Chapter 5 The Integumentary System -...
Transcript of Chapter 5 The Integumentary System -...
![Page 1: Chapter 5 The Integumentary System - Faribaultmail.faribault.k12.mn.us/~Laura_Childs/S01B3E075.2...A. Skin 1. Epidermis Roof Stratified squamous A. Stratum basale: Bottom Mitosis ~](https://reader033.fdocuments.in/reader033/viewer/2022042317/5f06a68a7e708231d4190d32/html5/thumbnails/1.jpg)
Chapter 5 The Integumentary System
bull Protects Sensation Vit D Temp regulatn excretn
A Skin
1 Epidermis Roof
Stratified squamous
A Stratum basale
Bottom
Mitosis ~ every 19 days
1 surfaces ~ 40-56 days later
B Stratum Granulosum
Fill whard protein keratin (die)
C Stratum Corneum
Upper
Protective cells coated wlipid
Top sloughs
Callus friction uarr cell layers
Corn thickened area over bone
2 Dermis
House
Collagen (strength)
Cleavage Lines (fiber orientation)
resist stretching
parallel incision heal best
striae stretch marks
Skin Cont
Few fat cellsmacrophages
Dermal papillae (upper)
Blood vessels (move matlrsquos temp reg)
Shape fingerft print
Grip of handft
B Skin Color 1 Melanin
Brnblk pigment molecule
UV protection
Melanocytes Produce melanin vesicles melanosomes Vesicles phagocytized by epidermal cells
Frecklesmoles most
Palmssoles least
of melanocytes
Same for all races
- Skin cells
Production based on
Genetics races
Albinism = recessive (no melanin)
Hormones pregnancy mask
UV light suntan
2 Tattoo bluish hue dermal fibers scatter light
3 Cyanosis bluish hue darr bld O2
Cyanosis of nail beds with extreme
pulmonary dysfunction
Irregular heart w Undersized lungs
4 Carotene yellowish lipid-soluble pigment (carrots)
5 Jaundice yellow wliver damage - bile in bld
Jaundice Yellow skin discoloration caused by hyperbilirubinemia Anatomy of the liver An obstruction in the bile duct may lead to jaundice
6 Birthmark congenital disorder of dermal BV
Capillary Malformation (Port-wine Stain)
Vascular birthmarks are caused by enlarged small blood vessels just beneath the skins surface The most common are known as angels kissesldquowhen located on the forehead or eyelids and as stork bites
when they appear on the back of the neck
Most birthmarks are no risk to health
C Hypodermis Foundation
Subcutaneous (under skin)
frac12 body fat (insulatepad) TBF count
Injection site
D Accessory Skin Structures 1 Hair
A Shaft (visible)
B Hair root (bulb) in follicle
Dermal papilla nourishes
Dividekeratinize
The cuticle or outer layer
of a healthy shaft of hair
A split end caused when
a hair shaft is pulled apart
Phases
Growth lash-30 dysscalp-3 yrs
Rest lash 105 dysscalp-1-2 yrs
C Pattern baldness sex linked (hormone)
D Color determined by bulb melanocytes
Age gray -darr activity white - no actvty
How does hair turn gray The melanocytes in hair stop producing melanins
2 Muscles arrector pili (smooth)
A Gooseflesh hair perpendicular
B Wfur insulatefierceness
3 Glands
A Sebaceous- sebum (oil) to follicle
B Eccrine- sweat (uarr H2O) to skin
C Apocrine- org-sweat to genitalaxillary follicle
Active wpuberty ndash bacterial decay=odor
An apocrine gland which
produces little sweat but is
responsible for the bodys
natural scent
E Physiology Temp reg (dermal BV) excretion (urea-sweat)
Vitamin D prodctn in skin wUV
Stimulates CaP uptake SI= musclebone health
F Diagnostic
Burns degree of depth
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
![Page 2: Chapter 5 The Integumentary System - Faribaultmail.faribault.k12.mn.us/~Laura_Childs/S01B3E075.2...A. Skin 1. Epidermis Roof Stratified squamous A. Stratum basale: Bottom Mitosis ~](https://reader033.fdocuments.in/reader033/viewer/2022042317/5f06a68a7e708231d4190d32/html5/thumbnails/2.jpg)
A Skin
1 Epidermis Roof
Stratified squamous
A Stratum basale
Bottom
Mitosis ~ every 19 days
1 surfaces ~ 40-56 days later
B Stratum Granulosum
Fill whard protein keratin (die)
C Stratum Corneum
Upper
Protective cells coated wlipid
Top sloughs
Callus friction uarr cell layers
Corn thickened area over bone
2 Dermis
House
Collagen (strength)
Cleavage Lines (fiber orientation)
resist stretching
parallel incision heal best
striae stretch marks
Skin Cont
Few fat cellsmacrophages
Dermal papillae (upper)
Blood vessels (move matlrsquos temp reg)
Shape fingerft print
Grip of handft
B Skin Color 1 Melanin
Brnblk pigment molecule
UV protection
Melanocytes Produce melanin vesicles melanosomes Vesicles phagocytized by epidermal cells
Frecklesmoles most
Palmssoles least
of melanocytes
Same for all races
- Skin cells
Production based on
Genetics races
Albinism = recessive (no melanin)
Hormones pregnancy mask
UV light suntan
2 Tattoo bluish hue dermal fibers scatter light
3 Cyanosis bluish hue darr bld O2
Cyanosis of nail beds with extreme
pulmonary dysfunction
Irregular heart w Undersized lungs
4 Carotene yellowish lipid-soluble pigment (carrots)
5 Jaundice yellow wliver damage - bile in bld
Jaundice Yellow skin discoloration caused by hyperbilirubinemia Anatomy of the liver An obstruction in the bile duct may lead to jaundice
6 Birthmark congenital disorder of dermal BV
Capillary Malformation (Port-wine Stain)
Vascular birthmarks are caused by enlarged small blood vessels just beneath the skins surface The most common are known as angels kissesldquowhen located on the forehead or eyelids and as stork bites
when they appear on the back of the neck
Most birthmarks are no risk to health
C Hypodermis Foundation
