Pathology The pathology involving the CNS arises from injuries, vascular insufficiency, tumors,...
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Transcript of Pathology The pathology involving the CNS arises from injuries, vascular insufficiency, tumors,...
Pathology
The pathology involving the CNS arises from injuries, vascular insufficiency, tumors, infections and disorders from other diseases. Neurological medical problems are due to interference with normal functioning of the affected cells
Nervous SystemAnatomy and PhysiologyReview
The nervous system acts as a coordinated unit both structurally and functionally
Communication network responsible for coordinating and organizing the functions of all body parts
The body’s link to the environment Works with the endocrine system to maintain
homeostasis Reacts in a split second
Functions
1.Regulates system 2. Controls communication 3. Coordinates Activities of body system
Divisions
Central nervous system ( CNS) : brain and spinal cord –interprets incoming sensory information and sends out instruction based on past experiences
Peripheral nervous system ( PNS) : Cranial and spinal nerves extending out from brain and spinal cord---carry impulses to and from brain and spinal cord
Neurological Terms
Anesthesia- complete loss of sensation Aphasia-loss of ability to use language Auditory/receptive aphasia- loss of ability to understand Expressive aphasia- loss of ability to use spoken or
written word Ataxia- uncoordinated movements Coma- state of profound unconsciousness Convulsion- involuntary contractions and relaxation of
muscles
Neurological terms
Delirium- mental state characterized by restlessness and disorientation
Diplopia- double vision Dyskeinesia- difficulty in voluntary movement Flaccidd- without tone- limp Neuralgia- intermittent, intense pain, along the
course of a nerve
Neurological terms
Neuritis- inflammation of a nerve or nerves Nystagmus- involuntary, rapid movements of the
eyeball Paresthesia- abnormal sensation without obvious
cause, with numbness and tingling Stupor- state of impaired consciousness with brief
response only to vigorous and repeated stimulation Vertigo- dizziness
Preparing a patient for a diagnostic test
Answer question that the patient may need clarification
Diet orders –NPO??? Special room or equipment
used Special medications required
for test An informed patient will be
more cooperative Nursing assessment
Baseline vital signs and neuro cks
Know level education to develop an individualized teaching plan
Determine awareness of actual or potential medical diagnosis
Determine previous experence with Dx test
Diagnostic test/ methods
A. Computerized Tomography- CT or CAT scan computer analysis of tissues as x-rays pass through them; has replaced many of the usual tests: no special preparation or care after test
CT scan
Nursing Interventions– Explain procedure – will be enclosed tunel– Written consent– Assess allergies to iodine– Remove wigs hair pins or clips, partial denture plates – Assess for pacemakers– NPO 4 hours before if oral contrast is administered– Encourage patient to drink fluids to avoid renal complications
and to promote excretion of the dye
Diagnostic test/ methods
B. lumbar puncture- spinal tap– Done under local anesthesia a puncture is made at
the junction of the third and fourth lumbar vertebrae to obtain a specimen of cerebrospinal fluid (CSF)
– CSF pressure measured – Used to inject medications- spinal anesthesia– Used to inject diagnostic materials –air or dye-
myelogram
Lumbar puncture
Nursing interventions– Written consent– Monitor vital signs– Have patient empty bowel and bladder – Position the patient– Label and number specimens– Keep patient supine 4-8 hours– Observe for headache and nuchal rigidity– Observe for mobility of extremities, pain, ability to void– Monitor site for leakage
Diagnostic test/ methods
Cerebral Angiography- intraarterial injection of radiopaque dye to obtain an xray film of the cerebrovascular circulation
Cerebral angiography
Nursing interventions– Written consent– Assess for allergy to iodine– NPO past midnight– Administer preprocedure medications– Observe arterial puncture site– Monitor extremity for adequate circulation- pain tenderness
bleeding temperature and color– Pedal pulses and vital signs q 1 hour– Provide ice pack to puncture site– Bedrest 12- 24 hours– Force fluids- to increase excretion of dye
Diagnostic test/ methods
Electroencephalography (EEG)- electrodes are placed on unshaven scalp with tiny needles and electrode jelly
EEG
