Adapted From Temple College EMS Professions. Poisons Substance which when introduced into body in...
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Transcript of Adapted From Temple College EMS Professions. Poisons Substance which when introduced into body in...
Poisons Substance which when introduced into
body in relatively small amounts causes in structural damage or functional disturbances
Suspect with: GI signs/symptoms (nausea, vomiting,
diarrhea, pain) Altered LOC, seizures, unusual behavior Pupil changes, salivation, sweating, other
signs/symptoms of disturbed autonomic nervous system function
Respiratory depression Burns, blisters of lips, mucous membranes Unusual breath odors
Treat Patient, Not Poison Proper support of ABCs is first step in
management Contact with Poison Control Center Priority action plan Symptomatic treatments Time management
Try to determine: What? How much? How long ago? What has already been done? Psychiatric history? Underlying illness?
When in doubt. . .
Assume containers were full Entire contents were ingested Patient may not be telling you the truth
If several patients involved. . .
Assume each ingested entire container contents
Triage Additional resources
Ingested Poisons
Prevent absorption of toxin from GI tract into bloodstreamPositioningRapid Transport to definitive treatment
center
Absorbed Poisons
Dry chemicalsdust skin, then wash
Liquid chemicalswash with large amounts of H20
avoid “neutralizing” agents
CAUTION Don’t accidentally expose
yourself!
Inhaled Poisons
Remove patient from exposure Maximize oxygenation, ventilation
CAUTION Don’t accidentally expose
yourself!
Injected Poisons
Attempt to slow absorption Venous constricting bands Dependent position Splinting of injected body part Cold packs (+) [May worsen local injury by
concentrating poison]
Substance Abuse
Self administration of a substance in a manner not in accord with approved medical or social practices
Substance Abuse
Psychological dependence Physical dependence Compulsive drug use Tolerance Addiction
Compulsive Drug Use
Use of drug and rituals/culture associated with its use become an overwhelming desire
Tolerance
Increasing amounts of drug needed to produce same effects
Tolerance contributes to addiction by keeping user “chasing the last high”
Addiction
Combination of psychological dependence, physical dependence, compulsive use, and tolerance
Patient becomes totally consumed with obtaining, using drug to exclusion of all other things
Ethyl AlcoholA CNS Depressant Drug
Decreased ReactionTime
Increased AccidentalTrauma Risk
Decreased SocialInhibitions
Increased IntentionalTrauma Risk
Potentiation of OtherCNS Depressants
Lethal Overdoses inCombination with OtherDrugs
Slowed GI TractActivity
Irritation, Gastritis,Ulcer Disease, GIBleeds
Toxic Overdose RespiratoryDepression, Shock
Ethanol Intoxication Signs
Breath odor Swaying, unsteadiness Slurred speech Nausea, vomiting Flushed face Drowsiness Violent, erratic behavior
Ethanol
Clouds signs, symptoms Complicates assessment Head trauma, diabetes, drug toxicity, CNS
infection can mimic EtOH intoxication and vice versa
Experience alcohol withdrawal syndrome if they reduce intake: Restlessness, tremulousness Hallucinations Seizures Delirium tremens--all of above plus tachycardia,
nausea, vomiting, hypertension, elevated body temperature
Alcohol Addicts
Life threatening condition! Occurs 1 days to 2 weeks after intake is
decreased 5 to 15% mortality Control airway, prevent aspiration, monitor
for hypovolemia
Delirium Tremens
Narcotics Withdrawal
AgitationAnxietyAbdominal painDilated pupils
–Sweating–Chills–Joint pains–Goose flesh
Resembles severe influenza
Not a life-threat
Induce sleepiness, state similar to EtOH intoxication
Medical usesAnestheticsSedativeHypnotics
Barbiturates
Barbiturate-like Non-barbiturates Doriden, Placidyl, Quaalude, Methyprylon Effects similar to barbiturates Overdose can cause sudden, very
prolonged respiratory arrest Withdrawal resembles ETOH; extremely
dangerous
Valium, Librium, Miltown, Equanil, Tranxene
Low doses relieve anxiety, produce muscle relaxation
High doses produce barbiturate-like effects
Tranquilizers
Overdose: Unlikely to cause respiratory arrest alone Extremely dangerous with EtOH
WithdrawalResembles EtOH withdrawalExtremely dangerous
Tranquilizers
CNS Stimulants: Amphetamines Dexedrine, Benzedrine, Methyl
amphetamine Relieve fatigue, promote euphoria,
reduce appetite
CNS Stimulants: Amphetamines Overdose
Restlessness, paranoiaTachycardiaHypertension CVA, Heart failureHyperthermia Heat stroke
WithdrawalLethargyDepression
Stronger stimulant effects than amphetamines
Can cause respiratory/cardiovascular failure, heat stroke, lethal arrhythmias
CNS Stimulants: Cocaine
“Snorting” can destroy nasal septum, cause massive nosebleed
Airway issue Withdrawal:
lethargydepression
CNS Stimulants: Cocaine
LSD, psilocybin, peyote, mescaline, DMT, MDMA
Enhance perception Wrong setting may induce “bad trips” with
extreme anxiety True toxic overdose rare
Hallucinogens
Phencyclidine
PCP, angel dust Produces bizarre, violent behavior Reduces pain sensation Patients may be capable of feats of
extreme strength Keep patient in quiet environment,
minimize stimulatin
Glue, paint, gas, light fluid, toluene Inhalation produces state similar to EtOH
intoxication Patient may asphyxiate if consciousness
lost while “sniffing”
Solvents