Gait, movements that produce locomotion
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Transcript of Gait, movements that produce locomotion
GAIT
movements that produces locomotion
Gait Cycle or Stride
� A single gait cycle or stride is defined:
� Period when 1 foot contacts the ground to when that
same foot contacts the ground again
� Each stride has 2 phases:
�Stance Phase
� Foot in contact with the ground
�Swing Phase
� Foot NOT in contact with the ground
2
Stance Phase
� Principal events during the stance phase
1.Heel strike
2. Foot-flat (followed by opposite heel-off)
3.Heel-rise (followed by opposite heel strike)
4. Toe-off
3
Principal events during the stance phase:
heel-strike, foot-flat, heel-off, toe-off
4
Activities occur in stance phase
Traditional Method
1. Heel Strike : double support
2. Foot flat : total contact
3. Mid-stance : total weight bearing
4. Heel-off : heel clears the ground
5. Toe-off : toe clears the ground
RLA Method
1. Initial contact : heel strike
2. Loading response : double support
3. Mid-stance : begins when contralateral L/E clears the ground & when the body come straight line to supporting limb
4. Terminal stance : end of mid stance to initial contact of CL L/E
5. Pre-swing : period of clearance from the ground
5
Initial Contact
� Phase 1
� The moment when the
red foot just touches the
floor.
� The heel (calcaneous) is
the first bone of the foot
to touch the ground.
� Meanwhile, the blue leg
is at the end of terminal
stance.
6
Static Positions at Initial Contact
� Shoulder is extended
� Pelvis is rotated left
� Hip is flexed and externally rotated
� Knee is fully extended
� Ankle is dorsiflexed
� Foot is supinated
� Toes are slightly extended
7
Loading Response
� Phase 2
� The double stance period
beginning
� Body wt. is transferred
onto the red leg.
� Phase 2 is important for
shock absorption,
weight-bearing, and
forward progression.
� The blue leg is in the
pre-swing phase. 8
Static Positions at Loading Response
� Shoulder is slightly
extended
� Pelvis is rotated left
� hip is flexed and slightly
externally rotated
� knee is slightly flexed
� ankle is plantar flexing to
neutral
� foot is neutral
� Toes are neutral
9
Mid-stance� Phase 3
� single limb support
interval.
� Begins with the lifting of
the blue foot and
continues until body
weight is aligned over the
red (supporting) foot.
� The red leg advances
over the red foot
� The blue leg is in its mid-
swing phase.10
Static Positions at Midstance
� Shoulder is in neutral
� Pelvis is in neutral rotation
� Hip is in neutral
� Knee is fully extended
� Ankle is relatively neutral
� Foot is pronated
� Toes are neutral 11
Terminal Stance
� Phase 4
� Begins when the red heel
rises and continues until
the heel of the blue foot
hits the ground.
� Body weight progresses
beyond the red foot
12
Static Positions at Terminal Stance
� Shoulder is slightly flexed
� Pelvis is rotated left
� Hip is extended and internally rotated
� Knee is fully extended
� Ankle is dorsi flexed
� Foot is slightly supinated
� Toes are neutral13
Pre-swing
� Phase 5
� The second double
stance interval in the gait
cycle.
� Begins with the initial
contact of the blue foot
and ends with red toe-
off.
� Transfer of body weight
from ipsilateral to
opposite limb takes
place. 14
Static Positions at Pre-swing� Shoulder is flexed
� Pelvis is rotated right
� Hip is fully extended and internally rotated
� Knee is fully extended
� Ankle is plantar flexed
� Foot is fully supinated
� Toes are fully
extended15
Swing phase
� Principal events during the Swing phase
1. Acceleration: ‘Initial swing’
2. Mid swing : swinging limb overtakes the limb in
stance
3. Deceleration: ‘Terminal swing’
16
Activities occur in swing phase
Traditional Method
1. Acceleration : starts
immediately from toe
off
2. Mid stance : swing
directly beneath body
3. Deceleration : knee
extension and prepare
for heel strike
RLA Method
1. Initial swing : max.
knee flexion
2. Mid swing : from max.
knee flxn. to verticl.
