Emphysema

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Chronic Obstructive Pulmonary Diseases (COPD) Chronic Airflow Limitation (CAL)

description

Chronic Obstructive Pulmonary Diseases (COPD) Chronic Airflow Limitation (CAL). Emphysema. Loss of lung elasticity Hyperinflation of the lung Formation of Bullae Small airway collapse and air trapping. Classifying Emphysema. - PowerPoint PPT Presentation

Transcript of Emphysema

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Chronic Obstructive Pulmonary Diseases (COPD) Chronic Airflow Limitation

(CAL)

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Emphysema

• Loss of lung elasticity• Hyperinflation of the lung• Formation of Bullae• Small airway collapse and air

trapping

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Classifying Emphysema

• Panlobular

• Centrolobular

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Clinical Manifestations

• Progressive dyspnea on exertion• Prolonged expiratory phase &

tachypnea• Increased work of breathing• Anorexia, weight loss• Barrel Chest• Flattened diaphragm

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Clinical Manifestations (cont)

• Formation of blebs and bullae• Hyperresonance• Polycythemia (pink puffer)• Chronic hypoxia• Pneumothorax• Hypercapnic to hypoxic drive• Chronic respiratory acidosis (end

stage)

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Pulmonary Function Tests

• Increased– residual volume– total lung capacity

• Decreased– Forced vital capacity

– FEV1

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Clubbing of Fingers

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Chronic Bronchitis

• Excessive production of mucus in the bronchi

• Productive cough– Persists 3 months of the year for 2

consecutive years

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Pathologic Changes

• Chronic inflammation• Hypertrophy and hyperplasia of

the mucus glands• Increased susceptibility to infection• V/Q changes

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Presentation

• May have same symptoms as emphysema

• Frequent respiratory infections• Cyanosis• Cor pulmonale• Polycythemia

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Asthma

• Not always listed as one of the diseases of COPD/CAL

• Asthma is usually a reversible process

• Involves periodic episodes

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Asthma Classifications

• Extrinsic (Allergic)– Antigen/antibody response– Childhood

• Intrinsic (Endogenous)– History recurrent RTI– adulthood

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Pathologic Changes

• Hypersensitivity response• Bronchoconstriction• May become chronic with

irreversible changes

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Presentation

• Bronchospasm• Increased mucus secretion• Dyspnea• Wheezing• Cough

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Consequences of CAL

• ABG’s– Initially normal ABG followed by

decreased PaO2 and O2 saturation

– Increased PaCO2 with an increase in HCO3 to compensate

• Compensated Respiratory Acidosis and Hypoxemia

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Polycythemia

• Related to decreased PaO2• What is the mechanism?

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Pulmonary Function Tests

• What do you expect?

TLC increasedFEV1 decreased

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Cor Pulmonale

COPD/CAL

Pulmonary Vascular Bed

Pulm Hypertension Hypoxemia

RV Failure PolycythemiaLV Failure

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Collaborative Management of CAL

• Medical management– Maximize oxygenation, ventilation

and perfusion

• Surgical management– Bullectomy– Lung volume reduction surgery

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Drug Therapy• Bronchodilators

– Sympathomimetics– Methylxanthines

• Anticholinergics• Steroids• Mast Cell Stabilizers• Leukotriene Antagonists• Expectorants• Antibiotics

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Nursing Diagnoses

• Impaired gas exchange• Ineffective airway clearance• Activity intolerance• Anxiety• Altered nutrition: less than body

requirements

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Nursing Interventions

• Maintain a patent airway• Safely administer oxygen• Use oxygen delivery systems

appropriately• Accurately assess the patient’s

breathing• Use positioning to improve

oxygenation

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Teach the Patient:

• Abdominal & Pursed lip breathing• Controlled coughing• Conservation of energy• Prevent secondary infection• Insure hydration• Nutrition• Therapeutic

communication/relaxation

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Metered Dose Inhaler (MDI)

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Peak Flow Meter