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B URN AND M ANAGEMENT OF DIFFERENT TYPES OF B URNS Dr Muath Mustafa Dept of Surgery, BMC HOD. Dr....
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Transcript of B URN AND M ANAGEMENT OF DIFFERENT TYPES OF B URNS Dr Muath Mustafa Dept of Surgery, BMC HOD. Dr....
![Page 1: B URN AND M ANAGEMENT OF DIFFERENT TYPES OF B URNS Dr Muath Mustafa Dept of Surgery, BMC HOD. Dr. Ashraf Balbaa.](https://reader034.fdocuments.in/reader034/viewer/2022051619/56649dbc5503460f94aad8b4/html5/thumbnails/1.jpg)
BURN AND MANAGEMENT OF DIFFERENT TYPES OF BURNS
Dr Muath Mustafa
Dept of Surgery, BMC
HOD. Dr. Ashraf Balbaa
![Page 2: B URN AND M ANAGEMENT OF DIFFERENT TYPES OF B URNS Dr Muath Mustafa Dept of Surgery, BMC HOD. Dr. Ashraf Balbaa.](https://reader034.fdocuments.in/reader034/viewer/2022051619/56649dbc5503460f94aad8b4/html5/thumbnails/2.jpg)
OVERVIEW
Burn Pathophysiology Initial Assessment & Management Airway Management & Inhalation
Injury Shock & Fluid Resuscitation Burn Wound Management Electrical Injuries Chemical Burns Pediatric Burns Other Topics
![Page 3: B URN AND M ANAGEMENT OF DIFFERENT TYPES OF B URNS Dr Muath Mustafa Dept of Surgery, BMC HOD. Dr. Ashraf Balbaa.](https://reader034.fdocuments.in/reader034/viewer/2022051619/56649dbc5503460f94aad8b4/html5/thumbnails/3.jpg)
SKIN ANATOMY
Epidermis
Dermis
Hypodermis
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FUNCTION OF NORMAL SKIN
Protection from infection & injury Prevention of loss of body fluid Regulation of body temperature Sensory contact with environment
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WHAT IS A BURN?
An injury to tissue from:
–Exposure to flames or hot liquids–Contact with hot objects–Exposure to caustic chemicals or
radiation–Contact with an electrical current
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PATHOPHYSIOLOGY OF BURN INJURY
Zone of Coagulation: Irreversible damage
Zone of Stasis: Impairment of blood
flowRecovery variable
Zone of Hyperemia:Prominent vasodilationUsually recovers
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OVERVIEW
Burn Pathophysiology Initial Assessment & Management Airway Management & Inhalation
Injury Shock & Fluid Resuscitation Burn Wound Management Electrical Injuries Chemical Burns Pediatric Burns Other Topics
![Page 8: B URN AND M ANAGEMENT OF DIFFERENT TYPES OF B URNS Dr Muath Mustafa Dept of Surgery, BMC HOD. Dr. Ashraf Balbaa.](https://reader034.fdocuments.in/reader034/viewer/2022051619/56649dbc5503460f94aad8b4/html5/thumbnails/8.jpg)
SEVERITY OF A BURN
Depends on: Depth of burn Extent of burn Location of injury Patient’s age Presence of associated
injury or diseases
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DEPTH OF A BURN
First Degree
Superficial Second
Deep Second
Third Degree
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DEPTH OF A BURN
First Degree
Epidermis only Erythematous Hypersensitive Classic sunburn Heals without scar
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DEPTH OF A BURNSecond Degree
Epidermis + part of dermis
SuperficialDeep
Blisters Edematous and red Very painful Scaring variable
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DEPTH OF A BURN
Third Degree Full thickness burn Can involve
underlying muscle, tendon, bone
Waxy white, leathery brown or charred black
Painless Heals with scar
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EXTENT OF A BURN
“Rule of Nines”
Most universal guide for initial estimate
Deviates in children due to larger head surface area
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“ROBYN’S RULE OF 4S”
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ABA BURN REFERRAL CRITERIA
2nd & 3rd degree burns of greater than 10% BSA in patients under 10 or over 50 yrs old
2nd & 3rd degree burns of greater than 20% BSA in other age groups
2nd & 3rd degree burns with functional or cosmetic implications
3rd degree burn of greater then 5% BSA
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ABA BURN REFERRAL CRITERIA
Significant electrical burn Chemical injury with functional or cosmetic
impairment Inhalation injury Circumferential burn of chest or extremity Burn injury with pre-existing medical disorder Any burn with concomitant trauma
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PRIMARY SURVEY
A – Airway B – Breathing C – Circulation / C-spine / Cardiac status D – Disability / Neurologic Deficit E – Exposure and Examination F – Fluid Resuscitation
