Dr. Hardi-critical Care in Burn Patient
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Transcript of Dr. Hardi-critical Care in Burn Patient
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CRITICAL CARE CRITICAL CARE IN BURN PATIENTIN BURN PATIENTCRITICAL CARE CRITICAL CARE
IN BURN PATIENTIN BURN PATIENT
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Bali, Bali, Oktober 2002Oktober 2002
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Mariott Hotel, JakartaMariott Hotel, JakartaAgustus 2003Agustus 2003
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Kuningan, Jakarta, Kuningan, Jakarta, September 2004September 2004
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CRITICAL CARE IN BURN PATIENT
BURN : • DEF/ :
INJURY OR DAMAGE OF TISSUE (SKIN AND TISSUE DEEPER THAN SKIN) CAUSE BY HEAT.
• ET/ :
FIRE,SCALD,CHEMIST,ELECTRIC,RADIATION
• MORBIDITY & MORTALITY SINCE INITIAL PHASE.
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PROBLEMS IN BURN • INITIAL PHASE/SHOCK PHASE/ACUTE: DISTURBANCES OF FLUID & ELECTROLITE
• SUB ACUTE PHASE (DAY 2 – DAY 21) - HYPERMETABOLISM - HIGH REACTION INFLAMATION
- HEALING PROCESS - INFECTION
- SIRS- MODS
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• LATE PHASE : HYPERTHROPIC SCAR,KELOID & CONTRACTURE PROBLEMS
CLASSIFICATION BASE ON DAMAGE THE TISSUE
GRADE I
GRADE II DEPEND ON AETIOLGY & CONTACT
GRADE III
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SKIN ANATOMY
SKIN ANATOMY
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ZONES IN BURN WOUND
1. COAGULATION ZONE :
PROTEIN COAGULATION (DIRECT)
2. STATIC ZONE :
DAMAGE OF ENDOTHEL,ERY,LEUCO,THROMBOCYT
CAPILER PERMEABILITY DISTURBANCE
3. HYPEREMIA ZONE:
VASODILATATION WITHOUT CELLULAR REACTION
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BURN WOUND ZONE
Zone 1
Zone 2
Zone 3
1
2
3
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CLASSIFICATION OF SEVERITY BURN
SEVERE BURN DEGREE II > 25 % TBSA DEGREE III > 10 % DEGREE III OR II CIRCUMFERENTIAL AT FACE, HAND, LEG. INHALATION INJURY
BURN WITH ASSOCIATED INJURYELECTRICAL BURN
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MODERATE BURN
DEGREE II 15 –25 %
DEGREE III < 10 % MINOR BURN
DEGREE II < 15 %
DEGREE III < 2 % ALL OF THE FIRST DEGREE
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PATOPHYSIOLOGIPATOPHYSIOLOGI
Release MediatorRelease Mediator InflamationInflamation
Increase MicrovascularIncrease MicrovascularPermeabilityPermeability
Decrease Myocardial Decrease Myocardial ContractilityContractility
Early ExcisionEarly ExcisionThermal InjuryThermal Injury
Capillary LeakCapillary Leak
Massive ExtravasasionMassive Extravasasion
hipovolemiahipovolemia edemaedema
Cellular perfusion Cellular perfusion disorderdisorder
Macro circullatoryMacro circullatorydisorderdisorder
acidosisacidosis
ischemiaischemia
necrosisnecrosis
hipoperfussionhipoperfussion
RenalIschemiaRenalIschemia
Cardiovascular collapsCardiovascular collaps
Fluid Fluid ressusciationressusciation
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PATOPHYSIOLOGICAL CHANGES
HYPERMETABOLISM
HORMONAL CHANGES : CORTISOL,INSULIN
IMMUNOLOGICAL IMPAIRMENT
INCREASED INTESTINAL PERMEABILITY
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Organ Injury
Inflamation“Fighting”
Pro Inflamation
Healing
Anti Inflamation(compentation)
ImmunosupressionSIRSSIRS
time
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HOW TO MANAGE THE BURN PATIENT
PREHOSPITAL
- PUT PATIENTS FAR TO SOURCE OF THE HEAT
- REMOVE ALL OF THE CLOTHES & ACCESORIES
- EARLY COOLING WITH WATER, NO ICE
- BURN MORE THAN MODERATE IN SEVERELY
SEND TO THE HOSPITAL
20 % TBSA IV LINE IF POSSIBLE
- THE PATIENT WITH CLEAN CLOTHES COVER
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PRIMARY ASSESMENTIDENTIFY IMMEDIATE LIFE THREATENING CONDITION
• AIRWAY : - OXYGEN
- PROGRESSIVE OBSERVATION
• BREATHING : - EXPANSION OF THE CHEST
- CIRCUMFERENTIAL BURN IN
CHEST ESCHAROTOMY
• CIRCULATION :
- PULSE AND BLOOD PRESSURE
• C – SPINE IMMOBILITATION
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CHEMISTRY BURN
• WATER IRRIGATION, DON’T NETRALISIR
ELECTRIC BURN• OBSERVATION VENTRICLE VIBRILATION
SECONDARY ASSESMENT
• WOUND EVALUATION
• FLUID RESUSCITATION
• CLOSE MONITORING
• GIT DECOMPRESSION : NGT
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WOUND EVALUATION• WOUND TOILET ASSESS DEPTH & EXTENT OF BURN• EXTREMITY
CHECK PULSATION ESCHAROTOMY• DEBRIDEMENT GENERAL ANESTHESIA
FLUID RESUSCITATION
• FORMULA DOESN’T ABSOLUTE.
• CLINICAL MONITORING FROM TIME TO TIME
• BAXTER : 4 ml/Kg BB/ % TBSA
• DIURESIS MONITORING
• BURN 40 % CVP
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NUTRITION
• 8 HOUR’S ENTERAL FEEDING NURITION : IF THE GUT WORK USE IT
• PARENTERAL NUTRITION (PN) ONLY IF THERE ISCONTRAINDICATION OR INTOLERANCE TO EN
• HIGH CALORY HIGH PROTEIN
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Normal Energy Intake
Energy needed in a normal activity
LimitedEnergy intake
Energy needed in a normal activity
Energy needed in a inflamation conditions
Energy needed in a regenera
tion process
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IMMUNONUTRITION• IS SUBSTANCES THAT ENHANCE THE IMMUNE SYSTEM EFFECTS :
- FEWER INFECTIONS COMPLICATION- SHORTER TIME IN VENTILATOR- LOW OF STAYING HOSPITAL
MOST IMPORTANT
• ARGININE, GLUTAMINE,NUCLEOTIDE
& OMEGA 3
• RECOMMENDED 7 DAYS, MINIMUM 3 DAYS
ADMINISTRATION
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HOW TO MANAGE BURN DISSASTER
• NO DIFFERENT IN SUPPORT & LIFE THREATENING• NEED MANY PROFESIONAL’S AND
HIGH SKILL IN BURN MANAGEMENT• NEED SPESIFIC SUPPORT FOR :
- ACCOMODATION EVACUATION - TECHNIQUE TO ATTACT THE FIRE - ABLS (ADVANCE BURN LIFE SUPPORT)
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THANK YOUTHANK YOU
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