Lecture 47 parekh sports f&a

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Foot and Ankle Sports Selene G. Parekh, MD, MBA Associate Professor of Surgery Partner, North Carolina Orthopaedic Clinic Department of Orthopaedic Surgery Adjunct Faculty Fuqua Business School Duke University Durham, NC 919.471.9622 http://seleneparekhmd.com Twitter: @seleneparekhmd

Transcript of Lecture 47 parekh sports f&a

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Foot and Ankle Sports

Selene G. Parekh, MD, MBAAssociate Professor of Surgery

Partner, North Carolina Orthopaedic ClinicDepartment of Orthopaedic Surgery

Adjunct Faculty Fuqua Business SchoolDuke University

Durham, NC919.471.9622

http://seleneparekhmd.comTwitter: @seleneparekhmd

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Ankle Problems in the Athlete

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Athletic Injuries

• Participation in organized/recreational sports continues to increase

• Injuries can be debilitating

• Recognition, prompt treatment and rehabilitation • Prevent long term disability

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Athletic Injuries

• 59% of adults participate in exercise

• 11% jog

• 25% run ≥ 2 miles/day

• 800 footstrikes/mile

• Impact 3-8 x body wt

• Running injuries vary between 37%-57%

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Athletic Injuries

• Risk Factors• Intrinsic

• Individual’s physical and personality traits

• Extrinsic• Training techniques

• Weekly running mileage greater than 40 miles

• Playing surfaces• Equipment

• Break-away bases

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Athletic Injuries

• Etiologic Factors• Biomechanical abnml

• Flexibility

• Strength

• Footwear & orthoses

• Playing surfaces

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Athletic Injuries: Etiology

• Biomechanical abnormalities

• Pelvic obliquity only predictor of injury

• <10% running injuries are due to biomechanical problems

• 70-80% runners treated with orthoses improve

• Need to wean use

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Athletic Injuries: Etiology

• Flexibility• Prevention

• Tight Achilles• Decreased DF

HV, turf toe, midfoot strain, ankle sprain, Achilles tendinitis, calf strain

• Decr. MTP ROM• HR dancers predispose to injury

• Hypermobility• Posterior ankle pain

• Pathologic laxity• Recurrent ankle sprain

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Athletic Injuries: Etiology

• Strength• Weakness injury

• >10% difference in extremity strength puts player at risk

• Soccer & volleyball players risk for injury and recurrence due to weakness

• Restore normal strength ratio though isotonic and isokinetic strengthening

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Athletic Injuries: Etiology

• Footwear and orthoses• Improper fit

• Tight/loose, neurapraxia• Load cushioning

• Reduced calcaneal stress fx in military recruits• Loss of sensory feedback

• Torque• Cleating ankle injuries• Too much, too little traction

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Athletic Injuries: Etiology

• Playing Surfaces• Concrete, synthetic track• Grass vs. artificial turf

• Role not completely defined

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Chronic Leg Pain

• Concurrent with foot/ankle complaints• Differential

• Chronic compartment syndrome• Medial tibial stress syndrome• Stress fractures• Muscle strain/tendinitis• Nerve entrapment• Vascular disease• Fascial herniation• Radiculopathy

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Chronic Leg Pain

• Incidence• 150 patients

• 33% chronic compartment syndrome• 25% stress fx• 14% muscle strain, 13% medial tibial stress

syndrome• 10% nerve entrapment, 4% venous disease

• Shin splints• Periostitis

• 10-15% all running injuries

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Exercise-Induced Compartment Syndrome

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Exercise-Induced Compartment Syndrome

• Incidence• Unclear

• 70% runners, 20% ball and puck• Clinical Features

• Pain predictable after certain distance, duration, speed

• Aching, cramping, stabbing, fullness• Neurologic symptoms

• Anterior, deep posterior compartments

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Exercise-Induced Compartment Syndrome

• Diagnosis• Clinical

• Rest: Minimal tenderness • Exercise to provoke sx• Muscle herniation 20-60% pts• DPN/SPN

• Radiologic• Stress fx• Occult tumor• Bone scan• MRI

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Exercise-Induced Compartment Syndrome

• Criteria for Diagnosis• Pressure measurements

• Resting, post-exercise measurements

• Secondary• Pressure >15mmHg pre-exercise• Pressure >30mmHg 1 min post-exercise• Pressure >15mmHg 5-10 min post-exercise

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Exercise-Induced Compartment Syndrome

