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    RAJIV GANDHI UNIVERSITY OF HEALTH SCIENCES

    BANGALORE, KARNATAKA

    PROFORMA FOR REGISTRATION OF SUBJECT FOR

    DISSERTATION

    Ms. DIMSEY. R. MARAK

    M. Sc. Nursing 1st

    year

    Obstetrics & Gynaecological Nursing

    Year 2009-2010

    PADMASHREE INSTITUTE OF NURSING

    BANGALORE

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    1

    RAJIV GANDHI UNIVERSITY OF HEALTH SCIENCES,

    BANGALORE, KARNATAKA.

    PROFORMA FOR REGISTRATION OF SUBJECTS FORDISSERTATION

    1 NAME OF THE

    CANDIDATE AND

    ADDRESS

    Ms Dimsey. R. Marak

    1st year M.Sc. Nursing

    Padmashree Institute of Nursing,

    Bangalore.

    2 NAME OF THE

    INSTITUTION

    Padmashree Institute of Nursing,

    Bangalore.

    3

    COURSE OF STUDY

    AND SUBJECT

    1st

    year M. Sc. Nursing

    Obstetrics & Gynaecological Nursing

    4DATE OF ADMISSION

    TO COURSE

    19th

    June 2009

    5 TITLE OF THE STUDY Assessment of effectiveness of Structured

    Teaching Programme on knowledge regarding

    gestational hypertension among primigravida

    mothers.

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    6. BRIEF RESUME OF THE INTENDED WORK

    6.1 INTRODUCTION

    Pregnancy is a time of great anticipation, when a woman is very aware that a

    new life is growing within her for the next nine months and hence they are highly

    motivated to remain as healthy as possible, so that she can give her baby the best

    start in life. Pregnancy is an excellent opportunity to develop good health habits;

    she can use these habits to keep the period of pregnancy safe and beautiful and also

    healthy for the rest of her life.1

    Certain physical conditions have specific implications for the pregnant

    women, her foetus and newborn. All pregnant women are at risk of complications

    like hypertension, anaemia, under nutrition, obesity, diabetes mellitus, abortion and

    intrauterine death, premature labour, Rh incompatibility in foetus and post

    maturity.

    Hypertension is one of the common complications met with in pregnancy and

    contributes significantly to maternal and foetal/neonatal morbidity and mortality.

    Hypertension in pregnancy is defined as a diastolic blood pressure of 90 or moreand systolic blood pressure level of 140 mm Hg or higher after 20 weeks of

    gestation in women with previously normal blood pressure.

    Hypertensive disorders predispose the women to serious complications,

    including abruptio placenta, thrombocytopenia, disseminated intravascular

    coagulation, acute renal failure, hepatic failure, pulmonary oedema, adult

    respiratory distress syndrome and cerebral haemorrhage2.

    The major classifications of hypertension in pregnancy are;

    Pre-eclampsia: It is a pregnancyspecific syndrome in which hypertensiondevelops after 20 weeks of gestation in a previously normotensive women,is

    characterized by the presence of hypertension and protienuria.

    Eclampsia: Preeclampsia when complicated with convulsion and or coma. Chronic hypertension: Chronic hypertensive disease is defined as the presenceof hypertension of any cause antedating or before the 20

    th

    week of pregnancy andits presence beyond the 42 days after delivery.

    http://www.pregnancy-period.com/pregnancy.htmlhttp://www.pregnancy-period.com/pregnancy.html
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    Preeclampsia or eclampsia superimposed on chronic hypertension: It is definedas protienuria developing during pregnancy in a woman with chronic hypertension.

    Gestational hypertension: Gestational hypertension is the onset of hypertensionwithout protienuria on at least two occasions at least 6 hours apart after the 20th

    week of gestation in women known to be normotensive before pregnancy3.

    Gestational Hypertension is a type of high blood pressure that occurs during

    pregnancy. It is a sustained rise in blood pressure to 140/90 mmHg or more on at

    least two occasions for four or more hours after 20 weeks of pregnancy to the first

    week after delivery. It is caused by an unknown set of factors that uniquely access

    during pregnancy and doesnt require free existing high blood pressure to develop.

    The incidence of gestational hypertension is higher in multipara and in twin

    pregnancy. Gestational hypertension before 30 weeks is frequently severe,

    advances to preeclampsia and has a guarded perinatal prognosis whereas after 34

    weeks it is usually a benign condition that rarely becomes severe, does not progress

    to preeclampsia, and therefore results in uniformly good prenatal outcome.

