Neurologic Nursing
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Transcript of Neurologic Nursing
Medical-Surgical Nursing Neurologic System Reviewer
Components of the Nervous System
1. Central nervous system(CNS) - consist of the brain and spinal cord 2. Peripheral nervous system(PNS)-consist of 12 pairs of cranial nerves and 31 pairs of spinal nerves
2 Division of the Peripheral Nerves
1. Somatic Division- Communicates with the skin and skeletal muscles.
2. Autonomic Division- communicates with smooth muscles, heart muscle and glands. 2 Groups of Nerves in the Autonomic Division
1. Sympathetic Nervous System 2. Parasympathetic Nervous System
S y m p a t h e t i c N e r v o u s S y s t e m ( S N S ) P a r a s y m p a t h e t ic N e r v o u s S y s t e m ( P N S )
“Fight” or aggression response “Flight” or withdrawal response
Also termed adrenergic or parasympatholytic response Also termed cholinergic or sympatholytic response
The neurotransmitter for the SNS is norepinephrine The neurotransmitter for the PNS is acetylcholine (Ach)
AA ll ll bb oo dd yy aa cc tt ii vv ii tt ii ee ss aa rr ee II NN CC RR EE AA SS EE DD ee xx cc ee pp tt GG II TT !! AA ll ll bb oo dd yy aa cc tt ii vv ii tt ii ee ss aa rr ee DD EE CC RR EE AA SS EE DD ee xx cc ee pp tt GG II TT !!
increased blood flow to brain, heart and skeletal muscles: These are the most important organs during times of stress
normalized blood flow to vital organs
increased BP, increased heart rate: To maintain perfusion to vital organs
decreased BP, decreased heart rate
bronchodilation and increased RR: To increase oxygen intake
bronchoconstriction, decreased RR
urinary retention FLUID VOLUME EXCESS Fluids are withheld by the body to maintain circulating volume
urinary frequency FLUID VOLUME DEFICIT
pupillary dilation: MYDRIASIS: To increase environmental awareness during aggression
pupillary constriction: MIOSIS [this is the correct spelling, not meiosis ]
decreased GIT activity: CONSTIPATION and DRY MOUTH: Blood flow is decreased in the GIT because it is the least important area in times of stress
Increased GIT: DIARRHEA and INCREASED SALIVATION
DRUGS WITH SNS effects: DRUGS WITH PNS effects:
Adrenergic/Parasympatholytic agents: Epinephrine [Adrenalin]
Antipsychotics: Haloperidol [Haldol], Chlorpromazine [Thorazine], etc.
Side effect of Thorazine: Atopic Dermatitis (eczema) and foul-smelling odor [recall: patients in NCMH are smelly] Side effect of all antipsychotics: Sx of PARKINSON’S DISEASE, therefore antipsychotics are given together with antiparkinson drugs
Anti-parkinsonians: Cogentin, Artane, etc.
Pre-operative drug: Atropine Sulfate (AtSO4) – given before surgery to decrease salivary and mucus secretions
Anti-hypertensives: Methyldopa – for pregnancy induced hpn (PIH)
-blockers (-olol): Propranolol [Inderal], atenolol, metoprolol
ACE inhibitors (-pril): Enalapril, Ramipril, Lisinopril, Benazepril, Captopril Side effect of ACE inhibitors: AGRANULOCYTOSIS and NEUTROPENIA (blood dyscracias… always asked in board!)
