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Marian Pieniak M.D., Ph.D. · Management of bradycardia Marian Pieniak M.D., Ph.D. Department and...
Transcript of Marian Pieniak M.D., Ph.D. · Management of bradycardia Marian Pieniak M.D., Ph.D. Department and...
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Management of bradycardia
Marian Pieniak M.D., Ph.D.
Department and Division of Internal Medicine and Cardiology Warsaw University of Medicine, Warsaw, Poland.
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Bradycardia
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50 mm/s
Bradyarrhythmia
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1. Anamnesis 2. Physical examination 3. Basic laboratory data 4. Electrocardiography 5. Holter monitoring 6. Echocardiography 7. Exercise stress test 8. Tilt-up test 9. Transesophageal Atrial Pacing (Sinus Node Recovery
Time. Sino-Atrial Conduction Time „Wenckebach Point”) 10. Invasive electrophysiological study 11. Electroencephalography 12. Doppler examination of jugular and vertebral arteries
Evaluation of bradycardia
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Pacemaker cell
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A-V junction ablation
His bundle automaticity
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Sinus node Atrio-ventricular node Automatic cells surrounding the A-V node and
coronary sinus Purkinje-like cells of internodal tracts and
Bachmann’s bundle His bundle His bundle branches Peripheral Purkinje fibers
Normal automaticity in the heart
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Among a variety of the secondary automatic centers in the heart only His bundle automaticity is sufficiently strong as a life-saving natural pacemaker. It means that this center is able to mantain a stable rhythm usually of more then 30 beats per minute needed to maintain cardiac output in sufficient level
REMEMBER:
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„Symptomatic bradycardia” is defined as a documented bradyarrhythmia that is directly responsible for the development of syncope or near-syncope, transient dizziness or light-headedness, and confusional states resulting from cerebral hypoperfusion attributable to slow heart rate
ACC/AHA guidelines for implantation of cardiac pacemakers and arrhythmia devices
(Circulation 1998, 97, 1325)
Fatigue, exercise intolerance and frank congestive heart failure may also result from bradycardia. These symptoms may occur at rest or with exertion
Definite correlation of symptoms with a bradyarrhythmia is a requirement to fulfill the criteria of symptomatic bradycardia
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Sinus bradycardia (1)
Acute (potentially reversible causes) 1. Extreme generalized hypoxia:
- respiratory failure of different mechanism - pulmonary embolism - carbone oxyde intoxication
2. Coronary ischemia and infarction 3. Drug intoxication
- digitalis - antiarrhythmics overdosage
CLASS I B and C (quinidine, procainamide, disopyramide, ajmaline, prajmaline, flecainide, lorcainide,) CLASS III (amiodarone, sotalol) CLASS IV (Ca entry blokers (verapamil, gallopamil)
- beta-adrenolytic drugs (CLASS II)
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Sinus bradycardia (2)
4. Carotide sinus syndrome 5. Vaso-vagal syndrome 6. Neurocardiogenic Vaso-Vagal syndrome 7. Reflex bradycardia (vagal reflex):
- endotracheal intubation - esophagoscopy, bronchoscopy - eye surgery - coronarography and interventional procedures
(PTCA, stenting)
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Sinus bradycardia
Chronic
1. Progressive fibrosis of sinus node 2. Chronic ischemia 3. Other causes (rare):
degenerative and autoimmunological processes myxoma
4. Physiological: trained individuals sleep
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Overdrive suppresion
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Positive result of the test: syncope or a symptomatic drop of the arterial presure (near syncope) and/or significant bradycardia
Positive result for cardiodepressive type of syncope: - ventricular asystole of 3 seconds or more or: - bradycardia less then 40 bpm for at least 10 seconds
Neurocardiogenic vaso-vagal syndrome TILT-UP test (Westminster Protocol, Angle 60°)
Types of syncope (VASIS classification) Mixed Cardiodepressive Vasodepressive
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Acute A-V blook
1. Myocardial infarction 2. Myocarditis
- rheumatic - viral - parasitic (Chagas disease)
3. Mechanical damage: - heart surgery - cardiac catheterization
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Chronic A-V block
Congenital Chronic fibrosis
- coronary atherosclerosis - hypertension - diabetes
Idiopathic - Lenegr’e disease - Lev’s disease
Chronic myocarditis Cardiomyopathy Degenerative and autoimmunological processes Progressive muscular dystrophy Emery-Dreifuss syndrome
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Recognizing bradycardia (site of damage within the conduction system)
1. Sinus node disease - disorders of impuls formation (body of the sinus node)
- cardiac arrest, bradycardia - disorders of sino-atrial conduction (perinodal tissue) - sino-atrial block
2. Disorders of atrio-ventricular and intraventricular conduction
- A-V node (Wenckebach type A-V block, MOBITZ I)
- Atrio-ventricular bundle (His bundle): (MOBITZ II type A-V block)
- Bundle branches and fascicles: bundle branch block bifascicular block trifascicular block (MOBITZ II typeA-V block)
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Atropine 1 to 2 mg i.v. (bolus) Isoprenaline Dopamine Dobutamine Adrenaline
Treatment of acute bradycardia
Cardiac pacing transthoracic intracardiac
i.v. infusion in saline (drop rate depending on the effect)
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Venous routs for introduction of permanent pacing electrodes
External jugular vein
Internal jugular vein
Subclavian vein
Cephalic vein
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