    Early gestational hypertension shares with preeclampsia a high incidence of

    poor placentation with histologic evidence of placental ischemia and hemodynamic

    changes characterized by vasoconstriction and decreased cardiac output. In late

    gestational hypertension the fundamental hemodynamic changes are increased

    plasma volume, increased cardiac output and normal peripheral vascular resistance.

    The fundamental problem behind early gestational hypertension is poor

    placentation, while late gestational hypertension corresponds to a poor maternal

    adaption to the physiologic changes of pregnancy.

    Gestational hypertension may resolve spontaneously after the baby is born, in

    which case the condition is referred to as transient hypertension. If the blood

    pressure remains elevated after delivery, the diagnosis becomes chronic

    hypertension4.

    The management of gestational hypertension include advices to the mother to

    change the life style with salt restricted diet, bed rest, proper exercises and avoiding

    stress. Antenatal mothers need to be advised to go for proper antenatal visit and

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    support for hospitalization in order to prevent from complications of hypertension

    and counsel the women and her family about the danger signs indicating

    preeclampsia5.

    6.2 NEED FOR THE STUDY:

    Hypertensive disorders are the most common medical complications reported

    during pregnancy. Women younger than 20 or older than 35 years, with gestational

    hypertension have significantly higher morbidity rates. Hypertension in pregnancy

    also increases the womens risk for caesarean birth. Hypertensive disorder constitute

    almost 7- 15% of all pregnancies, 22% of all perinatal deaths and 30% of all maternal

    deaths6.

    Gestational hypertension is the most frequent of the hypertensive conditions of

    pregnancy with prevalence rate between 6% - 15% in nulliparas and 2% - 4% in

    multiparas. The condition is more frequent in obese women and in women with

    multiple gestations, diabetes, and chronic hypertension and with a history of

    preeclampsia7.

    In rural India, women often report for antenatal care only in the latter half of

    pregnancy, hence their earlier blood pressure status is often not known. The majority

    of reports state that hemorrhage, hypertensive disorders in pregnancy, and sepsis are

    the chief indications for admission for critical care obstetric practice. Complicated

    hypertensive disorders in pregnancy account for over 20% of these admissions. The

    incidence of pre term births, birth asphyxia, and low Apgar scores are reported to be

    higher in the hypertensive group.

    Maternal and perinatal morbidity are increased in women with gestational

    hypertension. Also, women with mild gestational hypertension have an increased

    incidence of obstetrical interventions such as induction of labour and caesarean

    section. Women with severe gestational hypertension have a higher incidence of

    preterm birth and small for-gestational-age newborns than in those with mild

    preeclampsia8.

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    The most frequent complication of gestational hypertension is its progress to

    preeclampsia that is heralded by the development of protienuria. Approximately 15%-

    25% of women with gestational hypertension develop preeclampsia and this risk

    varies with gestational age.

    The concern is that gestational hypertension may develop into the more serious

    preeclampsia or eclampsia syndrome, therefore urine checking for protein should be

    done and blood pressure is monitored closely at each physician visit. If the blood

    pressure increases to a level that might endanger the women or her foetus, the

    physician will be prescribe antihypertensive drugs for the patient9.

    Kumar. Majhi et al., reporting from a referral hospital in Calcutta reported that

    the incidence of gestational hypertension was 44.6% of cases and primipara accounted

    for 88.7%, the caesarean section rate was 10.5% and maternal mortality rate was

    11.28% for gestational hypertension of these 48.7% was due to eclampsia. The

    perinatal mortality rate was 39.9% and the incidence of low birth weight and maternal

    outcome was better in the actively managed cases10

    .

    The researcher working as a staff nurse in the antenatal ward has seen patients

    having complications of delivery due to gestational hypertension. The researcher hasalso noted that these patients had inadequate knowledge regarding the problem and the

    complications that can arise if left untreated. All these prompted the researcher to

    select the following topic for study.

    Early identification of women at risk for Gestational hypertension may help

    prevent some complications of the disease. A health teaching programme should be

    developed together with the woman and her family to assist them in coping with the

    impact of high risk pregnancy Education about the warning symptoms, having regular

    prenatal visits and adapting to proper home care management are essential because

    early recognition may help women receive treatment and prevent worsening of the

    disease11

    .