Subcutaneous (under skin)
frac12 body fat (insulatepad) TBF count
Injection site
D Accessory Skin Structures 1 Hair
A Shaft (visible)
B Hair root (bulb) in follicle
Dermal papilla nourishes
Dividekeratinize
The cuticle or outer layer
of a healthy shaft of hair
A split end caused when
a hair shaft is pulled apart
Phases
Growth lash-30 dysscalp-3 yrs
Rest lash 105 dysscalp-1-2 yrs
C Pattern baldness sex linked (hormone)
D Color determined by bulb melanocytes
Age gray -darr activity white - no actvty
How does hair turn gray The melanocytes in hair stop producing melanins
2 Muscles arrector pili (smooth)
A Gooseflesh hair perpendicular
B Wfur insulatefierceness
3 Glands
A Sebaceous- sebum (oil) to follicle
B Eccrine- sweat (uarr H2O) to skin
C Apocrine- org-sweat to genitalaxillary follicle
Active wpuberty ndash bacterial decay=odor
An apocrine gland which
produces little sweat but is
responsible for the bodys
natural scent
E Physiology Temp reg (dermal BV) excretion (urea-sweat)
Vitamin D prodctn in skin wUV
Stimulates CaP uptake SI= musclebone health
F Diagnostic
Burns degree of depth
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
![Page 3: Chapter 5 The Integumentary System - Faribaultmail.faribault.k12.mn.us/~Laura_Childs/S01B3E075.2...A. Skin 1. Epidermis Roof Stratified squamous A. Stratum basale: Bottom Mitosis ~](https://reader033.fdocuments.in/reader033/viewer/2022042317/5f06a68a7e708231d4190d32/html5/thumbnails/3.jpg)
B Stratum Granulosum
Fill whard protein keratin (die)
C Stratum Corneum
Upper
Protective cells coated wlipid
Top sloughs
Callus friction uarr cell layers
Corn thickened area over bone
2 Dermis
House
Collagen (strength)
Cleavage Lines (fiber orientation)
resist stretching
parallel incision heal best
striae stretch marks
Skin Cont
Few fat cellsmacrophages
Dermal papillae (upper)
Blood vessels (move matlrsquos temp reg)
Shape fingerft print
Grip of handft
B Skin Color 1 Melanin
Brnblk pigment molecule
UV protection
Melanocytes Produce melanin vesicles melanosomes Vesicles phagocytized by epidermal cells
Frecklesmoles most
Palmssoles least
of melanocytes
Same for all races
- Skin cells
Production based on
Genetics races
Albinism = recessive (no melanin)
Hormones pregnancy mask
UV light suntan
2 Tattoo bluish hue dermal fibers scatter light
3 Cyanosis bluish hue darr bld O2
Cyanosis of nail beds with extreme
pulmonary dysfunction
Irregular heart w Undersized lungs
4 Carotene yellowish lipid-soluble pigment (carrots)
5 Jaundice yellow wliver damage - bile in bld
Jaundice Yellow skin discoloration caused by hyperbilirubinemia Anatomy of the liver An obstruction in the bile duct may lead to jaundice
6 Birthmark congenital disorder of dermal BV
Capillary Malformation (Port-wine Stain)
Vascular birthmarks are caused by enlarged small blood vessels just beneath the skins surface The most common are known as angels kissesldquowhen located on the forehead or eyelids and as stork bites
when they appear on the back of the neck
Most birthmarks are no risk to health
C Hypodermis Foundation
Subcutaneous (under skin)
frac12 body fat (insulatepad) TBF count
Injection site
D Accessory Skin Structures 1 Hair
A Shaft (visible)
B Hair root (bulb) in follicle
Dermal papilla nourishes
Dividekeratinize
The cuticle or outer layer
of a healthy shaft of hair
A split end caused when
a hair shaft is pulled apart
Phases
Growth lash-30 dysscalp-3 yrs
Rest lash 105 dysscalp-1-2 yrs
C Pattern baldness sex linked (hormone)
D Color determined by bulb melanocytes
Age gray -darr activity white - no actvty
How does hair turn gray The melanocytes in hair stop producing melanins
2 Muscles arrector pili (smooth)
A Gooseflesh hair perpendicular
B Wfur insulatefierceness
3 Glands
A Sebaceous- sebum (oil) to follicle
B Eccrine- sweat (uarr H2O) to skin
C Apocrine- org-sweat to genitalaxillary follicle
Active wpuberty ndash bacterial decay=odor
An apocrine gland which
produces little sweat but is
responsible for the bodys
natural scent
E Physiology Temp reg (dermal BV) excretion (urea-sweat)
Vitamin D prodctn in skin wUV
Stimulates CaP uptake SI= musclebone health
F Diagnostic
Burns degree of depth
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
![Page 4: Chapter 5 The Integumentary System - Faribaultmail.faribault.k12.mn.us/~Laura_Childs/S01B3E075.2...A. Skin 1. Epidermis Roof Stratified squamous A. Stratum basale: Bottom Mitosis ~](https://reader033.fdocuments.in/reader033/viewer/2022042317/5f06a68a7e708231d4190d32/html5/thumbnails/4.jpg)
C Stratum Corneum
Upper
Protective cells coated wlipid
Top sloughs
Callus friction uarr cell layers
Corn thickened area over bone
2 Dermis
House
Collagen (strength)
Cleavage Lines (fiber orientation)
resist stretching
parallel incision heal best
striae stretch marks
Skin Cont
Few fat cellsmacrophages
Dermal papillae (upper)
Blood vessels (move matlrsquos temp reg)
Shape fingerft print
Grip of handft
B Skin Color 1 Melanin
Brnblk pigment molecule
UV protection
Melanocytes Produce melanin vesicles melanosomes Vesicles phagocytized by epidermal cells
Frecklesmoles most
Palmssoles least
of melanocytes
Same for all races
- Skin cells
Production based on
Genetics races
Albinism = recessive (no melanin)
Hormones pregnancy mask
UV light suntan
2 Tattoo bluish hue dermal fibers scatter light
3 Cyanosis bluish hue darr bld O2
Cyanosis of nail beds with extreme
pulmonary dysfunction
