Cardiac arrest
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Transcript of Cardiac arrest
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Cardiac Arrest
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SUBMITTED TO:
Dr. Sonia Qandeel
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SUBMITTED BY
Nimra Iqbal Dph-fa10-100
Ammarah Siddique Dph-fa10-094
Talat Fatima Dph-fa10-102
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CONTENTS
Definition
Diagnosis
Causes
Symptoms
Management Approach
Medication used
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Definition of Cardiac arrest:
Sudden cessation of heartbeat and cardiac function, resulting in the loss of effective circulation.
or
Absence of systole; failure of the ventricles of the heart to contract (usually caused by ventricular fibrillation) with consequent absence of the heart beat leading to oxygen lack and eventually to death
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Diagnosis of cardiac arrest (TRIAD):
Loss of consciousness, unresponsiveness
Loss of normal breathing Apnea.
Loss of pulse and blood pressure {apical & central pulsations (carotid, femoral loss}
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CAUSES
Influx problems in the heart.
Congenital heart disease
Valvular heart disease
Enlarged heart (cardiomyopathy).
Heart attack
Coronary artery disease
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Symptoms of cardiac arrest
Symptomscardiac arrest symptoms are immediate and drastic.Sudden collapseNo pulseNo breathing (respiration arrest – may be in 30 seconds after cardiac arrest
Loss of consciousness
enlargement of pupils – may be in 90 seconds after cardiac arrest
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TREATMENT
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To find the best treatment methodsfor managing cardiac arrest, in orderto save more lives!
Our Ultimate Goal
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Delay Can Be Deadly
Patient delay is the biggest cause of not getting care fast.
Do not wait more than a few minutes—5 at the most
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Early Access to Care – Know the Signs Early CPR, Cardiopulmonary Resuscitation
especially with quality chest compressions Rapid defibrillation(with AEDs) (an electrical
shock to the heart) Effective paramedics (advanced life support ) Follow up care (post-cardiac arrest care)
“Chain of Survival”
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A…B…C…D…E…
The ABCDE approach to the critically ill patient
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ABCDE approachAirway
Recognition of airway obstruction Talking
Difficulty breathing, distressed, Shortness of breath
Noisy breathing
stridor, wheeze, gurgling See-saw respiratory pattern,
A
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ABCDE ApproachAirway
Treatment of airway obstruction
Oxygen
Airway opening
- i.e. head tilt, chin lift, jaw thrust Advanced techniques
- e.g. LMA, tracheal tube
A
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ABCDE approachBreathing
Recognition of breathing problemsLook
Inspect respiratory distress, cyanosis, respiratory rate, chest deformity,
Listen Auscultate breath sounds, noisy breathing
Feel palpat expansion, percussion, tracheal position
Pulse oxymetry
B
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ABCDE approachBreathing
Treatment of breathing problems
Airway
Oxygen
Treat underlying cause
- e.g. drain pneumothorax
- e.g . Nebulizers Support breathing if inadequate
- e.g. ventilate with bag valve mask
B
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ABCDE approachCirculation
Look at the patientPulse – central pulse (carotid) peripheral pulse Peripheral perfusion
capillary refill time ( normally <2 sec)Blood pressureMonitor
C
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ABCDE approachCirculation
IV access, take blood sample and lab investigations
Treat cause Give fluids Haemodynamic monitoring MONA if acute coronary
syndrome
Treatment
C
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ABCDE approachDisability
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ABCDE approachDisability
AVPU or GCS, and pupils
Treatment - ABC
Treat underlying cause
Blood glucose if < 3 mmol l-1 give glucose
D
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ABCDE approachExposure
Remove clothes to enable examination
- e.g. injuries, bleeding, rashes
Avoid heat loss
Maintain dignity
E
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Defibrillation
All moving away from stacked shocks to single shocks Reduces pauses in chest compressions
Still role for initial stacked shocks if cardiac arrest occurs in presence of defibrillator
All recommend immediate CPR after defibrillation (without rhythm or pulse check)
Different recommendations on joules (150-360J) Between guidelines
Between manufacturers
Between monophasic and biphasic
There may be a role for CPR before defibrillation in some Particularly if in VF for more than a few minutes
Right heart dilation an impediment to defibrillation
Confused?
