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    ANEURISMSBINAL J

    M. SC. N

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    ANEURYSM

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    DEFINITION

    Aneurysm is a localized, blood-filled balloon-like bulgeof a blood vessel. Aneurysms can commonly occur in

    the base of the brain (the circle of Willis) and an aortic

    occurs in the main artery carrying blood from the left

    the heart.

    When the size of an aneurysm increases, there is a sig

    of rupture, resulting in severe hemorrhage, other comp

    death.

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    DEFINITION: AORTIC ANEURISM

    Abnormal dilation of a blood vessel at a site of we

    or a tear in the vessel wall.

    Usually secondary to atherosclerosis.

    Most commonly affect the aorta

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    CLASSIFICATION

    TRUE

    ANEURISM

    FALS

    ANEUR

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    TRUE ANEURYSM

    A true aneurysm is one that involves all threof the wall of an artery (intima, media and

    adventitia).

    True aneurysms include atherosclerotic, syp

    and congenital aneurysms, as well as ventraneurysms that follow transmural myocardia

    infarction

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    FALSE ANEURYSM :

    A false aneurysm or pseudo-aneurys

    not primarily involve such distortion

    vessel.

    It is a collection of blood leaking comout of an artery or vein, but confined

    the vessel by the surrounding tissue.

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    FALSE ANEURYSMS

    May result from

    Trauma

    Infection

    After peripheral artery bypass graft surgery at siteanastomosis

    Arterial leakage after cannulae removal

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    MACROSCOPIC SHAPE AND SIZE :

    FUSIFORM

    ANEURISM

    SACCU

    ANEUR

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    FUSIFORM ANEURISM:

    Fusiform Fusiform ("spindle-shaped") an

    are variable in both their diameter and leng

    diameters can extend up to 20 cm (8 in). Th

    involve large portions of the ascendtransverse aortic arch, the abdominal aorta

    frequently the iliac arteries

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    Fusiform

    Most AAA are fusiform and 98% are below the

    renal artery

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    SACCULAR ANEURYSM

    Saccular aneurysms are spherical in

    and involve only a portion of the vess

    they vary in size from 5 to 20 cm (8

    diameter, and are often filled, either par

    fully, by thrombus.

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    ACCORDING TO LOCATION :

    Aortic ThoracicArterial and

    venousCerebral

    aneurysmPeripheral

    Aneurysms

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    CLINICAL MANIFESTATIONS

    Frequently asymptomatic

    May have sub sternal, neck or back pain

    Coughing, due to pressure placed on the windpipe (trachea)

    Hoarseness

    Difficulty swallowing Swelling (edema) in the neck or arms

    Myocardial infarction, or stroke due to dissection or rupture inbranches of the aorta

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    DIAGNOSIS

    X-rays- Most are diagnosed without

    symptoms on routine X-ray

    Chest - Demonstrate abnormal widening of

    thoracic aorta

    Abdomen -May show calcification within wall of

    AAA

    ECG -to rule out MI

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    DIAGNOSIS

    Echocardiography

    Assists in diagnosis of aortic valve insufficiency

    Related to ascending aortic dilation

    Ultrasonography Useful in screening for aneurysms

    Monitor aneurysm size

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    DIAGNOSIS

    CT scan

    Most accurate test to determine

    Anterior to posterior length

    Cross-sectional diameter

    Presence of thrombus in aneurysm

    MRI

    Diagnose and assess the location and severity

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    DIAGNOSIS

    Angiography

    Anatomic mapping of aortic system using contr

    Not reliable method of determining diameter or

    Can provide accurate info about involvement of

    intestinal, renal or distal vessels

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    AORTIC ANEURYSM

    CLINICAL MANIFESTATIONS

    May mimic pain associated with abdominal or back dis

    Pain correlates to the size- can be excruciating

    May spontaneously embolize plaque

    Causing blue toe syndrome patchy mottling of feet/toes withof palpable pedal pulses

    It can rupture, causing shock and death in 50% of ruptu

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    COMPLICATION

    AORTIC DISSECTION

    Blood invades or dissects the layers of the vessel wall- m

    thoracic

    Dissecting aneurysms are unique and life

    threatening. A break or tear in the tunica intima

    and media allows blood to invade or dissect thelayers of the vessel wall. The blood is usually

    contained by the adventitia, forming a saccular

    or longitudinal aneurysm.