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SECONDARY SURVEY
Complete head-to-toe examination Obtain as much information as possible
regarding injury:
A – Allergies M –Medications P – Past medical history L – Last meal or drink E – Events preceding injury
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MANAGEMENT PRINCIPLES
Stop the Burning Process Universal Precautions Airway Management Circulatory Management Insertion of a Nasogastric Tube Insertion of a Foley Catheter
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MANAGEMENT PRINCIPLES
Relieve Pain Assess Extremity Pulses Regularly Assess for Ventilatory Limitation Provide Emotional Support Suicide Management
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OVERVIEW
Burn Pathophysiology Initial Assessment & ManagementAirway Management & Inhalation
Injury Shock & Fluid Resuscitation Burn Wound Management Electrical Injuries Chemical Burns Pediatric Burns Other Topics
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INHALATION INJURY
Important determinant of morbidity & mortality
Manifests within the first 5 days after injury Present in 20-50% of pts admitted to burn
centers Present in 60-70% of pts who die in burn
centers
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INDICATORS OF INHALATION INJURY
Burned in closed space
Facial or intra-oral burns
Singed nasal hairs Soot in mouth,
nostrils, larynx Hoarseness or stridor Respiratory distress Signs of hypoxemia
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HISTORY OF EVENT
Is there a history of unconsciousness? Were there noxious chemicals involved? Did injury occur in closed space?
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TYPES OF INHALATION INJURY
Carbon Monoxide Poisoning
Inhalation Injury Above the Glottis
Inhalation Below the Glottis
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CARBON MONOXIDE POISONING
Colorless, odorless gas Binds to hemoglobin 200 times more than
oxygen Most immediate threat to life in survivors with
severe inhalation injury Toxicity related directly to percentage of
hemoglobin it saturates
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CARBON MONOXIDE POISONING
Signs & Symptoms of Carbon Monoxide Toxicity
Carboxyhemoglobin (%) Signs/Symptoms
0-10 None
10-30 Headache
30-50 Headache, nausea, dizziness, tachycardia
50-60 CNS dysfunction, coma
60+ Death
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SIGNS OF CO POISONING
Cherry red colorationNormal or pale skin with lip
colorationHypoxic with no apparent cyanosisPaO2 is unaffectedEssential to determine carboxyhemoglobin levels !
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CO POISONING: TREATMENT
100% oxygen until carboxyhemoglobin levels less than 15 Increases rate of CO diffusion from 4 hours to 45
minutes
Hyperbaric oxygen is of unproven value May be useful in isolated CO intoxication but
complicates wound care
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INHALATION INJURY ABOVE THE GLOTTIS
Most common inhalation injury
Results from heat dissipation into tissues
Commonly leads to obstruction
Edema lasts for 2-4 days
Dx by visualization of upper airways
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INHALATION INJURY ABOVE THE GLOTTIS:TREATMENT
Intubate!!!
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INHALATION INJURY BELOW THE GLOTTIS Chemical pneumonitis caused by toxic
products of combustion Ammonia, chlorine, hydrogen chloride,
phosgene, aldehydes, sulfur & nitrogen oxides Related to amount and type of volatile
substances inhaled Onset of symptoms is unpredictable
Close monitoring for first 24 hours
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INHALATION INJURY BELOW THE GLOTTIS:TREATMENT
Prior to transfer to burn center Intubation
to clear secretionsrelieve dyspneadeliver PEEP Improve oxygenation
Steroids not indicated Prophylactic antibiotics unjustified Circumferential chest burns:
escharotomies
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INHALATION INJURY IN THE PEDIATRIC PATIENT
Small airways: rapid onset of obstructionWell secured, appropriately sized, uncuffed
tube
Rib cage is not ossifiedMore pliablePt exhausts rapidly due to decrease in
compliance with circumferential chest burns
Escharotomies performed with first evidence of ventilatory impairment
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OVERVIEW
Burn Pathophysiology Initial Assessment & Management Airway Management & Inhalation
InjuryShock & Fluid Resuscitation Burn Wound Management Electrical Injuries Chemical Burns Pediatric Burns Other Topics
