Digestive Tract: Let’s Get to the Bottom of it By: Diana Blum RN MSN Metropolitan Community...
-
Upload
kurt-verry -
Category
Documents
-
view
213 -
download
0
Transcript of Digestive Tract: Let’s Get to the Bottom of it By: Diana Blum RN MSN Metropolitan Community...
Digestive Tract: Let’s Get to the Bottom of it
By: Diana Blum RN MSNMetropolitan Community College
Primary Role
Extract molecules essential for cellular function from fluids and food.
Ingestion, Digestion, Absorption, Elimination
Digestion: breakdown of food into simple nutrient molecules that can be used by cells Process requires:
1. 2. 3.
http://health.discovery.com/centers/digestive/machine.html
Digestive tract
Also called ___________tract muscular tube about 30 ft long Main parts
Mouth Pharynx Esophagus Stomach Small intestine Large intestine Anus
Acessory Organs
Salivary glands Liver Gallbladder Pancreas
Each of the above accessory organs secrete fluid that contain special enzymes that enable breakdown (metabolism) of food
Peritoneum lines the abdominal cavity and covers surface of organs Enables organs to moves without friction during breathing
and digestion
mouth
Teeth cut and grind food Salivary glands secrete saliva
Saliva: Amylase:
Tongue mixes saliva with food and when small enough- forces the food into the pharynx
Pharynx
Shared by digestive and respiratory tracts Joins mouth and nasal passages Contains the epiglottis
Covers the airway (like a trap door) to prevent food from entering respiratory tract
esophagus
Long muscular tube that passes through the diaphragm into the stomach
Gravity helps move the food but it is not essential
Circular, wave like contractions of the muscles propel food down the tract (peristalsis)
Stomach
Widest section of the GI tract Separated from esophagus by the cardiac sphincter Has 3 sections Unique muscle layers churn food by mixing it with gastric
secretions Rennin-starts breakdown of milk proteins Pepsin-partially digests protein HCL acid-partially digests protein Lipase-breaks down fat
Chyme: Pyloric sphincter- keeps food in stomach until it is mixed
properly
Small Intestine
Chyme leaves stomach and enters here Chemical digestion and absorption of
nutrients take place 20 feet long 3 sections
Duodenum-liver and pancreatic enzymes enter here
Jejunum Ileum
Small Intestine Continued
Bile- produced in the liver and stored in the GB break down large fat globs
Pancreatic enzymes-reduce the fat to glycerol and fatty acids to be easily absorbed
3 layers of tissue make up the wall Mucous membrane-secretes digestive enzymes Sucrase, lactase, maltase, lipase, etc. (see table 36-1) Inner layer- covered with Villi (microscopic projections).
Digestive food molecules are absorbed through the villi into the bloodstream
Muscle layers continue to contract moving the chyme into the large intestine.
