Asthma and COPD Lecture 4

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    Respiratory System

    Assessment & Disorders

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    Upper Respiratory System

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    Noses and Sinuses

    NoseBegin respiratory systemFilter and warm air

    SinusesOpenings in facial bonesLighten skull

    Assist in speechProduce mucus

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    Pharynx and Larynx

    PharynxNasopharynxOropharynxLaryngopharynx

    LarynxConnects laryngopharynx to tracheaRoutes air and food to properpassageway

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    Bronchi and Alveoli

    Trachea divides into right and leftmainstem bronchiBronchi continue to branch and getsmaller (bronchioles) and end asalveoli

    Air moves through passageways toalveoli where gas exchange occurs

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    Bronchioles and Alveoli

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    Pulmonary Circulation

    Pulmonary arteriesPulmonary veins

    Pulmonary capillary network

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    Pleura

    Double-layered membrane thatcovers lungs

    ParietalVisceral

    Hold lungs out to chest wall

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    Rib Cage and IntercostalMuscles

    Protect lungs12 pairs ribs

    Intercostal muscles are between ribs Assist with process of breathing

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    Ventilation

    Divided into inspiration and expirationNormal is 12 20 breaths per minute

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    Expiration

    Lasts 2 to 3secondsPassive

    Muscles relaxDiaphragm risesRibs descend

    Lungs recoilPressure in chestcavity increases

    (compressingalveoli)Pressure in lungshigher thanatmosphericcauses gases toflow out of the

    lungs

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    Factors Affecting Respiration

    Respiratory center of the brainChemoreceptors in the brain, aorticarch, and carotid arteries

    Airway resistanceCompliance

    ElasticitySurface tension of alveoli

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    Respiratory Changes Associated with Aging

    Cartilage that connects ribs tosternum and spinal cord calcifies

    Anterior-posterior diameter of chestincreasesRespiratory muscles weaker

    Cough and laryngeal reflexes lesseffective

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    Respiratory Changes Associated with Aging

    Size of lungs decreases Alveoli less elastic

    Older client at greater risk fordeveloping respiratory infections

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    Assessment

    SubjectiveCurrent complaint or existing conditionOnset or duration of symptoms

    Ability to maintain ADLNasal congestion, nosebleedsSore throat, difficulty swallowingChanges in voice qualityDifficulty breathing, orthopneaPain on breathing

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    Assessment (continued)

    SubjectivePresence of cough frequency, duration,productive or unproductiveSputum amount, color, and consistencyExposure to infections (colds orinfluenza)

    History of chronic lung conditionsOccupational exposure to chemicals,smoke, asbestos

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    Assessment (continued)

    SubjectiveHistory of previous respiratory problems

    Allergies to medication or environmentalallergensUse of tobacco, chewing tobacco,marijuana, cocaine, injected drugs, and

    alcohol

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    Assessment (continued)

    Objective Assess state of healthColorEase of breathingNote respiratory rate and patternObserve nasal flaringUse of accessory muscles for breathingListen for hoarseness in clients speech

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    Assessment (continued)

    ObjectiveInspect mucosa of nose, mouth, andoropharynxInspect neck, position of tracheaInspect anterior/posterior diameter ofchest

    Palpate lips for nodules, chest fortenderness or swelling

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    Pulse Oximetry

    Monitors oxygen saturation (SpO 2) Amount of arterial hemoglobin that iscombined with oxygen

    Nursing Care Apply to fingertip, forehead, earlobe, ornose

    Remove nail polish when using fingertip

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    Arterial Blood Gases

    Nursing care Apply pressure to site 2 5 minutesfollowing arterial puncture

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    Serum Alpha 1-Antitrypsin

    Deficiency in this serum proteincontributing factor in emphysema andCOPDNormal value in adults 150 350mg/dLFasting specimen obtained in clientwith elevated cholesterol ortriglycerides

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    Sputum and Tissue

    Throat or nose swabSputum specimen

    Culture and sensitivityGrams stain Acid-fast stain

    Cytology

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    Imaging Techniques

    X-raysCT scans

    Ventilation perfusion scansNursing care and client teachingIf contrast used remember to ask

    about allergies, especially iodine andseafood

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    Lung Volumes and Capacities

