DR.HINA ADNAN. 1. Abscesses of periodontium. 2. Necrotizing periodontal diseases. 3. Gingival...
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Transcript of DR.HINA ADNAN. 1. Abscesses of periodontium. 2. Necrotizing periodontal diseases. 3. Gingival...
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Acute periodontal conditions
DR.HINA ADNAN
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1. Abscesses of periodontium.2. Necrotizing periodontal diseases.3. Gingival disease of viral origin – herpes
virus.4. Recurrent aphthous stomatitis.5. Allergic reaction.
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Abscesses of periodontium1. Gingival abscess.2. Periodontal abscess.3. Pericoronal abscess.
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Periodontal Vs periapical abscessperiodontal periapical
• Vital tooth.• No Carious.• Pocket .• lateral radioluicency.• Mobility.• Percussion sensitivity
variable.• Sinus tract open via
keratinized gingiva.
• Non vital.• Carious.• No pocket.• Apical radioluicency.• No or minimal mobility.• Percussion sensitivity.• Sinus tract via alveolar
mucosa.
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Necrotizing periodontal diseases
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Gingival disease of viral origin – herpes virusAcute manifestation of viral infections of
oral mucosa characterized by redness and multiple vesicles that easy rupture to form painful ulcers affecting the gingiva.
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Primary herpetic gingivostomatitisClassic initial infection of herpes simples
virus type 1.Mainly in young children.90% of primary oral infection are
asymptomatic.
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Clinical features1. Painful severe gingivitis with ulcerations ,
edema and stomatitis.2. Vesicles rupture coalesce and form ulcers.3. Fever and Lymphadenopathy are classic
features.4. lesion usually resolve in 7-14 days.
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Treatment 1. Bed rest.2. Fluid.3. Nutrition.4. Antipyretics. 5. Pain relieve .6. Antiviral medications.
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Recurrent oral herpes ‘Fever blisters’ or ‘cold sores’.Oral lesion usually herpes simples type 1.Recurrent infections in 20%-40% of those
with primary infection. herpes labials are common.Less severe than primary infection.
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Clinical features 1. Prodromal syndrome.2. lesion start as vesicles and then rupture
leave ulcers. 3. A cluster of small painful ulcers on attached
gingiva or lip is characteristic.4. Can cause post operative pain following
dental treatment.
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Virus reactivation1. Fever .2. systemic infection.3. Ultraviolet reaction.4. Stress.5. Immune system changes.6. Trauma .7. Undefined cases.
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Treatment 1. Palliative care .2. Antiviral medication.
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Recurrent aphthous stomatitis Canker sores.Etiology unknown.10%-20% of population.Usually begins in childhood.Decreasing with age.
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Clinical features 1. Affect mobile mucosa.2. Three forms:I. Minor.II. Major.III. Herpitiform.
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Minor aphthousClinical features: 1. most common.2. Small shallow ulceration with slightly
raised erythematous borders.3. Central are covers by white-yellow
pseudomembrane. 4. Heal without scarring in 10-14 days.
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Major aphthous Clinical features:1. Herpetiform aphthae : small recurrent
ulcerations Lesion usually small than herpetic stomatitis.
2. Similar to herpetic stomatitis but no vesicles.
3. Heal with 7-10 days without scaring.
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Predisposing factors 1. Stress.2. Trauma.3. Food hypersensitivity.4. Previous viral infection.5. Nutritional deficiency.
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Allergic reactionDental restorative materials.( mercury,
nickel, gold , zinc).Toothpaste and mouthwashes.Foods ( peanut, red peppers )