Nerve and Pain Disorders

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    With Bells palsy, impulses from the

    seventh cranial nerve - the nerve

    responsible for motor innervations of

    the facial muscles are blocked.

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    Corneal ulceration

    Blindness

    Impaired nutrition

    Psychosocial problems

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    Pain on the affected side

    Difficulty eating on the affected side

    Difficulty speaking clearly

    Drooping mouth or drooling

    Distorted taste perception over the affectedanterior portion of the tongue.

    Inability to raise the eyebrow, smile, show

    teeth, or puff out the cheek on the affected

    side.

    Difficulty closing eye on the affected side; if

    attempted, the eye rolls upward (bells

    phenomenon) and shows excessive tearing.

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    Diagnosis is based on clinical

    presentation.

    Electromyography

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    Administration of corticosteroidsAnalgesics

    Moist heat applied to the affected side

    Lubricants or an eye ointment may beneeded to protect the eye; patching

    during sleep may also be necessary.

    Electrotherapy

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    Provide psychological support to the

    patient. Reassure him/her that he/shehasnt had s stroke. Tell him/her that

    spontaneous recovery usually occurs

    within 8 weeks. These should help

    decrease anxiety and help adjust to thetemporary change in his body image.

    Administer medications and monitor for

    adverse reactions.

    Monitor serum glucose levels during

    corticosteroid therapy.

    Apply moist heat to the affected side of

    the face, as ordered.

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    Massage the patients face with a gentleupward motion two to three times dailyfor 5 to 10 minutes; teach massage to thepatient.

    Apply a facial sling, if necessary, toimprove lip alignment.

    Provide frequent and complete mouth

    care, taking special care to removeresidual food that collects between thecheek and gums.

    Provide a soft, nutritionally balanced

    diet, eliminating hot foods and fluids.Provide preoperative and postoperative

    care, as appropriate.

    Provide appropriate education to the

    patient an his family before discharge.

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    Dysfunction affects cranialnerves III, IV, and VI. These

    nerves are responsible for

    innervating eye movements.

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    Diabetes neuropathy

    Trauma

    Pressure from an aneurysm or abrain tumor

    Other: vary depending on the cranial

    nerve involved

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    Diplopia

    Ptosis

    StrabismusNystagmus

    Ocular torticollis

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    Recent onset of diplopia

    Torticolliswith CN III palsy:

    Ptosis

    Extropia

    Papillary dilation and unresponsiveness to

    light and accommodation

    Inability to move the eye

    Inability to move the eye downward andupward

    Estropia

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    Diagnosis is based on neuro-

    opthalmologic examination

    Blood studies

    CT-scan, MRI, or skull x-rays

    Angiography

    C&s tests

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    Appropriate treatment varies depending

    on the cause, such as:Neurosurgery may be necessary for a

    brain tumor or an aneurysm.

    Massive doses of antibiotics may be

    appropriate for infection

    After treating the primary condition, the

    patient may need to perform exercises

    that stretch the neck muscles to correctacquired torticollis (wry neck).

    Other care and treatments depend on

    residual symptoms.

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    Provide emotional support to help

    minimize the patients anxiety about

    the cause of the motor nerve palsy.

    Provide treatment appropriate for

    the specific cause of the palsy.

    Encourage neck exercise if

    torticollitis is present.

    Provide appropriate education to the

    patient before discharge.

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    Also called multiple neuritis,peripheral neuropathy, andpolyneuritis, is the inflammatory

    degeneration of peripheral nervesthat primarily supply the distalmuscles of the extremities.

    It results in muscle weakness with

    sensory loss and atrophy anddecreased or absent deep tendonreflexes.

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    Exact cause is unknown; it is thought

    to be mediated by inflammation,ischemia, and demyelination of the

    larger peripheral nerves

    Drugs that may cause peripheralneuritis include:

    thalidomide (Talomid)

    metronidazole (Flagyl)phenytoin (Dilantin)

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    Peripheral neuritis is damage to

    nerves that run from the spinal cord

    to the rest of the body, which

    impairs function of the sensory,motor, and autonomic nerves.

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    Chronic pain

    Depression

    Drug dependence

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    Symptoms vary according to which

    type of nerve is affected and may

    include:

    Altered sensations or paresthesia

    Impaired balance when standing

    or walking

    Difficulty maintaining a grip on

    objects

    Muscle weakness

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    Patient history and physical

    examination delineate characteristic

    distribution of motor and sensory

    deficits.

