DYNAMIC AUSCULTATION
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Transcript of DYNAMIC AUSCULTATION
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DYNAMIC AUSCULTATION
DR RAJESH K F
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This is a technique of altering circulatory dynamics by means of a variety of physiological and pharmacological maneuvers and determining their effects on heart sounds and murmurs
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Interventions most commonly employed are Respiration Postural changes Isometric exercise Valsalva maneuver Premature ventricular contractions Vasoactive agents-
amyl nitrite ,methoxamine ,phenylephrine
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Splitting of S2
RESPIRATION
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Heart sounds
Accentuated duringInspiration
RVS3 and RVS4 Tricuspid OS
Expiration
LVS3 and LVS4 mitral OS
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Pulmonary ejection click Inspiration diminish intensity of valvular PEC PA diastolic pressure is very low Inspiration causes elevation of RV EDP RV late diastolic Pr > PA Pressure Causes partial presystolic opening of PV Less upward motion of valve during systole
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MURMURS Respiration exerts more pronounced and
consistent alterations on murmurs of right side than left side
Especially tricuspid murmurs 100% sensitivity, 88% specificity
Inspiration increases venous return to right side of heart
Expiration increases venous return to left side of heart
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Inspiration TS TR (Carvallo’s sign) PR Mild or moderate PS Severe PS no
further increase in gradient
Murmurs accentuated during
Expiration MS MR AS AR VSD Pericardial rub (AP
diameter)
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MVP MSC and systolic murmur occur earlier
during systole in inspiration Inspiratory reduction in LV size Increased redundancy of MV Increase valvular prolapse
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Effects of inspiration on auscultatory findings may be accentuated by Muller maneuver
Converse of Valsalva Maneuver Forced inspiration against closed glottis Forcibly inspires while the nose is held
closed and mouth is firmly sealed for about 10 sec.
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Widens split S2 and augments murmur and filling sound originating in right side of the heart.
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RAPID STANDING Decrease in venous return, thus stroke
volume
POSTURAL CHANGE
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Width of the splitting become reduced No change in patients with true fixed splitDecrease in intensity RVS3 and RVS4 LVS3 and LVS4
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Decrease in intensity
Semilunar valve stenosis AV valve regurgitation murmurs VSD Most functional systolic murmurs
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Since LV EDV is decreased
Increase in murmurs HOCM(95% sensitivity,
84% specificity) Early MSC and
murmur of MVP
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SQUATTING Sudden change from standing to squatting
position Increase venous return and systemic
resistance simultaneously Squatting abruptly increases ventricular
preload and afterload Arterial pressure rise may cause transient
reflex bradycardia
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Increase in stroke volume causes augmentation of S3 and S4(of both ventricles) Right sided murmurs MS AS
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Elevation of arterial pressure
Increase in aortic reflux AR Increase in MR volume Increase in LT to RT shunt in VSD Increase in blood flow through RVOT in TOF
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Combination of elevated arterial pressure and venous return Increase LV size and reduce LVOT
obstruction Decrease murmur in HOCM(95% sensitivity,
85% specificity) Click and murmur of MVP delayed
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LEFT LATERAL RECUMBENT POSITIONAccentuate intensity of S1 LVS3 and LVS4 OS of MS Murmurs of MS and MR Click and murmur of MVP Austin Flint murmur
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SITTING AND LEANING FORWARD Accentuate AR and PR murmur (mechanical)
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This can be carried out by using a calibrated handgrip device or a handball
Better to carryout bilaterally Should be sustained for 20 to 30 secs Valsalva maneuver during the handgrip
must be avoided Contraindicated in patients with myocardial
ischemia and ventricular arrhythmias
ISOMETRIC EXERCISE
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Isometric exercise results in significant increase in Systemic vascular resistance Arterial pressure Heart rate COP LV filling pressure Heart size
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Systolic murmur of AS diminished –reduction of pressure gradient across AV
Diastolic murmur of AR and systolic murmurs of rheumatic MR and VSD increases
LVS3 and LVS4 accentuated Diastolic murmur MS becomes louder –
increase in flow across valve