Postn. of tibia
3. Terminal swing : from
verticl. Postn. of tibia
to initial contact
17
Initial Swing� Phase 6
� Begins when the red foot
is lifted from the floor
and ends when the red
swinging foot is opposite
the blue stance foot.
� It is during this phase
that a foot drop gait is
most apparent.
� The blue leg is in mid-
stance.
18
Static Positions at Initial Swing
� Shoulder is flexed
� Spine is rotated left
� Pelvis is rotated right
� hip is slightly extended
and internally rotated
� Knee is slightly flexed
� Ankle is fully plantar
flexed
� Foot is supinated
� Toes are slightly flexed
19
Mid-swing
� Phase 7
� Starts at the end of the
initial swing and
continues until the red
swinging limb is in front
of the body
� Advancement of the red
leg
� The blue leg is in late
mid-stance.
20
Static Positions at Mid-swing
� Shoulder is neutral
� Spine is neutral
� Pelvis is neutral
� Hip is neutral
� Knee is flexed 60-90°
� Ankle is plantar flexed to
neutral
� Foot is neutral
� Toes are slightly
extended
21
Terminal Swing
� Phase 8
� Begins at the end of mid-
swing and ends when the
foot touches the floor.
� Limb advancement is
completed at the end of
this phase.
22
Static Positions at Terminal Swing
� Shoulder is extended
� Spine is rotated right
� Pelvis is rotated left
� Hip is flexed and
externally rotated
� Knee is fully extended
� Ankle is fully dorsi flexed
� Foot is neutral
� Toes are slightly
extended
23
DISTANCE VARIABLES
� Step length
� Stride length
� Width of walking base
� Degree of toe out
24
Step Length
� Distance between corresponding successive points
of heel contact of the opposite feet
� Rt step length = Lt step length (in normal gait)
25
Stride Length
� Distance between successive points of heel contact
of the same foot
� Double the step length (in normal gait)
26
Walking Base
� Side-to-side distance between the line of the two feet
� Also known as ‘stride width’
� Normal is 3.5 inches
27
Degree of toe out
� Represents the angle of foot placement
� It is the angle formed by each foot’s line of progression and a line intersecting the centre of the heel and the second toe
� Normal angle is 7°for men at free speed walking
28
TEMPORAL VARIABLES� Stance Time : Is the amount of time that elapses during the stance phase of one extremity in a gait cycle
� Single support time : is the amount of time that elapses during the period when only one extremity is on the supporting surface in a gait cycle
� Double support time : is the amount of time that a person spends with both feet on the ground during one gait cycle
� Stride duration : is the amount of time it takes to accomplish one stride
� Step duration : is the amount of time spent during a single step
29
TEMPORAL VARIABLES
� Cadence � Number of steps per unit time� Normal: 100 – 115 steps/min
� Cultural/social variations
� Velocity� Distance covered by the body in unit time
� Usually measured in cm/s
� Instantaneous velocity varies during the gait cycle
� Average velocity (m/min) = step length (m) x cadence (steps/min)
� Comfortable Walking Speed (CWS)� Least energy consumption per unit distance
� Average= 80 m/min (~ 5 km/h , ~ 3 mph)
30
DETERMINANTS OF GAIT
1. Lateral pelvic tilt
2. Knee flexion
3. Knee, ankle, foot interactions
4. Pelvic forward and backward rotation
5. Physiologic valgus of knee
� DGs represents the adjustments made by above components that help to keep movements of body’s COG to minimum.
� They are credited with decreasing the vertical and lateral excursions of the body’s COG and therefore decreasing energy expenditure and making gait more efficient
31
Lateral pelvic tilt
� During mid-stance the COG reaches the peak level & the total body supported by one lower extremity
� The pelvis slopes downwards laterally towards the leg which is in swing phase. i.e. rotation about an anterior-posterior axis
� Only anatomically possible if the swing leg can be shortened sufficiently (principally by knee flexion) to clear the ground.
� Where this is not possible (e.g. through injury), the absence of pelvic tilt and pronounced movements of the upper body are obvious.
32
Knee flexion
� Another DG which helps to reduce the COG during mid-stance
� As the hip joint passes over the foot during the support phase, there is some flexion of the knee.
