Pulpal Disorder Ppt

42
Pulpal Disorders Heidi Emmerling, RDH, PhD DHYG 138 Oral Pathology Fall 2007

Transcript of Pulpal Disorder Ppt

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Pulpal Disorders

Heidi Emmerling, RDH, PhD

DHYG 138 Oral Pathology

Fall 2007

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Heidi Emmerling, RDH, PhD DHYG 138 Oral PathFall 2007

PULPITIS

Inflammation of the pulp a general term, describing all pulpal

inflammation

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Heidi Emmerling, RDH, PhD DHYG 138 Oral PathFall 2007

General Information

Pulp is unique in that it is surrounded by rigid dentin and has no collateral circulation because the arteries are end arteries.

Pulp is connective tissue and the inflammation here is similar to inflammation of C.T. elsewhere in the body.

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Heidi Emmerling, RDH, PhD DHYG 138 Oral PathFall 2007

Pulpitis

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Heidi Emmerling, RDH, PhD DHYG 138 Oral PathFall 2007

General Information

The effects of an irritant on the pulp and how the tooth reacts has a lot to do with the resistance of the host.

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Heidi Emmerling, RDH, PhD DHYG 138 Oral PathFall 2007

General Information

Irritants: Microorganisms:

One of the most common forms of irritation.

Decay, fractures, etc. Trauma, Blows to tooth,

Bruxism

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Heidi Emmerling, RDH, PhD DHYG 138 Oral PathFall 2007

General Information

Irritants (Cont) Iatrogenic: A condition

produced by improper dentistry Drilling, polishing (heat),

others

Chemical Eugenol, restorative

materials, cements.

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Heidi Emmerling, RDH, PhD DHYG 138 Oral PathFall 2007

General Information

Irritants (Cont) Systemic disorders:

Diabetic infections and

pulp disease Sickle cell anemia

Lack of oxygen to the pulp.

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Heidi Emmerling, RDH, PhD DHYG 138 Oral PathFall 2007

General Information

Pain with pulpitis is due to pressure from inflammation within a confined chamber.

Progressive inflammatory changes in the pulp are: Hyperemia Acute Suppurative Pulpitis Acute Serous Pulpitis Chronic Pulpitis

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Heidi Emmerling, RDH, PhD DHYG 138 Oral PathFall 2007

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Heidi Emmerling, RDH, PhD DHYG 138 Oral PathFall 2007

HYPEREMIA

An increase in the amount of blood in the vessels of the pulp.

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Heidi Emmerling, RDH, PhD DHYG 138 Oral PathFall 2007

Etiology

Caries: most common cause Lost temporary or permanent restoration. Heat: High speed drill; polishing cup. Traumatic injury: Mild blow to tooth Occlusal trauma Chemical irritants: Bases, liners. Galvanic shock: An electrical current

caused from saliva and two different metals.

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Heidi Emmerling, RDH, PhD DHYG 138 Oral PathFall 2007

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Heidi Emmerling, RDH, PhD DHYG 138 Oral PathFall 2007

Symptoms

Pain during eating due to ordinary temperature changes. Sharp pain that lasts for a short period of time.

Pain from sweet and sour food that last for a short period of time.

X-rays normal; hyperemia is confined to the pulp PDL is normal; may show caries

Vitality Test: electric pulp tester: readings are lower than normal

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Heidi Emmerling, RDH, PhD DHYG 138 Oral PathFall 2007

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Heidi Emmerling, RDH, PhD DHYG 138 Oral PathFall 2007

Treatment:

Remove the irritant. Pain should go away.

If pain persists, then it's Acute Serous Pulpitis.

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Heidi Emmerling, RDH, PhD DHYG 138 Oral PathFall 2007

ACUTE SEROUS PULPITIS

Etiology: Same as Hyperemia Symptoms: Similar to Hyperemia, but more:

Severe Prolonged Pain may come and go without an apparent cause.

Vitality Test: EPT: Very sensitive and lower-than-normal readings. Ice: A quick response of pain. Heat: No noticeable response.

X-rays Everything appears normal.

Treatment: Remove the irritant

If pain persists, do a RCT.

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Heidi Emmerling, RDH, PhD DHYG 138 Oral PathFall 2007

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Heidi Emmerling, RDH, PhD DHYG 138 Oral PathFall 2007

ACUTE SUPPURATIVE PULPITIS

Etiology: Usually caries in close proximity to the pulp.

Symptoms: Pain that is excruciating, throbbing,

continuing, especially at night. Very tender to percussion.

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Heidi Emmerling, RDH, PhD DHYG 138 Oral PathFall 2007

ACUTE SUPPURATIVE PULPITIS

X-rays: everything appears normal Vitality Test:

Percussion: very tender EPT: confusing; may be low or

negative depending on stage of pulpitis early=low; late=dead

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Heidi Emmerling, RDH, PhD DHYG 138 Oral PathFall 2007

ACUTE SUPPURATIVE PULPITIS

Ice: relief of pain (reduces pressure caused

by gas from necrotic tissue Heat: pain Treatment: Open tooth; let drain Medication for a few days. Do a RCT

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Heidi Emmerling, RDH, PhD DHYG 138 Oral PathFall 2007

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Heidi Emmerling, RDH, PhD DHYG 138 Oral PathFall 2007

CHRONIC PULPITIS

Etiology A low grade infection. Pulp dies slowly with NO ACUTE

PAIN. Patient may experience pain off-and-on, but ignores this pain.