    Electromyography

    Nerve biopsy and nerve conduction

    tests

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    Supportive measures to relieve pain

    Adequate bed rest

    Physical, vocational, andoccupational therapy

    Orthopedic interventions to promote

    independence

    OTC analgesics or prescription pain

    medications may be needed to

    control nerve pain.

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    Anticonvulsants (phenytoin,

    carbamazepine) or tricyclic

    antidepressants

    duloxetine (Cymbalta) may beprescribed specially for the

    treatment of diabetic

    neuropathy

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    For patients with bladder

    dysfunction, manual expressionof urine (pressing over thebladder with the hands),

    intermittent catheterization, ormedications, may be necessary.

    Others:

    acupuncture,plasmapheresis andIV gamma globulin, electricalnerve stimulation, andbiofeedback.

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    Encourage participation in physicaltherapy. Provide ROM exercises, ifnecessary. Teach use of assistive

    device, if appropriate.Assess affected areas frequently for

    bruises, open skin areas, or otherinjuries and provide appropriatecare.

    Provide safety measure to preventinjury.

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    Reposition the patient every 2 hoursor teach to change position

    frequently if nerve damage preventsadequate sensation of pressure.

    Provide small, frequent meals ifappropriate.

    Assist the patient with bladderdysfunction with manual expressionof urine and intermittent

    catheterization, as necessary.Provide appropriate education to the

    patient and his family duringdischarge.

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    Also known as tic douloureux, is

    a disorder of one or morebranches of the 5th cranial

    nerve.

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    Typical trigeminal neuralgia (also

    known as classical, idiopathic, and

    essential trigeminal neuralgia)

    Atypical trigeminal neuralgia

    Pre-trigeminal neuralgia

    MS-related trigeminal neuralgia

    Secondary trigeminal neuralgia

    Post traumatic trigeminal neuralgia

    Failed trigeminal neuralgia

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    Intractable pain

    Lack of self-careDepression

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    Localized searing or burning that

    occurs lightning-like jabs and last from

    1-2 minutes (pain is usually initiated by

    a light touch to a hypersensitive area,

    such as the tip of the nose, cheeks, orgums. Pain may also follow a draft of

    air, exposure to heat or cold, eating,

    smiling, talking, or drinking hot or cold

    beverages)

    Constant, dull ache

    Splinting of the affected area

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    Skull x-rays, CT-scan, andMRI

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    carbamezipine(Tegretol)

    oxcarbezapine

    Nerve block injectionsPercutaneous surgery (through

    the cheek) to open skull surgery

    Pin-point radiation

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    Provide emotional support to the patientand family.

    Observe and record the characteristics ofeach attack, including the patientsprotective mechanism.

    Provide small, frequent meals at a room

    temperature to maintain adequatenutrition.Assist the patient in identifying factors

    that precipitate an attack, and urge thepatient to avoid stimulation (air, heat,cold) to trigger zone (lips, cheeks, gums).

    Administer medications, as ordered, andmonitor for adverse effects.

    Provide appropriate preoperative and

    postoperative care, as appropriate.

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    Also known as reflex sympathetic

    dystrophy (CRPS1) or causalgia

    (CRPS2) is a chronic pain disorderthat results from abnormal healing

    after an injury -either minor or

    major- to a bone, muscle, or nerve.

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    Stage I (acute)

    Stage II (subacute or dystrophic)

    Stage III (chronic or atrophic)

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    Specificity

    Intensity

    Pattern

    Neuromatrix

    Gate control

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    Chronic pain

    Depression

    Drug dependence

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    Severe and constant pain

    Altered blood flow to the affectedarea

    Discoloration, sweating, or swellingof the affected area (may also bewarm or cool to the touch)

    Skin, hair, and nail changesImpaired mobility and muscle

    wasting (if adequate treatment isdelayed)

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    Diagnosis is based on the

    patients history and clinical

    findingsBone x-rays

    Bone scans, nerve conduction

    studies, and thermography

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    Typically consists of acombination of therapies,

    including administration of anti-

    inflammatory, antidepressant,

    vasodilator, and analgesic agents

    Corticosteroids may beprescribed for some patients

    Physical therapy

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    Application of hot and coldUse of transcutaneous electrical

    nerve stimulation (TENS) unit

    Nerve or regional blocks

    Surgical sympathectomy

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    Administer medications, monitor

    their effects and adverse effects.

    Assist with ROM exercises. Provide

    rest period as needed.

    Provide emotional support.

    Consult a pain care specialist to

    provide additional options for the

    patient and help manage

    discomfort.