Irregular heart w Undersized lungs
4 Carotene yellowish lipid-soluble pigment (carrots)
5 Jaundice yellow wliver damage - bile in bld
Jaundice Yellow skin discoloration caused by hyperbilirubinemia Anatomy of the liver An obstruction in the bile duct may lead to jaundice
6 Birthmark congenital disorder of dermal BV
Capillary Malformation (Port-wine Stain)
Vascular birthmarks are caused by enlarged small blood vessels just beneath the skins surface The most common are known as angels kissesldquowhen located on the forehead or eyelids and as stork bites
when they appear on the back of the neck
Most birthmarks are no risk to health
C Hypodermis Foundation
Subcutaneous (under skin)
frac12 body fat (insulatepad) TBF count
Injection site
D Accessory Skin Structures 1 Hair
A Shaft (visible)
B Hair root (bulb) in follicle
Dermal papilla nourishes
Dividekeratinize
The cuticle or outer layer
of a healthy shaft of hair
A split end caused when
a hair shaft is pulled apart
Phases
Growth lash-30 dysscalp-3 yrs
Rest lash 105 dysscalp-1-2 yrs
C Pattern baldness sex linked (hormone)
D Color determined by bulb melanocytes
Age gray -darr activity white - no actvty
How does hair turn gray The melanocytes in hair stop producing melanins
2 Muscles arrector pili (smooth)
A Gooseflesh hair perpendicular
B Wfur insulatefierceness
3 Glands
A Sebaceous- sebum (oil) to follicle
B Eccrine- sweat (uarr H2O) to skin
C Apocrine- org-sweat to genitalaxillary follicle
Active wpuberty ndash bacterial decay=odor
An apocrine gland which
produces little sweat but is
responsible for the bodys
natural scent
E Physiology Temp reg (dermal BV) excretion (urea-sweat)
Vitamin D prodctn in skin wUV
Stimulates CaP uptake SI= musclebone health
F Diagnostic
Burns degree of depth
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
![Page 5: Chapter 5 The Integumentary System - Faribaultmail.faribault.k12.mn.us/~Laura_Childs/S01B3E075.2...A. Skin 1. Epidermis Roof Stratified squamous A. Stratum basale: Bottom Mitosis ~](https://reader033.fdocuments.in/reader033/viewer/2022042317/5f06a68a7e708231d4190d32/html5/thumbnails/5.jpg)
2 Dermis
House
Collagen (strength)
Cleavage Lines (fiber orientation)
resist stretching
parallel incision heal best
striae stretch marks
Skin Cont
Few fat cellsmacrophages
Dermal papillae (upper)
Blood vessels (move matlrsquos temp reg)
Shape fingerft print
Grip of handft
B Skin Color 1 Melanin
Brnblk pigment molecule
UV protection
Melanocytes Produce melanin vesicles melanosomes Vesicles phagocytized by epidermal cells
Frecklesmoles most
Palmssoles least
of melanocytes
Same for all races
- Skin cells
Production based on
Genetics races
Albinism = recessive (no melanin)
Hormones pregnancy mask
UV light suntan
2 Tattoo bluish hue dermal fibers scatter light
3 Cyanosis bluish hue darr bld O2
Cyanosis of nail beds with extreme
pulmonary dysfunction
Irregular heart w Undersized lungs
4 Carotene yellowish lipid-soluble pigment (carrots)
5 Jaundice yellow wliver damage - bile in bld
Jaundice Yellow skin discoloration caused by hyperbilirubinemia Anatomy of the liver An obstruction in the bile duct may lead to jaundice
6 Birthmark congenital disorder of dermal BV
Capillary Malformation (Port-wine Stain)
Vascular birthmarks are caused by enlarged small blood vessels just beneath the skins surface The most common are known as angels kissesldquowhen located on the forehead or eyelids and as stork bites
when they appear on the back of the neck
Most birthmarks are no risk to health
C Hypodermis Foundation
Subcutaneous (under skin)
frac12 body fat (insulatepad) TBF count
Injection site
D Accessory Skin Structures 1 Hair
A Shaft (visible)
B Hair root (bulb) in follicle
Dermal papilla nourishes
Dividekeratinize
The cuticle or outer layer
of a healthy shaft of hair
A split end caused when
a hair shaft is pulled apart
Phases
Growth lash-30 dysscalp-3 yrs
Rest lash 105 dysscalp-1-2 yrs
C Pattern baldness sex linked (hormone)
D Color determined by bulb melanocytes
Age gray -darr activity white - no actvty
How does hair turn gray The melanocytes in hair stop producing melanins
2 Muscles arrector pili (smooth)
A Gooseflesh hair perpendicular
B Wfur insulatefierceness
3 Glands
A Sebaceous- sebum (oil) to follicle
B Eccrine- sweat (uarr H2O) to skin
C Apocrine- org-sweat to genitalaxillary follicle
Active wpuberty ndash bacterial decay=odor
An apocrine gland which
produces little sweat but is
responsible for the bodys
natural scent
E Physiology Temp reg (dermal BV) excretion (urea-sweat)
Vitamin D prodctn in skin wUV
Stimulates CaP uptake SI= musclebone health
F Diagnostic
Burns degree of depth
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
![Page 6: Chapter 5 The Integumentary System - Faribaultmail.faribault.k12.mn.us/~Laura_Childs/S01B3E075.2...A. Skin 1. Epidermis Roof Stratified squamous A. Stratum basale: Bottom Mitosis ~](https://reader033.fdocuments.in/reader033/viewer/2022042317/5f06a68a7e708231d4190d32/html5/thumbnails/6.jpg)
Skin Cont
Few fat cellsmacrophages
Dermal papillae (upper)
Blood vessels (move matlrsquos temp reg)
Shape fingerft print
Grip of handft
B Skin Color 1 Melanin
Brnblk pigment molecule
UV protection
Melanocytes Produce melanin vesicles melanosomes Vesicles phagocytized by epidermal cells
Frecklesmoles most
Palmssoles least
of melanocytes
Same for all races
- Skin cells
Production based on
Genetics races
