Manual and IASTM Treatment featuring the FAKTR Concepts3.amazonaws.com/IASTM/PPT slides 2 per...
Transcript of Manual and IASTM Treatment featuring the FAKTR Concepts3.amazonaws.com/IASTM/PPT slides 2 per...
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Tom Hyde, DC, DACBSP, FRCCSS (Hon), ICSSP,
FAKTR®, ART®, GT, CRTP, CKTP, CSTI, FICC,
Adjunct Faculty UWS
Manual and IASTM Treatment featuring the FAKTR Concept
Cyriax – Friction Massage
Simple yet effective
Can be time consuming
Requires a functional exam
Tender area is not necessarily the source of pain
Hammer WI, Functional soft tissue examination and treatment by manual methods: the extremities, Aspen
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Cyriax
Because friction massage involves pressure and movement directed across the scar tissue, most of the theories as to why friction works are based on theories concerning the effect of motion on healing tissue. Hammer
Cyriax
…mentioned transverse motion across the involved tissue and the resultant traumatic hyperemia as the chief healing factors of friction massage.
Hammer
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Cyriax
Felt moving across the fibers at a right angle would not injure the normal healing tissue but would prevent the formation of or break down abnormal scar tissue.
Hammer
Cyriax
In the acute phase, only necessary to use a light force
In the chronic stages a deep, stronger friction is necessary.
Hammer
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Cyriax
Light friction used to aid promotion of normal orientation of collagen, maintain mobility and prevent future adhesions.
Recommends immediate treatment of ligament lesions to prevent adhesions.
Hammer
Cyriax treatment
Frictioning begins lightly until a level of anesthesia begins; then the pressure is increased until a deeper level of anesthesia is reached. With this method it should be possible to arrive at the deepest tissue level within a few visits. Hammer
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Cyriax treatment time
A minimum of 10 minutes is usually required
Depending on the thickness of the scar and the length of time the problem has existed, up to 20 minutes might be required
Hammer
Pin & Stretch Technique (ART®):
Hands-on soft-tissue technique applied to repetitive strains, adhesions, tissue hypoxia (decreased oxygen supply), and joint dysfunction
Frequently used in the treatment of Cumulative trauma disorders
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How does P&S work?
Used in the treatment of fascial restrictions
Breaks down adhesions running perpendicular to tendon, ligament, muscle and neural tissue
Allows for proper glide of adjacent soft tissue structures
P&S Treatment:
Utilizes passive and whenever possible active motions with a specific hand contact
Passive motions are used when muscle guarding prevents proper treatment when performed actively
Active motions are used whenever possible– Patient has greater control over treatment
– Simulates normal motions between adjacent soft tissues and the neurological feed back that controls motion
– Modifies pain impulses via the lateral spinal-thalamic tract via movement patterns
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P&S Treatment:
Frequently symptoms are reproduced during treatment
Treated tissue function should improve following treatment in most cases, but tissue remodeling and stabilization can take much longer
Pin & Stretch Technique (ART®):
A broad-based contact is taken on the area to be treated in a shorten position. Treatment can be initiated by having the clinician move the area from a shortened position to a lengthened position with a broad based contact (thumb) or the patient can actively move from the concentric to the eccentric position.
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FAKTR Concepts
Functional – begin to think about treating patients during function, not simply in a static posture – determine if the patient’s condition is worse with load or no load and if with load, treat with load
Kinetic – look at the kinetic chain when assessing for any soft tissue injury
FAKTR Concepts
Treatment – use your own choice of soft tissue treatment, P&S, MRT, Nimmo, PNF, Mattes - AIS, Mulligan, IASTM, Cyriax, Muscle Energy, PIR, etc
Rehab – why not consider using low tech rehab equipment during the treatment process in addition to post treatment
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FAKTR Concepts
Provocation – place the patient into a position of provocation, i.e., if back pain is present when seated, treat the patient seated
Motion – evaluate the patient to determine if the pain is worse with or without motion, if with motion, treat with motion
FAKTR Concepts
Once you determine all components of FAKTR, you begin your treatment protocol
Before initiating any form of treatment, a complete history and physical examination must be performed that leads you to the determination that your patient will benefit from whatever type of care you plan to utilize always remembering that your treatment MUST beat the natural history of that disorder
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Bringing Soft Tissue and Exercise Rehabilitation together
Rehab / proprioceptive techniques
Functional movement patterns utilizing the kinetic chain
Movements and/or positions of provocative to recreate symptoms, referral patterns, imbalances, instabilities, weaknesses, functional loss or injuries.
