skin and areas around pins or
tongsTurn at least q2h and use kinetic table
or other specialty care devices. Insure adequate nutritional intake INFORM family and client about risk of
pressure ulcers
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ConstipationRelated to location of injury, fluid
intake, diet, immobility AEB Lack of BM in over 2 days bowel sounds Palpable impaction Hard stool or incontinence
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ConstipationAuscultate bowel sounds and monitor
abdominal distentionNote and report any nausea and vomitingBegin bowel program when BS return and
teach to client and familyAdminister suppositories and stool
softenersEnsure appropriate fluid and fiber intake
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Bowel program for SCINeeds to be consistentGive suppository after meal and place
on toilet approx 30 minutes after.Do this at same time each day!Fiber, fluids and activity are importantConstipation leads to AUTONOMIC
DYSREFLEXIA!!!
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Urinary RetentionRelated to injury and limited fluid intake
as evidenced by Decreased output Bladder distention Involuntary emptying of bladder
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Urinary RetentionPalpate bladder every shiftDuring acute phase, insert indwelling
catheterBegin intermittent cath program when
appropriateKeep I and O and end fluidsMonitor BUN and creatinineCrude (pronounced croo-DAY)
manuever when voiding/cathing
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Risk for AUTONOMIC DYSREFLEXIA
Assess for HTN, bradycardia, headache, sweating, blurred vision, flushing, nasal stuffiness/congestion
Reduce or eliminate noxious stimuli such as impaction, urine retention, tactile stimulation and skin lesions or pain!
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Autonomic dysreflexiaElevate HOB 43 degrees Identify cause and eliminateTake BP and pulseAdminister antihypertensives as
ordered if hypertensive.Call physician if interventions not
effectiveTEACH CLIENT AND CARGIVERS
HOW TO PREVENT THIS!
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Other diagnoses Impaired physical mobilityAltered nutrition: < body requirementsSexual dysfunctionRisk or injury r/t sensory deficitsAltered family processesRisk for ineffective individual copingBody image disturbance
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Acute intervention
ImmobilizationCrutchfield tongsHalo vestStryker bedRoto-rest bed (side to side)
Motion sickness a problem with these.
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Respiratory dysfunction Intubation if injury is high Decreased tidal volume and shallow
breathing lead to pneumonia and atelectasis
CPT and pain management Prone position may be risky Count to 10 test QUAD COUGH technique to assist with
ineffective abdominal muscles
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Fluids and nutritionParalytic ileus common in 48-72 hoursWhen bowel sounds return:
High calorie, high protein, high fiber diet Evaluate SWALLOWING before feeding!
EATING CAN BECOME A POWER STRUGGLE!
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Bowel and Bladder mgmt. Indwelling catheter initially Intermittent catheterization when ableMonitor pH of urine (should be acetic!)Ascorbid acid and Mandelamine (an
antiseptic) given to keep down bacteria
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Temperature controlNO vasoconstriction, piloerection or
heat loss through sweating below level of injury
Do not over cool or over heat client. They only have the remaining upper portion of their bodies, generally, for temperature adjustment
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