Large Intestine
No Villi No digestive enzymes Chyme enters through the ileocecal valve Water is absorbed and remaining waste=feces 5 sections
Cecum-1st section..appendix is here Ascending colon-up right abdomen Transverse colon- across abdomen just below waist Descending colon-down the left abdomen
Sigmoid colon-the part of the descending colon between iliac crest and rectum
Rectum-the last 6-8 inches of the large intestine Anus – where waste leaves the body
Age related changes
Teeth mechanically worn down Illness causes increased risk for problems with digestion/elimination Gingiva recedes Tooth loss from caries and periodontal disease Loss of taste buds Xerostomia (dry mouth) is common Walls of esophagus and stomach are thinner with lessened secretions HCL Acid and digestive enzyme production decreases Gastric motor activity slows Delayed gastric emptying Hunger contractions diminish In the large intestine- muscle layer and mucosa atrophy Smooth muscle tone and blood flow decreases Connective tissue increases Constipation is frequent More laxative use
Nursing Assessment
Hx of illness: weight loss, indigestion, change in bowel habit PMH: surgery, trauma, infection, burns, hepatitis, ulcers,
cancer, stomas, meds, allergies Fam Hx: diabetes, CA, ETOH, polyps, obesity, ulcers, GB
Dx System Review: flatus, dyspepsia (indigestion), skin
changes, caries, diff chewing, abd distention, pain, elimination
Functional: nutrition, activity, meal times, likes/dislikes, food beliefs
Physical exam: mucous membranes, condition of mouth/teeth, abd distention, bowel tones, palpation, percussion, rectum/anus for lesions, color, hemorrhoids
diagnostics
Imaging/radiographs: NPO, allergy (iodine, dye, shellfish), consent UGI Barium swallow/enema Endoscope
Upper Lower
Hemmocult-looks for blood
NG
Salem Sump
Tube feedings
Assist pt into fowlers to reduce aspiration. Remains this way for 30 minutes after
Pt remains up at least 30 degree during continuous feeding Check placement for tube in stomach or duodenum prior to
use Air bolus and residual
Check to make sure you have the correct formula Stop feeding if nausea or pain Rinse tube with 30 cc fluid after each bolus Administration
Remove plunger Pinch tube while inserting syringe to avoid stomach content leak Hold barrel about 12 inches above stomach and allow gravity to
infuse Flush after bolus complete
GI decompression
Ng with suction removes fluid and gas
To use Attach to sxn as ordered
Generally low, intermittent is used for single tube Low continuous for dual lumen tubes
Check patency Irrigate routinely Monitor output Assess for flatus Provide comfort measures Once tube in place- securely tape it to nose
feedings
TPN Deliver nutrients directly
into bloodstream via central line
Use sterile technique for dressings and care
Monitor flow rate Monitor blood glucose Label lines
PPN Same as TPN except
goes through peripheral line
Anorexia
Lack of appetite Causes
Nausea Physical/emotional disturbances Environment Decreased sense of smell
Tests: weight, physical, hemoglobin, iron, electrolytes, thyroid
Nursing diagnosis
Imbalanced nutrition less than requirements r/t anorexia
Goal: improved appetite and adequate food intake
AEB: increase in intake, stable or increased wt
Interventions: provide antiemetics prior to meals, remove the bed pan and emesis basin from sight, conceal drains and collection devices, deodorize room
clients with Feed problems
Paralyzed Confused Severe arthritis CVA Visually impaired Etc
FEEDER is demeaning and can threaten self esteem
Interventions for feed problem
Position properly Specially enhanced utensils Open sealed products Cut meats Butter bread Season food after asking client their
preferences See page 751
Role play
Practice feeding classmate a simple meal then reverse.
The person being fed can not speak but understands what is being said
1.How did it feel to be fed?2. What steps did you use?3. How did the feeder feed?4. What did you learn?
Stomatitis
Inflammation of the oral mucosa Mechanical trauma (poor fitting dentures) Irritation 2nd to smoke and ETOH Poor hygiene Radiation Drug therapy