![Page 36: B URN AND M ANAGEMENT OF DIFFERENT TYPES OF B URNS Dr Muath Mustafa Dept of Surgery, BMC HOD. Dr. Ashraf Balbaa.](https://reader034.fdocuments.in/reader034/viewer/2022051619/56649dbc5503460f94aad8b4/html5/thumbnails/36.jpg)
SHOCK & FLUID RESUSCITATION
Goal:
To maintain vital organ function while avoiding the complications of inadequate or excessive therapy
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SYSTEMIC EFFECTS OF BURN INJURY Magnitude & duration of response
proportional to extent of surface burned Hypovolemia
Decreased perfusion & oxygen delivery Initial increase in PVR & decrease in CO
Neurogenic & humoral effects Corrected with adequate fluid resuscitation
Prevent shock & organ failure
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CELLULAR RESPONSE TO BURN INJURY
Severity dependant on temperature exposed and duration of exposure
“Zone of Stasis”: recovery of injured cells dependant on prompt resuscitation
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RESUSCITATION FLUID NEEDS
Related to: extent of burn (rule of nines) body size (pre-injury weight estimate)
Delivered through large bore peripheral IV Attempt to avoid overlying burned skin Can use venous cut down or central line
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RESUSCITATION FLUID NEEDS:FIRST 24 HOURS
Parkland Formula: Adults: 2-4 ml RL x Kg body weight x % burn Children: 3-4 ml RL x Kg body weight x % burn
First half of volume over first 8 hours, second half over following 16 hours Hypovolemia, decreased CO Increased capillary permeability Crystalloid fluid is keystone, colloid not useful
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RESUSCITATION FLUID NEEDS:SECOND 24 HOURS
Capillary permeability gradually returns to normal Colloid fluids started to minimize volume
Only necessary in patients with large burns (greater than 30% TBSA) 0.5 ml of 5% albumin x Kg body weight x % burn
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MONITORING OF RESUSCITATION
Actual volume infused will vary from calculates according to physiologic monitoring
Optimal regimen: minimizes volume & salt loading prevents acute renal failure low incidence of pulmonary & cerebral edema
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MONITORING OF RESUSCITATION
Urinary output is a reliable guide to end organ perfusion Adults: 30-50 ml per hour Children (less than 30 Kg): 1 ml/Kg per hour
Infusion rate should be increased or decreased by 1/3 if u/o falls or exceeds limits by more than 1/3 for 2-3 hours
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MANAGEMENT OF MYOGLOBINURIA & HEMOGLOBINURIA
High voltage electrical injury and mechanical trauma
Maintain urine output of 75-100 ml per hour Add 12.5 gm of Mannitol to each liter of fluid
Urine output not sustained Urine pigment does not clear
Sodium bicarbonate 1 amp (50 meq) per liter of fluid Heme pigments more soluble in alkaline urine
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MONITORING RESUSCITATION
Blood pressure:Can be misleading due to progressive
edema & vasoconstriction Heart Rate:
Tachycardia commonly observed Hemaglobin & hematocrit:
Not a reliable guide Transfusion not to be used for resuscitation
Baseline serum chemistries & arterial blood gasesBaseline to be obtained in burns of >30%
BSA
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MONITORING RESUSCITATION
CXR: daily for first 5-7 days Normal study in first 24 hours does not r/o
inhalation injury ECG:
All electrical injuries Pre-existing cardiovascular disease
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FLUID RESUSCITATION IN THE PEDIATRIC PATIENT
Require greater amounts of fluid Greater surface area per unit body mass
More sensitive to fluid overload Lesser intravascular volume per unit surface
area burned
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OVERVIEW
Burn Pathophysiology Initial Assessment & Management Airway Management & Inhalation
Injury Shock & Fluid ResuscitationBurn Wound Management Electrical Injuries Chemical Burns Pediatric Burns Other Topics
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DEPTH OF BURN
Partial ThicknessFirst degreeSuperficial second
degreeDeep second
degree
Full ThicknessThird degree
Ack 361
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TAR BURNS
Contact burns Bitumen is non-toxic Immediate cooling of
molten with cold H20 Removal of tar not an
emergency Cover with petroleum
based product & dressed to emulsify tar
Please Pass the Mayo!