� This reduces vertical movements at the hip, and therefore of the trunk and head.
33
Knee, ankle, foot interactions
� KAF interaction prevent abrupt hike in COG from heel strike to foot flat
� After heel strike huge upward displacement of COG occurs
� This is reduced by Knee flexion, ankle plantar flexion & foot pronation.
� From mid stance to heel off there is sudden drop in COG
� Ankle plantar flexion, knee extension and foot supination maintain this
34
Pelvic forward and backward rotation
� Forward rotn. occurs in swing phase
� It starts during acceleration and ends in deceleration
� During mid-swing pelvis comes to neutral position
� Forward and backward rotation help to prevent further reduction in COG which started from mid-stance
� During deceleration both lower extremities lengthens
� This prevents in further reduction of COG
35
Physiologic valgus of knee
� Is minimised by having a narrow walking base i.e. feet closer together than are hips.
� Therefore less energy is used moving hip from side to side (less lateral movement needed to balance body over
stance foot)
� Reduced lateral pelvic displacement
36
Efficiency, and energy considerations
• Walking is very energy-
efficient: little ATP is required.
• This is because of various
mechanisms that ensure the
mechanical energy the body has
is passed on from one step to
the next.
• The two forms of mechanical
energy involved are
•kinetic energy (energy due
to movement
•potential energy (energy
due to position)
Economy (J m-1)
37
A conventional pendulum –
energy interconversion
P.E. – Potential energy
K.E. – Kinetic energy
Three points on a pendulum
swing are illustrated.
As the pendulum swings away
from the midpoint, in either
direction, KE is progressively
converted into PE
At the extreme points in the
swing, there is no KE at all and
all the energy is present as PE38
Conventional pendulum action
during the swing phase
� The legs move as conventional pendulums during the swing
phase (with a little assistance from the hip flexors).
� This reduces the amount of muscle energy needed to move
the swinging leg forward
� It also accounts for the “natural” frequency of gait that has
optimal energy efficiency
� Although the legs swing forwards much like pendulums, they
are prevented from swinging backwards by foot strike.
� During the stance phase, the leg can be viewed as an
“inverted pendulum”. This action also involves inter-
conversion of potential and kinetic energy39
An “inverted” pendulum
The pendulum
“bounces”
backwards and
forwards, using
the springs.
40
“Inverted” pendulum action during the stance phase
� During the stance phase, the leg can be viewed as an “inverted
pendulum”.
� The forward momentum of the body gives it the necessary
initial angular velocity of rotation (taking the place of the
“spring” on the previous slide).
� “Inverted” pendulum action also involves inter-conversion of
potential and kinetic energy, but in this case (unlike a
conventional pendulum) KE reaches a minimum at the
midpoint of the motion, and PE is highest at that point.
� When reaching the endpoint of its “inverted swing” the stance
leg does not swing back, as a real inverted pendulum would,
because the foot is taken off the floor, the fulcrum transfers
from the foot to the hip, and the leg swings again as a
conventional pendulum. 41
Positive & negative Work� At a cadence of 105-112 steps/minute
� between heel strike and foot flat
1. a brief burst of positive work (energy generation) occurs as the hip extensors contract concentrically
2. while the knee extensors perform negative work (energy absorption) by acting eccentrically to control knee flexion
� from foot flat through mid-stance
1. Negative work is done by plantar flexors as the leg rotates over the foot during the period of stance
2. Positive work of the knee extensors occurs during this period to extend the knee
� late stance and in early swing
1. Positive work of plantar flexors and hip flexors increase the energy level of the body
� In late swing
1. negative work is performed by the hip extensors as they work eccentrically to decelerate the leg in preparation for initial contact
42
Forces
� The principal forces are:� body weight (BW)
� ground reaction force (GRF)
� muscle force (MF)
� BW and GRF are external forces; so the movement of the centre of mass (CoM) can be predicted from them alone.
� MF must be examined however if we wish to consider either of the following:
� movements of individual limbs or body segments,
� why GRF changes in magnitude and direction during the gait cycle.