Why Dynamic activities during soft tissue treatments?
Through the use of active motions, functional positioning, and exercise rehabilitation during the soft tissue treatment it is possible to immediately initiate proper alignment of collagen synthesis and soft tissue remodeling through fibroblastic activity
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Why Dynamic activities during soft tissue treatments?
Dynamic activities force fascial planes to move against each other
If there is an adhesion preventing proper fascial, muscle, ligament, soft tissue function why would we ever treat statically
We can use dynamic activities to assist us in the manner in which new fibroblasts are laid down with our soft tissue treatment
INSTRUMENT-ASSISTED CROSS FIBER MASSAGE ACCELERATES KNEE LIGAMENT HEALING: Dept of
anatomy and cell biology, Indiana University. Loghmani, MT et al., 2006
Controlled study: 20 rats underwent surgical bilateral transection of the MCL.
7 days postoperatively GT was used on the left MCL for one minute 3x per week for 3 weeks.
Results: “Ligaments treated with IACFM were found to be 31% stronger and 34% stiffer than untreated ligaments.”
Article will appear in JOSPT.
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M. TERRI LOGHMANI, PT, MS.PT, PhD.Instrument-Assisted Cross-Fiber MassageAccelerates Knee Ligament Healing
506-514 | july 2009 | volume 39 | number 7 | journal of orthopaedic & sports physical therapy
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Before Treatment: Irregularly
oriented and diminished amount of
fibroblasts
20L 506004 4wk treated 20L PH2 4wk
treated 20R 506004 4wk untreated 20R PH2
The treated appears to have increased
cellularity and more regularly oriented,
elongated fibroblasts.
Effect of IASTM on Ligamentous Healing
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Robert Schleip states that “Fascia forms a continuous tensional network throughout the human body, covering and connecting every single organ, every muscle, and even every nerve or tiny muscle.”
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Muscles hardly ever transmit their full force directly via tendons into the skeleton but rather distribute a large portion of their contractile or tensional forces onto fascialsheets. In turn, these fascial sheets transmit there forces to both synergistic as well as antagonistic muscles when means they stiffen not only the respective joint, but may even affect regions several joints further away.
Schleip
Fascia is densely innervated with many sensory nerve endings including mechanoreceptors and nociceptors, which can become the source for acute myofascialpain. Fascia also has the ability to contract. Fascia has a blood supply in addition to a nerve supply and lymphatics. These coupled with it’s ability to contract raises the possibility for classification as an “organ.”
Schleip
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With the FAKTR-PM concept, we place focus on areas or regions rather than on origins and insertions. We look at regions and how these regions overlap or interconnect, rather than treating isolated, individual muscles. One important aspect of what we stress is how groups of muscles and fascia work together.
Example: The shoulder.
FAKTR Protocols and other manual and IASTM methods:
Following a complete history and examination, if appropriate, initiate FAKTR
Treat
Re-evaluate
Re-treat up to 3 times
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Caveat:
If you treat a patient with FAKTR and they exhibit excellent results but those results are short lived (1-2 days), and this scenario is repeated for 2-3 treatments, there is a strong possibility that the patient does not have a MSK disorder but some non-MSK. Please reassess, send for a second opinion or consider diagnostic workups.
Treatment is initiated as follows:
1. Site of pain:1. Pain that is local or distal to what is perceived to be
the involved structure
2. Loss of ROM
3. Loss of function
4. Tightness:Felt by clinician and patient
On evaluation, tissue does not feel “normal” to either the patient or the clinician
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Eccentric Training for Tendiopathies
Eccentric Training for Treatment of Tendiopathies, Murtaugh, Bryan and Ihm, Joseph M. Training, Prevention, and Rehabilitation: Current Sports Medicine Reports, 2013 Am College of Sports Med; 1537-890X/1203/175-182
Research has shown that treating tendinopathies in the eccentric phase can be effective.
Studies exist currently on the following body parts:Achilles tendonPatellar tendonProximal lateral elbowRotator cuff
Literature Review
• Medial collateral ligament (MCL) -most commonly injured knee ligament – Mechanism of a lateral to medial force causing
excessive valgus displacement
• Athletes involved in contact sports have a higher incidence of this injury (Dressendorfer 2014).
• Management of injuries is under debate – Surgical intervention to conservative management.