Calcium channel blockers (Calcium antagonists) Nifedipine [Procardia], Verapamil [Isoptim], Dialtiazem [Cardizem]
NURSING ALERT: Anti-hypertensives are not given to patients with CHF or cardiogenic shock (Drug will cause a further decrease in heart rate Death)
Rx for Myasthenia Gravis: Pyridostigmine [Mestinon] Neostigmine [Prostigmin]
NEURONS -the basic unit of structure and function of the nervous system 3 characteristics of neurons:
1. Excitability – Neurons are affected by changes in the environment 2. Conductivity – Neurons transmit wave of excitations 3. Permanent cells – Once neurons are destroyed, they are not capable of regeneration
NEUROGLIA Function: support and protection of neurons Clinical significance: Majority of brain tumors arise from neuroglia Types:
Astrocytes Microglia Oligodendrocytes Ependymal cells
Note: Astrocytoma is the #1 type of brain tumor ASTROCYTES – maintain the integrity of the BLOOD-BRAIN BARRIER
Central Nervous System SPINAL CORD - begins at the base of the skull and extends throughout most of the backbone, or vertebral column Regions:
• Cervical -8 • Thoracic - 12 • Lumbar -5 • Sacral - 5
BRAIN -mass of billions of neurons - receives messages from and sends messages to all organs and tissues of the body
Three main parts 1. Brain stem 2. Cerebellum 3. Cerebrum
Lobes of Cerebrum
1. Frontal 2. Temporal 3. Parietal 4. Occipital
Neurologic Assessment COMPREHENSIVE NEUROLOGIC EXAM A. Purpose
1. To know exact neuro deficit 2. To localize lesion 3. For rehabilitation 4. For guidance in nursing care
B. Survey of Mental Status 1. LOC Conscious – awake Lethargy – sleepy/drowsy/obtunded Stupor – only awakened by vigorous stimulation
General body weakness
Decreased body defenses Coma
Light – (+) to all painful stimuli
Deep – (-) to all painful stimuli PAINFUL STIMULATION
Deep Sternal Stimulation/Pressure
Orbital Pressure
Pressure on Great Toes
Nail bed pressure
Corneal/Blinking Reflex a. Conscious – wisp of cotton b. Unconscious – institute/drop of saline solution (coma if positive reaction, deep coma if negative)
2. Test of memory (consider educational background) Short term memory (ask what the pt ate for breakfast)
(+) anterograde amnesia lead to temporal lobe damage Long term memory (ask birthday)
(+) retrograde amnesia lead to damage to Rhinencephalon (Limbic system) C. Levels of Orientation (time, person and place) D. CN Assessment E. Motor Assessment F. Sensory Assessment
1. PAIN - Gingerbread test
100% very painful
75% tolerable pain
25% moderate pain
0% no pain 2. TOUCH – Stereognosis
Identifying familiar object placed on clients hands
Astereognosis – if patient cannot identify object; damage in parietal lobe 3. PRESSURE AND TOUCH – Graphesthesia
Identify numbers or letters written on client’s palm
Agraphesthesia if (-), damage to parietal lobe
G. Cerebellar Test 1. Romberg’s Test
Instruct patient to close eyes, assume a normal anatomical position for 5-15 minutes; two nursesat right and left side
Normal is (-)
If (+) ataxia
2. Finger-to-nose Test 3. Alternate Pronation and Supination
Dysmetria – inability of a client to stop a movement at a desired point H. DTRs I. Autonomics Glasgow Coma Scale
Cranial Nerves
DEMYELLENATING DISEASES
ALZHEIMER’S DISEASE– atrophy of brain tissue due to a deficiency of acetylcholine.
Signs and Symptoms A – amnesia – loss of memory A – apraxia – unable to determine function & purpose of object A – agnosia – unable to recognize familiar object A – aphasia – - Expressive – brocca’s aphasia – unable to speak - Receptive – wernickes aphasia – unable to understand spoken words Common to Alzheimer – receptive aphasia Drug of choice – ARICEPT (taken at bedtime) & COGNEX. Management: Supportive & palliative
MULTIPLE SCLEROSIS-Chronic intermittent disorder of CNS – white patches of demyelenation in brain & spinal cord