43
Vitally important point:
Muscle forces can only influence
the movement of the body as a
whole indirectly, by their effects
on the GRF
44
Walking as a “controlled fall”� One way of beginning to understand the mechanics of walking is to view the movements as a “controlled fall”
� When starting a walk, we lean forward, overbalancing from the equilibrium position.
� This gives the upper part of the body forwards (and downwards) motion
� As the body falls forwards, a leg is extended forwards and halts the fall
� At the same time, the other leg “kicks off” in order to keep the body moving forwards.
� This forward momentum carries the body forward into the next forward fall, i.e. the start of the next step
45
Walking as a controlled fall: forces
involved
� When starting to move, we lean forward (MF)
� As the body starts to fall (BW), a leg is extended
forwards and halts the fall (MF; GRF)
� At the same time, the other leg “kicks off, upwards and
forwards” (MF; GRF) in order to keep the body
moving forwards.
� This forward momentum carries the body forward into
the next forward fall, i.e. the start of the next step
46
Body weight
� Always acts vertically downwards from the CoM
� If its line of action does not pass through a joint, it
will produce a torque about that joint
� The torque will cause rotation at the joint unless it is
opposed by another force (e.g. muscle, or ligament)
� BW contributes to GRF
47
Ground reaction force
� The force that the foot exerts on the floor due to gravity & inertia is opposed by the ground reaction force
� Ground reaction force (RF) may be resolved into horizontal (HF) & vertical (VF) components.
� Understanding joint position & RF leads to understanding of muscle activity during gait
� Forces are typically resolved into:1. Vertical Compression (z)2. Anterior-Posterior Shear (y)3. Medial-Lateral Shear (x)
48
Muscle force
In gait, as in all human movement, muscle activation generates internal joint moments (torques) that: � Contribute to ground reaction force
� Ensure balance
� Increase energy economy
� Allow flexible gait patterns
� Slow down and/or prevent limb movements
Much muscle activity during gait is eccentric or isometric, rather than concentric
49
Center Of Pressure (COP)
� Represents the centroid of foot forces on the floor
� This is an idealization, because pressures are distributed all over
� It is important, because we want to know where the GRF is applied to the body
� When measured by a force plate, it is more correctly called the point of application of the GRF
� Plotting the COP as it moves under the foot:
1. Normal Path: Center of the calcaneus or slightly lateral, curving laterally and then medial (pronation) and ending between the 1st and second toes
2. Variable: Normal individuals can have many COP trajectories, just by changing the footgear
50
Muscles, Joints, and Forces
� Eccentric
� Concentric
� Ground Reaction Force
51
Sagittal Plane Analysis
Initial Contact
� Hip 30° of flexion,
� knee is extended
� ankle is neutral
� GRF
� Ant. to hip, drives the hip into
flexion
� Ant. to knee drives the knee
into extension
� Ankle into plantar flexion
� Hip: hamstrings, gluteus maximus,
and adductor magnus (i to e)
� Knee: quadriceps (c to e)
� Tibiotalar joint: tibialis anterior (e)
� Subtalar joint: anterior and lateral
compartment muscles (e)
52
Sagittal Plane Analysis
Loading Response
� Hip extension 25°
� Knee flexion to 20°
� Ankle plantar flexion to 10°
� Contralateral pelvis rotates
anterior
� GRF
� Ant. to hip
� posterior to knee
� posterior to ankle
� Hip : extensors (e),
Abductors (e) limit contralateral
drop to 5°
� Knee : Quadriceps fire (c)
� Ankle :Tibialis anterior (e)
53
Sagittal Plane Analysis
Mid Stance
� GRF through hip, knee, and ankle
� Muscular activity terminates
� Hip and knee stability provided by ligamentous restraints
� GRF
� Posterior to hip
� Anterior to knee and ankle
� Gastrosoleus complex fires to initiate knee flexion
� Pelvis continues to rotate, abductors continue to resist pelvic drop