• Conservative treatment shows beneficial return to pre-injury levels over long-term measurement (Brotzman 2011).
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Case Review
• Player injured Oct 18
• Player fell into lateral leg, forcing valgus collapse
• Initial assessment by University med staff– Grade 2; possible 3
– 8 to 10 week RTP
– Season over
• Presents Oct 19, non-weight bearing on left knee
18-Y.O. Soccer player with MCL Tear
• 7/10 pain (L knee)• Mild, medial joint effusion• Significant medial pain with
palpation• (-) Lachman’s• (-) Post. Drawer• (+) Valgus Stress
– 10◦ deviation• MRI report indicates severe
Grade 2– Possibly Grade 3
• Other knee ligaments intact, including meniscus
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Intervention
• Goal was to return player to competition in 4 weeks for NCAA tournament
– Pending clearance by the university’s team physician.
• Primary treatment method was IASTM utilizing FAKTR-brand instruments and FAKTR protocol
Following assessment, initiation of the FAKTR Concepts was initiated
The athlete was having difficulty going up and down stairs secondary to loss of ROM and pain
Gait on flat surface was also significantly reduced and painful with loss or ROM
Treatment was first given in the position of provocation
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Length of each treatment was approximately 30-45 seconds
Following each treatment, the athlete was returned to a neutral, non-painful position, then asked to assume a position of provocation once more
This process continued and included having the athlete attempt to flex or extend the knee as far as possible, while also receiving treatment with motion, in and out of flexion and extension
Various low tech items were used during treatment such as:Therabands, BOSU, stability pad, iJoyBoard
The 5 Concepts indluded:
Static - valgus, flexion, extension
Motion - in and out of valgus, flexion and extension, climbing and descending stairs, medial and lateral movements so simulate a kick with zero resistance and very slowly
Resistance – with elastic bands attached with athlete attempting to kick a ball with the medial and lateral side of foot
Function – squatting, kneeling, walking, climbing and descending stairs
Proprioception – BOSU, Stability pad, iJoyBoard, single leg stance
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Treatment was both manual and with Instrument Assisted Soft Tissue Mobilization (IASTM)
FAKTR Instruments were used with an emollient to reduce friction
Cross Fiber Friction described by Cyriax was performed except friction was given in all directions, not just 90 degrees to the fiber
Pin and Stretch or Myofascial Release also given
Kinesiology tape was utilized in an attempt to offer a feeling of stability, aid in ROM and reduce pain.
Intervention• Weeks One & Two
– IASTM with active, provocative movement (daily)
– Class IV laser therapy (daily)
– Aquatic therapy (2x daily)
– Myofascial release (as needed)
• Note:
– No bracing was used during rehab (taping only)
– 8 treatments in office
– 3/10 pain, normal gait, body weight squat, single leg stance
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Intervention
Weeks One & Two
IASTM with active, provocative movement (daily)
Class IV laser therapy (daily)
Aquatic therapy (2x daily)
Myofascial release (as needed)
Note:
No bracing was used during rehab (taping only)
8 treatments in office
3/10 pain, normal gait, body weight squat, single leg stance
Intervention• Weeks One & Two
– IASTM with active, provocative movement (daily)
– Class IV laser therapy (daily)
– Aquatic therapy (2x daily)
– Myofascial release (as needed)
• Note:
– No bracing was used during rehab (taping only)
– 8 treatments in office
– 3/10 pain, normal gait, body weight squat, single leg stance
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Intervention• Weeks 3 & 4
– Rehabilitation focusing on:• Functional strength• Sprinting• Cutting• Kicking• Plyometrics• RTP
• IASTM used with provocative exercise as needed
• Laser used as needed• 8 treatments in office
Outcomes
• Return to play assessment Nov 16– 0/10 pain– All provocative testing normal– Agility, strength, sprinting & sport-
specific skills normal with no pain– Referred back to team MD
• Cleared by MD Nov 17
– 35 days total
• Competed in 2 games of NCAA tourney– No complaints or injury
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Lateral Ankle Sprain
Ankle Anatomy
Ankle Joints– Talocrural (ankle Mortice)– Subtalar– Distal Tib/Fib
Ligaments Peroneals Tibialis Anterior Ext Hallucis Longus Ext Digitorum Gastroc/Soleus Flex Hallucis Flex Digitorum Tibialis Posterior