Symptoms: usually asymptomatic occasional sharp pain

X-rays: may be negative or may have a radiolucency at the apex

depending on stage of pulpitis Vitality Test:

EPT: high readings or no response at all Hot/cold:no response

Treatment: rct

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Heidi Emmerling, RDH, PhD DHYG 138 Oral PathFall 2007

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Heidi Emmerling, RDH, PhD DHYG 138 Oral PathFall 2007

PULP STONES

Large calcification in the pulp. Of no significance unless a RCT is

needed.

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Heidi Emmerling, RDH, PhD DHYG 138 Oral PathFall 2007

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Heidi Emmerling, RDH, PhD DHYG 138 Oral PathFall 2007

CHRONIC HYPERPLASTIC PULPITIS (Pulp Polyp)

An excessive proliferation of chronically inflamed dental pulp tissue.

Occurs in teeth with large, open carious lesions in children.

Primary and permanent teeth A red or pink nodule of tissue, filling the entire cavity

of the tooth and protrudes from the pulp chamber. The tissue is granulation tissue. Treat by extraction or RCT.

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Heidi Emmerling, RDH, PhD DHYG 138 Oral PathFall 2007

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Heidi Emmerling, RDH, PhD DHYG 138 Oral PathFall 2007

SEQUELEA TO PULPITIS

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Heidi Emmerling, RDH, PhD DHYG 138 Oral PathFall 2007

PERIAPICAL ABSCESS

This abscess is composed of purulent exudate or pus. The patient will be in pain due to the pressure of the exudate. The pus will seek a path of escape by going toward the path-

of-least resistance, and forming a fistula, or spreading to other tissues.

The tooth is quite painful and extruded slightly from its socket. It will be in hyperocclusion.

Treatment is to establish drainage if possible, by opening the tooth through the pulp chamber, or extraction.

If the abscess invades other tissue, an incision may be necessary.

Antibiotics are also indicated in many cases.

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Heidi Emmerling, RDH, PhD DHYG 138 Oral PathFall 2007

PERIAPICAL GRANULOMA

A localized mass of chronic granulation tissue at the apex of the tooth due to a chronic stimulus.

The tooth is usually asymptomatic for the most part. (Some slight discomfort at times; may be slightly extruded.)

Treatment is to extract or do a RCT. X-rays will show a slight thickening of the

periodontal ligament space and/or a diffuse radiolucent area at the apex.

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Heidi Emmerling, RDH, PhD DHYG 138 Oral PathFall 2007

RADICULAR CYST (PERIAPICAL CYST)

A true cyst, a pathological cavity lined by epithelium.

The cyst develops from a granuloma when the granuloma proliferates epithelial rest of Malassez.

As the cells in the center of the granuloma become more distant from the lining, they die and liquify, forming a liquid center.

A granuloma and a cyst are identical except for the epithelial lining of the cyst.

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Heidi Emmerling, RDH, PhD DHYG 138 Oral PathFall 2007

RADICULAR CYST (PERIAPICAL CYST)

No symptoms are apparent. X-rays show a radiolucent area with a well

circumscribed, not diffuse margin. It is difficult to determine a granuloma from a

cyst. The radiopaque lining of the cyst may be the determining factor in diagnosis.

Treatment is to extract the tooth or perform a RCT. Then the cyst must be curetted from the apical area.

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Heidi Emmerling, RDH, PhD DHYG 138 Oral PathFall 2007

TOOTH RESORPTION

Roots are resorbed in the same manner as bone. If it resorbs from the outside, it is called external

root resorption This occurs during eruption of permanent teeth on

the roots of the deciduous teeth. It also occurs to 2nd molars during eruption of third molars.

Can occur with improper forces during orthodontic treatment.

Can occur in response to pressure from granulomas and cysts.

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Heidi Emmerling, RDH, PhD DHYG 138 Oral PathFall 2007

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Heidi Emmerling, RDH, PhD DHYG 138 Oral PathFall 2007

Tooth Resorption

If the root resorbs from the inside, it is called internal root resorption Causes cannot always be seen. It has something to do with the inflammatory process. If internal resorption occurs in the crown, it can often be

seen during the oral exam. The tooth looks pink. If in the root, it can only be seen on x-rays. The pulp

canals will be unequally larger in certain areas. Treatment: If treated early—RCT; if late: the tooth

will be lost

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Heidi Emmerling, RDH, PhD DHYG 138 Oral PathFall 2007

CONDENSING OSTEITIS

A change in bone, near the apices of the teeth due to a low-grade infection.

Radiopaque area extending beyond the apex of the tooth.

Most common tooth effected is the mandibular first molar.

Treatment is not necessary. Biopsy may be needed to rule out other pathology.

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Heidi Emmerling, RDH, PhD DHYG 138 Oral PathFall 2007