54
Sagittal Plane Analysis
Terminal Stance
� No change in GRF
� Free forward fall
� Strong activation of
gastrosoleus complex
55
Sagittal Plane Analysis Pre-Swing
� Hip 20° of hyperextansion
� Knee 30° of flexion
� Ankle 20° of plantarflexion
� Toes 50° of hyper extension
� GRF
posterior to hip, knee
anterior to ankle
� Rapid flexion of knee from rapid
heel rise and unweighting of limb
� Rectus femoris initiates hip flexion
� Adductor longus fires
� Hip : iliopsoas, adductor magnus,
adductor longus
� Knee : Quadriceps
� Ankle :Gastrosoleus complex
� Toes : Ab.hal., FDB, FHB,
Introssei, lumb. 56
Sagittal Plane Analysis
Initial Swing
� Hip 0-30° of flexion
� Knee from 30-60° of flexion and extension from 60-30°
� Ankle 20° of plantarflexion to neutral
� Foot clearance is passive due to rapid hip flexion, unless gait is very slow
� In slow gait, tibialis anterior and hamstrings fire to help
� Gait cadence (speed) governed by accelerations of hip flexion during this phase
� Hip flexion
� Rectus femoris
� Iliacus
� Adductor longus
� Gracilis
� Sartorius
� Rest of limb is passive pendulum
57
Sagittal Plane Analysis
Mid Swing
� Tibialis anterior fires to
maintain foot position
� Knee extension and hip
flexion continue by inertia
58
Sagittal Plane Analysis
Terminal Swing
� Decelerate knee extension
and hip flexion
� Hamstrings
� Gluteus max
� Quads co-contract
� Tibialis anterior maintains
ankle position
59
Frontal Plane Analysis
IC to LR
� Pelvis : forward rotn.
� Hip : med. rotn. of femur
� Knee : increased valgus
� Ankle : increased pronation
� Thorax : Post. postion at
initial contact and begins
moving forward
� Shoulder : extended and
moving forward
� Gracilis, vastus medialis,
semitendinosus, LH of
biceps femoris
� Tibialis post. to control
valgus thrust
60
Frontal Plane Analysis
LR to Mid-stance
� Pelvis : Rt. side rotating backward to reach neutral. Lat. tilt towards the swinging extremity
� Hip : Med. rotn. of femur continues to neutral.
� Knee : Reduction in valgusand tibia begins to rotate laterally
� Ankle-foot : neutral at mid-stance
� Thorax : Rt. side move forward
� Shoulder : Move forward
� Hip abductors are active
� Tibialis post. produce
supination
61
Frontal Plane Analysis
Mid-stance to TS
� Pelvis : Rt. side move Posteriorly
� Hip : Lat. rotn. of femur and adduction
� Knee : lat. rotn. of tibia
� Ankle & foot : supination of subtalar jt. increases
� Thorax : Rt. side move forward
� Shoulder : Rt. shoulder move forward
� Hip : inconsistent adductor activity
� Ankle plantar flexor activity
62
Frontal Plane AnalysisTS to PS
� Pelvis : Lt. side move forward, lat. tilting to swing side ends as double support begins
� Hip : Abduction as wt. shifted to opp. extremity, Lat. rotn. of femur
� Knee : Lat rotn. of tibia
� Foot/Ankle : Wt. shifted to toes. Supination of sub talarjoint
� Thorax : Translation to the left
� Shoulder : Moving forward
� Hip adductors control pelvis
� Plantar flexion
63
Frontal Plane Analysis
IS to MS
� Pelvis : Lat. pelvic tilt to the rt. Right side move forward
� Hip : Lat. rotn to med. rotn.
� Knee : From lat. to med. rotn
� Foot/Ankle : NWBing subtalar joint returns to supination
� Thorax : Rt. side move Posteriorly
� Shoulder : Rt. side move Posteriorly
� Left gluteus medius on pelvis
64
Frontal Plane Analysis
MS to TS
� Pelvis : Rt. side move anteriorly
� Hip : Lat. tilting to the left med. rotn.
� Knee : Med. rotn.
� Ankle/Foot :
� Thorax : Rt. side move posteriorly
� Shoulder : Rt. shoulder move posteriorly
� Right gluteus medius
65
A word on running
� Walking is biomechanically like a pendulum, KE to PE to KE
� Running is biomechanically like a spring
� No double leg stance phase
� Aerial phase or float period
� Ground Reaction Force during stance phase loads spring (quads, achilles)
� Unloading in preparation for aerial phase is passive recoil from tendons and connective tissue and dynamic concentric muscular contraction
66
Running: swing phase
� Muscular rather than pendularmotion at hip.