Nursing Inventions– Anticipate patient’s fears about electrocutions– Explain procedure– Written consent– Hair should be clean– Do not give stimulants/ depressants before test /consult with M.D. about meds– Administer sedatives or hypnotics if ordered– No smoking or caffeinated beverages before the test– Eat full meal before the test –hypoglycemia may alter brain waves– Stress need for restful sleep before the test sleep deprivation may cause
abnormal brain waves– Wash hair and scalp after test– Ensure safety precautions until effects of meds wear off
Diagnostic test/ methods
Brain Scan-after injection of a radioisotope, abnormal brain tissue will absorb more rapidly than normal tissue: this can be detected with a Geiger counter to diagnose brain tumors
Brain Scan
Nursing interventions– NPO 4 hours before test– Remove wigs, hair clips or pins,– Assess for iodine allergies– If ordered give sedation– Encourage fluids after test to increase excretion of
dye
Diagnostic test/ methods
Magnetic Resonance Imaging- ( MRI)
uses combination of radio waves and a strong magnetic field to view soft tissue ( does Not use x-rays or dyes) ; produces a computerized picture that depicts soft tissues in high –contrast color
MRI
Nursing interventions– Written consent– Explain procedure- will have to remain perfectly still
in the narrow cylinder-shaped machine . No pain or discomfort but no room for movement
– Assess for any metal contraindications-pacemaker, surgical clips, hair clips, belts
– Empty bladder before test
Diagnostic test/ methods
Myelogram- injection of a radiopaque dye into the subarachnoidd space via a lumbar puncture: performed to locate lesions of the spinal column or ruptured vertebral disk
Myleogram
Nursing interventions– Written consent– Prepare for LP– NPO for 4 hours before test– Positioning for LP– Vital signs– Observe for photophobia, fever stiff neck, occipital headaches, nausea ,
dizziness, and possibly seizures– Force fluids to promote dye excretion dehydration will result in severe
headache– Check with M.D. when withheld medications prior to test may be restarted– Observe site for leakage of CSF– Bedrest
Nursing Diagnosis and Interventions
Identify the patients needs Neuro checks Assessment of history from family Patient history Nursing observations
Impaired Physical Mobility
Neuro checks q2-4h Explain the need for regular
exercise program ROM to all joints q2-4h
foundations pg 243-244 Use assistive devices Protect the affect side from
injury Protection from falling Turn q2h
Risk for injury/infection related to fixed eyes ( no blinking)
Protect with eye shields Remove dry exudate
with warm saline Close eyes Inspect for inflammation
Ineffective breathing pattern related to neuromuscular impairment
Maintain patent airway Suction as needed Elevate HOB 30-60-
degrees Have trach set ready Provide O2 with humidity V/S with neuro cks q2h Oral hygiene q2h Lubricate lips
Maintain bed rest Keep unconscious pt in
lateral position to allow secretion drainage
Monitor for S/S pulmonary emboli
– Chest pain, SOB, Monitor ability to swallow
Risk for alteration in body temperature
Asses rectal temp q2h Use external heating or
cooling blankets
Risk for aspiration
Maintain NPO Position Pt on side: turn
q2h Provide N/G feedings Monitor IV fluid
Altered patterns of urinary elimination
1. Oligura-urinary retention
– Provide indwelling catheter
– Monitor I&O qh
– 2. Incontinence Wash dry and inspect
skin Implement measures to
prevent decubitus ulcers Implement bladder
training
Bowel incontinence/constipation
Incontinence
wash dry and inspect skin
Implement measures to prevent decubitus ulcers
Implement bowel training
Constipation
-Record bowel movements
-Provide stool softners, laxatives and enemas
-Check for impaction
-Increase fluid intake
-Increase Fiber in diet
-Increase activity
Altered Nutrition: less than body requirements related to dysphagia and fatigue
Prepare for N/G feedings
Check gag reflex Provide mouth care,
clean and care for dentures
Place food in patients visual field do patient can see food
Diet low salt low cholesterol
consult dietary Wt daily
Impaired Communication
Assess communication patterns
Provide calm environment with minimal distraction
Use touch to increase attention
Use familiar music to enhance recall
Simple verbal commands
Communication boards Pen and paper Gestures eye blinks
Fluid Volume deficit
Inability to meet needs:Coma
COMA-Unconscious state in which the Pt is unresponsive to verbal or painful stimuli: this occurs with many primary diseases: the Pt depends on the nurse for maintenance of all basic human needs, nourishment, bathing, elimination, respiration, prevention of complications and assessment and provision of care for problems