Albinism = recessive (no melanin)
Hormones pregnancy mask
UV light suntan
2 Tattoo bluish hue dermal fibers scatter light
3 Cyanosis bluish hue darr bld O2
Cyanosis of nail beds with extreme
pulmonary dysfunction
Irregular heart w Undersized lungs
4 Carotene yellowish lipid-soluble pigment (carrots)
5 Jaundice yellow wliver damage - bile in bld
Jaundice Yellow skin discoloration caused by hyperbilirubinemia Anatomy of the liver An obstruction in the bile duct may lead to jaundice
6 Birthmark congenital disorder of dermal BV
Capillary Malformation (Port-wine Stain)
Vascular birthmarks are caused by enlarged small blood vessels just beneath the skins surface The most common are known as angels kissesldquowhen located on the forehead or eyelids and as stork bites
when they appear on the back of the neck
Most birthmarks are no risk to health
C Hypodermis Foundation
Subcutaneous (under skin)
frac12 body fat (insulatepad) TBF count
Injection site
D Accessory Skin Structures 1 Hair
A Shaft (visible)
B Hair root (bulb) in follicle
Dermal papilla nourishes
Dividekeratinize
The cuticle or outer layer
of a healthy shaft of hair
A split end caused when
a hair shaft is pulled apart
Phases
Growth lash-30 dysscalp-3 yrs
Rest lash 105 dysscalp-1-2 yrs
C Pattern baldness sex linked (hormone)
D Color determined by bulb melanocytes
Age gray -darr activity white - no actvty
How does hair turn gray The melanocytes in hair stop producing melanins
2 Muscles arrector pili (smooth)
A Gooseflesh hair perpendicular
B Wfur insulatefierceness
3 Glands
A Sebaceous- sebum (oil) to follicle
B Eccrine- sweat (uarr H2O) to skin
C Apocrine- org-sweat to genitalaxillary follicle
Active wpuberty ndash bacterial decay=odor
An apocrine gland which
produces little sweat but is
responsible for the bodys
natural scent
E Physiology Temp reg (dermal BV) excretion (urea-sweat)
Vitamin D prodctn in skin wUV
Stimulates CaP uptake SI= musclebone health
F Diagnostic
Burns degree of depth
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
![Page 7: Chapter 5 The Integumentary System - Faribaultmail.faribault.k12.mn.us/~Laura_Childs/S01B3E075.2...A. Skin 1. Epidermis Roof Stratified squamous A. Stratum basale: Bottom Mitosis ~](https://reader033.fdocuments.in/reader033/viewer/2022042317/5f06a68a7e708231d4190d32/html5/thumbnails/7.jpg)
B Skin Color 1 Melanin
Brnblk pigment molecule
UV protection
Melanocytes Produce melanin vesicles melanosomes Vesicles phagocytized by epidermal cells
Frecklesmoles most
Palmssoles least
of melanocytes
Same for all races
- Skin cells
Production based on
Genetics races
Albinism = recessive (no melanin)
Hormones pregnancy mask
UV light suntan
2 Tattoo bluish hue dermal fibers scatter light
3 Cyanosis bluish hue darr bld O2
Cyanosis of nail beds with extreme
pulmonary dysfunction
Irregular heart w Undersized lungs
4 Carotene yellowish lipid-soluble pigment (carrots)
5 Jaundice yellow wliver damage - bile in bld
Jaundice Yellow skin discoloration caused by hyperbilirubinemia Anatomy of the liver An obstruction in the bile duct may lead to jaundice
6 Birthmark congenital disorder of dermal BV
Capillary Malformation (Port-wine Stain)
Vascular birthmarks are caused by enlarged small blood vessels just beneath the skins surface The most common are known as angels kissesldquowhen located on the forehead or eyelids and as stork bites
when they appear on the back of the neck
Most birthmarks are no risk to health
C Hypodermis Foundation
Subcutaneous (under skin)
frac12 body fat (insulatepad) TBF count
Injection site
D Accessory Skin Structures 1 Hair
A Shaft (visible)
B Hair root (bulb) in follicle
Dermal papilla nourishes
Dividekeratinize
The cuticle or outer layer
of a healthy shaft of hair
A split end caused when
a hair shaft is pulled apart
Phases
Growth lash-30 dysscalp-3 yrs
Rest lash 105 dysscalp-1-2 yrs
C Pattern baldness sex linked (hormone)
D Color determined by bulb melanocytes
Age gray -darr activity white - no actvty
How does hair turn gray The melanocytes in hair stop producing melanins
2 Muscles arrector pili (smooth)
A Gooseflesh hair perpendicular
B Wfur insulatefierceness
3 Glands
A Sebaceous- sebum (oil) to follicle
B Eccrine- sweat (uarr H2O) to skin
C Apocrine- org-sweat to genitalaxillary follicle
Active wpuberty ndash bacterial decay=odor
An apocrine gland which
produces little sweat but is
responsible for the bodys
natural scent
E Physiology Temp reg (dermal BV) excretion (urea-sweat)
Vitamin D prodctn in skin wUV
Stimulates CaP uptake SI= musclebone health
F Diagnostic
Burns degree of depth
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
![Page 8: Chapter 5 The Integumentary System - Faribaultmail.faribault.k12.mn.us/~Laura_Childs/S01B3E075.2...A. Skin 1. Epidermis Roof Stratified squamous A. Stratum basale: Bottom Mitosis ~](https://reader033.fdocuments.in/reader033/viewer/2022042317/5f06a68a7e708231d4190d32/html5/thumbnails/8.jpg)
Production based on
Genetics races
Albinism = recessive (no melanin)
Hormones pregnancy mask
UV light suntan
2 Tattoo bluish hue dermal fibers scatter light
3 Cyanosis bluish hue darr bld O2
Cyanosis of nail beds with extreme
pulmonary dysfunction
Irregular heart w Undersized lungs
4 Carotene yellowish lipid-soluble pigment (carrots)
5 Jaundice yellow wliver damage - bile in bld
Jaundice Yellow skin discoloration caused by hyperbilirubinemia Anatomy of the liver An obstruction in the bile duct may lead to jaundice
