Otitis Media Akuta
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Transcript of Otitis Media Akuta
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OTITIS MEDIA AKUTA
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OTITIS MEDIA AKUTA
OTITIS MEDIA AKUTA : RADANG AKUT MUKOSA TELINGA TENGAH YANG DIIKUTI PEMBENTUKAN PUS DI KAVUM TIMPANI OTITIS MEDIA AKUTA PADA BAYI DAN ANAK SERING MERUPAKAN LANJUTAN INFEKSI SAL NAFAS ATAS:
RHINITIS AKUTA SINUSITIS AKUTA
ADENOIDITIS
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• TRAUMA• FRAKTUR BASIS KRANII • LEDAKAN ATAU LUKA TUSUKAN• HEMATOGEN
KUMAN PENYEBAB:• STREPTOKOKUS• HEMOPHILUS INFLUENZA• PNEUMOKOKUS• PSEUDOMONAS• BACTERIODES FRAGILIS
PENYEBAB LAIN :
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OTITIS MEDIA AKUTA DENGAN SEBAB RHINOGEN 4 STADIUM
1. STADIUM KATARALIS (OKLUSIO TUBA)
2. STADIUM SUPURASI3. STADIUM PERFORATA4. STADIUM RESOLUSI
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1. STAD. KATARALIS (OKLUSIO TUBA)
INFEKSI SAL. NAFAS ATAS ↓
UDEM MUKOSA TUBA ↓ FUNGSI TERGANGGU ↓ VAKUM TELINGA TENGAH ↓ PERMIABILITAS MENINGKAT ↓ HYDROP EX VAKUO
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KLINIS : - OTALGI RINGAN - TELINGA GREBEG-GREBEG (TERASA BERAIR) - PENDENGARAN MENURUN - GEJALA DARI URI
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OTOSKOPI MEMBRANA TIMPANI RETRAKSI HIPEREMI KADANG-KADANG AIR FLUID LEVEL
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• TERAPI :– DEKONGESTAN ORAL ( PSEUDOEFEDRIN) – DEKONGESTAN LOKAL :
GLUKOPHEDRIN DEWASA 1% ANAK 1/2 % BAYI 1/4 %
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• ANTIBIOTIKA :– AMPISILIN DEWASA 4 X 500 MG,
ANAK 4 X 25 MG/KG/HARI ATAU– AMOKSISILIN DEWASA 3 X 500 MG,
ANAK 3 X 10 MG/KG/HARI ATAU– ERITROMISIN DEWASA 4 X 500 MG,
ANAK 4 X 10 MG/KG/HARI SELAMA 7 HARI
• SIMTOMATIS : PANAS ANTIPIRETIK
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2. STADIUM SUPURASI
( B0MBANS ) PATOFISIOLOGI:VAKUM TRANSUDASI
PENETRASI KUMAN EKSUDASI
BOMBANS
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• KLINIS :– OTALGI HEBAT– FEBRIS TINGGI– URI– KONVULSI– DIARE
OTOSKOPI :
-BOMBANS
-HIPEREMIA
TERAPI :
-PARASINTESA
-DEKONGESTAN
-ANTIBIOTIKA
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3. STADIUM PERFORATA
BOMBANS TAK DIPARASENTESA
PECAH SPONTANPERFORASI
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• KLINIS:– TEK. MENURUN :
• OTALGI BERKURANG
• PANAS BERKURANG
– OTORE +– GANGGUAN
PENDENGARAN– URI
OTOSKOPI :
-PERF. SEKRET MOLOR
-HIPEREMI PULSASI
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• TERAPI :– BOMBANS PARASENTESA– ANTIBIOTIKA– DEKONGESTAN
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4. STADIUM RESOLUSI (PENYEMBUHAN)
PATOLOGI :– PROSESE SEMBUH :
•OEDEM ↓, •HIPEREMI ↓
– GANGG. FUNGSI TUBA BERKURANG
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• KLINIS : – KELUHAN
MENURUN
• OTOSKOPI :– POSISI
MEMBRAN KEMBALI NORMAL
– WARNA PUTIH SEDIKIT HIPEREMI,
– PERFORASI. +
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TERAPI: • OBAT TIDAK PERLU • HINDARI !! :
– URI– KEMASUKAN AIR, KOREK-2
• OMA SEMBUH : 10 – 14 HARI • PERF. KECIL–DPT. MENUTUP/SIKATRIK• FUNGSI PEND. NORMAL : 1 – 2 BLN.
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KOMPLIKASI OMA
1. MASTOIDITIS AKUT2. MENINGITIS3. EXTRADURAL ABSES4. PETROSITIS5. FASIALIS PARESE6. LABIRINTITIS7. ABSES OTAK
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MASTOIDITIS AKUT
• INFEKSI SELULAE MASTOID AKIBAT • PENJALARAN INFEKSI KAV.TIMPANI• ETIOLOGI :
– KUMAN PENYEBAB :•S. PNEUMONIA •S. AUREUS•H. INFLUENZAE •PS. AERUGENOSA•BASILUS FRAG
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• OMA UMUMNYA SEMBUH• SEBAGIAN KECIL MAST. AKUT• FAKTOR BERPENGARUH :
– VIRULENSI / RESISTENSI KUMAN– ANATOMI ANTRUM, PNEUMATISASI– RESISTENSI MUKOSA (IMUNOLOGI)– DAYA TAHAN PENDERITA
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DIAGNOSE :I. ANAMNESA :
1. TELINGA KELUAR CAIRAN 6-8 MINGGU2. NYERI BELAKANG TELINGA
II. PEMERIKSAAN:1. PERUBAHAN POSISI AURIKEL2. HIPEREMIA DAN NYERI TEKAN MASTOID3. PENURUNAN DDNG POST. MAE
(SAGGING)4. X – FOTO MASTOID: DESTRUKSI
SELULAE MASTOID
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ABSES RETROAURIKULER
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3. DIAGNOSA BANDING :FURUNKEL MEATUS EKSTERNUS
INFEKSI MELUAS :•ABS. SUBPERIOSTAL•PARESE N. FASIALIS•LABIRINTITIS•INTRAKRANIAL
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TERAPI
1. MASTOIDEKTOMI SIMPEL 2. ANTIBIOTIKA3. SIMPTOMATIS
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