Coma : nursing interventions
Include family in nursing care and planning Note LOC q15 minutes Nero Ck q 15 minutes Demonstrate respect for Pt presence Provide quite restful environment Speak to Pt, use proper name, introduce self,
explain all care Provide privacy
Patient with paralysis
Paraplegia-paralysis of the lower extremities There may be no motion or sensory function or reflexes There may be uncontrollable muscle spasms Perspiration ceases then becomes profuse Loss of bowel and bladder control Anxiety, fear, depression, anger, and embarrassment May be totally dependant
Patient with paralysis
Quadriplegia- paralysis of all four extremities Same problems as paraplegia
Nursing interventions : Paralysis
Take measures to prevent complications of immobility Bowel and bladder training Prevent deformity: maintain joint mobility: correct
alignment Increase fluid intake Provide high protein diet Teach independence according to ability Work with health care team for rehabilitation Include family in planning and care
Increased intracranial pressure( ICP)
Fluid accumulation or a lesion takes up space in the cranial cavity, producing ICP: the brain is gradually compressed, or life-sustaining functions cease: may be sudden or progress slowly
ICPCauses
Tumors Hematoma Edema from trauma Abscesses from infection
ICPsigns and symptoms
Headache, restless, anxiety Vomiting,recurrent, projectile,
and not related to nausea or meds
Change in pupil response to light
Seizures Respiratory difficulty;
irregular, Cheyne-Stokes or Kussmaul
BP elevates ,with wide pulse pressure
Pulse Increases at first then slows to 40- 60
Alter LOC,lethargic, speech slows, confused, decrease level of response
Visual disturbances,diplopia and blurred vision
Progressive weakness or paralysis
Loss of consciousness,coma death
ICPTreatment
Depends on cause Craniotomy Meds
– Steroids– Anticonvulsants– Mannitol– dexamethasone
ICPNursing interventions
Elevate HOB to semi-Fowler’s Never place in Trendelenburg V/S and neuro cks q15 minutes Prevent aspiration Place Pt on Side Maintain airway- O2 Observe pupillary response ( usually unequal and may not react to light) Report changes in LOC immediately Seizure precations Provide care for Coma Pt Monitor IV fluids– Do not overhydrate NPO or fluid limited by M.D. I & O q1h
Convulsive disorders
Frequently a convulsion or seizure is not a disease but a symptom of a neurologic disorder:
Epilepsy is a disease characterized by a disposition for seizures;
Types of seizures
Generalized or grand mal Aura- There may be a premonition or sign The Pt cries out Loss of consciousness Enters tonic phase- the body is rigid and the jaw is clenched Then the clonic phase- jerking movements of muscles Cessation of respiration Fecal and urinary incontinence Lasts 1-2 minutes Followed by short period of unresponsiveness
Types of seizures
Partial or petit Mal Loss of consciousness that last 5- 30
seconds Normal activities may or may not ceas There may be amnesia concerning the time
Types of seizures
Jacksonian or Motor A focal seizure that may precede a grand mal
seizure
Convulsive Disorders Causes
May be secondary to another condition– CVA, head injury, brain tumor, elevated temp,
toxins, electrolyte imbalance
Epilepsy may have no known cause– Onset is usually during childhood or before age 30
Convulsive DisordersDiagnostic test
EEG CT scan MRI
Convulsive DisordersTreatment
Treat and remove cause Anticonvulsant drugs Surgery – sterotactic- electrical stimulation to
locate and reset ( destroy) epileptogenic focus
Convulsive DisordersNursing Interventions
Provide accurate observation and documentation Aura Time of onset Whether seizure is generalized or focal Specific parts of body involved Progression of seizure Eye movements Loss of consciousness Loss of bowel or bladder Condition after seizure Memory loss Weakness Any injury caused by seizure
Convulsive DisordersNursing interventions
Encourage Pt to wear medical alert tag Have suction available During seizure maintain airway Prevent head injury Place pt on side Protect extremities from injury Do not restrain Loosen clothing Remove pillows Maintain safety until fully conscious
Transient Ischemic AttacksTIA
Altered cerebral tissue perfusion related to a temporary neurologic disturbance
Manifested by sudden loss of motor or sensory function
Lasts for a few minutes to a few hours Caused by temporarily diminished blood supply
to an area of the brain High risk for stroke
TIATreatment
Control hypertension Low sodium diet Possible anticoagulant therapy Stop smoking