� Knee flexion, and ankle dorsiflexion, bring CoM of the leg closer to the hip. This reduces moment of inertia and increases angular velocity.
� Knee movements largely passive (i.e not due to muscle activity), and result from transfer of momentum from thigh.
� Depending on the speed of running, initial ground contact may be with heel, whole foot, or ball of foot.
67
Running: support phase
� Hip: slight flexion followed by extension. Gluteus maximusactivity initially eccentric
� Knee: degree of flexion increases with speed; that of extension decreases. Quadriceps active at knee, initially eccentrically
� Ankle : dorsiflexion followed by plantarflexion. Gastrocnemius and soleus active during whole phase, particularly so at the end.
� Stretch shortening/energy
storage activity occurs at all
three joints
68
STAIR GAITStair Ascent� Stance Phase
1. Weight acceptance
2. Pull up
3. Forward continuance
� Swing Phase
1. Foot clearance
2. Foot Placement
� Ascending stairs involves a large amount of positive work that is accomplished by concentric action of the rectus femoris, vastus lateralis, soleus and medial gastrocnemeus
69
STAIR GAITStair Descending� Swing Phase
1. Foot clearance
2. Foot Placement
� Stance Phase
1. Weight acceptance
2. Pull up
3. Forward continuance
� Descending stairs is achieved mostly through eccentric activity of same muscles and involves energy absorption
� The support moments exhibit similar pattern in stair and level gait
� But magnitude is greater in stair gait
70
Sagittal Plane analysis of Stair gait
WA to PU
� Hip : Extension from 60-
30° of flexion
� Knee : Extension from
80-35° of flexion
� Ankle : DF 20-25° of
DF, PF 25-15° of DF
� Hip : Gluteus maximus,
Semitendinosus, Gluteus
medius
� Knee : Vastus Lateralis,
Rectus femoris
� Ankle : Tibialis anterior,
soleus, gastronemius
71
Sagittal Plane analysis of Stair gait
PU to FC
� Hip : extension 30 - 5° of
flexion, flexion 5-20° of
flexion
� Knee : Extension 35-10°
of flexion, flexion 5- 10°
of flexion
� Ankle : PF 15°of DF to
15-10° of PF
� Hip : Gluteus maximus,
gluteus medius,
semitendinosus
� Knee : Vastus lateralis,
rectus femoris
� Ankle : Soleus,
gastronemius, tibialis
anterior
72
Sagittal Plane analysis of Stair gait
Foot clearance through foot placement
� Hip : flexion 10-20° to 40-60° of flexion, extension 40-60° of flexion to 50° of flexion
� Knee : Flexion 10° of flexion to 90-100° of flexion, extension 90-100° of flexion to 85° of flexion
� Ankle : DF 10° of PF to 20°of DF
� Hip : Gluteus medius
� Knee : semitendinosus,
vastus lateralis, rectus femoris
� Ankle : Tibialis anterior
73
FACTORS INFLUENCING GAIT
� Age
� Gender
� Assistive devices
� abnormalities
74
FACTORS INFLUENCING GAIT
Age� A toddler has a higher COG, wider BOS, decreased single leg support time, a shorter step length , a slower velocity and a higher cadence in comparison to adult
� 3-5 year old showed increase in stride length adjusted to leg length, step length and a faster speed in gait
� From 6-13 years ROM of L/E were almost identical to adults. However linear displacement, velocities and accelerations are larger.
