Angina & Dysrhythmias

62
Angina & Dysrhythmias

description

Angina & Dysrhythmias. A & P OF THE CARDIAC SYSTEM. Cardiac output CO=SV(stroke volume) X HR(heart rate) Preload Volume of blood in the ventricles at the end of diastole Afterload - PowerPoint PPT Presentation

Transcript of Angina & Dysrhythmias

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Angina & Dysrhythmias

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A & P OF THE CARDIAC SYSTEM

Cardiac output CO=SV(stroke volume) X HR(heart rate)

Preload Volume of blood in the ventricles at the end of

diastole Afterload

Peripheral resistance against which the left ventricle must pump. Affected by the size of the ventricle, wall tension and arterial blood pressure.

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ANGINA PECTORIS

Definitions Signs and Symptoms Stable vs Unstable Silent Ischemia Precipitating factors Diagnostic studies

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Lewis Page 812

Precipitating

Factors of Angina

PRECIPITATING FACTORS OF ANGINA

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CARDIAC DRUGS

Nitrates

Beta Blockers

Calcium Channel blockers

Ace inhibitors

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Lewis fig 33-13

Decision tree

Collaborative care

Coronary Artery Disease Collaborative care decision tree

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MYOCARDIAL INFARCTION

Definition Signs and Symptoms Complications Diagnostics PTCA (Percutaneous Coronary

Angioplasty) CABG (Coronary Artery Bypass)

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MYOCARDIAL INFARCTION

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NURSING DIAGNOSIS

Acute pain Ineffective Tissue perfusion (cardiac) Anxiety Activity intolerance Ineffective Therapeutic regimen

management

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PTCA

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CORONARY ARTERY BYPASS GRAFT

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CARDIOMYOPHATHY

Dilated (congestive)

Hypertrophic

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Types of cardiomyopathies

See differences in ventricle muscles

Types of Cardiomyopathies

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CARDIAC TRANSPLANT

Drug therapies

Lifestyle changes

Support Systems

Teaching

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INFLAMMATORY HEART DISEASE NURSING DIAGNOSIS

Hyperthermia Decreased Cardiac Output Activity Intolerance Ineffective Health Maintenance Pain Anxiety

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Layers of the heart Page 886

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INFLAMMATORYHEART DISEASES

Infective EndocarditisClinical

manifestationsDiagnostic

studies

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INFLAMMATORY HEART DISEASE

ACUTE PERICARDITISEtiology and PathophysiologyClinical ManifestationsComplicationsCollaborative Care

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MYOCARDITIS

Etiology

Clinical Manifestations

Diagnostic Studies

Nursing Management

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VALVULAR DISEASE

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NURSING DIAGNOSIS

Activity Intolerance Excess Fluid Volume Decreased Cardiac

Output Ineffective

Therapeutic Regimen Management

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MITRAL VALVEStenosis and Regurgitant

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MITRAL VALVE STENOSIS

Etiology Clinical Manifestations Teaching Diagnostic findings

ECGEchocardiogramCardiac Catheterization

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RELATIONSHIP OF ECG TO CARDIAC CYCLE

Page 767, Chapter 31

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ATRIAL STENOSIS & REGURGIATION

Etiology Clinical Manifestation Diagnostic Findings

ECGEchocardiogramCardiac Catheterization

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VALVULAR HEART DISEASECOLLABORATIVE CARE (PTBV) Percutaneous transluminal balloon

valvuplasty

Surgical therapy

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TYPES OF PROSTHETIC AND TISSUE VALVES

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ARRHYTHMIAS

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Chapter 31, pages 758 & 759

Normal sinus rhythm

NORMAL SINUS RHYTHM

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NORMAL SINUS RHYTHMTIMING

Page 865

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Normal Sinus Rhythm in Lead II

Artifact, loose leads or movement

Rate of conduction systems

NSR, ARTIFACT, CONDUCTION SYSTEM RATES

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Calculating Rhythm and RateUsing 6 second cardiac strip

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REGULAR RHYTHM & RATE

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Sinus bradycardia

Sinus Tachycardia

Premature Atrial Contraction (PAC)

Paroxysmal SVT

SINUS BRADYCARDIATACHYCARDIA, PAC, SVT

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HEART BLOCKS PAGE 871

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Atrial Flutter

Atrial Fibrillation

Page 870What medications would you anticipate the patient to be taking chronically for this rhythm? Why?

ATRIAL FLUTTER AND FIBRILLATION

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Multifocal PVC

PVC’s couplets, runs

PVC bigeminy

PVC Trigeminy

What would you assess first?

PVC, BIGEMINY, TRIGEMINY

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Ventricular Tachycardia

Ventricular

Fibrillation

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V-TACH, V-FIB

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Pulseless Electrical Activity

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PORTABLE MONITOR

Contains Monitor,

Defibrillator,

Transcutaneous

Pacemaker,

Page 875

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Placement of defibrillation pads

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Implantable Defibrillator (ICD) and Dual Chamber Pacemaker

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Temporary Pacemaker Insertion

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