- Remission & exacerbation - Common in women, 15 – 35 years old - cause – unknown
Predisposing factor
1. Slow growing virus 2. Autoimmune – (supportive & palliative treatment only) Normal Resident Antibodies:
Ig G – can pass placenta – passive immunity. Short acting. Ig A – body secretions – saliva, tears, colostrums, sweat Ig M – acute inflammation Ig E – allergic reactions IgD – chronic inflammation
Signs and Symptoms: (everything down)
1. Visual disturbances a. Blurring of vision b. Diplopia/ double vision c. Scotomas (blind spots) – initial sx 2. Impaired sensation to touch, pain, pressure, heat, cold a. Numbness b. Tingling c. Paresthesia 3. Mood swings – euphoria (sense of elation ) 4. Impaired motor function: a. Weakness b. Spasiticity –“ tigas” c. Paralysis –major problem 5. Impaired cerebellar function Charcots Triad I – intentional tremors N – nystagmus – abnormal rotation of eyes A – Ataxia & Scanning speech 6. Urinary retention or incontinence 7. Constipation 8. Decrease sexual ability
Diagnostic
1. CSF analysis thru lumbar puncture - Reveals increase CHON & IgG 2. MRI – reveals site & extent of demyelination
3. Lhermitte’s response is (+). Introduce electricity at the back. Theres spasm & paralysis at spinal cord. Nursing Management 1. Administer medications as ordered
Acute Exacerbation -ACTH (Adrenocorticotropic hormone) reduces edema at site of demyelinatiothereby preventing paralysis; compression on spinal cord will lead to paralysis
Baclofen (Lioresal), Dantrolene Na – to reducmuscle spasticity
Interferons
Immunosuppressives
Diuretics
PROPHANTHELENE BROMIDE (PRO-BANTHENE) anti-cholinergic for urinary incontinence 2. Provide for Relaxation
DBE, biofeedback, yoga 3. Retain side rails 4. Prevent complications of immobility – TTS Q2h, Q1 for elderly, 20 minutes only on affected side 5. Increase OFI, high fiber diet (for constipation), acid ash in diet to acidify urine to prevent bacteriamultiplication (cranberry juice, prunes, grape juicevitamin c, plums, orange and pineapple juice.) 6. Provide catheterization for urinary retention
INCREASED ICP – increase ICP is due to increase in 1 of the Intra Cranial components Predisposing Factors
a. Head injury b. Tumor c. Localized abscesses d. Cerebral edema e. Hydrocephalus f. Hemorrhage g. Inflammatory conditions
-Meningitis -Encephalitis
Signs and Symptoms a. Early signs 1. Decreased or change in LOC 2. Restlessness to confusion 3. Disorientation 4. Lethargy to stupor 5. Stupor to coma b. Late signs 1. Changes in the vital signs
-Elevated BP (SBP rising, DBP constant) N Pulse Pressure: 40 mmHG - HR decreased - RR decreased (Cheyne-Stokes respiration: normal rhythmic respiration followed by periods of apnea) - Elevated temperature
2. Headache, papilledema, projectile vomiting 3. Abnormal posturing- decorticate (flexion) – damage to corticospinal tract (spinal cord and cerebral cortex) (extension): upper brain stem damage – pons, midbrain,cerebellum 4. Unilateral dilation of pupil (ANISOCORIA) –indicates uncal brain herniation; if bilateral dilatation:tentorial herniation 5. possible seizures 6. Cushing’s reflex (hypertension with bradycardia)
-SHOCK – inadequate tissue perfusion - HYPOXIA – inadequate tissue oxygenation
Nursing Management 1. maintain patent airway and adequate ventilation by: - prevention of hypoxia( cerebral edema increased ICP) and hypercarbia (CO2 retention)
cerebral vasodilation increased ICP decreased tissue perfusion possible shock Early signs of hypoxia Restlessness Agitation Tachycardia Late signs of hypoxia Bradycardia Extreme restlessness Dyspnea Cyanosis
- Increased CO₂ – most potent respiratory stimulant in the normal person (irritates medulla oblongata) -Decreased O₂ – stimulates respiration in CRDS -Suctioning should only last for 10 -15 seconds and application of suction should be done upon withdrawal of catheter in a circular fashion.