Cerebrovascular AccidentCVA Stroke
Decreased blood supply to a part of the brain caused by rupture , occlusion, or stenosis of the blood
vessels Onset may be sudden or gradual Symptoms and patient problems depend on location and
size of area of brain with reduced or absent blood supply right CVA results in Left side involvement often
associated with safety/ judgment Left CVA results in Right side involvement often
associated with speech problems
Cerebrovascular AccidentCVA Stroke
Symptoms related to location and size of brain area affected
Approximately 50% of survivors permanently disabled High proportion experiencing recurrence within weeks
to years Chances for complete recovery depending an
circulation returning to normal soon after the initial stroke
Third most common cause of neurological disability
Predisposing factors-CVA
History TIA’s Hypertension Arrhythmias Atherosclerosis Rheumatic Heart
Disease MI DM
High serum triglyceride levels
Lack of exercise Cigarette smoking Family history
CVACauses
Incidence increased with aging Atherosclerosis Embolism Thrombosis Hemorrhage from ruptured cerebral aneurysm hypertension
CVASigns and Symptoms
Altered LOC Change in mental status Decreased attention span Decreased ability to think and reason Difficulty following simple directions Communication; motor and sensory aphasia difficulty
with reading ,writing, speaking, or understanding Bowel and bladder dysfunction retention impaction or
incontinence
CVASigns and Symptoms
Seizures Limited motor function; paralysis, dysphgia, weakness ,
hemiplegia, loss of function Loss of sensation/ perception Headaches and syncope Loss of temp regulation elevated TPR and BP Absent of gag reflex ( aspiration) Unusual emotional responses; depression, anxiety,
anger, verbal outburst, and crying: emotional lability Problems related with immobility
CVADiagnostic test
Physical assessment Pt and family history EEG CT scan Lunbar puncture Cerebral angiogram Carotid ultrasonogram
CVATreatments
Remove cause, prevent complications, and maintain function, rehabilitation to restore function
Meds– Antihypertensives– Anticoagulants– Stool softners
Surgical removal of clot, repair of aneurysm, carotid endarterectomy or balloon agioplasty
CVANursing Interventions
Patent airway Maintain bedrest Provide complete care Use turn sheet Footboard Firm mattress Pillow and torchanter rolls Maintain proper body
alignment
Place items within reach Reposition q2h ROM passive and active Place in chair Flotation mattress or
sheepskin Skin assessment
CVANursing Interventions
O2 with humidity C,T, DB q2h Suction PRN Keep head turned to
side Place in semi- fowler’s
Assess nutrition daily with I&O, WT, %diet, calorie count
Provide N/G feedings if needed
Maintain IV fluids Progress to soft diet prn TPN as ordered Aspiration precautions Dietary consult & Speech for
swallowing
CVANursing interventions
Establish means of communication
Nonverbal gestures Speak slowly Explain all care Speech therapy
Encourage family participation
CVANursing Interventions
Assess LOC Maintain safety Use side rails Restrain only as
necessary Observe for ICP V/S & Neuro CKS q 4 h Seizure precations
Ensure elimination Assess bowel sounds Monitor bowel
movements I & O Indwelling catheter prn Bowel and bladder
training
CVANursing interventions
Family support Begin discharge
teaching early Physical therapy Speech therapy
Brain Tumor
A benign or malignant growth that grows a nd exerts pressure on vital centers of the brain decreasing function and causing increased intracranial pressure
Cause is unknown
Brain TumorSigns and Symptoms
Personality changes, fear and anxiety H/A , dizziness and visual disturbances Seizures Pituitary dysfunction ICP Local paralysis or anesthia Aphsia Problems with coordination
Brain tumorDiagnostic test
History Physical exam Neurologic assessment EEG CT Angiogram MRI
Brain tumortreatment
Surgical removal –craniotomy Combination of radiation or chemotherapy
Brain tumornursing interventions
Neuro cks q 1-4 hours depending on pt status
Safety Seizure precautions express fears and
feelings
POST OP care– Maintain airway– Seizure precautions– Regulate body temp– Position on unoperated side– Elevate HOB ONLY under
MD orders– Inspect dressing q30min– V/S neuro cks q 15 min
progress to q4h– Coma care
Head injuries
Trauma to scalp, skull, or brain. A fracture to skull may result either a simple break in the bone or bone fragmentation that penetrates the brain tissue, can also cause hemorrhage, concussion, or contusion
Head injuries
Cerebral concussion- injury to the head, patient may be dazed; or unconscious for a few minutes: some function(memory) may be impaired for as long as several weeks
