Ncm103 6th Cv II

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    Care of Clients with Problems In Oxygenation,

    Fluids and Electrolytes, Metabolism and Endocrine

    (NCM103)

    Cardiovascular System: Coronary Vascular Disease

    Coronary Atherosclerosis

    Definition: Abnormal accumulation of fatty substances Creates BLOCKAGE Repetitious inflammatory response

    o Obstruction + Inflammation = Blood FlowMI or APAtherosclerosis Arteriosclerosis

    1. Definition Lumen Lumen2. Cause Deposition of fatty substances Elasticity of blood vessels3. Age Toddlers Aging process4. Layer Tunica intima Tunica media

    Risk Factors:

    Blood lipid level Smoking lumen; vasoconstriction Hypertension Narrowing down of blood vessels Diabetes / Hyperlipidemia Obesity Metabolic Syndrome

    o Hyperglycemia FBS, Glucose Tolerance Testo Hypertensiono Level of lipids in bloodo Presence of elevation CRP

    Clinical Manifestations:

    - Depends on1. Location and degree of

    narrowing

    2. Thrombus formation andobstruction of blood

    CHEST PAIN~ Asymptomatic~

    Prevention:

    Control of cholesterol level Get lipid profile once ever 5 years (20

    y/o and above)

    a. Dietb. Physical Activityc. Medications Anti-lipid Drugsd. Promoting cessation of tobacco

    Nicotine!! Viscosity

    of blood

    Vasoconstriction of Blood

    Vessels

    e. Managing Hypertension Religious intake of

    DRUGS

    Regular exercise Diet Alcohol intake

    Topics Discussed Here Are:1. Coronary Atherosclerosis2. Angina Pectoris3. Myocardial Infarction

    LOOKY

    HERE

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    NUTRIENT CONTENT OF THE THERAPEUTIC LIFESTYLE CHANGES (TLC) DIET

    Nutrient Recommended IntakeTotal calories Balance intake and expenditure to maintain desirable weight

    Total fat

    Saturated fat Polyunsaturated fat

    Monounsaturated fat

    25% - 35% of total calories

    Less than 7% of total caloriesUp to 10% of total calories

    Up to 20% of total calories

    Carbohydrate 50% - 60% of total calories

    Dietary fiber 20 30 g/day

    Protein Approximately 15% of total calories

    Cholesterol Less than 200 mg/day

    Angina PectorisFactors: Blood Flow due to Lumen

    Non-modifiable Factors

    1. Age2. Sex3. Race4. Family History

    Modifiable Factors

    5. Diet ( Na, Fat, Nicotine)6. Alcohol7. Obesity8. Sedentary activity9. Stress

    Coronary

    Insufficiency

    Coronary occlusion

    (Continuation of coronary insufficiency)

    LumenCHEST

    PAIN On and Off Bearable

    Short Duration

    Angina Pectoris

    CONSTANTSEVERE

    LONGER

    Myocardial

    Infarction

    Ischemia

    What is Angina Pectoris? Associated With:

    Outside factors Mental / Emotional problems Relieved With: Rest Coronary vasodilators

    Levines SignTypes of Angina Pectoris

    1. Stable Angina Predictable and consistent pain Relieved by rest and / nitroglycerin

    2. Unstable Angina / Preinfarction Angina / Crescendo Angina Symptoms increase in frequency May not be relieved by rest and nitroglycerin

    3.

    Variant Angina (Prinzmetals Angina) Pain at rest Reversible ST-segment elevation

    4. Silent Ischemia Objective evidence of ischemia But NO PAIN :o

    Management:1. Medical Management

    a. Rest Minimum of 2 hours

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    b. Drugs1. Coronary Vasodilators

    Dilates lumen of blood vessels in the myocardium Drugs:

    o Nitroglycerin 1 Tab, SL q5 min PRN for chest pain Maximum of 3 Tablets; if not resolved considered

    MI

    Take the medications beforeany stressful event

    Loses potency whenexposed in the sun; store in

    dark colored bottles

    If head gets big, warm, andflushing of face it is

    NORMAL, not SIDE EFFECT or ADVERSE

    EFFECT

    2. Anti-platelet Drugs Aspirin (Produces GIT disturbance; Coated Aspirin New) Heparin

    o Side Effect = Bleeding (Petechiae, Ecchymosis, Hematoma)o WOF: Tea colored urine for HEMATURIAo

    WOF: BLACK STOOLS NO PARENTERAL ADMINISTRATION OF DRUGS; if needed,apply pressure after!!

    Dont use dental floss3. Beta Blockers To reduce O2 demand of myocardium4. Ca+ Channel Blocking Agent Relaxes the Blood Vessel to BP

    c. Take 30 60 cc of BRANDY/WHISKY To reduce chest pain by vasodilation Amount = Vasoconstriction Amount = Vasodilation

    2. Prevention of Succeeding Attacks to Angina Pectorisa. Diet

    Fat Diet (THE FOLLOWING FOODS ARE NOT ALLOWED OR MODERATE) 4 Legged animals Foods with wings (Moderate) White meat Chicken (NO SKIN) Intake of food in WATER; but grows in SOIL

    Na Diet (2 gm Na) AVOID THE FF FOODS 1. Na Nitrate Food colorings, cold cuts, preserved food2. Na Nitrite Tocino, Tapa3. Na HCO3 Baking Soda, bread, biscuit, pastries

    8:00 1st

    Tablet

    8:05 2nd

    Tablet

    8:10 3rd Tablet

    8:00 1st

    Tablet

    8:05 2nd

    Tablet

    3:30 1st

    Tablet

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    4. NaCl Fish sauce, soy sauce, dried fish5. Na Saccharin

    b. Must be with the patient all the time!! Coronary Vasodilators

    DONT EXPOSE IN THE SUN!! DONT LEAVE IN THE CAR!!