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    6.3 STATEMENT OF THE PROBLEM

    A study to assess the effectiveness of Structured Teaching Programme on

    knowledge regarding gestational hypertension among primigravida mothers in a

    selected Maternity Hospital, Bangalore.

    6.4 OBJECTIVES

    1. To assess the pre-test knowledge regarding gestational hypertension amongprimigravida mothers.

    2. To assess the post-test knowledge regarding gestational hypertension amongprimigravida mothers.

    3. To compare the mean pre-test and posttest knowledge regarding gestationalhypertension among primigravida mothers.

    4. To associate the pre test knowledge regarding gestational hypertension amongprimigravida mothers with their selected demographic variables.

    6.5 OPERATIONAL DEFINITIONS

    1. EffectivenessIt refers to the outcome of structured teaching program in improving the

    knowledge regarding gestational hypertension.

    2. Structured teaching programmeA systematically arranged teaching programme regarding gestational

    hypertension- its definition, aetiology and risk factors, clinical features,

    complications, management and prevention provided by means of instructional

    aids.

    3. KnowledgeIt refers to the level of understanding and awareness regarding

    gestational hypertension.

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    4. Gestational hypertensionIt refers to a sustained rise of blood pressure to 140/90 mm of Hg or

    more on at least two occasions four or more hours apart beyond the 20th week

    of pregnancy or during the first 24 hours after delivery in a previously

    normotensive woman.

    5. Primigravida motherIt refers to a mother who is pregnant for the first time.

    6.6 ASSUMPTIONS

    1. The primigravida mothers may have inadequate knowledge regarding

    gestational hypertension.

    2. The structured teaching programme may improve the level of knowledge

    regarding gestational hypertension among primigravida mothers.

    3. The primigravida mothers knowledge regarding gestational hypertension may

    vary with their selected demographic variables.

    6.7 RESEARCH HYPOTHESIS

    H1:There will be a significant difference between the mean pre test and post test

    knowledge regarding gestational hypertension among primigravida mothers.

    H2: There will be a significant association between pre test knowledge regarding

    gestational hypertension among primigravida mothers with their selected

    demographic variables.

    6.8 REVIEW OF LITERATUREA prospective study was conducted with an objective to evaluate the perinatal

    outcome amongst mothers admitted with hypertensive disease during pregnancy at the

    Department of Pediatrics and Obstetrics of Maharaja Yeshwantrao Hospital, Indore

    over a period of one year. The study sample comprised of 405 cases admitted as

    hypertensive disorders of pregnancy and 100 cases admitted as full term normal

    pregnancy in labor and without any complication during pregnancy were taken as

    controls. The outcome findings revealed that the frequency of hypertensive disorders

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    in pregnancy was 7.49%. Twenty two (5.4%) mothers had twin deliveries. Out of

    these 405 cases, majority were due to toxaemia of pregnancy, i.e ., pre-eclampsia 204

    (50.4%) and eclampsia-43 (10.6%). One hundred and thirty two (30.9%) births needed

    intervention in the form of forceps application or caesarean section and foetal distress

    was the indication in 47 (35.6%) of these cases and low birth weight prevalence was

    high. Hence they concluded that there exist a relation between frequency of

    gestational hypertension and perinatal complications12

    .

    A descriptive study was conducted among 153 antenatal mothers attending the

    OPD of a co-operative hospital, Malappuram, Kerala to determine the knowledge

    level of antenatal mothers regarding gestational hypertension and its complications

    arising for both mother and the foetus. Samples who were selected using convenient

    sampling technique were administered a structured questionnaire to evaluate the

    knowledge level. The study findings showed that among the respondents, only few

    mothers (21%) were having good knowledge regarding gestational hypertension, its

    manifestations (19%), need for medical intervention (37%), its complications (11%)

    and its prevention (9%).The mothers with adequate knowledge were educated than the

    others. And it was concluded that an extensive health teaching is required to improvethe level of knowledge about this condition among the antenatal population

    13.

    A retrospective cohort study was conducted to assess the effects of gestational

    hypertension on infant mortality in triplets stratified by gestational age at birth, using

    the linked 19952000 US birth/infant death database. Generalized estimating

    equations were used to evaluate the observed association. The results revealed that

    gestational hypertension was associated with lesser neonatal mortality (95%), post

    neonatal mortality (95%) and infant mortality (95%) in triplets. whereas the

    association was not significant in late preterm or in full-term triplets. The study

    concluded that gestational hypertension is associated with a decreased risk of infant

    mortality in triplets. This effect varies with gestational age at birth14

    .