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Increase LV volume Systolic murmur of HOCM decreased Click and murmur of MVP delayed
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Forced expiration against a closed glottisStandard test consists of asking the patient to blow against an aneroid manometer and maintain a pressure of 40mmhg for 30seconds
VALSALVA MANEUVER
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Relatively deep inspiration followed by forced exhalation against a closed glottis for 10 to 20 seconds
Physician has to keep flat of the hand on the abdomen to provide the patient a force to breathe against
Normal response has four phases
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PHASE1 Intrathoracic pressure rises Transient increase in LV output and SBP
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PHASE II STRAINING PHASE Systemic venous return decrease Filling of right and then left side reduced Stroke volume reduced Mean arterial and pulse pressures falls Reflex tachycardia
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A2-P2 interval narrowsAttenuation of S3 and S4 AS & PS MR & TR AR & PR TS & MS
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Since LV volume is reduced Murmur of HOCM increased(65% sensitivity,
95% specificity) Systolic click and murmur of MVP
commence earlier
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PHASEIII VALSALVA RELEASE During first two cycles following release
murmurs and sounds(S3 and S4) right side of heart return to normal
After six to eight cycles sounds and murmurs originating from left side of heart returns to normal
A2-P2 split increases Decrease SBP
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PHASE IV OVERSHOOT PHASE Murmurs and heart sounds transiently
augmented
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Followed by a significant pause
Increase in ventricular filling Augmentation of cardiac contractility- post
extra systolic potentiation
POSTPREMATURE VENTRICULAR CONTRACTIONS
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During postpremature beat – augmented are
ESM of AS and PS ^volume ^contractility HOCM ^contractility-increase dynamic
LVOT obstruction ^volume-decrease LVOT obstruction net increase gradient
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PSM of MR and of VSD - not altered(relatively little further increase in mitral valve flow or change in the LV-LA gradient) (ventricle has has 2 openings aorta and LA in MR not in AS)
Systolic murmur of papillary muscle dysfunction diminish
Increase in LV size delays systolic click and murmur of MVP (depend mainly on volume)
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Similar auscultatory changes follow prolonged diastolic pauses in AF
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AMYL NITRITE INHALATION
Crush ampoule in towel take 3-4 deep breaths over 10 – 15 secs
First 30 secs– Systemic art pressure decrease
30 to 60 secs– Reflex Tachycardia > 60 secs -CO,HR and Velocity of BF increase
PHARMACOLOGICAL AGENTS
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S1 augmented A2 diminished OS mitral and tricuspid valve become louder A2 OS interval shortens RVS3 and LVS3 augmented –rapidity of
ventricular filling LVS3 associated with MR diminished(MR
reduced)
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Systolic murmurs accentuated are HOCM AS PS TR Functional systolic murmurs
Increased ventricular contractility and SV
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Due fall in systemic arterial pressure murmurs diminished are PSM of MR PSM of VSD EDM of AR Austin flint murmur Continuous murmur of PDA Continuous murmur of AVF
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Systolic ejection murmur of TOF diminished Decrease in arterial pressure Increase right to left shunt Decrease blood flow in RVOT
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Reduction cardiac size leads to Early appearance of click and murmur of
MVP Murmur intensity show variable response
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Amyl nitrate response useful in distinguishing
Systolic murmur of AS(^)and MR(v) Systolic murmur of TR(^) and MR(v) Systolic murmur of PS(^) and TOF(v) Systolic murmur of PS(^) and VSD(v) Diastolic murmur of MS(^) and Austin
flint(v) EDM of PR(^) and AR(v)
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METHOXAMINE AND PHENYL EPHRINE Increase systemic arterial pressure Reflex bradycardia and decreased
contractility and COP Contraindicated in CHF and HTN
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Methoxamine 3-5 mg IV increase arterial pressure by 20-40 mm Hg for 10 to 20 min
Phenylephrine 0.5mg IV elevates systolic pressure around 30mm Hg for 3-5min
Phenylephrine preferred due to shorter duration action
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S1 reduced A2 becomes louder A2 OS prolonged S3 and S4 response variable
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Increase in arterial pressures cause following murmurs louder EDM of AR PSM of MR VSD TOF Continuous murmurs of PDA and AVF
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Systolic murmur of HOCM softens(^ LV size) Click and murmur of MVP delayed(^ LV size)Decrease in COP diminish ESM of AS Functional systolic murmurs MDM of MS