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Talocrural Joint
Dome of the talus Medial Malleolus Lateral Malleolus Distal Tibia
Transmits torque from the lower leg to the foot– Internal and external
rotation at leg– Pronation and Supination
at foot
Ligaments of Talocrural Joint
Joint Capsule Anterior talofibular ligament
– Lateral malleolus to talus– Prevents anterior displacement,
resists inversion and internal rotation
– Demonstrates the lowest maximal load and energy failure
Calcaneofibular Ligament– Lateral malleolus to calcaneus– Resists supination, internal
rotation, inversion (most taut with these motions and dorsiflexion)
Posterior Talofibular ligament– Lateral malleolus to talus– Resists inversion and internal
rotation
Deltoid Ligament
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Subtalar joint
Anterior Subtalar– Calcaneus (sustentaculum
tali)
– Talus
– Navicular
Posterior– Calcaneus
– Talus
Converts torque from leg to foot
Ligaments ofSubtalar Joint
Deep Ligaments Cervical Anterior “cruciate”
Resists supination
Interosseous Posterior “cruciate”
Resists pronation and supination
Extensor Retinacula Lateral root (within
sinus tarsi)
Cervical Ligament
Interosseous Ligament
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Ligaments ofSubtalar Joint
Calcaneofibular Ligament– Resists inversion and
internal rotation
Lateral talocalcaneal ligament– Resists supination
Fibulotalocalcaneal ligament– Resists supination
LTCL
Distal Tibiofibular Joint
Syndesmosis
Accessory gliding
“high ankle sprain”
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Ligaments of Distal Tib/Fib Joint
Interosseous Membrane
Anterior Inferior Tibulofibular ligament– Injured in supination and
external rotation
– High ankle sprain
– Weaker of the two
Posterior Inferior Tibulofibular ligament– Injured in pronation and
external rotation
Muscular Anatomy
Lateral Compartment– Peroneus longus– Peroneus Brevis
Anterior Compartment– Tibialis Anterior– Extensor Hallucis Longus– Extensor Digitorum
Superficial Posterior Compartment– Gastroc– Soleus
Deep Posterior Compartment– Tibialis Posterior– Flexor Hallucis Longus– Flexor Digitorum
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Lateral Compartment
Peroneus Longus– Attachments
Lateral Head of Fibula and shaft
Base of First Metatarsal and lateral border of medial cuneiform
– Nerve Supply Superficial Peroneal
Nerve (L5,S1)
– Action Plantar Flexion and
Eversion
Lateral Compartment
Peroneus Brevis– Attachments
Distal 2/3 Fibular shaft
Base of 5th Metatarsal (styloid process)
– Nerve Supply Superficial Peroneal
Nerve (L5,S1)
– Action Plantar Flexion and
Eversion
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Anterior Compartment
Tibialis Anterior
– Attachments Inferior Lateral Tibial Condyle,
Lateral 2/3 tibial shaft
Medial surface of Medial Cuneiform and medial base of 1st Metatarsal
– Nerve SupplyDeep Peroneal (L4,5)
– ActionDorsiflexion and Inversion
Anterior Compartment
Extensor Hallucis Longus
Attachments Middle 2/4th of the Fibular shaft
Dorsal surface of the distal phalanx of the hallux
Nerve Supply Deep Peroneal (L5,S1)
Action Dorsiflexion of Hallux and foot
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Anterior Compartment
Extensor Digitorum– Attachments
Lateral Tibial Condyle, Upper 3/4th of the Fibular shaft
Dorsal surface of the middle and distal phalanx of the lateral 4 toes
– Nerve SupplyDeep Peroneal (L4,5,S1)
– Action Extension of lateral 4 toes and
foot and assist in Dorsiflexionof foot
Mechanism of Injury
Acute
Chronic
– Mechanical Instability Pathologic Laxity
Arthrokinematic Impairments
Synovial and Degenerative Changes
– Function Instability Impaired proprioception and sensation
Impaired Neuromuscular Firing Patterns
Postural Impairments
Strength Deficits
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Acute Ankle Sprain
Excessive supination of the rear foot about an externally rotated lower leg after initial contact of the rear foot during gait or landing from a jump
ATFL first and most commonly injured of the lateral ligaments
CFL 2nd most commonly injured
PTFL only sprained in severe injuries (often accompanied by fractures &/or dislocations
Grading of Ankle Sprains
Grade I:– Decreased ROM, strength WNL, mild swelling, tenderness over ATFL, slight
antalagia, negative Anterior Drawer test– Usually mild injury to ATFL
Grade II:– Decreased ROM with painful ROM, pain on MMT of everters, mild to
moderate swelling, tenderness over ATFL & CFL, antalgic gait, positive Anterior Drawer test
– Involvement of ATFL and CFL