6 Birthmark congenital disorder of dermal BV
Capillary Malformation (Port-wine Stain)
Vascular birthmarks are caused by enlarged small blood vessels just beneath the skins surface The most common are known as angels kissesldquowhen located on the forehead or eyelids and as stork bites
when they appear on the back of the neck
Most birthmarks are no risk to health
C Hypodermis Foundation
Subcutaneous (under skin)
frac12 body fat (insulatepad) TBF count
Injection site
D Accessory Skin Structures 1 Hair
A Shaft (visible)
B Hair root (bulb) in follicle
Dermal papilla nourishes
Dividekeratinize
The cuticle or outer layer
of a healthy shaft of hair
A split end caused when
a hair shaft is pulled apart
Phases
Growth lash-30 dysscalp-3 yrs
Rest lash 105 dysscalp-1-2 yrs
C Pattern baldness sex linked (hormone)
D Color determined by bulb melanocytes
Age gray -darr activity white - no actvty
How does hair turn gray The melanocytes in hair stop producing melanins
2 Muscles arrector pili (smooth)
A Gooseflesh hair perpendicular
B Wfur insulatefierceness
3 Glands
A Sebaceous- sebum (oil) to follicle
B Eccrine- sweat (uarr H2O) to skin
C Apocrine- org-sweat to genitalaxillary follicle
Active wpuberty ndash bacterial decay=odor
An apocrine gland which
produces little sweat but is
responsible for the bodys
natural scent
E Physiology Temp reg (dermal BV) excretion (urea-sweat)
Vitamin D prodctn in skin wUV
Stimulates CaP uptake SI= musclebone health
F Diagnostic
Burns degree of depth
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
![Page 9: Chapter 5 The Integumentary System - Faribaultmail.faribault.k12.mn.us/~Laura_Childs/S01B3E075.2...A. Skin 1. Epidermis Roof Stratified squamous A. Stratum basale: Bottom Mitosis ~](https://reader033.fdocuments.in/reader033/viewer/2022042317/5f06a68a7e708231d4190d32/html5/thumbnails/9.jpg)
2 Tattoo bluish hue dermal fibers scatter light
3 Cyanosis bluish hue darr bld O2
Cyanosis of nail beds with extreme
pulmonary dysfunction
Irregular heart w Undersized lungs
4 Carotene yellowish lipid-soluble pigment (carrots)
5 Jaundice yellow wliver damage - bile in bld
Jaundice Yellow skin discoloration caused by hyperbilirubinemia Anatomy of the liver An obstruction in the bile duct may lead to jaundice
6 Birthmark congenital disorder of dermal BV
Capillary Malformation (Port-wine Stain)
Vascular birthmarks are caused by enlarged small blood vessels just beneath the skins surface The most common are known as angels kissesldquowhen located on the forehead or eyelids and as stork bites
when they appear on the back of the neck
Most birthmarks are no risk to health
C Hypodermis Foundation
Subcutaneous (under skin)
frac12 body fat (insulatepad) TBF count
Injection site
D Accessory Skin Structures 1 Hair
A Shaft (visible)
B Hair root (bulb) in follicle
Dermal papilla nourishes
Dividekeratinize
The cuticle or outer layer
of a healthy shaft of hair
A split end caused when
a hair shaft is pulled apart
Phases
Growth lash-30 dysscalp-3 yrs
Rest lash 105 dysscalp-1-2 yrs
C Pattern baldness sex linked (hormone)
D Color determined by bulb melanocytes
Age gray -darr activity white - no actvty
How does hair turn gray The melanocytes in hair stop producing melanins
2 Muscles arrector pili (smooth)
A Gooseflesh hair perpendicular
B Wfur insulatefierceness
3 Glands
A Sebaceous- sebum (oil) to follicle
B Eccrine- sweat (uarr H2O) to skin
C Apocrine- org-sweat to genitalaxillary follicle
Active wpuberty ndash bacterial decay=odor
An apocrine gland which
produces little sweat but is
responsible for the bodys
natural scent
E Physiology Temp reg (dermal BV) excretion (urea-sweat)
Vitamin D prodctn in skin wUV
Stimulates CaP uptake SI= musclebone health
F Diagnostic
Burns degree of depth
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
![Page 10: Chapter 5 The Integumentary System - Faribaultmail.faribault.k12.mn.us/~Laura_Childs/S01B3E075.2...A. Skin 1. Epidermis Roof Stratified squamous A. Stratum basale: Bottom Mitosis ~](https://reader033.fdocuments.in/reader033/viewer/2022042317/5f06a68a7e708231d4190d32/html5/thumbnails/10.jpg)
3 Cyanosis bluish hue darr bld O2
Cyanosis of nail beds with extreme
pulmonary dysfunction
Irregular heart w Undersized lungs
4 Carotene yellowish lipid-soluble pigment (carrots)
5 Jaundice yellow wliver damage - bile in bld
Jaundice Yellow skin discoloration caused by hyperbilirubinemia Anatomy of the liver An obstruction in the bile duct may lead to jaundice
6 Birthmark congenital disorder of dermal BV
Capillary Malformation (Port-wine Stain)
Vascular birthmarks are caused by enlarged small blood vessels just beneath the skins surface The most common are known as angels kissesldquowhen located on the forehead or eyelids and as stork bites
when they appear on the back of the neck
Most birthmarks are no risk to health
C Hypodermis Foundation
Subcutaneous (under skin)
frac12 body fat (insulatepad) TBF count
Injection site
D Accessory Skin Structures 1 Hair
A Shaft (visible)
B Hair root (bulb) in follicle
Dermal papilla nourishes
Dividekeratinize
The cuticle or outer layer
of a healthy shaft of hair
A split end caused when
a hair shaft is pulled apart
Phases
Growth lash-30 dysscalp-3 yrs
Rest lash 105 dysscalp-1-2 yrs
C Pattern baldness sex linked (hormone)
D Color determined by bulb melanocytes
Age gray -darr activity white - no actvty