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OVERVIEW
Burn Pathophysiology Initial Assessment & Management Airway Management & Inhalation
Injury Shock & Fluid Resuscitation Burn Wound ManagementElectrical Injuries Chemical Burns Pediatric Burns Other Topics
![Page 52: B URN AND M ANAGEMENT OF DIFFERENT TYPES OF B URNS Dr Muath Mustafa Dept of Surgery, BMC HOD. Dr. Ashraf Balbaa.](https://reader034.fdocuments.in/reader034/viewer/2022051619/56649dbc5503460f94aad8b4/html5/thumbnails/52.jpg)
ELECTRICAL INJURY Occurs when electricity is converted to heat
as it travels through tissue Divided into:
High voltage – greater than 1000 V Low voltage – less than 1000
Hands & wrists are common entrance wounds
Feet are common exit wounds
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ELECTRICAL INJURY
Extremely difficult to evaluate clinically Greatest tissue damage occurs under and
adjacent to contact points Superficial tissues cool more rapidly than the
deeper tissue Accounts for non-viable tissue beneath viable,
more superficial muscle
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TYPES OF TISSUE INJURY
Cutaneous Burn with no underlying tissue damage No passage of current through patient
Cutaneous Burn plus deep tissue damage Involving fat, fascia, muscle and/or bone
Muscle damage associated with myoglobin release Urine may be light red to “port wine” color Risk of kidney damage
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LIGHTNING INJURY
Direct current of >100 000 000 volts and up to 200 000 amps
Injury results from: Direct strike Side flash
Flow of current between person & nearby object Often travels on surface of body
Burns typically superficial “splashed on” spidery pattern
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MANAGEMENT OF ELECTRICAL INJURY
ABC’s Assess Injury
History LOC, cardiac arrythmia, other trauma
Physical Exam neuro exam, long bone #, dislocations, cervical spine
Maintain Patency of Airway Cardiac Monitoring:
Standard 12 lead ECG on admissionContinuous cardiac monitoring for first 24 hours
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MANAGEMENT OF ELECTRICAL INJURY:FLUID RESUSCITATION
Administer Ringer’s Lactate in amounts estimated with Parkland FormulaWill underestimate required volume due to
underlying tissue damage Increase fluids as per urine output
Examine urine for pigmentMaintain urine output 75-100 ml/hr until
clearAdd 1 amp (50 meq) per liter of RL to
alkalize urineMannitol 12.5 mg/liter to maintain urine
output
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MANAGEMENT OF ELECTRICAL INJURY: PERIPHERAL CIRCULATION Hourly monitoring of skin color, sensation,
capillary refill and peripheral pulses Remove all rings, watches, jewelry Surgical correction of vascular compromise
Decompression by escharotomy or fasciotomy Upper limb-volar & dorsal incisions with
protection of ulnar nerve Lower limb-medial & lateral incisions
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ELECTRICAL BURNS IN THE PEDIATRIC PATIENT
Low voltage accidents most common Generally household
(faulty insulation, frayed cords, insertion of metal object into wall socket)
Cutaneous injury, no muscle damage
Oral commisure injury Look worse than they are No initial debridement
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OVERVIEW
Burn Pathophysiology Initial Assessment & Management Airway Management & Inhalation
Injury Shock & Fluid Resuscitation Burn Wound Management Electrical InjuriesChemical Burns Pediatric Burns Other Topics
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CHEMICAL BURNS: CLASSIFICATION
AlkalisHydroxides, carbonates and caustic sodas of
sodium, ammonium, lithium, barium & calciumOven & drain cleaners, fertilizers, industrial
cleaners Acids
HCl, oxalic, muriatic & sulfuric acidsCommon in household & swimming pool cleaners
Organic CompoundsPhenols, creosote, petroleum productsContact chemical burns & systemic effects
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CHEMICAL BURNS
Factors That Determine Severity: Agent Concentration Volume Duration of contact
(delay in treatment)
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TREATMENT OF CHEMICAL BURNS Wear gloves and protective clothing Remove saturated clothing Brush skin if agent is a powder Irrigate, irrigate, irrigate!