Treatment: soft bland diet, antiviral agents, antibiotics
Vincent’s infection (aka Trench Mouth)
Vincent’s infection
Caused by bacteria Called trench mouth b/c occurred in WWI
field S/S: metallic taste foul breath. Bleeding
ulcers, increased saliva, general infection signs, anorexia
TX: topical antibiotics, mouthwash, rest, nutritious diet, good oral hygiene
Herpes Simplex
Herpes Simplex
Caused by Herpes simplex virus type 1 S/S: ulcers and vesicles in mouth and on lips Other name is cold sore or fever blister Common with people who have upper
respiratory infections, excessive sun exposure, or are stressed
TX: Camphor, topical steroids, antiviral agents
Aphthous Stomatitis (aka canker sore)
Caused by virus S/S: ulcer on lips or
mouth that recur at intervals
TX:topical or systemic steroids
Candidas AlbicansAKA yeast like fungus
Other names: thrush or candidiasis
S/S: bluish white lesions on mucous membrane of mouth
Those at risk: steroid users, long term antibiotic users
TX: oral medications, topical antifungal agents, vaginal nystatin tablets can be used like lozenges
Care and intervention
CARE Usually tx outpt Look at pt symptoms
Onset of symptoms, meds, radiation, habits, diet, ETOH use, and smoking
Describe pain (location, onset, precipitating factors)
INTERVENTION Gentle oral hygiene Prescribed mouthwash Use soft bristle tooth
brush Instruct to take meds as
prescribed (swish and spit, or swish and swallow)
Teach flossing techniques
Dental Caries
Destructive process of tooth decay
Caused by plaque Plaque is made from
bacteria, saliva, and cells that stick to tooth surface
In time if untreated the canal will erode causing intense pain and death of pulp
TX: fluoride, good nutrition
Gingivitis
Beginning of periodontal dx
Inflammation of the gums
s/s: red inflammed tissue of gums, pain, bleeds easily
More frequent in those with missing teeth or whose teeth don’t close properly, vitamin deficiency, anemia
Care and Intervention
CARE Assess pain and
soreness Assess diet and
examinations Examine mouth care
practices
INTERVENTION Minimize pain Gentle mouth care
several times a day Teach client proper
technique Page 752
Oral Cancer
Most life threatening condition of mouth
2 types: Squamous Basal cell
S/S: tongue irritation, loose teeth, tongue pain, ulcerations, leukoplakia (hard white spots), decreased appetite, diff swallowing, weight loss, change in denture fit, hemoptysis
TX: biopsy, surgery, radiation, chemo
Care and Intervention
CARE Assess sun exposure, smoking
habits, ETOH use, fam hx of oral ca,
Interventions Radiation=edema
Dry mouth is issue Good hygiene Special rinses see pg 753 Monitor respirations Suction if ordered Stay on top of pain Soft or liquid diet Monitor I/O Use communication board to talk
with pt BE PATIENT BE A GOOD LISTENER Monitor for infection If graft: monitor color and temp
Parotitis
Inflamed parotid glands S/S: painful swelling near
low jaw, pain increases with mastication
Suseptible: those unable to drink liquids, those weak, no resistance to infection
TX: antibiotics, mouthwash, warm compress
Complications: gland ruptures, surgical drainage or removal may be necessary
Achalasia
Progressive worsening dysphagia
Low esophageal muscles do not relax
Unknown cause TX:dilation, surgery,
botulism toxin, isosorbide dinitrate
Esphageal cancer
Not common Poor prognosis No known cause At risk: smokers, ETOH users,
chronic trauma, poor oral hygiene, spicy food eaters
S/S: progressive dysphagia, substernal pain, epigastric pain, neck/back pain,sore throats, choking, obstruction, weight loss
Esophageal treatment
Esophagectomy Esophagogastrostomy Esophagoenterostomy Dilitation of esophagus Stent Laser tx Chemo Radiation Photodynamic therapy
See page 756
Interventions
Treat pain Daily weight Strict I/O Calorie count Quiet relaxed environment Erect position Chin tuck maneuvers for swallowing Feeding tubes TPN If post op---do not irrigate or reposition Assess pt knowledge Monitor for infection Monitor respirations
N/V
Nausea: feeling of queasiness Pain, pallor, perspiration, cold, clammy skin Causes: irritating foods, infection, radiation, meds, inner ear disorders,