� Elderly demonstrate a decrease in natural walking speed, shorter stride and step length, longer duration of double support periods and smaller swing to support phase ratios
75
FACTORS INFLUENCING GAIT
Gender
Men Women
�Joint angle increases as speed
increases
�Gait speed faster i.e.. 118-134
cm/s
�Step length larger
� Not much joint angle increase
as compared to men
�Gait speed slower i.e.. 110-129
cm/s
�Step length smaller
�Increased hip flexion and
decreased knee extension during
gait initiation
�Increased knee flexion in pre-
swing
�Increased stride length
�Greater cadence 76
FACTORS INFLUENCING GAIT
Assistive devices
� Canes are typically been used on the contralateral side to an affected limb to reduce forces acting at the affected limb
� Use of cane on the contralateral side increase the BOS and decrease muscle, GRF forces acting at the affected hip and hip abductor & gluteus maximus activity was reduced to 45%
� Walker gait
77
COMMON GAIT ABNORMALITIES
A. Deformity(Contracture)
B. Muscle Weakness
C. Sensory Loss
D. Pain
E. Impaired Motor Control(Spasticity)
78
Ankle and Foot Gait Deviation
79
Knee Abnormal Gait
80
Knee Abnormal Gait
81
Knee Abnormal Gait
82
Knee Abnormal Gait
83
Hip Abnormal Gait
84
Hip Abnormal Gait
85
Pelvis and Trunk Pathological Gait
86
Pelvis and Trunk Pathological Gait
87
Pelvis and Trunk Pathological Gait
88
Pelvis and Trunk Pathological Gait
89
COMMON TYPES OF ABNORMAL GAITS
� Scissor gait
� Antalgic gait
� Cerebellar ataxia
� Festinating gait
� Pigeon gait
� Propulsive gait
� Steppage gait
� Stomping gait
� Spastic gait
� Myopathic gait
� Magnetic gait
� Trendelenburg gait
90
Scissor gait
� Hypertonia in the legs, hips and pelvis means these areas
become flexed, to various degrees, giving the appearance of
crouching, while tight adductors produce extreme adduction,
presented by knees and thighs hitting or crossing in a scissors-
like movement, while the opposing muscles, the abductors,
become comparatively weak from lack of use. Most common in
patients with spastic cerebral palsy,
usually diplegic and paraplegic varieties. The individual is forced
to walk on tiptoe unless the dorsiflexor muscles are released by
an orthaepedic surgical procedure.
91
Antalgic gait
� Person tries to avoid pain associated with the ambulation. Often quick, short and soft foot steps.
92
Ataxic gait
� Spinal - proprioceptive pathways of the spine or brainstem are interrupted. There is loss of position and motion sense. The person will walk with a wide base of gait with foot slap at heel contact. Often watch feet as they walk.
� Cerebellar - coordinating functions of the cerebella are interfered with, so the person tends to walk with a wide base of gait with an unsteady irregular gait, even if watching feet.
93
Festinating gait
� The patient has difficulty starting, but also has difficulty stopping after starting. This is due to muscle hypertonicity. The patient moves with short, jerky steps.
94
Pigeon gait
� In-toe gait is a very common problem among children and even adults. Fortunately, most in-toeing that is seen in children is a growth and developmental condition and will correct itself without medical or surgical intervention.
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Propulsive gait
� Disturbance of gait typical of Parkinsonism in which, during walking, steps become faster and faster with progressively shorter steps that pass from a walking to a running pace and may precipitate falling forward.
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Steppage gait
� A manner of walking in which the advancing foot is lifted high so that the toes clear the ground. Steppage gait is a sign of foot-drop.
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Stomping gait
� Sensory ataxia presents with an unsteady "stomping" gait with heavy heel strikes, as well as postural instability that is characteristically worsened when the lack of proprioceptive input cannot be compensated by visual input, such as in poorly lit environments.
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Spastic gait
� An unbalanced muscle action of certain muscle groups leads to deformity. Prime example is "Scissor gait" - adduction and internal rotation of the hips with an equinus of the feet and flexion of the knee.
� the legs are held together and move in a stiff manner, the toes seeming to drag and catch.
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Myopathic gait
� The "waddling" is due to the weakness of the proximal muscles of the pelvic girdle.
� The patient uses circumduction to compensate for gluteal weakness.
� exaggerated alternation of lateral trunk movements with an exaggerated elevation of the hip.
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Magnetic gait
� Normal pressure hydrocephalus (NPH) gait disturbance is often characterized as a "magnetic gait," in which feet appear to be stuck to the walking surface until wrested upward and forward at each step. The gait may mimic a Parkinsonian gait, with short shuffling steps and stooped, forward-leaning posture, but there is no rigidity or tremor. A broad-based gait may be employed by the patient in order to compensate for the ataxia.