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TRANSIENT ARTERIAL OCCLUSION Transient external compression of both
brachial arteries By bilateral cuff inflation to 20 mm Hg
greater than peak systolic pressure Augments the murmurs of MR, VSD, and AR
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Inspiration, Sudden standing Dec pulmonary venous return, Reduces LAP MDM reduced OS softens A2-OS gap widen Three sequential sounds (A2, P2, and OS)
may be audible
Exercise ,Squatting ,Amyl Nitrate MDM accentuated
MS
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Varies little with respiration
Decrease murmur Sudden standing Valsalva Amyl Nitrate
Augments the murmur Squatting Isometric Exercise
MR
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Murmur increases on Post PVC beat squatting
Reduces AS murmur Valsalva Standing handgrip
AS
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EDM increases on sitting up and leaning forward Squatting Isometric exercise Vasopressors
Decreases with Amyl Nitrate Valsalva
AR
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Murmur and click earlier(intensity decreases)LV Volume decrease Standing Valsalva Murmur and click laterLV Volume increase Squatting Post ectopic Isometric Exercise (intensity increases)
MVP
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Increase murmur in Valsalva Standing Post ectopic
Decrease murmur in Sustained Handgrip squatting Methoxamine
HOCM
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AS X HOCM squatting (^/v) valsalva/standing (v/^)
AS x MR handgrip (v/^) phenyl ephrine (v/^) post pvc (^/v) amyl nitrate (^/v)
Dynamic auscultation helpful in
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MS X TS respiration MR X TR respiration MS X AUSTIN FLINT amyl nitrate(^/v) PS X AS respiration PS X Small VSD amyl nitrate (^/v) phynylephrine (v/^) respiration PR X AR squatting (_/^) sus handgrip (-/^)
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THANK U
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MCQS
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1 . During phase 2 of valsalva A2-P2 intervalA. IncreaseB. DecreaseC. No changeD. Any of the above
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2 .Intensity of murmur in MVP during isometric handgripA. IncreaseB. DecreaseC. No changeD. Increase then decrease
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3. Rheumatic MR murmur increase with all the following exceptA. Sudden squattingB. Isometric handgripC. Phenyl ephrine D. Amyl nitrate
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4. After amyl nitrate systolic murmur of VSD A. IncreaseB. DecreaseC. No changeD. Any of the above
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5 .After squatting AS murmurA. IncreaseB. DecreaseC. No changeD. Increase then decrease
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6. PS murmur following handgripA. IncreaseB. DecreaseC. No changeD. Increase then decrease
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7 .HOCM murmur increase following post ectopic beat due toA. Increase LV volume B. ContractilityC. Decrease LV volumeD. Decrease gradient
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8. A2 OS gap during standingA. IncreaseB. DecreaseC. No changeD. Increase then decrease
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9 .Amyl nitrate is A. liquid silverB. Venodilator C. Arterial dilatorD. both
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10. MDM of MS increase inA. Left lateral positionB. CoughingC. HandgripD. All the above
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1 . During phase 2 of valsalva A2-P2 intervalA. IncreaseB. DecreaseC. No changeD. Any of the above
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2 .Intensity of murmur in MVP during isometric handgripA. IncreaseB. DecreaseC. No changeD. Increase then decrease
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3. Rheumatic MR murmur increase with all the following exceptA. Sudden squattingB. Isometric handgripC. Phenyl ephrine D. Amyl nitrate
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4. After amyl nitrate systolic murmur of VSD A. IncreaseB. DecreaseC. No changeD. Any of the above
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5 .After squatting AS murmurA. IncreaseB. DecreaseC. No changeD. Increase then decrease
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6. PS murmur following amyl nitrateA. IncreaseB. DecreaseC. No changeD. Increase then decrease
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7 .HOCM murmur increase following post ectopic beat due toA. Increase LV volume B. ContractilityC. Decrease LV volumeD. Decrease gradient
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8. A2 OS gap during standingA. IncreaseB. DecreaseC. No changeD. Increase then decrease
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9 .Amyl nitrate is A. liquid silverB. Venodilator C. Arterial dilatorD. both
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10. MDM of MS increase inA. Left lateral positionB. CoughingC. HandgripD. All the above