motion sick Vomiting: forceful expulsion of stomach content through the mouth Regurgitation: gentle ejection of fluid or food w/o nausea or retching
TACHYCARDIA AND INCREASED SALIVA are common before vomiting
Complications: loss of fluid and electrolytes, dehydration, metabolic alkalosis, weakness, aspiration
TX: antiemetics, iv fluids, NG tube Interventions: maintain cool room, remove unpleasant stimuli, place in
comfortable position, provide emesis basin, cool damp cloth on head/neck, slow deep breaths, offer mouth care after vomiting, clear liquids
Hiatal Hernia Protrusion of stomach and and lower esophagus up
thru the diaphragm and into chest 2 types:
Sliding: gastroesophageal junction is just above the hiatus. Stomach slides when patient reclines (associated with GERD)
Rolling: gastroesophageal junction remains in place but a portion of the stomach herniates up throu diaphragm through a 2nd ary opening
Complications: ulcerations, bleeding, aspiration Strangulated hernia is one that becomes trapped
without blood flow Causes: asymptomatic to fullness, dysphagia,
eructation (belching), regurgitation, heartburn TX: meds(antacids, H2 receptor blockers, etc), diet,
avoid intra abd pressure, surgery Interventions: stay on top of pain, no food or fluid 2-
3 hours before bed, wooden blocks under top of bed, monitor wt, small frequent meals, avoid fatty foods, caffeine, ETOH, and spicy foods
GERD
Back flow of gastric content from the stomach into the esophagus
Key find: inappropriate relaxation of the low esophagus sphincter
Causes: abnormalities in the LES, ulcers, esophageal surgery, prolonged vomiting, gastric intubation
S/S: can be sudden or gradual, painful burning that moves up and down (common after meals) resolve after antacids, dysphagia, belching
Diagnosis: Based on s/s, raqdiographic studies, endoscopy, bx
Tx: H2 receptor blockers (zantac), prokinetic agents (reglan), proton pump inhibitors (prilosec), surgery
Gastritis
Inflammation of the stomach lining
Mucosal barrier that normally protects stomach breaks down
H pylori is cause S/S: N/V, anorexia, fullness,
pain, hemorrhage Tx: npo until resolve, IVF, Bx,
medication,, bland diet, surgery
Peptic Ulcer
Loss of tissue from digestive lining Caused by pepsin and HCL injure
unprotected tissue LOCATION, LOCATION, LOCATION
Either gastric or duodenal Causes: drugs, infection, stress. S/S: burning pain, nausea, anorexia,
wt loss Complication: hemorrhage, perforation,
obstruction, Tx: meds, diet, stress management
Drug therapy Used to relieve symptoms Antacids are first line of defense
Diet Avoid coffee, tea, meat broth, alcohol, spicy food Frequent small feedings
Management NG tube to sxn if hemorrhage suspected Saline lavage after NG procedure on page 769-772 Vasopressin may help control hemorrage
Table 38-6 discusses surgery tx of peptic ulcer dx
Stomach cancer
25,000 dx each year Most common in men, african
americans, people over 70, low socioeconomic status
S/S: no early signs Late signs: vomiting, ascites, liver
enlargement, abd mass 5 yr survival: 10% No known cause Risk factors: pernicious anemia,
chronic atrophic gastritis, achlorhydria (lack of HCL), smoking, high salt starch pickled food nitrate diet
obesity
Excess body fat Causes: heredity, body build,
metabolism, psychosocial, caloric intake
Complications: heart/lung problems, DM, polycythemia, cholelithiasis, infertility, endometrial cancer, DJD
Tx: wt reduction diet, exercise, medication (pg775), surgery,
malabsorption 1 or more nutrients not absorbed/digested Causes: bacteria, bile salt and digestive enzyme deficiency, alterations in
intestinal mucosa 2 types:
Celiac sprue (tropical, nontropical)- genetic, Non-Tropical: changes in mucosa, impaired absorption Tropical: infectious agent
Lactose intolerance Inherited or aquired Causes: IBS, gastroenteritis, sprue syndrome
S/S: steatorrhea (fatty stools), foul stools, wt loss, decreased libido, easy bruising, edema, anemia, bone pain
Tx: diet, meds, elimate gluten for celiac dx Tropical sprue: oral folate, antibx, vit B12 injections Lactose: no milk or milk products, lactase enzyme, monitor vitamin levels