2. Assist in mechanical ventilation 3. Elevate head of bed 30-45 degrees with neck in neutral position when contraindicated to promote venous drainage 4. Limit fluid intake to 1.2-1.5 l per day (Forced fluids =2-3 L/day) 5. Monitor VS, NVS, I/O strictly 6. Prevent complications of immobility 7. Prevent further increase in ICP
Provide comfortable environment
Avoid use of restraints will cause fractures 8. Keep side rails up 9. Avoid valsalva maneuver
Straining of stools (give laxatives/stool softeners)
Excessive vomiting (give Metoclopramide (plasil) – anti-emetic)
Lifting of heavy objects
Bending or stooping 10. Administer medications as ordered
Osmotic Diuretics – Mannitol (Osmitol) – cerebral diuresis
Monitor VS especially BP (SE: Hypotension resulting from hypovolemia)
Monitor I/O qH
Given via side drip, fast drip to avoid precipitate formation
Instruct client that a flushing sensation will be felt as drug is introduced Loop Diuretics via IV push – Furosemide
BP
Monitor 1/0 q1, notify if <30cc/hr
IV push Lasix effect in 10-15 minutes, max 6 hours; best given in AM to preventsleep interruption Corticosteroids
Dexamethasone (decadron)
Steroids administered 2/3 in AM to mimic diurnal rhythm
Hydorcortisone
Prednisone Mild Analgesic
Codeine sulfate Anti-Convulsant
Pheytoin (Dilantin)
PARKINSONS DISEASE
(parkinsonism) - chronic, progressive disease of CNS characterized by degeneration of dopamine producing cells in substancia nigra at mid brain & basal ganglia Predisposing Factors 1. Poisoning (lead & carbon monoxide). Antidote for lead = Calcium EDTA 2. Hypoxia 3. Arteriosclerosis 4. Encephalitis 5.High doses of the ff: a. Reserpine (serpasil)
b. Methyldopa (aldomet) c. Haloperidol (Haldol)- anti psychotic d. Phenothiazide- anti psychotic
Over meds of anti psychotic drugs – Neuroleptic Malignant Syndrome characterized by tremors,tachycardia,tachypnea,fever
Signs and Symptoms 1. Pill rolling tremors of extremities – early sign 2. Bradykinesia – slow movement 3. Over fatigue 4. Rigidity (cogwheel type)
a. Stooped posture b. Shuffling – most common
c. Propulsive gait 5. Mask like facial expression with decrease blinking eyes 6. Monotone speech 7. Difficulty rising from sitting position 8. Mood labilety – always depressed – suicide
Nursing priority: Promote safety 9. Increase salivation – drooling type 10. Autonomic signs:
- Increase sweating - Increase lacrimation - Seborrhea (increase sebaceous gland) - Constipation - Decrease sexual activity
Nursing Mangement 1.)Maintain siderails 2.) Prevent complications of immobility
- Turn pt. every 2h - Turn pt. every 1h – elderly
3.)Assist in passive ROM exercises to prevent contractures 4.)Maintain good nutrition
CHON – in am CHON – in pm – to induce sleep – due Tryptopan – Amino Acid 5.)Increase fluid intake, high fiber diet to prevent constipation 6.)Assist in surgery – Sterotaxic Thalamotomy
Complications 1.) Subarachnoid hemorrhage 2.) aneurism 3.) encephalitis
MYASTHENIA GRAVIS (MG)
- disturbance in transmission of impulses from nerve to muscle cell at neuro muscular junction. -Common in Women, 20 – 40 y/o, unknown cause or idiopathic -Autoimmune – release of cholenesterase – enzyme -Cholinesterase destroys ACH (acetylcholine) = Decrease acetylcholine -Descending muscle weakness Nursing Priority
1) Airway 2) aspiration 3) immobility
Signs and Symptoms 1. Ptosis – drooping of upper lid ( initial sign)
-Check Palpebral fissure – opening of upper & lower lids = to know if (+) of MG. 2. Diplopia – double vision 3. Mask like facial expression 4. Dysphagia – risk for aspiration! 5. Weakening of laryngeal muscles – hoarseness of voice 6. Respiratory muscle weakness – lead respiratory arrest. Prepare at bedside tracheostomy set 7. Extreme muscle weakness during activity especially in the morning
Diagnostic
Tensilon test (Edrophonium Hcl) – temporarily strengthens muscles for 5 – 10 mins. Short term- cholinergic. PNS effect. Nursing Management
1. Maintain patent airway & adequate ventilation by: a.) Assist in mechanical vent – attach to ventilator b.) Monitor pulmonary function test. Decrease vital lung capacity. 2. Monitor VS, I&O, neurologic check, muscle strength or motor grading scale (4/5, 5/5, etc) 3. Siderails 4. Prevent complications of immobility. Adult-every 2 hrs. Elderly- every 1 hr. 5. NGT feeding 6. Administer meds –
a.) Cholinergics or anticholinesterase agents Mestinon (Pyridostigmine)
Neostignine (prostigmin) – Long term b.) Corticosteroids – to suppress immune respiration
Decadron (dexamethasone)
Monitor for 2 types of Crisis:
Myasthenic Crisis Cholinergic Crisis Causes undermedication, stress, infection overmedication
Signs and Symptoms (-)seeing, swallowing,speaking,breathing PNS, increased salivation will lead to aspiration
Treatment Administer cholinergic agents as ordered anticholinergic agents, atropine sulfate
7. Assist in surgical procedure – Thymectomy- Removal of thymus gland. Thymus secretes auto immune antibody 8. Assist in plasmaparesis – filter blood 9. Prevent complication – respiratory arrest -Prepare tracheostomy set at bedside.