How does hair turn gray The melanocytes in hair stop producing melanins
2 Muscles arrector pili (smooth)
A Gooseflesh hair perpendicular
B Wfur insulatefierceness
3 Glands
A Sebaceous- sebum (oil) to follicle
B Eccrine- sweat (uarr H2O) to skin
C Apocrine- org-sweat to genitalaxillary follicle
Active wpuberty ndash bacterial decay=odor
An apocrine gland which
produces little sweat but is
responsible for the bodys
natural scent
E Physiology Temp reg (dermal BV) excretion (urea-sweat)
Vitamin D prodctn in skin wUV
Stimulates CaP uptake SI= musclebone health
F Diagnostic
Burns degree of depth
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
![Page 11: Chapter 5 The Integumentary System - Faribaultmail.faribault.k12.mn.us/~Laura_Childs/S01B3E075.2...A. Skin 1. Epidermis Roof Stratified squamous A. Stratum basale: Bottom Mitosis ~](https://reader033.fdocuments.in/reader033/viewer/2022042317/5f06a68a7e708231d4190d32/html5/thumbnails/11.jpg)
4 Carotene yellowish lipid-soluble pigment (carrots)
5 Jaundice yellow wliver damage - bile in bld
Jaundice Yellow skin discoloration caused by hyperbilirubinemia Anatomy of the liver An obstruction in the bile duct may lead to jaundice
6 Birthmark congenital disorder of dermal BV
Capillary Malformation (Port-wine Stain)
Vascular birthmarks are caused by enlarged small blood vessels just beneath the skins surface The most common are known as angels kissesldquowhen located on the forehead or eyelids and as stork bites
when they appear on the back of the neck
Most birthmarks are no risk to health
C Hypodermis Foundation
Subcutaneous (under skin)
frac12 body fat (insulatepad) TBF count
Injection site
D Accessory Skin Structures 1 Hair
A Shaft (visible)
B Hair root (bulb) in follicle
Dermal papilla nourishes
Dividekeratinize
The cuticle or outer layer
of a healthy shaft of hair
A split end caused when
a hair shaft is pulled apart
Phases
Growth lash-30 dysscalp-3 yrs
Rest lash 105 dysscalp-1-2 yrs
C Pattern baldness sex linked (hormone)
D Color determined by bulb melanocytes
Age gray -darr activity white - no actvty
How does hair turn gray The melanocytes in hair stop producing melanins
2 Muscles arrector pili (smooth)
A Gooseflesh hair perpendicular
B Wfur insulatefierceness
3 Glands
A Sebaceous- sebum (oil) to follicle
B Eccrine- sweat (uarr H2O) to skin
C Apocrine- org-sweat to genitalaxillary follicle
Active wpuberty ndash bacterial decay=odor
An apocrine gland which
produces little sweat but is
responsible for the bodys
natural scent
E Physiology Temp reg (dermal BV) excretion (urea-sweat)
Vitamin D prodctn in skin wUV
Stimulates CaP uptake SI= musclebone health
F Diagnostic
Burns degree of depth
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
![Page 12: Chapter 5 The Integumentary System - Faribaultmail.faribault.k12.mn.us/~Laura_Childs/S01B3E075.2...A. Skin 1. Epidermis Roof Stratified squamous A. Stratum basale: Bottom Mitosis ~](https://reader033.fdocuments.in/reader033/viewer/2022042317/5f06a68a7e708231d4190d32/html5/thumbnails/12.jpg)
6 Birthmark congenital disorder of dermal BV
Capillary Malformation (Port-wine Stain)
Vascular birthmarks are caused by enlarged small blood vessels just beneath the skins surface The most common are known as angels kissesldquowhen located on the forehead or eyelids and as stork bites
when they appear on the back of the neck
Most birthmarks are no risk to health
C Hypodermis Foundation
Subcutaneous (under skin)
frac12 body fat (insulatepad) TBF count
Injection site
D Accessory Skin Structures 1 Hair
A Shaft (visible)
B Hair root (bulb) in follicle
Dermal papilla nourishes
Dividekeratinize
The cuticle or outer layer
of a healthy shaft of hair
A split end caused when
a hair shaft is pulled apart
Phases
Growth lash-30 dysscalp-3 yrs
Rest lash 105 dysscalp-1-2 yrs
C Pattern baldness sex linked (hormone)
D Color determined by bulb melanocytes
Age gray -darr activity white - no actvty
How does hair turn gray The melanocytes in hair stop producing melanins
2 Muscles arrector pili (smooth)
A Gooseflesh hair perpendicular
B Wfur insulatefierceness
3 Glands
A Sebaceous- sebum (oil) to follicle
B Eccrine- sweat (uarr H2O) to skin
C Apocrine- org-sweat to genitalaxillary follicle
Active wpuberty ndash bacterial decay=odor
An apocrine gland which
produces little sweat but is
responsible for the bodys
natural scent
E Physiology Temp reg (dermal BV) excretion (urea-sweat)
Vitamin D prodctn in skin wUV
Stimulates CaP uptake SI= musclebone health
F Diagnostic
Burns degree of depth
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
![Page 13: Chapter 5 The Integumentary System - Faribaultmail.faribault.k12.mn.us/~Laura_Childs/S01B3E075.2...A. Skin 1. Epidermis Roof Stratified squamous A. Stratum basale: Bottom Mitosis ~](https://reader033.fdocuments.in/reader033/viewer/2022042317/5f06a68a7e708231d4190d32/html5/thumbnails/13.jpg)
C Hypodermis Foundation
Subcutaneous (under skin)
frac12 body fat (insulatepad) TBF count
Injection site
D Accessory Skin Structures 1 Hair
A Shaft (visible)
B Hair root (bulb) in follicle
Dermal papilla nourishes
Dividekeratinize
The cuticle or outer layer
of a healthy shaft of hair
A split end caused when
a hair shaft is pulled apart
Phases
Growth lash-30 dysscalp-3 yrs
Rest lash 105 dysscalp-1-2 yrs
C Pattern baldness sex linked (hormone)
D Color determined by bulb melanocytes