diarrhea
Loose liquid stools Causes: spoiled foods, allergies,
infection, diverticulosis, cancer, malabsorption, impactions, tube feedings, medications
S/S:cramps, abd pain, urgency Complications: dehydration,
electrolyte imbalance Tx: anti diarrheal drugs, clear
liquids vs npo, possible TPN
constipation
Hard dry infrequent stools Causes: ignoring urge, laxative
use, inactivity, inadequate fluid intake, drugs, brain/spinal cord injury, colon diseases, surgery,
Tx: laxatives, stool softeners,
megacolon
Large intestine looses ability to contract to move feces to rectum
Pts need regular enemas
Fecal impaction
Retention of large amount of stool in the rectum
Some liquid passes around TX: Digital exam/extraction
Intestinal obstruction
Causes by strangulated hernia, tumor, ileus, stricture, volvulus (twisting of bowel)
S/S: vomiting (bile, blood, feces), abd pain, constipation
Complications: electrolyte imbalances, gangrene, perforation, shock, death
TX: gastric decompression, IVF, surgery
appendicitis
Blind patch in the cecum Inflammation of opening of
appendix-bacteria related s/s: pain especially at
mcburney’s point (1/2 way b/w umbilicus and iliac crest), fever, n/v, elevated WBC
Tx: NPO, cold pack
peritonitis
stomach contents enter Abd cavity Complications: fluid shift, abscesses,
adhesions, septicemia, hyovolemic shock, ileus, organ failure
S/S: abd distention, increased pulse and RR, n/v, fever, rigid abd, shock
TX: NG for gastric decompression, IVF, antibiotics, pain meds, surgery
IBS 2 types:
Ulcerative colitis: Begins in rectum, expands to cecum
Crohn’s: regional enteritis Affects all GI tract Most common= terminal ileum
Causes: unknown S/S of IBS: constipation, diarrhea, bloody stools, abd
cramping, wt loss S/S crohn’s: variable, n/v, pain, cramping, abd
tenderness, fever, night sweats, malaise, joint pain Complications:hemorrhage, obstruction, perforation,
abscess, fistulas, megacolon, colon cancer, joint inflammation, diarrhea, stones, liver dx, electrolyte imbalances
Tx: meds (page 786), low roughage diet without milk, nicotine patches, surgery with possible removal of intestine
Diverticulosis
Small sac like pouches in intestinal wall Most in sigmoid colon Risk factors: lack of dietary factors, age,
constipation, obesity, emotional tension S/S: asymptomatic, constipation,
diarrhea, pain, rectal bleed, n/v, urinary problems
Complications: bleed, obstruct, perforation, peritonitis, fistula
Tx: high residue diet, no spicy foods, no seedy food, stool softener, meds, page 788, surgery
Colorectal CA
3rd most common in women High fat low fiber diet is risk factor Most found in rectum or low
sigmoid S/S: depend on location,
cramping, anemia, weakness, fatigue, left sided= more obvious changes
TX: surgery, colostomy, chemo, radiation
Polyps
Small benign growths that can become malignant
Multiple polyps called gardner’s syndrome or familial polyposis
S/S:asymptomatic Complications: bleed, obstruction Tx: removal, colectomy
hemorrhoids
Dilated veins in rectum May be internal or external Risk factors: increased
pressure in rectal blood vessels from constipation, pregnancy, prolonged sit or stand
S/S: pain, bleed, itching, TX : surgery, ice followed by
heat, medication
Anorectal abscess
Infection in the tissue around rectum
S/S: pain, swelling, redness, tenderness, diarrhea, bleeding, itching, discharge
Tx: antibx, incision, drainage, surgery, ice packs, pt education r/t to cleansing
Anal fissure
Laceration b/w anus and perianal skin
r/t constipation, diarrhea, crohn’s, TB, leukemia, trauma, childbirth
S/S: pain with defecation, bleeding, itching, urinary frequency, urinary retention, dysuria
Tx: heal spontaneously, sitz bath, stool softeners, pain meds, surgery
Anal fistula
Abnormal opening b/w anal canal and perianal skin
Causes: abscess, IBD, TB S/S: pruritis, discharge Tx: sitz bath, surgery, temporary
colostomy, pain meds
Pilonidal cyst
Painful and swollen
May form abscess
Surgery may be needed to fix
PT EDUCATION
Handwashing Proper food handling Food poisoning Stress management When to call doctor Page 793
THE END