• Conservative treatment• Dependent on athletes goals• Activity modification• Rest gradual increase in exercise• Ice, NSAID’s• Insoles, orthoses

• Surgical treatment• Fasciotomies

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Exercise-Induced Compartment Syndrome

• Results• Good with proper patient and techniques• 80-95% excellent results• 20% decrease in strength

• Complications• Swelling• Hematoma• Infection• Herniation• Nerve entrapment• Recurrence

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Stress Fractures: Tibia & Fibula

• Abnormal repetitive loads causing imbalance of resorption over formation

• Running > basketball > soccer > skating > aerobics > ballet

• Athletes with decreased mineral content • Amenorrheic females

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Stress Fractures: Tibia & Fibula

• Clinical features• Recent change in routine• Insidious, localized tenderness, swelling

• Diagnosis• X-ray initially negative

• (2-3 weeks) radiodense haze, periosteal rxn• Transverse cortical lucency• Bone scan

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Stress Fractures: Tibia & Fibula

• Conservative Treatment• Rest, cessation of activity• NWB PWB to WBAT

• As symptoms allow• Swimming, pool running, cycling

• Resume competition full motion, strength, minimal tenderness

• Surgical Treatment• Excision, bone grafting• PEMF bone stimulation• Intramedullary fixation

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Gastrocnemius-Soleus Strain

• Common injury• Racquet sports, basketball, running, skiing• Plantaris tear

• Clinical Features• Sudden knee extension while crouched w/ ankle DF• Sudden sharp pain

• Difficulty with toe-off

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Gastrocnemius-Soleus Strain

• Treatment• Casting/immobilization

• RICE

• WBAT• Active dorsiflexion• Gentle passive stretching more functional

recovery

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Nerve Entrapment Syndromes

• Superficial Peroneal Nerve• Neuritic pain, burning, tingling, radiation• Tenderness over area of compression• R/o compartment syndrome, double-crush• Tx

• Injections, modalities, thermogesic creams, neuroactive medications, shoe modifications

• Surgical nerve release, fasciotomy

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Popliteal Artery Entrapment Syndrome

• Rare, mimics pain chronic compartment syndrome• Recognition important as may lead to amputation

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Popliteal Artery Entrapment Syndrome

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Popliteal Artery Entrapment Syndrome

• Clinical• Cramping pain, intermittent claudication

• 67% bilaterality

• Pulses diminished or absent• Knee hyperextended, ankle dorsiflexed

• Knee warm, foot cool• Ischemia, thrombosis; palpable mass

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Popliteal Artery Entrapment Syndrome

• Diagnostics• Provocative testing (treadmill) absent pulses• Doppler, U/S flow, aneurysm• Biplanar arteriography after exercise

• Deviation, occlusion, dilatation

• Treatment• Release• Bypass grafting

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Venous Disease

• Effort thrombosis• Pain, edema, distended veins, discoloration• Venography• Tx: anticoagulation heparin, coumadin

• Thrombophlebitis• Immobilization after injury• Air travel• Dehydration• Oral contraceptives• Alcohol/drug use

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Delayed-Onset Muscle Soreness

• Unaccustomed to activity

• Eccentric muscle contraction microinjury, subcellular level• Inability to generate maximal force

• Pain, fatigue• Loss of performance 24-48hrs• Resolve 5-7 days

• Tx• Symptomatic treatment

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Medial Ankle Sprains

• Rare, associated with lateral, bony injuries

• 2 portions:• Superficial deltoid• Deep deltoid

DeepSuperficial

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Medial Ankle Sprains

• Biomechanics of deltoid• Prohibits abduction

• Secondary restraint against anterior translation ankle

• Deep deltoid • Greatest restraint lateral translation (after fibula)• Highest load to failure

• Valgus tilt superficial & deep rupture

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Medial Ankle Sprains

• Clinical Evaluation• Lateral injury, fracture, syndesmotic injury• Abduction, eversion• Other injuries

• Maisonneuve, FDL, PTT, FHL, tibial & saph n

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• Radiographs• Assoc fx, AP stress valgus tilt, normal with

incomplete injury• MRI

Medial Ankle Sprains

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• Conservative Treatment• Dependent upon associated injuries• Walking cast, boot 6-8wks• Functional treatment