Age gray -darr activity white - no actvty
How does hair turn gray The melanocytes in hair stop producing melanins
2 Muscles arrector pili (smooth)
A Gooseflesh hair perpendicular
B Wfur insulatefierceness
3 Glands
A Sebaceous- sebum (oil) to follicle
B Eccrine- sweat (uarr H2O) to skin
C Apocrine- org-sweat to genitalaxillary follicle
Active wpuberty ndash bacterial decay=odor
An apocrine gland which
produces little sweat but is
responsible for the bodys
natural scent
E Physiology Temp reg (dermal BV) excretion (urea-sweat)
Vitamin D prodctn in skin wUV
Stimulates CaP uptake SI= musclebone health
F Diagnostic
Burns degree of depth
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
![Page 14: Chapter 5 The Integumentary System - Faribaultmail.faribault.k12.mn.us/~Laura_Childs/S01B3E075.2...A. Skin 1. Epidermis Roof Stratified squamous A. Stratum basale: Bottom Mitosis ~](https://reader033.fdocuments.in/reader033/viewer/2022042317/5f06a68a7e708231d4190d32/html5/thumbnails/14.jpg)
D Accessory Skin Structures 1 Hair
A Shaft (visible)
B Hair root (bulb) in follicle
Dermal papilla nourishes
Dividekeratinize
The cuticle or outer layer
of a healthy shaft of hair
A split end caused when
a hair shaft is pulled apart
Phases
Growth lash-30 dysscalp-3 yrs
Rest lash 105 dysscalp-1-2 yrs
C Pattern baldness sex linked (hormone)
D Color determined by bulb melanocytes
Age gray -darr activity white - no actvty
How does hair turn gray The melanocytes in hair stop producing melanins
2 Muscles arrector pili (smooth)
A Gooseflesh hair perpendicular
B Wfur insulatefierceness
3 Glands
A Sebaceous- sebum (oil) to follicle
B Eccrine- sweat (uarr H2O) to skin
C Apocrine- org-sweat to genitalaxillary follicle
Active wpuberty ndash bacterial decay=odor
An apocrine gland which
produces little sweat but is
responsible for the bodys
natural scent
E Physiology Temp reg (dermal BV) excretion (urea-sweat)
Vitamin D prodctn in skin wUV
Stimulates CaP uptake SI= musclebone health
F Diagnostic
Burns degree of depth
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
![Page 15: Chapter 5 The Integumentary System - Faribaultmail.faribault.k12.mn.us/~Laura_Childs/S01B3E075.2...A. Skin 1. Epidermis Roof Stratified squamous A. Stratum basale: Bottom Mitosis ~](https://reader033.fdocuments.in/reader033/viewer/2022042317/5f06a68a7e708231d4190d32/html5/thumbnails/15.jpg)
Phases
Growth lash-30 dysscalp-3 yrs
Rest lash 105 dysscalp-1-2 yrs
C Pattern baldness sex linked (hormone)
D Color determined by bulb melanocytes
Age gray -darr activity white - no actvty
How does hair turn gray The melanocytes in hair stop producing melanins
2 Muscles arrector pili (smooth)
A Gooseflesh hair perpendicular
B Wfur insulatefierceness
3 Glands
A Sebaceous- sebum (oil) to follicle
B Eccrine- sweat (uarr H2O) to skin
C Apocrine- org-sweat to genitalaxillary follicle
Active wpuberty ndash bacterial decay=odor
An apocrine gland which
produces little sweat but is
responsible for the bodys
natural scent
E Physiology Temp reg (dermal BV) excretion (urea-sweat)
Vitamin D prodctn in skin wUV
Stimulates CaP uptake SI= musclebone health
F Diagnostic
Burns degree of depth
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
![Page 16: Chapter 5 The Integumentary System - Faribaultmail.faribault.k12.mn.us/~Laura_Childs/S01B3E075.2...A. Skin 1. Epidermis Roof Stratified squamous A. Stratum basale: Bottom Mitosis ~](https://reader033.fdocuments.in/reader033/viewer/2022042317/5f06a68a7e708231d4190d32/html5/thumbnails/16.jpg)
C Pattern baldness sex linked (hormone)
D Color determined by bulb melanocytes
Age gray -darr activity white - no actvty
How does hair turn gray The melanocytes in hair stop producing melanins
2 Muscles arrector pili (smooth)
A Gooseflesh hair perpendicular
B Wfur insulatefierceness
3 Glands
A Sebaceous- sebum (oil) to follicle
B Eccrine- sweat (uarr H2O) to skin
C Apocrine- org-sweat to genitalaxillary follicle
Active wpuberty ndash bacterial decay=odor
An apocrine gland which
produces little sweat but is
responsible for the bodys
natural scent
E Physiology Temp reg (dermal BV) excretion (urea-sweat)
Vitamin D prodctn in skin wUV
Stimulates CaP uptake SI= musclebone health
F Diagnostic
Burns degree of depth
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
![Page 17: Chapter 5 The Integumentary System - Faribaultmail.faribault.k12.mn.us/~Laura_Childs/S01B3E075.2...A. Skin 1. Epidermis Roof Stratified squamous A. Stratum basale: Bottom Mitosis ~](https://reader033.fdocuments.in/reader033/viewer/2022042317/5f06a68a7e708231d4190d32/html5/thumbnails/17.jpg)
D Color determined by bulb melanocytes
Age gray -darr activity white - no actvty
How does hair turn gray The melanocytes in hair stop producing melanins
2 Muscles arrector pili (smooth)
A Gooseflesh hair perpendicular
B Wfur insulatefierceness
3 Glands
A Sebaceous- sebum (oil) to follicle
B Eccrine- sweat (uarr H2O) to skin
C Apocrine- org-sweat to genitalaxillary follicle
Active wpuberty ndash bacterial decay=odor
An apocrine gland which
produces little sweat but is
responsible for the bodys
natural scent
E Physiology Temp reg (dermal BV) excretion (urea-sweat)
Vitamin D prodctn in skin wUV
Stimulates CaP uptake SI= musclebone health
F Diagnostic
Burns degree of depth
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
