1228 Class 17 Cad

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    Nursing Management: Coronary

    Artery Disease

    NURS 1228

    Class 17

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    Terms to Know

    Angina Pectoris

    Laser Angioplasty

    Atherectomy Plaque

    Atherosclerosis

    Prinzmetals Angina

    Collateral Circulation

    PTCA

    Coronary Artery

    Disease

    Stent Placement

    Low fat diet

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    Medications: Familiarize

    Yourself! AntiplateletAggregation

    Nitrates

    Beta-adrenergic

    blockers

    (b adrenergic blocker)

    Calcium Channel

    Blockers

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    Diagnostic Tests: Familiarize

    Yourself! Chest X-ray Lipid Level

    ECG Echocardiography

    Exercise Stress Test

    CK

    Nuclear Imaging

    Studies

    LDH

    Position Emission

    Tomography

    Cardiac Troponin

    Coronary Angioplasty

    Studies

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    Coronary Artery Disease

    What is it? How does it Happen?

    Atherosclerosis:

    Stages of development:Fatty Streak

    Raised Fibrous Plaque

    (Smooth muscle cellproliferation)

    Complicated Lesion

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    Plaque Build Up

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    Collateral Circulaton

    What is it?

    How is it formed?

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    Coronary Circulation

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    Risk Factors

    Modifiable

    Diet: Serum lipid levels

    Smoking (You would

    guess this one, wouldnt

    you??!) Hypertension (stress does

    it, too!!)

    Physical Inactivity (sorry,

    student nurses)

    Obesity

    Unmodifiable

    Age and gender

    Family history and

    heredity

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    Modifiable ContributingRisk

    Factors

    Diabetes Mellitus

    Stress and behavior patterns This guys an MI and a cardiac

    arrest waiting for a chance to

    happen!! (To say nothing of a

    CVA which is not discussed inthis class).

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    Health Promotion

    Identify and Manage High Risk Persons

    Exercise: Begin young

    Drug Therapy Questran and Colestid

    B vitamins

    Lopid, Zocor, Lipitor, etc.

    See table 32-7 on p.852

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    CAD: What does it look like?

    Insufficient blood supply to the heart itself

    The heart muscle does not receive theoxygenated blood it requires

    Atherosclerotic stenosis

    Coronary artery spasm

    Coronary thrombosis

    All result in Myocardial IschemiaEither silent or symptomatic (angina)

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    Angina

    When an occlusion occurs:

    The myocardial cells do not get the glucose needed for

    aerobic metabolism

    Anaerobic metabolism takes over; lactic acidproduced (hence the pain)

    Myocardial cells can live about 20 minutes without

    oxygen. When oxygen is restored, aerobic

    metabolism returns and things return to normal. See p. 852 for precipitating factors that may be

    connected with myocardial ischemia

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    Types of Angina

    Stable Angina

    Infrequent and controlled with medication; predictable

    Unstable Angina

    Unpredictable with less pattern discernable Associated with seterioration of once stable

    atherosclotic plaque.

    Thrombus formation occurs; can progress to an MI

    (Myocardial Infarction) Treated with ASA and anticoagulants along with nitrates and

    Beta Blockers.

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    Types of Angina Continued

    Prinzmetals Angina

    Occurs at rest; major coronary artery goes intospasm.

    Patient may not have known CAD

    May occur at night especially during REMsleep

    May be cyclicalConfirmed with Coronary Angiography

    (detects obstruction of coronary arteries)

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    Types of Angina Continued

    Nocturnal Angina

    Occurs only at night

    Awake or asleepSitting up or laying down

    Angina Decubitus

    While the patient is lying downRelieved by standing or sitting

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    What does it look like?

    Pain:

    Substernal

    Referred

    GI Feeling of anxiety

    Shortness of breath, weakness, cold sweat, etc.

    Prinzmetals: longer in duration; may wakepatient from sleep

    Can cause dysrhythmias, decreased myocardialcontractility

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    Other Diagnostic Studies

    Chest x-ray

    Detects:_________________________________________

    ________________________

    Lab tests: Serum Lipids,Cardiac enzyme values ECG, ECG stress test

    Ambulatory ECG Monitoring (Holter Monitor)

    Angiography (Cardiac Catheterization)

    Nuclear imaging (Thallium scan, Sestimbi) Positron Emission Tomography (PET scan): identifies ischemia and

    infarcted areas.

    Stress echocardiogram

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    Myocardial Infarction

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    Treatment Options

    PercutaneousTransluminalCoronaryAngioplasty

    Stent placement

    Atherectomy

    Laser

    Angioplasty Coronary

    Artery BypassGraft

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    Drug Therapy

    Antiplatelet

    aggregation therepy

    (aspirin!)

    Nitratesvasodiltors

    Nitroglycerine

    b-adrenergic

    blockers Calcium Channel

    blockers

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    NURSING

    IMPLEMENTATION Health Promotion and Education

    As discussed earlier

    Acute Needs

    Assessment: of pain, history, activity Pain: deep or superficial? Diffuse or well located?

    Care during anginal attack:

    O2 therapy

    Vitals, ECG

    Pain relief with Nitrate (& narcotic analgesic if ordered)

    Assessment of heart and breath sounds

    Patient comfort

    Assessment of patient response to therapy

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    Instructions regarding Nitrate

    Therapy

    Storage of medicaton

    Sub lingual administraton

    Immediate side effects and experience ofmedication working should be discussed

    with the patient.

    Sitting and standingpostural hypotension # of tablets to take to obtain relief of pain

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    End of Class

    Drive Carefully!