GBS – Guillain Barre Syndrome - Disorder of CNS - Bilateral symmetrical polyneuritis - Ascending paralysis - Cause – unknown, idiopathic
Signs and Symptoms Initial :
1. Clumsiness 2. Ascending muscle weakness – lead to paralysis 3. Dysphagia 4. Decrease or diminished DTR (deep tendon reflexes)
-Paralysis 5. Alternate HPN to hypotension – lead to arrhythmia - complication 6. Autonomic changes –
increase sweating
increase salivation
increase lacrimation
Constipation Diagnostic CSF analysis thru lumbar puncture reveals increase in : IgG & CHON (same with MS) Nursing Management 1. Maintain patent airway & adequate ventilation a. Assist in mechanical vent b. Monitor pulmonary function test 2. Monitor VS, I&O,neurolohic check, ECG tracing due to arrhythmia 3. Siderails 4. Prevent complication 5. Assist in passive ROM exercises 6. Institute NGT feeding
7. Administer meds as ordered: 1. Anti cholinergic – atropine SO4
2. Corticosteroids – to suppress immune response 3. Anti arrhythmic agents a.) Lidocaine /Xylocaine –SE confusion = VTach b.) Bretyllium c.) Quinines/Quinidine – anti malarial agent. Give with meals. Toxic effect – Cinchonism Side Effect – anorexia, nausea/vomiting, headache, vertigo, visual disturbances
8. Assist in plasmaparesis 9. Prevent complications – arrhythmias, respiratory arrest
MENINGITIS – inflammation of meningitis & spinal cord Etiology
Meningococcus
Pneumococcus
Hemophilous influenza – child
Streptococcus – adult meningitis Mode of Transmission direct transmission via droplet nuclei Signs and Symptoms
1. Stiff neck or nuchal rigidity (initial sign) 2. Headache 3. Projectile vomiting – due to increase ICP 4. Photophobia 5. Fever chills, anorexia 6. Gen body malaise 7. Wt loss 8. Decorticate/decerebration – abnormal posturing 9. Possible seizure 10. Signs of meningeal irritation
nuchal rigidity or stiffness
Opisthotonus- rigid arching of back Pathognomonic sign (+) Kernig’s - leg pain (+) Brudzinski sign - neck pain Diagnostic 1. Lumbar puncture – lumbar/ spinal tap – use of hallow spinal needle – sub arachnoid space L3 & L4 or L4 & L5
Nursing Management 1. Obtain informed consent 2. Empty bladder and bowel to promote comfort 3. Instruct client to arch back to clearly visualize L3, L4
Nursing Management(Post lumbar) 1. Flat on bed for 12-24hto prevent spinal headache & leak of CSF 2. Force fluid 3. Check punctured site for drainage, discoloration & leakage to tissue 4. Assess for movement & sensation of extremeties
CSF analysis will reveal 1. Increased CHON and WBC 2. Decreased Glucose 3. Increased CSF opening pressure 4. N = 50-160 mmHg 5. (+) cultured microorganisms
- These confirm presence of meningitis
2. Complete blood count CBC – reveals increase WBC Nursing Management 1. Administer medications a.) Broad-spectrum antibiotic penicillin
Side Effects: 1. GIT irritation – take with food 2. Hepatotoxicity, nephrotoxcicity 3. Allergic reaction 4. Super infection – alteration in normal bacterial flora
b.) Antipyretic c.) Mild analgesic 2. Strict respiratory isolation 24h after start of antibiotic therapy 3. Provide comfortable & dark room due to photophobia & seizure 4. Prevent complications of immobility 5. Maintain fluid and electrolyte balance 6. Monitor VS, I&O, neurologic check 7. Provide client health teaching & discharge plan a. Nutrition – increase calcium, CHO, CHON-for tissue repair-Small frequent feeding b. Prevent complication: hydrocephalus, hearing loss or nerve deafness 8. Prevent seizure 9. Rehabilitation for neurological deficit it can lead to mental retardation or a delay in psychomotor development