![Page 18: Chapter 5 The Integumentary System - Faribaultmail.faribault.k12.mn.us/~Laura_Childs/S01B3E075.2...A. Skin 1. Epidermis Roof Stratified squamous A. Stratum basale: Bottom Mitosis ~](https://reader033.fdocuments.in/reader033/viewer/2022042317/5f06a68a7e708231d4190d32/html5/thumbnails/18.jpg)
2 Muscles arrector pili (smooth)
A Gooseflesh hair perpendicular
B Wfur insulatefierceness
3 Glands
A Sebaceous- sebum (oil) to follicle
B Eccrine- sweat (uarr H2O) to skin
C Apocrine- org-sweat to genitalaxillary follicle
Active wpuberty ndash bacterial decay=odor
An apocrine gland which
produces little sweat but is
responsible for the bodys
natural scent
E Physiology Temp reg (dermal BV) excretion (urea-sweat)
Vitamin D prodctn in skin wUV
Stimulates CaP uptake SI= musclebone health
F Diagnostic
Burns degree of depth
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
![Page 19: Chapter 5 The Integumentary System - Faribaultmail.faribault.k12.mn.us/~Laura_Childs/S01B3E075.2...A. Skin 1. Epidermis Roof Stratified squamous A. Stratum basale: Bottom Mitosis ~](https://reader033.fdocuments.in/reader033/viewer/2022042317/5f06a68a7e708231d4190d32/html5/thumbnails/19.jpg)
3 Glands
A Sebaceous- sebum (oil) to follicle
B Eccrine- sweat (uarr H2O) to skin
C Apocrine- org-sweat to genitalaxillary follicle
Active wpuberty ndash bacterial decay=odor
An apocrine gland which
produces little sweat but is
responsible for the bodys
natural scent
E Physiology Temp reg (dermal BV) excretion (urea-sweat)
Vitamin D prodctn in skin wUV
Stimulates CaP uptake SI= musclebone health
F Diagnostic
Burns degree of depth
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
![Page 20: Chapter 5 The Integumentary System - Faribaultmail.faribault.k12.mn.us/~Laura_Childs/S01B3E075.2...A. Skin 1. Epidermis Roof Stratified squamous A. Stratum basale: Bottom Mitosis ~](https://reader033.fdocuments.in/reader033/viewer/2022042317/5f06a68a7e708231d4190d32/html5/thumbnails/20.jpg)
E Physiology Temp reg (dermal BV) excretion (urea-sweat)
Vitamin D prodctn in skin wUV
Stimulates CaP uptake SI= musclebone health
F Diagnostic
Burns degree of depth
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
![Page 21: Chapter 5 The Integumentary System - Faribaultmail.faribault.k12.mn.us/~Laura_Childs/S01B3E075.2...A. Skin 1. Epidermis Roof Stratified squamous A. Stratum basale: Bottom Mitosis ~](https://reader033.fdocuments.in/reader033/viewer/2022042317/5f06a68a7e708231d4190d32/html5/thumbnails/21.jpg)
F Diagnostic
Burns degree of depth
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
![Page 22: Chapter 5 The Integumentary System - Faribaultmail.faribault.k12.mn.us/~Laura_Childs/S01B3E075.2...A. Skin 1. Epidermis Roof Stratified squamous A. Stratum basale: Bottom Mitosis ~](https://reader033.fdocuments.in/reader033/viewer/2022042317/5f06a68a7e708231d4190d32/html5/thumbnails/22.jpg)
Burns degree of depth
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
![Page 23: Chapter 5 The Integumentary System - Faribaultmail.faribault.k12.mn.us/~Laura_Childs/S01B3E075.2...A. Skin 1. Epidermis Roof Stratified squamous A. Stratum basale: Bottom Mitosis ~](https://reader033.fdocuments.in/reader033/viewer/2022042317/5f06a68a7e708231d4190d32/html5/thumbnails/23.jpg)
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
![Page 24: Chapter 5 The Integumentary System - Faribaultmail.faribault.k12.mn.us/~Laura_Childs/S01B3E075.2...A. Skin 1. Epidermis Roof Stratified squamous A. Stratum basale: Bottom Mitosis ~](https://reader033.fdocuments.in/reader033/viewer/2022042317/5f06a68a7e708231d4190d32/html5/thumbnails/24.jpg)
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
![Page 25: Chapter 5 The Integumentary System - Faribaultmail.faribault.k12.mn.us/~Laura_Childs/S01B3E075.2...A. Skin 1. Epidermis Roof Stratified squamous A. Stratum basale: Bottom Mitosis ~](https://reader033.fdocuments.in/reader033/viewer/2022042317/5f06a68a7e708231d4190d32/html5/thumbnails/25.jpg)
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
![Page 26: Chapter 5 The Integumentary System - Faribaultmail.faribault.k12.mn.us/~Laura_Childs/S01B3E075.2...A. Skin 1. Epidermis Roof Stratified squamous A. Stratum basale: Bottom Mitosis ~](https://reader033.fdocuments.in/reader033/viewer/2022042317/5f06a68a7e708231d4190d32/html5/thumbnails/26.jpg)
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
![Page 27: Chapter 5 The Integumentary System - Faribaultmail.faribault.k12.mn.us/~Laura_Childs/S01B3E075.2...A. Skin 1. Epidermis Roof Stratified squamous A. Stratum basale: Bottom Mitosis ~](https://reader033.fdocuments.in/reader033/viewer/2022042317/5f06a68a7e708231d4190d32/html5/thumbnails/27.jpg)
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
![Page 28: Chapter 5 The Integumentary System - Faribaultmail.faribault.k12.mn.us/~Laura_Childs/S01B3E075.2...A. Skin 1. Epidermis Roof Stratified squamous A. Stratum basale: Bottom Mitosis ~](https://reader033.fdocuments.in/reader033/viewer/2022042317/5f06a68a7e708231d4190d32/html5/thumbnails/28.jpg)
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
![Page 29: Chapter 5 The Integumentary System - Faribaultmail.faribault.k12.mn.us/~Laura_Childs/S01B3E075.2...A. Skin 1. Epidermis Roof Stratified squamous A. Stratum basale: Bottom Mitosis ~](https://reader033.fdocuments.in/reader033/viewer/2022042317/5f06a68a7e708231d4190d32/html5/thumbnails/29.jpg)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)