    Myocardial Infarction Brought about by lack of blood supply going to the myocardium A.k.a. Coronary occlusion or Heart Attack

    Pain:

    - CRUSHING- TIGHTNESS- Sudden onset- Substernal- Severe- Unrelieved by Nitroglycerin- May radiate to: Back, neck, jaw, shoulders, arm-

    Dyspnea- Syncope ( BP)- Nausea / Vomiting- Extreme weakness- Diaphoresis- Denial is common- HR

    Treatment:

    - O2 IV Medications- Dietary restrictions- Na, Cholesterol, Caffeine- Surgery? Pacemaker?

    Causes:

    1. Atheroma Associated with body metabolism2. O2 / Blood demand Patients with hyperthyroidism3. O2 Supply (Anemia, BP)4. Vasospasm Sudden constriction / narrowing of coronary arteries

    How to Diagnose Clients With MI1. Presenting Manifestations

    Chief complaint Previous illnesses ECG: Done in 10 minutes PRIOR to admission

    P

    Q

    R

    S

    T

    ANG HIRAP MAG DROWING NG ECG PQRST SA PC XDComponents

    1. P Wave Atrial Contraction / Depolarization (0.08 seconds)2. PR Interval Time for impulses to travel from Artery to Ventricle (0.16 seconds)3. QRS Complex Ventricular Contraction / Depolarization (0.08 seconds)4. ST Segment Length of time for the ventricle to relax (0.12 seconds)5. T Wave Ventricular relaxation / Repolarization (0.16 seconds)

    0.08

    seconds

    2

    seconds

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    Diagnosing Myocardial Infarction Inverted T Wave

    Injury becomes ischemic, myocardial Repolarization is altered and delayed Which causes the T Wave to invert

    Elevated ST-segment Injured myocardial cells depolarize normally, but repolarize more rapidly than normal

    cells

    Causing the ST-segment to rise at least 1 mm above the isoelectric line Laboratory Results

    Creatinine Kinase (CK-MB) Lactic Dehydrogenase Myoglobin Troponin T and I

    Management1. Minimize Myocardial damage

    a. Cardiac overload Place patient on absolute bed rest

    NO VISITORS ALLOWED (NEAREST RELATIVE ONLY) Health team should be calm

    b. Get baseline Vital Signs TPR

    Temperature = Due to cell death Pulse = Respiration = Blood Pressure =

    Baseline ECG Basic serum electrolytes

    c. Administration of Drugs Thrombolytic Drugs (Alteplase)

    1st Drug To dissolve the blood clot The faster it is given, it is better

    Analgesics To relieve chest pain Morphine - Pain and Anxiety related to MI

    ACE Inhibitors To prevent BP

    2. Cardiac Rehabilitation Main objective: To extend the life of the patient and improve the quality of life of

    patients

    Consists of different activities3 Phases of Cardiac Rehabilitation

    1. Phase I Time of diagnosis of atherosclerosis Found out by lipid profile Management:

    Preventive measures1. Level of activities (Walking)2. Initial education of patient and family (For self care)3. Quit Smoking!4. DIET > NO FATTY FOODS

    If patient is not compliant, there

    would be repeated attacks of

    angina pectoris and myocardialinfarction.

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    2. Phase II Time the patient has been discharged from the hospital All activities are supervised; support from an outside person Based on the stress test of the client Dietitian would make a list of foods to be eaten and not eaten

    3. Phase III Maintain cardiovascular stability Patient can still be on diet and exercise, but no longer supervised

    Reperfusion Therapy- Procedures wherein to further improve the blood flow to the myocardium such as:

    o Angioplasty: Repair of blood vessel PTA (Percutaneous Transluminal Angioplasty) PTCA (Percutaneous Transluminal Coronary Angioplasty) It is the inflation of a balloon pushing atheromas to the side, done not to attain

    100%

    o Coronary Stent Inserted through the femur Placement of a spring Can cause thrombus formation But eventually becomes part of the endothelial tissue

    o Atherectomy Removal of atheromas Hollow cylinder Diamond chip-blade Rotation: Scrape atheromas Not full scraping of atheromas

    o Coronary Artery Bypass Graft (CABG) They cannot perform not until there are still very minimal amount of blood to

    the myocardium

    Harvest a Graft Artery Vein

    o Common blood vessel used Internal mammary artery Saphenous vein

    Nursing Care Patient should be VERY compliant

    Drugs be taken religiously Thrombolytics Beta blockers Digitalis

    Compliance to diet FAT Na

    Ambulation Gradually increasing

    Sexual Intercourse Patient should be able to make 5 7 steps in the stairs without fatigability