Regurgitant Systolic Murmurs Chapter 15 Are G. Talking, MD, FACC Instructor Patricia L. Thomas, MBA,...
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Transcript of Regurgitant Systolic Murmurs Chapter 15 Are G. Talking, MD, FACC Instructor Patricia L. Thomas, MBA,...
![Page 1: Regurgitant Systolic Murmurs Chapter 15 Are G. Talking, MD, FACC Instructor Patricia L. Thomas, MBA, RCIS.](https://reader036.fdocuments.in/reader036/viewer/2022062417/551c2532550346ad4f8b5d53/html5/thumbnails/1.jpg)
Regurgitant Systolic MurmursChapter 15
Are G. Talking, MD, FACC
Instructor
Patricia L. Thomas, MBA, RCIS
![Page 2: Regurgitant Systolic Murmurs Chapter 15 Are G. Talking, MD, FACC Instructor Patricia L. Thomas, MBA, RCIS.](https://reader036.fdocuments.in/reader036/viewer/2022062417/551c2532550346ad4f8b5d53/html5/thumbnails/2.jpg)
Outline
• Mitral Regurgitation
• Tricuspid Regurgitation
• Ventricular Septal Defect
• Patent Ductus Arterious
• Acute Ventricular Septal Perforation
• Papillary Muscle Rupture
• Mitral Valve Prolapse Syndrome
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Introduction
• Regurgitant Murmurs are caused by retrograde flow across AV valves
• TR heard at the lower left sternal border
• MR heard at the apex
• Holosystolic Murmurs suggest MR, TR, VSD’s
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Chronic Mitral Regurgitation
• Continues as long as LV pressure > that of the enlarged LA
• Begins at S1 and extend through S2
• Large high pitched, blowing holosystolic/pansystolic murmur
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Acute Mitral Regurgitation
• Loud Grade IV or >, diamond shaped
• Pressure in the normal nondilated LA increases rapidly because of regurgitant flow in early systole and = LV pressure in late systole
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Mitral Regurgitation Causes
• Rheumatic Heart Disease
• Papillary Muscle Dysfunction
• Mitral Valve Prolapse
• Rupture Chordae Tendineae
• Calcified mitral Annulus
• LV Dilatation
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Tricuspid Regurgitation
• The holosystolic murmur of MR engulfs A2 but stops before P2 whereas the murmur of TR persists through and engulfs P2
• Increases with inspiration (Carvallo sign) & does not radiate well to the axillary region
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• Mild TR– Infective Endocarditis
seen with IV drug abuse may be mid-systolic of low intensity, heart only with inspiration
– S4 may be present
• Advance TR– May not increase with
inspiration or may be absent
– Tricuspid honk or whoop (highly musical)
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Causes• Tricuspid Insufficiency is commonly secondary to
dilatation of the right ventricle • Severe Right Heart Failure secondary to mitral
stenosis• Pulmonary Heart Disease with pulmonary
hypertension• Congenital deformity (Epstein's Anomaly),
Rheumatic Valve disease, or Infective Endocarditis
• Listen with the diaphragm of the stethoscope along the lower left sternal border (third interspace)
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Ventricular Septal Defect
• Holosystolic, loud, & harsh; S2 is loud & widely split; possible palpable thrill
• Begins with ventricular systole S1, when the rise in LV pressure exceeds that of the RV & continues until S2 when left ventricular pressure falls
• Listen with the diaphragm of the stethoscope from the mid-to lower left sternal border
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• Patent Ductus Arteriosus– Continuous murmur
• Acute Ventricular Septal Perforation– Caused by acute MI– Loud short systolic murmur, grade IV– Listen with diaphragm of stethoscope
• Papillary Muscle Rupture– mid-to late systolic murmur, thrill– Listen with diaphragm for the stethoscope
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Mitral Valve Prolapse Syndrome• Mid-to-late systolic, late systolic, or holosystolic• Moderate Prolapse
– 1/3 or ½ into systole & increases its intensity until A2
– Valve is competent in early systole & prolapse in LA in late systole
• Severe Prolapse– Loud S1, holosystolic murmur– Fusion of a click with S1, Sound is louder
• Click – In < ½ of patients marks onset of the murmur “click
murmur syndrome”
• Cause– Mitral insufficiency
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THE ENDOF
CHAPTER 15
Tilkian, Ara MD Understanding Heart Sounds and Murmurs,
Fourth Edition, W.B. Sunders Company. 2002, pp. 180-196