Asfiksia & Hie
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ASFIKSIA & HIE
Kelompok Kerja PerinatologiRSAB Harapan Kita
Jakarta
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newly born newborn infant
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KEMATIAN BAYIKEMATIAN BAYI
( < 1 ( < 1 tahuntahun ))
8 8 jutajuta
KEMATIAN NEONATUSKEMATIAN NEONATUS
( ( 28 28 harihari ))
5 5 jutajuta
KEMATIAN NEONATUS DINIKEMATIAN NEONATUS DINI
( ( 7 7 harihari ))
3,4 3,4 jutajuta
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KEMATIAN NEONATUS DI INDONESIA
ANGKA KEMATIAN BAYI ( IMR )ANGKA KEMATIAN BAYI ( IMR )
45,7 / 1000 45,7 / 1000 lahirlahir hiduphidup
50 %50 %
NeonatusNeonatus dinidini
47 %47 %
neonatusneonatus
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PENYEBAB KEMATIAN NEONATUS
Asfiksia + trauma lahir29%
Prematur24%
Kongenital10%
Lain-lain5%
Infeksi32%
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BAYI BARU LAHIR
masa transisiIntra uterin ekstra uterin
respirasikardiovaskuler
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Definisi asfiksia keadaan bayi baru lahir yang gagal
bernapas spontan dan teratur segerasetelah lahir
Hutchinson 1967
disertai hipoksia, hiperkapnia dan asidosis
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KESULITAN TRANSISI Bayi tidak bernapas dengan kuatparu
tak terisi udara dan oksigen tak masukdarah
Kehilangan darah dan bradikardia karenahipoksia menyebabkan hipotensi sistemik
Konstriksi arteriol karena hipoksia akanmenyebabkan hipertensi pulmonalpersisten
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POLA KLINIS ASFIKSIA APNOE PRIMER: setelah pernapasan
awal yg cepat,disertai penurunanfrekuensi jantung, tekanan darah normal dan perangsangan dapat memulaipernapasan
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POLA KLINIS ASFIKSIA APNOE SEKUNDER:setelah napas
megap2, disertai penurunan frekuensijantung dan tekanan darah; rangsangansaja tak cukup untuk memulai pernapasan
Dawes 1968
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TANDA KLINIS BAHAYA SIANOSIS : karena kekurangan oksigen
darah BRADIKARDIA: karena kekurangan
oksigen di otot jantung dan otak Tekanan darah yang rendah karena
kekurangan oksigen dan kehilangan darah
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Perubahan pentingpada asfiksia
Hipoksia Hiperkapnia Asidosis respiratorik dan metabolik Glikogenolisis untuk metabolisme anaerob Perubahan sistem kardiovaskular
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BAYI LAHIR DENGAN ASFIKSIA
menunjukkan satu / > tanda klinis : Sianosis Bradikardi Hipotensi Depresi pernapasan Hipotoni
Apnoe primer dan apnoe sekunder
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tatalaksana
RESUSITASI Adekuat Jangan tunda
PENCEGAHAN antisipasi oksigen antenatal pada ibu persalinan emergensi konseling
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prognosis
Tergantung lamanya bayi tidak bernafas Organ yg dipengaruhi
Otak Hati Ginjal Paru Usus
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HYPOXIC-ISCHAEMIC ENCEPHALOPATHY
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sinonim
HIE Newborn encephalopathy Neonatal encephalopathy NE HIE-NE Birth asphyxiation Hypoxic-ischaemic brain injury BILIRUBIN ENCEPHALOPATHY mirip NE
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NCB cortex cerebri nuclei cerebri
NKB substantia alba IVH
Terjadi fetal,antental,intranatal
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National Colaborative Perinatal Project
Aktifitas yg kurang setelah hari pertama Perlu inkubator lebih dari 3 hari Problem minum Refleks isap buruk Gangguan pernapasan
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patologi
perfusi dan oksigenisasi otak dibawah batas kritis
hipoksemia dan hiperkarbia gangguan miokard iskemia neuron
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patofisiologi
Hiposemia perinatal primer Iskemi perinatal
diperburuk oleh hipoglikemia & hipertermia Gangguan autoregulasi sirkulasi otak Excitotoxicity
glutamat
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riwayat
problem persalinan dan melahirkan kesulitan resusitasi gangguan kesadaran gangguan ventilasi kejang
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NKB (terutama KMK) PVL hipotensi ibu kehamilan kembar aritmia ibu/janin gangguan kardiorespirasi; TD< 30 asidosis gagal jantung
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klasifikasi
SARNAT n SARNAT Stage1: kurang tidur, simpatis yg aktif, reaksi
berlebih thd stimuli, refleks isap kurang walau tonus otot baik, kelainan EEG (1 hari)
Stage 2: hipotonia,letargi, kejang, parasimpatis aktif, FJ < 120,peristaltik naik, kelainan EEG (2-14 hari)
Stage 3: stupor, posisi deserebrasi, hipotonia, refleks menghilang, refleks primitif jelas
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klasifikasi..
MILLER Dgn sistem skor. Makin besar makin buruk Yg dinilai: minum, kesadaran, tonus, status
pernafasan, refleks dan kejang Skor pada hari 3 dipakai sebagai faktor
prognostik Lipper postasphyxiation index: usia 48 jam
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tatalaksana
mencegah asfiksia, hipoperfusi tatalaksana cairan: hindari overload bantuan respirasi atasi kejang atasi hipokalsemia dan hipoglikemia
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