• Surgical treatment• Inability to reduce medial clear space• Direct repair

• Suture, suture anchors

• Stirrup brace 6 months after surgery

Medial Ankle Sprains

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Sinus Tarsi Syndrome

• Related to subtalar instability

• Nebulous condition w/ pain in lateral ankle & sinus tarsi

• Pathologic anatomy not clearly defined• Scarring or degenerative changes to soft tissues

within sinus tarsi• Loss of nerve function loss of proprioception

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Sinus Tarsi Syndrome

• Diagnosis• Clinical Evaluation

• Pain lateral ankle, sinus tarsi• Prior inversion injury• Instability rare

• Radiologic Evaluation• X-rays- normal• Subtalar arthrography

• Absence of microrecesses, interdigitation• MRI

• Fibrosis, fluid collections

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Sinus Tarsi Syndrome

• Conservative treatment• Injection

• Surgical treatment• Exploratory, sx may persist• Resect fibrofatty tissue & extensions from IER• Subtalar arthroscopy

• Results• 96 cases; 69% excellent, 25% good, 6% failures

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Os Trigonum/Posterior Impingement

• First Described Rosenmuller

• First Described as Fx Shepherd 1822

• Asymptomatic• Separate

Ossification Center• Age 8-11• Incidence 1.7 - 7%

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Os Trigonum/Posterior Impingement

• No side predominence• Vary in size• Normal anatomic variation

• Mild extension• Thumb shaped projection - 1 cm (Stieda’s

Process)

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Os Trigonum/Posterior Impingement

• Symptoms• Pain with PF• Young Athlete/Dancer

• Spectrum• Acute – Fx• Gradual - FHL Tendinitis

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Os Trigonum/Posterior Impingement

• Conservative• Immobilization• NSAID

• Surgical – Excision

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Prone Ankle Scope

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Evaluation

• 54% of talar dome

• Os trigonum

• Haglund’s

• Achilles insertion site

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Foot Problems in the Athlete

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Goals & Objectives

• Specific problems• Midfoot

• Forefoot

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Os Peroneum

• Ossification• Complete• Less than complete• Multipartite

• Location• Lateral border calc & plantar lateral cuboid

• Useful marker for ruptured PL

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Painful Os Peroneum Syndrome (POS) & PL Disruption

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POPS/Peroneus Long. Tendonitis/Tear

• Conservative treatment• Cast immobilization (20% success)• Physical therapy

• Surgical treatment• Os peroneum excision/tendon repair• Peroneal tenodesis

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Forefoot Sprains

•First MTP joint/ “turf toe”

•Etiology•Hard artificial surface, flexible shoes•True incidence not defined, but many reports indicate a fairly common entity

•Functional disability•Push-off impaired•Compromised forward drive and running•More missed games & practices than other injuries

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Forefoot Sprains

• Mechanisms of injury• Hyperextension of the MTP joint

• Foot dorsiflexed, forefoot fixed, heel raised• Joint capsule tears, compression injury to the dorsal articular

surface with extremes of dorsiflexion

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Forefoot Sprains

• Etiologic factors• Shoe-surface interface

• Flexible shoes

• Hardness of surface• Loss of resiliency with time

• Enhanced friction• Fixes foot allows extremes of motion

• Excessive DF• In those with limited DF

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Forefoot Sprains

• Radiographs• R/o fx, avulsions, impaction• Other methods usually not necessary

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Forefoot Sprains

• Conservative treatment• RICE, NSAID’s• Taping, boot• Increase stiffness in shoe

• Custom molded insert/Morton’s extension

• Rehab once decrease in sx allows

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Forefoot Sprains

• Surgical treatment• Rarely necessary

• Instability• Loose bodies, osteochondral injury• Ligamentous disruption• Sesamoid retraction

• Repair plantar plate, FHB, long flexor transfer

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Metatarsal Stress Fractures

• Etiology• Injury to opposite limp

increased stress to normal limb

• Muscle fatigue increase in loading

• Hard surfaces, poorly cushioned shoes, heavy heel strike

• Rigid cavus feet decrease shock absorption of the midfoot increase stress on MT’s

• Hypermobile first ray or long 2nd MT

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Stress Fractures

• High index of suspicion • Focal tenderness • Min/no swelling• X-rays nl < 3-4 wks • Bone scan, MRI, & CT scans

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Treatment

• Activity modifications• Non-impact sports• Avoid casting if possible

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Treatment

• Shoe modifications• Rigid cavus foot

• Flexible, cushioned arch support• Hyperpronation/hypermobile 1st ray

• Soft medial arch support• Long 2nd MT

• MT pad

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RE ECT

the ankle

the foot