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Trendelenburg gait
� The Trendelenburg gait is an abnormal gait caused by
weakness of the abductor muscles of the lower
limb, gluteus medius and gluteus minimus.
� During the stance phase, the weakened abductor
muscles allow the pelvis to tilt down on the opposite
side. To compensate, the trunk lurches to the weakened
side to attempt to maintain a level pelvis throughout the
gait cycle. The pelvis sags on the opposite side of the
lesioned superior gluteal nerve.
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Hemiplegic Gait Demonstration
� The patient has unilateral
weakness and spasticity with
the upper extremity held in
flexion and the lower
extremity in extension. The
foot is in extension so the leg
is "too long" therefore, the
patient will have to
circumduct or swing the leg
around to step forward. This
type of gait is seen with a
UMN lesion.
� This girl has a right
hemiparesis. Note how she
holds her right upper
extremity flexed at the elbow
and the hand with the thumb
tucked under the closed
fingers (this is "cortical
fisting"). There is
circumduction of the right
lower extremity.
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Diplegic Gait Demonstration
� The patient has spasticity in the lower extremities greater than the upper extremities. The hips and knees are flexed and adducted with the ankles extended and internally rotated. When the patient walks both lower extremities are circumducted and the upper extremities are held in a mid or low guard position. This type of gait is usually seen with bilateral periventricular lesions. The legs are more affected than the arms because the corticospinal tract axons that are going to the legs are closest to the ventricles.
� This man has an UMN lesion affecting both lower extremities. He has spasticity and weakness of the legs and uses a walker to steady himself. There is bilateral circumduction of the lower extremities.
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Neuropathic Gait Demonstration
� This type of gait is most often seen in peripheral nerve disease where the distal lower extremity is most affected. Because the foot dorsiflexors are weak, the patient has a high stepping gait in an attempt to avoid dragging the toe on the ground.
� This girl has weakness of the distal right lower extremity so she can't dorsiflex her foot. In order to walk she has to lift her right leg higher then the left to clear the foot and avoid dragging her toes on the ground.
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Myopathic Gait Demonstration
� With muscular diseases, the proximal pelvic girdle muscles are usually the most weak. Because of this the patient will not be able to stabilize the pelvis as they lift their leg to step forward, so the pelvis will tilt toward the non-weight bearing leg which results in a waddle type of gait.
� This young boy has pelvic girdle weakness, which produces a waddling type of gait. Note the lumbar hyperlordosis with the shoulders thrust backwards and the abdomen being protuberant. This posture places the center of gravity behind the hips so the patient doesn't fall forward because of weak back and hip extensors.
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Parkinsonian Gait Demonstration
� This type of gait is seen
with rigidity and
hypokinesia from basal
ganglia disease. The
patient's posture is
stooped forward. Gait
initiation is slow and
steps are small and
shuffling; turning is en
bloc like a statue.
� This man's gait is
bradykinetic and his
steps are smaller then
usual. There is also the
pill-rolling tremor in his
hands. He turns en bloc
and there is decreased
facial expression
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Choreiform Gait Demonstration
� This is a hyperkinetic gait
seen with certain types of
basal ganglia disorders. There
is intrusion of irregular, jerky,
involuntary movements in
both the upper and lower
extremities.
� Note the involuntary,
irregular, jerky movements of
this woman's body and
extremities, especially on the
right side. There are also
choreiform movements of
the face. A lot of her
movements have a writhing,
snake-like quality to them,
which could be called
choreoathetoisis.
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Ataxic Gait Demonstration
� The patient's gait is wide-
based with truncal instability
and irregular lurching steps
which results in lateral
veering and if severe, falling.
This type of gait is seen in
midline cerebellar disease. It
can also be seen with severe
lose of proprioception
(sensory ataxia)
� This woman's gait is wide-
based and unsteady. She has
to use a walker or hold on to
someone in order to maintain
her balance (note how hard
she has to work with the
hand that she's holding on
with in order to maintain her
balance). Her ataxia is even
more apparent when she tries
to turn.
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