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Defibrillation
We (St John CMG) recommend a simple approach Start with one round of stacked shocks if cardiac
arrest occurs in presence of defibrillator, then go to single shocks
Always use maximum joules
Opt for defibrillation first
Round kids off to nearest 10kg and use 5J/kg
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Defibrillation
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Starting and stopping
These decisions can be difficult
A resuscitation attempt should begin in most patients Except where the patient is clearly dead (livedo, rigor
mortis)
Or where they are clearly dying and it would be inappropriate
A competent patient can decline therapy but neither a patient nor their family can demand therapy that is medically inappropriate
Some scenarios have >99% mortality rates Unwitnessed cardiac arrest with initial rhythm of
asystole
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Starting and stopping
The chances of survival fall rapidly with time Exponential falling curve
There is no absolute cut off when mortality becomes zero
Resuscitation attempts requiring longer than 20 minutes of CPR have a very high mortality rate We recommend stopping at around 20 minutes
unless there is a clinical reason to continue for longer
Transport to hospital with CPR enroute usually has no role
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Automated External Defibrillator
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An ICD monitors the heartbeat and delivers shock when
it detects lethal dysrhythmia.
Implantable Cardioverter Defibrillator
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Ventricular Fibrillation (VF)
What VF looks like on an EKG
Shock “converts” VF to better rhythm
Defibrillation (electrical shock) is the primary solution (cannot be used in other lethal heart rhythms)
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Automated External Defibrillators may be used
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Manual Defibrillator
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Drugs used commonly during resuscitation
Epinephrine (Adrenaline)
Atropine
Amiodarone
Magnesium Sulphate
Lidocaine (Lignocaine)
Sodium Bicarbonate
Calcium
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Epinephrine (Adrenaline)
First line cardiac arrest drug, given after every 3 minutes of CPR
Dose 1mg (10ml of 1 in 10,000) IV
Causes vasoconstriction, increased systemic vascular resistance increasing cerebral and coronary perfusion
Increases myocardial excitability, when the myocardium is hypoxic or ischaemic
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Atropine
Given for asystole or pulseless electrical activity with a rate less than 60 beats per minute
3mg is given as a single intravenous dose
It blocks the activity of the vagus nerve on the SA and AV nodes, increasing sinus automaticity and facilitating AV node conduction
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Amiodarone
For Refractory VF/VT; haemodynamically stable VT and other resistant tachyarrhythmias
If VF or pulseless VT persists after the first 3 shocks then Amiodarone 300mg is considered.
If not pre-diluted, must be diluted in 5% dextrose to 20ml. (Will crystallise is mixed with saline)
Should be given centrally but in an emergency can be given peripherally
Increases the duration of the action potential in the atrial and ventricular myocardium
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Magnesium Sulphate
For refractory VF when hypomagnesaemia is possible; ventricular tachyarrhythmias when hypomagnesaemia is possible
In refractory VF – 1 to 2g (2-4ml of 50% magnesium sulphate) peripherally over 1 to 2 minutes.
Other circumstances 2.5g (5ml of 50% magnesium sulphate) over 30 minutes
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Lidocaine (Lignocaine)
For Refractory VF/ pulseless VT (when Amiodarone is unavailable
100mg for VF/ pulseless VT that persists after three shocks. Another 50mg can be given if necessary
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Sodium Bicarbonate
Given for severe metabolic acidosis and Hyperkalaemia
50mmol (50ml of 8.4% solution), where there is an acidosis or cardiac arrest associated with Hyperkalaemia
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Calcium
Administered when pulseless electrical activity caused by:
Hyperkalaemia
Hypocalcaemia
Overdose of Calcium channel blocking
drugs
Dose 10ml of 10% calcium chloride repeated according to blood results
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Controllable Risk Factors
Smoking
Diabetes
High blood cholesterol
High blood pressure – especially stroke
Overweight/obesity
Physical inactivity
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Lifestyle Changes
Reduce intake of fatty foods and eat more fruits and vegetables
Walk 30 minutes a day
Exercise prevents stroke, heart disease and other conditions
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Questions