CEREBRO VASCULAR ACCIDENT ( stroke, brain attack or cerebral thrombosis, apoplexy)
-Partial or complete disruption in the brains blood supply, usually in Middle cerebral artery and Internal carotid artery(2 largest & common arteries) -Common to male – 2 – 3x high risk
Predisposing factor
1. Thrombosis – clot (attached) 2. Embolism – dislodged clot – pulmonary embolism
Signs and Symptoms of Pulmonary embolism 1. Sudden sharp chest pain 2. Unexplained dyspnea, Shortness of breath 3. Tachycardia, palpitations, diaphoresis & mild restlessness
Signs and Symptoms of Cerebral embolism
1. Headache, disorientation, confusion & decrease in LOC 2. Femur fracture – complications: fat embolism – most feared complication w/in 24hrs 3. Yellow bone marrow – produces fat cells at meduallary cavity of long bone 4. Red bone marrow – provides WBC, platelets, RBC found at epiphisis
3.) Hemorrhage 4.) Compartment syndrome – compression of nerves/ arteries
Risk factors
1. HPN 2. DM 3. MI 4. artherosclerosis, 5. valvular heart dse - Post heart surgery – mitral valve replacement 6. Lifestyle
a. Smoking – nicotine – potent vasoconstrictor b. Sedentary lifestyle c. Hyperlipidemia – genetic d. Prolonged use of oral contraceptives
- Macro pill – has large amt estrogen - Mini pill – has large amt of progestin - Promote lipolysis (breakdown of lipids/fats) – artherosclerosis – HPN - stroke
e. Type A personality -Deadline driven person -2 – 5 things at the same time -Guilty when not doing anything
7. Diet – increase saturated fats 8. Emotional & physical stress 9. Obesity Signs and Symptoms 1. Transient Ischemic attack- warning signs of impending stroke attacks
Headache (initial sign), dizziness/ vertigo, numbness, tinnitus, visual & speech disturbances, paresis or plegia,Increase ICP possible, cheyne-stokes respirations
2. Stroke in evolution – progression of Signs and Symptoms of stroke 3. Complete stroke – resolution of stroke
1. Headache
2. Cheyne-Stokes Resp 3. Anorexia, nausea/vomiting 4. Dysphagia 5. Increase BP 6. (+) Kernig’s & Brudzinski – signs of hemorrhagic stroke 7. Focal & neurological deficit
a. Phlegia b. Dysarthria – inability to vocalize, articulate words c. Aphasia d. Agraphia diff writing e. Alesia – diff reading
f. Homonymous hemianopsia – loss of half of field of vision
Diagnostic
1. CT Scan – reveals brain lesion 2. Cerebral arteriography – site & extent of malignant occlusion
- Invasive procedure due to inject dye - Allergy test
Post Dx 1.) Force fluid – to excrete dye is nephrotoxic 2.) Check peripheral pulses - distal
Nursing Management 1. Maintain patent a/w & adequate vent
- Assist mechanical ventilation - Administer O2
2. Restrict fluids – prevent cerebral edema 3. Elevate head of bed 30-45 degrees angle. Avoid valsalva maneuver. 4. Monitor vs., I&O, neuro check 5. Prevent complications of immobility by:
a. Turning client every 2hours, for elderly every 1hour
To prevent decubitus ulcer
To prevent hypostatic pneumonia – after prolonged immobility b. Egg crate mattress or H2O bed c. Sand bag or foot board- prevent foot drop
6. NGT feeding – if pt can’t swallow 7. Passive ROM exercise every 4 hours 8. Alternative means of communication
Non-verbal cues
Magic slate or picture board not paper and pen because it is tiring for the patient
If (+) to hemianopsia – approach on unaffected side 9. Administer medications as ordered
a. Osmotic diuretics – Mannitol b. Loop diuretics – Lasix/ Furosemide c. Corticosteroids – dextamethazone d. Mild analgesic e. Thrombolytic/ fibrolitic agents
Streptokinase
Urokinase