    A retrospective case control study was performed over a period of 5 years

    whose data were retrieved from hospital records. All teenage mothers (aged 1319

    completed years at delivery) delivering in the University Hospital were taken as cases.

    The results showed that majority of the teenagers were primigravida (83.2%).

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    Complications like gestational hypertension (11.4%), pre-eclamptic toxemia (PET)

    (4.3%) eclampsia (4.9%) and premature onset of labor (26.1%) occurred more

    comonly in teenagers compared to controls. The study concluded that teenage

    pregnancy was associated with a significantly higher risk of gestational hypertension,

    PET, eclampsia, premature onset of labor, fetal deaths and premature delivery15

    .

    The prospective cohort study was conducted to investigate whether gestational

    hypertension is more common in first pregnancies. 763 primiparous mothers who had

    their first births between 2004- 2007 were taken for study. The result showed that the

    risk of gestational hypertension was 4.1% in the first pregnancy and 1.7% in later

    pregnancies overall. The study concluded that early onset gestational hypertension

    might be associated with a reduced likelihood of a future pregnancy and with more

    recurrences than late onset gestational hypertension when there are further

    pregnancies16

    .

    The potential study was conducted to evaluate potential associations between

    maternal shift work and long working hours during pregnancy and gestational

    hypertension. The stratified systematic sampling was used to 24 200 post-partum

    women from the Taiwan in 2005. The study findings revealed that there was noassociation between employment status and gestational hypertension. The study

    concluded that there was no convincing evidence that maternal shift work or long

    working hours had a higher risk of gestational hypertension17

    .

    A case-control study of 250 pregnant women with hypertension and 400

    pregnant women without this complication who presented to Safdarjang Hospital in

    New Delhi, India was conducted. 200 cases (80%) and 372 controls (93%) were

    primigravidae. As expected, the result findings were that prevalence of adverse

    outcomes was significantly higher in cases than controls: preterm delivery, 28.8% vs.

    3%; need for labor induction, 52.8% vs. 3.25%; caesarean section delivery, 14.8% vs.

    3.5%; and need for special nursery care, 40% vs. 6.75%. 4.8% of infants of mothers

    with hypertension were stillborn, compared with 0.25% of infants of controls; overall

    perinatal mortality rates were 14.8% and 1%, respectively. Hence the study concluded

    that maternal hypertension contributes to an estimated 22% of all perinatal deaths and

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    this risk could be reduced by optimum antenatal care and timely increased use of

    obstetric interventions18

    .

    A retrospective cohort study was conducted to evaluate the effect of different

    types of pregnancy-induced hypertension on foetal growth. The study which had 128

    samples examined the effect of the various types of hypertension in pregnancy, on

    gestational age, preterm birth, birth weight, low birth weight, and intrauterine growth

    restriction. The results showed that gestation was 0.6 week shorter in women with

    gestational hypertension than in normotensive women (P < .01). The study concluded

    that preeclampsia increases the risk of intrauterine growth restriction and low birth

    weight19

    .

    The prospective study was conducted to observe the effect of the antioxidant

    lycopene (carotenoid found in tomatoes and berries) on the occurrence of gestational

    hypertension and intrauterine growth retardation (IUGR) in primigravida women

    among 251 primigravida women selected by randomized controlled trial. The study

    revealed that gestational hypertension developed significantly less in women in the

    lycopene group than in the placebo group (8.6% vs. 17.7%, P=0.043 by chi-square

    test), mean fetal weight was significantly higher in the lycopene group. The results ofthe present study concluded that the antioxidant lycopene reduces the development of

    gestational hypertension and intrauterine growth retardation in primigravida women20

    .

    A cohort prospective study was carried out in l96 patients with the diagnosis

    of gestational hypertension at the time of the interruption of the pregnancy to

    determine how many patients progressed to chronic hypertension. The results showed

    that thirteen patients (6.6%) with diagnosis of gestational hypertension progressed to

    chronic hypertension. It was found that advanced age , high body mass index and the

    antecedent of hypertensive disease in a previous pregnancy were significantly

    associated with the progression to chronic hypertension. The study concluded that

    overweight, advanced maternal age and the antecedent of hypertensive disorder in a

    previous pregnancy are the variables associated with the progression from gestational

    hypertension to chronic hypertension21

    .