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    Direct Visualization

    Direct or indirect laryngoscopyUsed to identify and evaluate laryngealtumors

    Nursing care and client teachingMake sure consent form has beensignedRemove dentures, partial plates, bridgesprior to procedureNPO before procedureNPO after procedure until gag reflexreturns

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    Bronchoscopy

    Visualize trachea, bronchi andbronchioles

    Tumors and structural disorders

    Obtain tissue biopsyObtain sputum specimen

    Removal of foreign bodyNursing care and teaching

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    25/10/2010 34

    Asthma and COPD

    Dr Ibrahim Bashayreh, RN, PhD.

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    Asthma

    Asthma is achronicinflammatory

    pulmonary disorderthat ischaracterized byreversible

    obstruction of theairways

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    Asthma

    Asthma is a chronic (long-term)disease that makes it hard to breathe.

    Asthma can't be cured, but it can bemanaged. With proper treatment,people with asthma can lead normal,active lives.

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    Normal bronchiole/ Asthmatic bronchiole

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    How asthma works

    If you have asthma, your airways(breathing passages) are extra sensitive.When you are around certain things, your

    extra-sensitive airways can: Become red and swollen - your airwaysget inflamed inside. They fill up withmucus. The swelling and mucus make yourairways narrower, so it's harder for the airto pass through.

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    Cont.

    Become "twitchy" and go intospasm - the muscles around yourairways squeeze together and tighten.

    This makes your airways narrower,leaving less room for the air to passthrough.The more red and swollen yourairways are, the more twitchy theybecome.

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    What are the Triggering Factors?

    Domestic dustmites Air pollution

    Tobacco smokeOccupationalirritants

    Animal with furPollen

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    Asthma: Early Clinical

    ManifestationsExpiratory & inspiratory wheezingDry or moist non-productive coughChest tightnessDyspnea

    Anxious &AgitatedProlonged expiratory phaseIncreased respiratory & heart rate

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    Asthma: Early ClinicalManifestations

    Wheezing

    Chest tightness

    DyspneaCoughProlonged expiratory phase [1:3 or

    1:4]

    h l l

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    Asthma: Severe ClinicalManifestations

    HypoxiaConfusionIncreased heart rate & blood pressureRespiratory rate up to 40/minute & pursed lipbreathingUse of accessory muscles

    Diaphoresis & pallorCyanotic nail bedsFlaring nostrils

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    Asthma: Diagnostic Tests

    Pulmonary Function TestsFEV1 decreased

    Increase of 12% - 15% after bronchodilator indicative ofasthma

    PEFR decreased

    Symptomatic patienteosinophils > 5% of total WBC

    Increased serum IgEChest x-ray shows hyperinflation

    ABGsEarly: respiratory alkalosis, PaO2 normal or near-normal

    severe: respiratory acidosis, increased PaCO2,

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    Asthma: Nursing Diagnoses

    Ineffective airway clearance r/tbronchospasm, ineffective cough,excessive mucus

    Anxiety r/t difficulty breathing, fear ofsuffocationIneffective therapeutic regimenmanagement r/t lack of information aboutasthmaKnowledge deficit

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    Medical Management of AsthmaticPatient

    Limit exposure triggering agentsMedications such as: inhaledcorticosteroids, inhaled beta 2 adrenergic agonist, and cromolynsodium

    A h M di i A i

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    Asthma Medications: Anti-inflammatory

    CorticosteroidsNot useful for acute attackBeclomethasone: vanceril,beclovent, qvar

    Cromolyn & nedocromilInhibits immediate responsefrom exercise and allergens

    Prevents late-phase responseUseful for premedication forexercise, seasonal asthmaIntal, Tilade

    Leukotriene modifiersInterfere with synthesis orblock action of leukotrienes

    Have both bronchodilationand anti-inflammatorypropertiesNot recommended for acuteasthma attacksShould not be used as onlytherapy for persistentasthma

    Accolate, Singulair, Zyflo

    A h M di i

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    Asthma Medications:Bronchodilators

    2-adrenergic agonists Rapid onset: quick relief of bronchoconstriction

    Treatment of choice for acute attacksIf used too much causes tremors, anxiety, tachycardia,palpitations, nausea

