Asfiksia & Hie

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  • ASFIKSIA & HIE

    Kelompok Kerja PerinatologiRSAB Harapan Kita

    Jakarta

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  • newly born newborn infant

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  • KEMATIAN BAYIKEMATIAN BAYI

    ( < 1 ( < 1 tahuntahun ))

    8 8 jutajuta

    KEMATIAN NEONATUSKEMATIAN NEONATUS

    ( ( 28 28 harihari ))

    5 5 jutajuta

    KEMATIAN NEONATUS DINIKEMATIAN NEONATUS DINI

    ( ( 7 7 harihari ))

    3,4 3,4 jutajuta

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  • KEMATIAN NEONATUS DI INDONESIA

    ANGKA KEMATIAN BAYI ( IMR )ANGKA KEMATIAN BAYI ( IMR )

    45,7 / 1000 45,7 / 1000 lahirlahir hiduphidup

    50 %50 %

    NeonatusNeonatus dinidini

    47 %47 %

    neonatusneonatus

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  • PENYEBAB KEMATIAN NEONATUS

    Asfiksia + trauma lahir29%

    Prematur24%

    Kongenital10%

    Lain-lain5%

    Infeksi32%

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  • BAYI BARU LAHIR

    masa transisiIntra uterin ekstra uterin

    respirasikardiovaskuler

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  • Definisi asfiksia keadaan bayi baru lahir yang gagal

    bernapas spontan dan teratur segerasetelah lahir

    Hutchinson 1967

    disertai hipoksia, hiperkapnia dan asidosis

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  • KESULITAN TRANSISI Bayi tidak bernapas dengan kuatparu

    tak terisi udara dan oksigen tak masukdarah

    Kehilangan darah dan bradikardia karenahipoksia menyebabkan hipotensi sistemik

    Konstriksi arteriol karena hipoksia akanmenyebabkan hipertensi pulmonalpersisten

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  • POLA KLINIS ASFIKSIA APNOE PRIMER: setelah pernapasan

    awal yg cepat,disertai penurunanfrekuensi jantung, tekanan darah normal dan perangsangan dapat memulaipernapasan

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  • POLA KLINIS ASFIKSIA APNOE SEKUNDER:setelah napas

    megap2, disertai penurunan frekuensijantung dan tekanan darah; rangsangansaja tak cukup untuk memulai pernapasan

    Dawes 1968

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  • TANDA KLINIS BAHAYA SIANOSIS : karena kekurangan oksigen

    darah BRADIKARDIA: karena kekurangan

    oksigen di otot jantung dan otak Tekanan darah yang rendah karena

    kekurangan oksigen dan kehilangan darah

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  • Perubahan pentingpada asfiksia

    Hipoksia Hiperkapnia Asidosis respiratorik dan metabolik Glikogenolisis untuk metabolisme anaerob Perubahan sistem kardiovaskular

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  • BAYI LAHIR DENGAN ASFIKSIA

    menunjukkan satu / > tanda klinis : Sianosis Bradikardi Hipotensi Depresi pernapasan Hipotoni

    Apnoe primer dan apnoe sekunder

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  • tatalaksana

    RESUSITASI Adekuat Jangan tunda

    PENCEGAHAN antisipasi oksigen antenatal pada ibu persalinan emergensi konseling

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  • prognosis

    Tergantung lamanya bayi tidak bernafas Organ yg dipengaruhi

    Otak Hati Ginjal Paru Usus

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  • HYPOXIC-ISCHAEMIC ENCEPHALOPATHY

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  • sinonim

    HIE Newborn encephalopathy Neonatal encephalopathy NE HIE-NE Birth asphyxiation Hypoxic-ischaemic brain injury BILIRUBIN ENCEPHALOPATHY mirip NE

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  • NCB cortex cerebri nuclei cerebri

    NKB substantia alba IVH

    Terjadi fetal,antental,intranatal

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  • National Colaborative Perinatal Project

    Aktifitas yg kurang setelah hari pertama Perlu inkubator lebih dari 3 hari Problem minum Refleks isap buruk Gangguan pernapasan

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  • patologi

    perfusi dan oksigenisasi otak dibawah batas kritis

    hipoksemia dan hiperkarbia gangguan miokard iskemia neuron

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  • patofisiologi

    Hiposemia perinatal primer Iskemi perinatal

    diperburuk oleh hipoglikemia & hipertermia Gangguan autoregulasi sirkulasi otak Excitotoxicity

    glutamat

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  • riwayat

    problem persalinan dan melahirkan kesulitan resusitasi gangguan kesadaran gangguan ventilasi kejang

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  • NKB (terutama KMK) PVL hipotensi ibu kehamilan kembar aritmia ibu/janin gangguan kardiorespirasi; TD< 30 asidosis gagal jantung

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  • klasifikasi

    SARNAT n SARNAT Stage1: kurang tidur, simpatis yg aktif, reaksi

    berlebih thd stimuli, refleks isap kurang walau tonus otot baik, kelainan EEG (1 hari)

    Stage 2: hipotonia,letargi, kejang, parasimpatis aktif, FJ < 120,peristaltik naik, kelainan EEG (2-14 hari)

    Stage 3: stupor, posisi deserebrasi, hipotonia, refleks menghilang, refleks primitif jelas

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  • klasifikasi..

    MILLER Dgn sistem skor. Makin besar makin buruk Yg dinilai: minum, kesadaran, tonus, status

    pernafasan, refleks dan kejang Skor pada hari 3 dipakai sebagai faktor

    prognostik Lipper postasphyxiation index: usia 48 jam

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  • tatalaksana

    mencegah asfiksia, hipoperfusi tatalaksana cairan: hindari overload bantuan respirasi atasi kejang atasi hipokalsemia dan hipoglikemia

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