Copious amounts of water Continued until pain or burning has decreased
Neutralization of agent contraindicated Generation of heat may lead to further injury
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OVERVIEW
Burn Pathophysiology Initial Assessment & Management Airway Management & Inhalation
Injury Shock & Fluid Resuscitation Burn Wound Management Electrical Injuries Chemical BurnsPediatric Burns Other Topics
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PEDIATRIC BURNS
Scald burns most common burn in < 3 years
Flame burns most common in children > 3 years
Always consider child abuse
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PEDIATRIC BURNS:PATHOPHYSIOLOGY Greater surface area per pound of body
weight Greater fluid needs Greater evaporative water loss Greater heat loss
Disproportionately thin skin Burns may be deeper than initially assessed Requires less exposure time to result in burn
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PEDIATRIC BURNS:AIRWAY
Intubation performed by someone experienced Larynx more cephalad
More acute angulation of the glottis Incuffed tube always used Cricothyroidotomy is never indicated Large bore needle placed through cricothyroid
membrane may be used in emergency cases
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PEDIATRIC BURNS:CIRCULATORY STATUS
Burn > 10% BSA should be hospitalized
IV Ringer’s Lactate is administered as per formula Must also add maintenance fluid (4-2-1
rule) NG tube Urinary catheter to monitor urine
output:<30 Kg: 1ml/Kg per hour>30 Kg: 30-50 ml per hour
If hypoglycemic, add 5% glucose to RL solution
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PEDIATRIC PATIENT:WOUND CARE Stop burning process Remove all clothing Topical antibiotics not indicated before
transfer Conserve heat with thermal blankets Escharotomy
Chest: ventilatory impairment Limb: vascular compromise
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OVERVIEW
Burn Pathophysiology Initial Assessment & Management Airway Management & Inhalation
Injury Shock & Fluid Resuscitation Burn Wound Management Electrical Injuries Chemical Burns Pediatric BurnsOther Topics
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RADIATION INJURY
Effects reproductive mechanism of certain tissue cells
Mature cells suffer less damage Stem cells are more vulnerable to injury Large doses of radiation (> 2000 RAD) may
lead to acute mortality
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OUTCOMES ASSOCIATED WITH RANGES OF WHOLE BODY RADIATION
Whole Body Dose(RAD) Response
20-100 Change in # of leukocytes
200-400 Severe reduction in leuks, N/V, hair loss, death due to infection
600- 1 000 Destruction of mone marrow, diarrhea, 50% mortality within 1 month
1 000-2 000 GI ulceration, death within 2 weeks
2 000+ Death within hours due to severe damage to CNS
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TYPES OF IONIZING RADIATION
Alpha particles Large, highly charged particles Associated with decay of natural radioactive
elements Penetrate only a few microns of tissue
Beta particles Positive electrons or negatively charged particles Penetrate approximately 1 cm of tissue
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TYPES OF IONIZING RADIATION
Gamma and X-rays Radioactive decay or x-ray machines Penetrate deeply Once removed from source, no further radiation
injury occurs Poses no threat to attendants
Protons, Deuterons, Neutrons, Mesons and Heavy Nuclei Produced by equipment for medical and industrial
use
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RADIATION BURNS Identical in appearance to thermal burns
Treat as you would a non-contaminated burn Differ from thermal burns from time between
exposure and clinical manifestation
SKIN RESPONSE TO RADIATION
200-300 (RADS) Epilation
300 Erythema
1000-2000 Transdermal Injury
2000 Radionecrosis
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COLD INJURIES:FROSTBITE
Formation of ice crystals in the tissue fluids Occurs in areas that lose heat rapidly Three degrees of frostbite:
First degree: painful white or yellow firm plaque Second degree: painful superficial clear or milky
blisters Third degree: deep red or purple blisters or skin
color that is markedly changed Severity influenced by both patient &
environment factors
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COLD INJURIES:TREATMENT OF FROSTBITE
Rapid re-warming in 4O degree water bath Avoid mechanical trauma - No massaging! Tetanus prophylaxis Escharotomy if vascularity compromised Tissue injury is often underestimated
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COLD INJURIES:HYPOTHERMIA
Defined as a core temperature < 34 degrees C Signs are vague & non-specific
May mimic other disease states Treatment:
Limit stimulation of patient –V.Fib easily induced Rapid re-warming in warm water bath Intubation to administer warm air Central administration of warm Ringer’s solution
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COLD INJURIES:HYPOTHERMIA
Monitor for systemic acidosis with serial ABGs Treat with sodium bicarbonate
Cardiopulmonary bypass Cardiac monitoring
Ventricular dysrhythmia Patients not to be declared dead until rewarmed
Continue CPR until core temperature> 36 degrees C. Secondary assessment for contributing diseases
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HYPERTHERMIA:CLINICAL SYNDROMES
Heat Cramps Result from excessive loss of salt by evaporation Experiences severe pain & cramping in muscles Tx: oral replacement of salt & water
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HYPERTHERMIA:CLINICAL SYNDROMES
Heat Exhaustion Consequence of inappropriate cardiovascular
response to stress of heat Diversion of blood to skin is not accompanied by
vasoconstriction to other areas or by volume expansion
Present with postural hypotension, profuse sweating, pallor, nausea, light-headedness
Tx: oral replacement or IV normal saline if severe
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HYPERTHERMIA:CLINICAL SYNDROMES
Heat Stroke Failure of body cooling mechanism
severe hyperpyrexia Setting of physical exercise w/o acclimatization Present with temperature>103, no sweating, decreased
LOC Tx: rapid cooling until temperature <102 deg If shivering develops, slowly give IV Thorazine DIC frequently reported
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THE END!