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    7. MATERIAL AND METHODS OF STUDY

    7.1 SOURCE OF DATA

    The data will be collected from the primigravida mothers in a selected

    Maternity Hospital, Bangalore.

    7.2 METHODS OF DATA COLLECTION

    i. Research designQuasi experimentalOne group pre test and post test design

    22.

    ii.

    Research variable

    Dependent variable: Knowledge regarding gestational hypertension among

    primigravida mothers.

    Independent variable: Structured teaching programme regarding gestational

    hypertension among primigravida mothers.

    Demographic variable: Age, education, religion, occupation, income, food

    pattern, type of family, gestational weeks and sources of information

    iii. SettingThe study will be conducted in a selected Maternity Corporation

    Hospital, Bangalore.

    iv. PopulationThe population of the study will be the primigravida mothers who are

    admitted in a selected Maternity Hospital, Bangalore.

    v. SampleThe sample consists of 60 primigravida mothers who fulfill the inclusion

    criteria in a selected maternity hospital, Bangalore.

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    vi. Criteria for sample selection:Inclusion criteria

    The study includes

    1. Primigravida mothers with gestational hypertension admitted in theselected Maternity Hospital, Bangalore.

    2. Primigravida mothers who can understand Kannada or English.Exclusion criteria

    The study excludes

    1. Primigravida mothers who are not available at the time of data collection.2. Primigravida mothers who are not willing to participate in the study.

    vii. Sampling technique

    Non probability convenience sampling technique

    viii. Tool for data collection

    A structured questionnaire will be developed as a tool for data

    collection. It will consist of the following sections.

    Section AConsists of demographic variables which give baseline information

    such as age, education, religion, occupation, income, food pattern, type of

    family, gestational weeks and sources of information.

    Section BStructured questionnaire related to knowledge regarding gestationalhypertension among primigravida mothers.

    ix. Methods of data collection:After obtaining the official permissions from the hospital authorities and

    informed consent from the samples, the investigator will collect data pertaining

    to demographic variables. The remaining data will be collected in the following

    three phases-

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    Phase I- Pre-test will be conducted to assess the knowledge regarding

    gestational hypertension among primigravida mothers.

    Phase II- On the same day, structured teaching programme will be conducted

    regarding gestational hypertension among primigravida mothers by using

    instructional aids.

    PhaseIII- Post test will be conducted with the same questionnaire after 7 days,

    of planned teaching programme.

    x. Plan for data analysis

    The data collected will be analysed using descriptive and inferentialstatistics.

    Descriptive statistics:

    Frequency and percentage distribution will be used to describe the

    demographical variables. Mean and standard deviation will be used to analyze

    the knowledge among primigravida mothers.

    Inferential statistics:

    Pairedt test will be used to compare the pre test and post test knowledge

    regarding gestational hypertension among primigravida mothers.

    Chi-square test will be used to analyze the pre test knowledge regarding

    gestational hypertension among primigravida mothers with their selected

    demographic variables23

    .

    x. Projected outcome

    The Structured Teaching Programme will improve knowledge regarding

    gestational hypertension among primigravida mothers. It will help

    primigravida mothers for early prevention and detection of gestationalhypertension.

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    7.3 Does the study require any investigations or interventions to the

    patients or human beings or animals?

    Yes, Structured Teaching Programme will be administered regarding

    gestational hypertension as intervention for the primigravida mothers.

    7.4Has ethical clearance been obtained from your institution?

    Yes, permission will be taken from the concerned authorities in the selected

    Maternity Hospital. Informed consent from the samples will be obtained.

    Confidentiality and privacy of data will be maintained.

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    2. D.C. Dutta. Text book of obstetrics.6th ed.Calcutta: New central bookagency;2004.

    3. Fernando Arias. Shirish N Daftary. Amarnath G Bhide. Practical guideto high-risk pregnancy and delivery. 3

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    7. N.Jayne Klossner.Introductory maternity nursing. 1st ed .Philadelphia:Lippincott Williams & Wilkins;2006.