Tissue plasminogen activating -(Monitor bleeding time) f. Anticoagulants –
Heparin – monitor PTT partial thromboplastin time if prolonged – bleeding give Protamine SO4- antidote
Coumadin –Long term. monitor PT prothrombin time if prolonged- bleeding give Vit K – Aquamephyton- antidote
Given together because coumadin will take effect after 3 days still Health Teaching 1. Avoidance of modifiable lifestyle
- Diet, smoking 2. Dietary modification
- Avoid caffeine, decrease Na & saturated fats
3.Rehabilitation for focal neurologic weakness
4.Importance of ffup care and strict compliance to medications
CONVULSIVE Disorder (CONVULSIONS) - disorder of the CNS characterized by paroxysmal seizures with or without loss of consciousness, abnormal motor activity, alteration in sensation & perception & change in behavior -Febrile seizures are normal for children below 5 years only; can be outgrown -Febrile seizures in children >5 yo = abnormal SEIZURE – first convulsive attack EPILEPSY – series of seizure activity Predisposing Factor
1. Head injury due birth trauma 2. Toxicity of carbon monoxide 3. Brain tumor 4. Genetics 5. Nutritional & metabolic deficit 6. Physical stress 7. Sudden withdrawal to anticonvulsants will bring about status epilepticus 8. Status epilepticus – drug of choice: Diazepam & glucose
Signs and Symptoms Types of Seizures I. Generalized Seizure –
a.) Grand Mal / (tonic clonic seizures) With or without aura – warning symptoms of impending seizure attack- Epigastric pain- associated with olfactory, tactile, visual, auditory sensory experience
- Epileptic cry – fall - Loss of consciousness 3 – 5 minutes - Tonic clonic contractions - Direct symmetrical extension of extremities-TONIC. Contractions-CLONIC - Post ictal sleep -state of lethargy or drowsiness (unresponding sleep after tonic clonic)
b.) Petimal seizure – (same as daydreaming) or absent seizure - Blank stare - Decrease blinking eye - Twitching of mouth - Loss of consciousness – 5 – 10 secs (quick & short)
II. Localized/partial seizure
a.) Jacksonian seizure or focal seizure – tingling/jerky movement of index finger/thumb & spreads to shoulder & 1 sideof the body with janksonian march
b.) Psychomotor/ focal motor - seizure -Automatism – stereotype repetitive & non-purposive behavior - Clouding of consciousness – not in control with environment - Mild hallucinatory sensory experience
III. Status epilecticus – continuous, uninterrupted seizure activity, if untreated, lead to hyperprexia – coma – death -DOC: Valium, Glucose Diagnostic
1. CT scan – brain lesion 2. EEG electroencephalography
- Hyperactivity brain waves Nursing Management 1. Maintain patent a/w & promote safety Before seizure:
a. Remove blunt/sharp objects b. Loosen clothing c. Avoid restraints
d. Maintain siderails e. Turn head to side to prevent aspiration f. Tongue guard or mouth piece to prevent biting of tongue-BEFORE SEIZURE ONLY! Can use spoon at home. g. Avoid precipitating stimulus – bright glaring lights & noises h. Administer meds
a. Dilantin (Phenytoin) –( toxicity level – 20 ) Side effects:
Gingival hyperplasia H-hairy tongue A-ataxia N-nystagmus b. (Tegretol) Carbamasene- given also to Trigeminal Neuralgia
Sideeffect: arrythmia c. Phenobarbital (Luminal)
Side effect: hallucinations
2. Institute seizure & safety precaution. Post seizure: Administer O2. Suction apparatus ready at bedside 3. Monitor and document the ff: onset & duration
Type of seizure
Duration of post ictal sleep. The longer the duration of post ictal sleep, the higher chance of having status epilepticus 4. Assist in surgical procedure- Cortical resection