Grade III:– Moderate to severe swelling, tenderness over ATFL, CFL, PTFL, antalgic gait,
positive anterior drawer test and talar tilt test, instability – Involvement of ATFL, CFL, PTFL, associated with fractures and/or dislocation
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Pathological Laxity
Result of ligamentous damage
Chronic ligamentous injury results in joint instability during functional activities resulting in eventual injury to the joint structures– Most often at the talocrural and subtalar joints
– ATFL ligament injury the talus is able to excessively supinate
Arthrokinematic Impairments
Fibular head displacement– Mulligan reports with chronic ankle instability the
fibular head displaces anteriorly and inferiorlyWith this positioning, the talus can go through a greater
range of supination before the ATFL becomes taut
Decreased ankle Dorsiflexion– Prevents the talocrural joint from reaching its closed-
pack position allowing for greater internal and inversion motions
– Increases pronation at the subtalar joint– Posterior mobilization of the talus on the tibia
recover dorsiflexion more quickly after a sprain
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Impaired Neuromuscular-Firing Patterns
Decreased reflexive response times of the peroneal muscles to inversion or supination
– Impaired proprioception
– Slowed nerve-conduction velocity
– Central impairments in neuromuscular-recruitment strategies
Deficits in Bilateral Gluteus medius recruitment with severe unilateral ankle sprain
Impaired Postural Control
Impaired single leg stance demonstrated after both acute and those with a history of repetitive ankle sprains
Combination of impaired proprioception and neuromuscular control
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Impaired Postural Control
Ankle Strategy– Foot pronates and supinates to maintain body’s
center of gravity during single leg stance
– Those with chronic instability demonstrate hip strategyLess efficient
Due to changes in central neural control in presence of ankle dysfunction
Strength Deficits
Demonstrated in both eversion and inversion
Debate as to reason
– Weakness due to muscle damage or impaired neuromuscular recruitment
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PHYSICAL EXAMINATION
Range of Motion
Passive and Active Range of Motions
– Flexion
– Extension
– Inversion
– Eversion
– Internal Rotation
– External Rotation
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A-P Drawer
ATFL sprain/tear
– Pain
– Laxity
Inversion Stress Test
CFL sprain/tear
– Pain
– Laxity
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Eversion Stress Test
Deltoid Ligament sprain/tear
– Pain
– laxity
Internal Rotation Stress Test
Posterior Deltoid Ligament and ATFL
– Pain, Laxity
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External Rotation Stress Test
PTFL and Anterior Deltoid Ligament
– Pain, laxity
Stresses Distal Tib/Fib Syndesmosis
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Bibliography (text books)
Evans RC. Illustrated essentials in orthopedic physical assessment. Mosby; 1994.
Hyde TE, Gengenbach MS. Conservative management of sports injury. Williams and Wilkins; 1997.
Hammer WI. Functional Soft Tissue Examination and Treatment by Manual Methods. Aspen; 1999.
Bibliography (ankle sprain)
Hertel J. Functional anatomy, pathomechanics, and pathophysiology of lateral ankle instability. Journal of Athletic Training. Dec 2002;37(4):364-375
Osborne MD, et al. The effect of ankle disk training on muscle reaction time in subjects with a history of ankle sprain. Amer Jour Sports Medicine. Sept 2001;29(5):627
Green T, et al. Randomized controlled trial of passive accessory joint mobilization of acute ankle inversion sprains. Physical Therapy. April 2001;81(4):984
Bassewitz HL, et al. Persistent pain after ankle sprain: targeting the causes. The Physician and Sportsmedicine. Dec 1997;25(12):48
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Bibliography (ankle sprain)
Hertel J. Functional anatomy, pathomechanics, and pathophysiology of lateral ankle instability. Journal of Athletic Training. Dec 2002;37(4):364-375
Osborne MD, et al. The effect of ankle disk training on muscle reaction time in subjects with a history of ankle sprain. Amer Jour Sports Medicine. Sept 2001;29(5):627
Green T, et al. Randomized controlled trial of passive accessory joint mobilization of acute ankle inversion sprains. Physical Therapy. April 2001;81(4):984
Bassewitz HL, et al. Persistent pain after ankle sprain: targeting the causes. The Physician and Sportsmedicine. Dec 1997;25(12):48