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    http://images.google.com/imgres?imgurl=http://www.brown.edu/Courses/Digital_Path/systemic_path/cardio/CV74.JPG&imgrefurl=http://www.brown.edu/Courses/Digital_Path/systemic_path/cardio/iliac.html&h=504&w=600&sz=133&hl=en&start=7&um=1&tbnid=bTl0DNh435wf0M:&tbnh=113&tbnw=135&prev=/images?q=aortic+dissection&ndsp=20&svnum=10&um=1&hl=en&rlz=1T4ADBS_enUS220US220&sa=N
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    Affects men more often thanwomen

    Occurs most frequently between

    fourth and seventh decades of

    lifeAcute and life threatening

    Mortality rate 90% if not

    medically or surgically treated

    M if t ti f A ti D i

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    Manifestations of Aortic D isse

    Aneurysm Abrupt, severe, ripping or

    tearing pain in area of

    aneurysm Mild or marked

    hypertension early

    Weak or absent pulses andblood pressure in upperextremities

    S yncope

    C omplications: hemorrhage,ischemic kidneys (renalfailure), MI, heart failure,cardiac tamponade, sepsis ,weakness or paralysis oflower extremities .

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    AORTIC DISSECTION

    COLLABORATIVE CARE

    Initial goal BP and myocardial contractility to diminish pulsatile forces within a

    Drug therapy

    IV -adrenergic blocker

    Esmolol (Brevibloc)

    Other hypertensive agents Calcium channel blockers

    Sodium Nitroprusside

    Angiotensin-converting enzyme

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    AORTIC DISSECTION

    Surgical therapy

    When drug therapy is ineffective

    or

    When complications of aortic dissection are present

    Heart failure, leaking dissection, occlusion of an artery

    Surgery may be delayed to allow edema to decrease

    and permit clotting of blood.

    Even with prompt surgical intervention

    30-day mortality of acute aortic dissections remains high(10%-28%)

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    Stent Graft Repair

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    NURSING MANAGEMENT: ASSESSME

    Cardiovascular status

    Continuous ECG monitoring

    Electrolyte monitoring

    Arterial blood gas monitoring

    Oxygen administration

    Antidysrhythmic/pain

    medications

    Renal perfusionstatus

    Urinary output

    Fluid intake

    Daily weight

    CVP/PA pressu

    Blood urea

    nitrogen/Creatin

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    ASSESSMENT

    Infection

    Antibiotic admin

    Assessment of btemperature

    Monitoring of WAdequate nutriti

    Observe surgicafor signs of infec

    Gastrointestinal status

    Nasogastric tube

    Abdominal assessment

    Passing of flatus is key

    sign of returning bowel

    function

    Watch for manifestations

    of bowel ischemia

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    Neurologic status

    Level of consciousness

    Pupil size and response to light

    Facial symmetry

    Speech

    Ability to move upper

    extremities

    Quality of hand grasps

    Peripheral perfu

    Pulse assessme

    Mark pulse lofelt-tip pen

    Extremity asses

    (5Ps) Tempecapillary refillsensation anof extremities

    ASSESSMENT

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    NURSING DIAGNOSIS

    Risk for IneffectiveTissue Perfusion

    Risk for Injury

    Anxiety

    Pain

    Knowledge Deficit

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    NURSING MANAGEMENT

    Ambulatory and Home Care

    Encourage patient to express concerns

    Patient instructed to gradually increase activities

    No heavy lifting

    Educate on signs and symptoms of complications Infection

    Neurovascular changes

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    1.Ultrasound is extremely effective at detecting AAAs.The UPreventive Services Task Force (USPSTF) recommends that 65 to 75 who has ever smoked undergo a one-time ultrasouscreening for AAA

    2.Prevent atherosclerosis

    3.Treat and control hypertension

    4.Diet- low cholesterol, low sodium and no stimulants

    5.Careful follow-up if less than 5cm. It can grow .5cm /year

    PREVENTION

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    OTHER COMPLICATIONS

    Rupture- signs of ecchymosis (triad) Back pain

    Hypotension

    Pulsating mass

    Thrombi

    Renal Failure

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    RUPTURE TRIAD

    Back

    pain

    HypotensionPulsatinghematoma

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