Management of Bone Defects in TKA Dr. Mohammad Hossein Dehghani Isfahan Jesus Hospital.
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Transcript of Management of Bone Defects in TKA Dr. Mohammad Hossein Dehghani Isfahan Jesus Hospital.
Causes of bone defects in primary TKA
• erosion secondary to angular arthritic change, • inflammatory arthritis,• osteonecrosis, • and fracture.
Bone defects in primary TKA
• typically :asymmetrical & peripheral,
• although contained deficiencies caused by cyst formation may occur.
Bone defects in primary TKA
• In primary TKA ,base of contained and peripheral defects : condensed sclerotic bone,
• In revision surgery, removal of components often leaves osteopenic surfaces
Major Concern
• Diminish of subchondral bone strength distal to the subchondral plate.
• Solution:the level of lateral tibial resection should not exceed 1 centimeter to avoid compromising implant durability,– others have demonstrated that proximal tibial
bone strength is adequate to 20 mm
Management
1. translation of the component away from a defect,
2. lower tibial resection, 3. cement filling, 4. autologous bone graft,5. allograft, 6. wedges or augments, 7. custom implants..
Management(Use of stems )
• in primary TKA:– necessary when bone grafting is required – or when the bone defect compromises fixation
and renders the resurfacing component unstable without the added support of intramedullary fixation
Management (Lateral Translation)
• Lateralizing a smaller tibial component : effectively eliminates bony defect by removing contact of implant with defect
• However, the largest tibial tray size and polyethylene insert should always be favored to create the largest reasonable contact surface to distribute load.
Management (Lower Tibial Resection)
• is often effective • limit of a lower tibial resection is : insertion of
the ITB and infrapatellar ligament. (Gerdy)• Additionally, a lower tibial resection will
complicate component fit because of the natural taper of the tibia, necessitating the use of a smaller tibial component or tapered tibial augments.
Management (Cement Filling)
• Lotke &Ritter demonstrated satisfactory long-term results with cement fill provided: – tibial bone defects are no deeper than 20 mm – and involve less than 50% of either plateau.
But– cement fill with or without screw reinforcement is an
inferior method of defect management & radiolucent lines are commonly observed under defects filled with cement.
– larger volumes of cement introduce the risk of thermal necrosis of the cement-bone interface
Management(autologous bone graft)
• readily available in primary TKA.• high rates of incorporation• osteoinductive properties • lack of potential disease transmission• typically used when the size criteria for
cement fill are exceeded.
Management (criteria that promote improved outcome)
1. creating viable/bleeding bed of host bone, 2. proper fit and finish of graft in host bed, 3. complete coverage of graft by the component to
avoid graft resorption secondary to stress shielding, 4. optimal alignment of components for even load
distribution, 5. limited weight bearing when larger grafts are used
to allow for graft union, 6. and grafts protected with stems when required .
Management
• Contained defects : easily filled with bone graft,
• Peripheral defects : more challenging.
Management(Custom Prostheses and Metal Wedge Augmentation)
• Metal wedge : intraoperative construction of a custom implant to address a bone defect,
• Defects of less than 25 mm
• Custom prostheses : for dealing with larger defects ( > 25mm)
• limitations of practicality and cost
Metal Wedge Augmentation
• available in triangular and rectangular shapes,
• both cemented and cementless options.
• load transfer across a larger defect: a rectangular block and stem augmentation.
– good results using wedges attached with screw fixation
Distal Femoral Defects• frequently observed in valgus deformities
when the lateral femoral condyle is dysplastic.• As with the tibia, defects can be managed with
cement, bone graft, and metal augments.
A. Contained Defects: managed in the same manner as
contained tibial defects.B. Peripheral Defects :
1. affecting the chamfer cuts, 2. affecting the distal surface, or 3. causing major bone loss.
Femoral deficiencies
• increasing stages of bone loss:– Stage 1• when the femoral osteotomy includes a portion of the
lateral distal femur, but contouring to accommodate the femoral component results in chamfer “air cuts” anteriorly and posteriorly. • cement fill is acceptable for filling anterior and
posterior spaces between bone and prosthesis. The sclerotic bone surface should be prepared to accept cement interdigitation.
Femoral deficiencies
– Stage 2• occurs when the level of the femoral osteotomy passes
distal to the lateral femoral condyle even without chamfer cuts.• In this situation, cement fill typically is unsatisfactory
unless combined with a femoral stem extension. • Even in this instance, a metal augment to the distal
femur is preferred.
Femoral deficiencies
– Stage 3 • refers to massive bone loss of one femoral condyle. • Substantial bone loss can be managed with allograft or
metal block augmentation, • Allograft requires a period of non–weight bearing
postoperatively and a femoral stem extension. The advantage of allograft is that if a revision is required, bone stock may be partially restored. • Metal augments allow quicker rehabilitation without
restricted weight bearing .
Femoral deficiencies
• In general, optimized collateral ligament stability and restoration of normal anatomy is preferable to the use of constrained prostheses.