    Too-frequent use indicates poor control of asthmaShort-acting

    Albuterol[proventil]; metaproterenol [alupent]; bitolterol [tornalate];pirbuterol [maxair]

    Long-acting

    Useful for nocturnal asthmaNot useful for quick relief during an acute attackSalmeterol [serevent]

    A th M di ti

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    Asthma Medications:Bronchodilators cont

    Methylxanthines Less effective than beta-adrenergics

    Useful to alleviatebronchoconstriction ofearly and late phase,nocturnal asthmaDoes not relievehyperresponsivenessSide effects: nausea,headache, insomnia,tachycardia, arrhythmias,seizuresTheophylline,aminophylline

    Anticholinergics Inhibit parasympatheticeffects on respiratorysystem

    Increased mucus

    Smooth musclecontractionUseful for pts w/adversereactions to beta-adrenergics or incombination w/beta-adrenergics

    Ipratropium [atrovent]

    Ipratropium + albuterol[Combivent]

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    Management of Asthmatic Patient

    Identify and assess status Avoid precipitating factorsBring inhaler for each appointmentDrug considerations: Avoid ASA, NSAIDs,barbiturates, and narcoticsDrug interactions with asthmatic medications (ex.Theophylline vs. Antibiotics, Cimetidine)Chronic corticosteroid users may require steroid

    supplementationFor sedation, nitrous oxide/oxygen and/or smalldoses of oral diazepam is recommended

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    Asthma: Client Teaching

    Correct use of medicationsSigns & symptoms of an attack

    Dyspnea, anxiety, tight chest, wheezing, cough

    Relaxation techniquesWhen to call for help, seek treatmentEnvironmental control

    Cough & postural drainage techniques

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    COPD

    Chronic obstructive pulmonarydisease is a slowly progressivedisease that is characterized by agradual loss of lung functionCOPD includes chronic bronchitis,chronic obstructive bronchitis, or

    emphysema, or combinations of theseconditions

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    Epidemiology

    20.3 million Americans report havingasthma5,000 deaths annually from asthma12.1 million Americans reported beingdiagnosed with COPD119,000 deaths annually from COPDCOPD is the 4 th leading cause ofdeath in the U.S.

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    Chronic Bronchitis

    Inflammation of themain airway passages(bronchi) to the lungs,which results in the

    production of excessmucous, a reduction inthe amount of airflowin and out of the lungs,

    and shortness ofbreath

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    Emphysema

    A respiratorydiseasecharacterized by

    breathlessnessbrought on by theenlargement, orover-inflation of,

    the air sacs(alveoli) in thelungs

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    Signs and symptoms

    WheezingCoughingSputum productionShortness of breathChest tightness

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    Nursing DX

    Ineffective breathing pattern r/tmusculoskeletal impairment , decreasedenergyInability to sustain spontaneousventilation r/t muscle fatigue

    Activity intolerance r/t imbalance of O2supply

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    Clinical Features of COPD Patients

    Mild COPD: no abnormal signs, smokerscough, little or no breathlessnessModerate COPD: breathlessness

    with/without wheezing, cough with/withoutsputumSevere COPD: breathlessness on anyexertion/at rest, wheeze and coughprominent, lung inflation usual, cyanosis,peripheral edema, and polycythemia inadvanced disease

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    Diagnosis

    SpirometryBreathing test which measures the amount and rate atwhich air can pass through the airways

    Bronchodilator Reversibility Testing

    Relaxing tightened muscles around the airways andopening up airways quickly to ease breathing

    Other pulmonary function testingDiffusion capacity

    Chest X-ray Arterial Blood Gas

    Shows oxygen level in blood

    Medical Management of COPD

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    Medical Management of COPDPatient

    Smoking cessation and elimination ofenvironmental pollutantsPalliative measure such as regularexercise, good nutrition, flu andpneumonia vaccinesBronchodilators, corticosteroids,anticholinergics, and NSAIDs

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    Management of COPD Patient

    Review history for concurrent heart disease Avoid treatment if upper respiratory tract infection ispresentTreat in upright position

    Avoid rubber dam in severe casesUse pulse oximetry (if pulse ox

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