    8. Annamma Jacob .A comprehensive textbook of mifwifery.1st ed. NewDelhi:Jaypee Brothers;2005.

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    10. Deitra Leonard Lowdermilk,Shannon E.Perry. Maternity & Womens HealthCare.9 ed Missouri : Mosby Elsevier.2007

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    13. X.K.Chenab, S.W. Wenabc, G.N. Smithd, Q. Yangab, M.C. Walkerabc.Pregnancy-induced hypertension and infant mortality in triplets 2007. Volume

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    22. P.S.S. Sundar Rao and J Richard. Introduction to biostatistic and researchmethods. 4

    thedition. New Delhi: Prentice-Hall of India; 2008.

    http://www.ijgo.org/article/S0020-7292(07)00148-8/abstracthttp://www.ijgo.org/article/S0020-7292(07)00148-8/abstract#aff2http://www.ijgo.org/article/S0020-7292(07)00148-8/abstracthttp://www.ijgo.org/article/S0020-7292(07)00148-8/abstracthttp://www.ijgo.org/article/S0020-7292(07)00148-8/abstract#aff2http://www.ijgo.org/article/S0020-7292(07)00148-8/abstracthttp://www.ijgo.org/article/S0020-7292(07)00148-8/abstract#aff2http://www.ijgo.org/article/S0020-7292(07)00148-8/abstract#aff2http://www.ijgo.org/articlehttp://www.ncbi.nlm.nih.gov/pubmedhttp://www.ncbi.nlm.nih.gov/pubmedhttp://www.ncbi.nlm.nih.gov/pubmed%20%20/19700491?ordinalpos=52&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_DefaultReportPanel.Pubmed_RVDocSumhttp://www.ijgo.org/article/S0020http://www.springerlink.com/contentmailto:[email protected]:Romero%20Guti%C3%A9rrez%20G,%20Muro%20Barrag%C3%A1n%20SA,%20Ponce%20de%20Le%C3%B3n%20AL.Unidad%20M%C3%A9dica%20de%20Alta%20Especialidad,%20Hospital%20de%20Gineco-Pediatr%C3%ADa%20No.%2048,%20Leon,Guanajuato,%20M%C3%A9xico.%[email protected]:Romero%20Guti%C3%A9rrez%20G,%20Muro%20Barrag%C3%A1n%20SA,%20Ponce%20de%20Le%C3%B3n%20AL.Unidad%20M%C3%A9dica%20de%20Alta%20Especialidad,%20Hospital%20de%20Gineco-Pediatr%C3%ADa%20No.%2048,%20Leon,Guanajuato,%20M%C3%A9xico.%[email protected]:Romero%20Guti%C3%A9rrez%20G,%20Muro%20Barrag%C3%A1n%20SA,%20Ponce%20de%20Le%C3%B3n%20AL.Unidad%20M%C3%A9dica%20de%20Alta%20Especialidad,%20Hospital%20de%20Gineco-Pediatr%C3%ADa%20No.%2048,%20Leon,Guanajuato,%20M%C3%A9xico.%[email protected]:[email protected]://www.springerlink.com/contenthttp://www.ijgo.org/article/S0020http://www.ncbi.nlm.nih.gov/pubmed%20%20/19700491?ordinalpos=52&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_DefaultReportPanel.Pubmed_RVDocSumhttp://www.ncbi.nlm.nih.gov/pubmedhttp://www.ijgo.org/articlehttp://www.ijgo.org/article/S0020-7292(07)00148-8/abstract#aff2http://www.ijgo.org/article/S0020-7292(07)00148-8/abstract#aff2http://www.ijgo.org/article/S0020-7292(07)00148-8/abstracthttp://www.ijgo.org/article/S0020-7292(07)00148-8/abstracthttp://www.ijgo.org/article/S0020-7292(07)00148-8/abstract#aff2http://www.ijgo.org/article/S0020-7292(07)00148-8/abstracthttp://www.ijgo.org/article/S0020-7292(07)00148-8/abstracthttp://www.ijgo.org/article/S0020-7292(07)00148-8/abstracthttp://www.ijgo.org/article/S0020-7292(07)00148-8/abstracthttp://www.ijgo.org/article/S0020-7292(07)00148-8/abstract#aff2http://www.ijgo.org/article/S0020-7292(07)00148-8/abstract#aff2http://www.ijgo.org/article/S0020-7292(07)00148-8/abstracthttp://www.ijgo.org/article/S0020-7292(07)00148-8/abstract
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    9. Signature of the candidate :

    10. Remark of the guide :

    11. Name and designation of guide: Mrs. M. Sundaram

    Associate Professor

    11.2 Signature :

    11.3 Co guide : Ms. Subashini. GAssistant Professor

    11.4 Signature :

    11.5 Head of the department : Mrs. M. Sundaram

    11.6 Signature :

    12.1 Remarks of the principal :

    12.2 Signature :

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