Cerebral contusion- head injury causing bruising of brain tissue> person experiences stupor, confusion or loss of consciousness: if severe may go into coma
Head injuries
Cerebral laceration- a break in continuity of brain tissue
Causes– Blow to head– MVA– Fall
Head injuries Signs and Symptoms and diagnostic test
Nausea & vomiting Lethargic: increasing
loss of consciousness to impending coma
Disorientation Drainage of CSF from
ear or nose ICP
History and physical exam
X-ray of head Angiogram, doppler
studies CT head, MRI PET
Head injuriesTreatment
Anticonvulsulants Corticosteriods Mannitol Maintain fluid balance surgery
Head injuriesNursing interventions
Care for ICP COMA care Neuro cks & V/S q 15
min to q1h Maintain airway Seizure precations Observe ears and nose
for CSF
Multiple Sclerosis
A chronic progressive disease of the brainand spinal cord: lesions cause degeneration of the myelin sheath and interfere with conduction of motor nerve impulses: there are periods of remissions and exacerbations: onset occures in young adult: it has an unpredictable progression
Cause: unknown< exacerbates with stress
Multiple SclerosisSigns ands symptoms
Ataxia Paresthesia Weakness and loss of
muscle tone Loss of sense of position Vertigo Blurred vision –progress
to blindness
Inappropriate emotions– Euphoria, apathy,
depression
Dysphagia Slurred speech Bowel and bladder
dysfunction Sexual dysfunction spasticity
Multiple SclerosisDiagnostic test and treatments
History Physical exam Neuro Cks Ct MRI Exam of CSF
Treatment is symptomatic
Corticosteriods during acute excerbation
Multiple SclerosisNursing interventions
Prevent Complications of immobility
Encourage independence Patient should participate in
plan of care High calorie, vitamin, protein
diet Family education
Bowel and bladder training Safety Express feelings regarding
dependence and disabilities Avoid precipitating factors for
exacerbationsFatigue, cold, heat, infections,
stress
Parkinson’s Disease
A progressive , degenerative disease causing destruction of nerve cells in the basal ganglia of the brain caused by a deficiency of dopamine: limbs become rigid, fingers have characteristic pill rolling movement, and head has to and for movement: the patient has a bent position and walks in short, shuffling steps: facial expressions become blank with wide open eyes and infrequent blinking ( parkinson’s Mask)
Intelligence is NOT affected
Parkinson’s DiseaseSigns and symptoms
Tremor Voluntary movement is slow
and difficult Coordination is poor- ataxia Impaired chewing and eating Excessive salivation and
drooling Speech is slow Patient is soft spoken Written communication is
difficult
Excessive sweating Emotional changes
– Depression , confusion
dependency
Parkinson’s DiseaseDx test and treatments
History Physical exam Neuro cks
Many pt s respond to drug therapy and the disease is controlled with meds for the reminder of their lives
Others have no response to meds - invalidism
Parkinson’s Diseasenursing interventions
Foster independence ADL’s
Avoid social withdrawal –involve in work, social and diversional activities
Aviod embarrassment while eating
– Use straws, wipe drool, use bib, keep clothing clesn, use large handle grips
Soft diet Daily walking—safety Avoid fatigue Physical, Speech and
Occupational therapy Avoid constipation-stool
softner
Parkinson’s Diseasenursing interventions
Bowel and bladder training
Be patient when patient is slow and clumsy
Establish a means of communication
Reorientation Prevent pneumonia
Mouth care q4h Family participation
Spinal Cord Impairment
The vertebral column houses the spinal cord. A small cartilage disk acts as a cushion between the vertebrae. All sensory and motor nerves to the neck, trunk, and extremities branch out from the spinal cord. The degree of disability and patient problems is related the part of the body controlled by the injured or disease nerves
Spinal Cord Injuries
Trauma to spinal cord may cause complete or partial severing of the spinal cord
If severing is complete there is permanent paralysis of body parts below site of injury
When there is partial damage edema may cause a temporary paralysis
Spinal Cord Injuries
Cause : accident ,MVA diving, shooting S&S individual to site, respiratiory distress,
paralysis DX test: physical exam Treatment: immobilization
– Crutchfield tongs.halo traction.brace.body cast– Surgery corticosteroids, mannitol
Spinal Cord InjuriesNursing interventions
Care for paralysis patient Observe for complications of spinal shock Maintain airway and respiratory function